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Patellar Instability and Patellar Dislocations: A Comprehensive Guide


1. ANATOMY AND BIOMECHANICS

The patellofemoral joint depends on both bony containment and soft tissue constraints for stability:
Bony stabilizers:
  • The femoral trochlear groove guides the patella through a cam-like mechanism
  • The lateral trochlear facet is normally more prominent than the medial, resisting lateral patellar displacement
  • The patella begins engaging the trochlea at approximately 20-30 degrees of knee flexion - below this arc, soft tissues dominate
Soft tissue stabilizers:
  • Medial Patellofemoral Ligament (MPFL): The primary medial restraint (50-60% of resistance to lateral translation). Runs from the superomedial patella to the medial femoral epicondyle (Schottle's point)
  • Vastus Medialis Obliquus (VMO): Active dynamic medial stabilizer
  • Lateral retinaculum: Normally thicker/stronger - imbalance predisposes to lateral dislocation
  • Patellotibial and patellomeniscal ligaments: Secondary restraints
Key measurements for instability assessment:
  • Q angle: Angle between ASIS-patella line and patella-tibial tubercle line; normal <15° men, <20° women
  • TT-TG distance (tibial tubercle to trochlear groove): Normal 9-13 mm; >20 mm = high risk for instability
  • Patella alta: Caton-Deschamps index >1.2 or Insall-Salvati ratio >1.2
  • Trochlear sulcus angle: Normally <140° on axial views

2. CLASSIFICATION OF PATELLAR INSTABILITY

A. Functional Classification (Lyon/Dejour School)

Three major groups, as described by H. Dejour:
GroupDefinition
Objective Patellar Instability (OPI)At least one documented episode of lateral dislocation + identifiable anatomical risk factor. The most common and clinically relevant group. Subtypes: acute, recurrent (>3 events), habitual (dislocation consistently in early flexion), permanent (persistent displacement throughout ROM)
Potential Patellar Instability (PPI)Anatomical risk factors present + knee discomfort, but NO documented dislocation episode. Risk of future instability
Painful Patellar Syndrome (PPS)Knee pain without anatomical risk factors or documented subluxation/dislocation
  • Patellar instability: current approach, PMC 2026

B. Types of Patellar Dislocation by Direction

TypeDescriptionMechanismFrequency
Lateral dislocationPatella displaced laterally out of trochlear grooveKnee flexion + valgus stress + external tibial rotation; or direct medial blowBy far the most common (~95%)
Medial dislocationPatella displaced medially - almost always iatrogenic (after overzealous lateral release or realignment surgery)Surgical over-correctionRare; iatrogenic
Superior dislocationPatella locks on the anterior femoral margin in full extensionHyperextension; osteophyte entrapment in osteoarthritic kneesVery rare; <50 cases reported
Intraarticular/horizontal dislocationPatella rotates 90° within jointViolent direct traumaExtremely rare
Habitual dislocationOccurs consistently at a specific knee flexion angle during normal gaitUnderlying dysplasia; congenital laxityUncommon
Permanent/obligate dislocationPatella remains dislocated throughout entire range of motionSevere dysplasia; congenital anomalyRare

C. Trochlear Dysplasia: Dejour Classification

Trochlear dysplasia is the strongest anatomical risk factor for patellar instability. Classified by lateral radiograph and axial CT/MRI:
Dejour Type A - shallow trochlea with crossing sign
Dejour Type A: Crossing sign on lateral radiograph; shallow trochlea with sulcus angle >145°
Dejour Type B - flat trochlea with supratrochlear spur
Dejour Type B: Supratrochlear spur; flat trochlea
Dejour Type C - lateral convexity with medial hypoplasia
Dejour Type C: Double contour sign on lateral X-ray; lateral convexity + medial hypoplasia
TypeLateral X-ray signMorphology
ACrossing sign onlyShallow trochlea (sulcus angle >145°)
BCrossing sign + supratrochlear spurFlat/convex trochlea
CCrossing sign + double contourLateral convexity + medial hypoplasia
DCrossing sign + spur + double contourCliff pattern - most severe
Updated Dejour V3.0 (2024 - MRI-based):
  • Type 0 (no dysplasia): Sulcus angle <157°; lateral trochlear inclination (LTI) >14°
  • Type 1 (low-grade): Sulcus angle >157°; LTI <14°; central bump <5 mm
  • Type 2 (moderate): Sulcus angle/LTI unmeasurable; central bump <5 mm
  • Type 3 (high-grade): Sulcus angle >157° or unmeasurable; LTI <14° or unmeasurable; central bump >5 mm

3. RISK FACTORS AND PREDISPOSING ANATOMY

FactorDetail
Trochlear dysplasiaMost important; significantly reduces bony containment
Patella altaDelayed entry into the trochlear groove; more time unstabilized
Increased TT-TG distanceLateral pull vector on patella; >20 mm = high risk
MPFL incompetenceTraumatic rupture (most commonly at patellar insertion)
VMO weakness/atrophyReduced active medial stabilization
Generalized ligamentous laxityHypermobility syndromes (Ehlers-Danlos, Down syndrome)
Femoral anteversionIncreases effective valgus alignment
Genu valgumIncreases Q angle and lateral force vector
Female sexHigher incidence; greater physiological laxity
Age 10-17 yearsPeak incidence; incidence 29/100,000 in this group
Family historyGenetic predisposition to dysplasia
"Miserable malalignment syndrome"Femoral anteversion + genu valgum + pronated feet - exacerbates symptoms, especially in adolescents
  • Miller's Review of Orthopaedics 9th Ed, p. 365
  • Campbell's Operative Orthopaedics 15th Ed, p. 1861

4. SIGNS AND SYMPTOMS

History

  • Mechanism: Pivoting/twisting injury with foot planted (non-contact); external tibial rotation with knee in slight flexion; or direct blow to the medial aspect of the patella
  • Subjective dislocation event: Patient may report seeing or feeling the kneecap "slip out," followed by spontaneous reduction as the knee extends
  • Pop: Often heard/felt - can mimic ACL tear; in patients aged 9-14 years, this mechanism is more likely a patellar instability event than ACL injury
  • Previous episodes: Recurrence rate 15-60% after first dislocation; younger patients and those with dysplasia at higher risk

Symptoms

SymptomDescription
Acute painImmediate, severe; localised anteriorly around the patella
Swelling/effusionDevelops hours post-injury; hemarthrosis common (especially with osteochondral fracture)
Inability to weight bearDifficulty walking and flexing the knee
Visible deformityPatella displaced laterally if not yet reduced
Catching/lockingParticularly with loose osteochondral bodies
Giving wayEpisodes of buckling - hallmark of recurrent instability
StiffnessEspecially in early morning or after activity
Subluxation sensationPatella "threatening to give way" without complete dislocation

Physical Examination Findings

Inspection:
  • Knee held in flexion (guarding)
  • Visible lateral displacement of the patella (if not spontaneously reduced)
  • Swelling/effusion
  • Bruising (ecchymosis) developing within hours, particularly medially (from medial retinaculum/MPFL tear)
Palpation:
  • Medial tenderness: Tenderness along the medial retinaculum, medial patellar border, and MPFL origin at the medial femoral condyle - highly specific for MPFL rupture
  • Joint effusion: Ballottement sign; wave sign
  • Hemarthrosis: Suggests osteochondral fracture
  • Defect: Palpable defect at medial retinaculum
Special Tests:
TestTechniquePositive Finding
Patellar apprehension testPatient supine, knee at 30°; examiner gently translates patella laterallyPatient grabs examiner's hand, exhibits fear/anxiety or contracts quadriceps - highly specific for patellar instability
Patellar glide testDivide patellar width into quarters; assess lateral and medial translation>3 quadrants lateral glide = lateral hypermobility; <1 quadrant = medial tightness
J-signWatch the patella track during active knee extension from 90°Patella jumps laterally like a "J" as it exits the trochlea near full extension = suggests dysplasia/alta
Tilt testExaminer lifts lateral patellar edge while patient relaxed<0° tilt = lateral retinacular tightness
Clarke's testCompress patella, patient contracts quadricepsPain = patellofemoral chondromalacia (non-specific)
Vastus medialis assessmentLook for VMO atrophyIndicates chronic quadriceps imbalance
  • Miller's Review of Orthopaedics 9th Ed, p. 365
  • Rosen's Emergency Medicine, p. 721

5. INVESTIGATIONS AND EVALUATION

Radiographs (First-Line)

Views Required:
ViewWhat It Shows
AP (anteroposterior)Gross alignment, fractures, loose bodies, patella position
True lateral (30° flexion)Patella alta/baja, trochlear morphology, crossing sign, supratrochlear spur, double contour sign
Axial/Merchant view (30-45° flexion)Patellar tilt, subluxation, sulcus angle, congruence angle; difficult to obtain pre-reduction
Sunrise view (post-reduction)Patella centering in trochlear groove, confirmed reduction, osteochondral fragments
Expected X-ray findings with dislocation:
  • Empty trochlear groove (if not yet reduced)
  • Displaced patella (lateral)
  • Joint effusion (radiopaque soft tissue swelling)
  • Osteochondral fracture fragment - most commonly at the medial inferior patella edge or lateral femoral condyle
  • Lipohemarthrosis (on horizontal beam lateral) - suggests intra-articular fracture
Lateral radiograph trochlear signs:
  • Crossing sign: Trochlear groove line crosses the anterior femoral condyle outline = dysplasia present
  • Supratrochlear spur: Bony bump proximal to trochlea = Type B dysplasia
  • Double contour sign: Hypoplastic medial condyle creates second contour = Type C/D dysplasia

CT Scan

  • Gold standard for measuring TT-TG distance (most accurate)
    • Normal: 9-13 mm
    • 15-20 mm: borderline
    • 20 mm: significantly associated with instability
  • Quantifies trochlear morphology in cross-section
  • Detects osseous loose bodies
  • Identifies torsional deformities (femoral anteversion, tibial torsion)
  • Dynamic CT can assess patellar tracking in different flexion angles
TT-TG distance measurement diagram
TT-TG distance: measured on overlaid CT slices comparing the tibial tubercle (TT) to the deepest point of the trochlear groove (TG). Values >20 mm indicate significant lateralization.

MRI (Essential in Acute Dislocation with Effusion)

Recommended when effusion is present after patellar instability event to rule out osteochondral injury.
MRI findingSignificance
Lateral femoral condyle bone bruisePathognomonic "kissing contusions" pattern - confirms dislocation mechanism
Medial patellar bone bruiseFrom lateral wall impact during reduction
MPFL tearMost commonly at patellar insertion; seen as disruption of medial soft tissues
Osteochondral fractureHigh incidence (~95% have cartilage injury); common on medial patellar facet and lateral femoral condyle
VMO injuryAssociated disruption
Loose bodiesFree fragments requiring arthroscopic removal
TT-TG on MRIUnderestimates true distance vs CT; CT preferred for this measurement
The International Patellofemoral Study Group recommends MRI for patients with large hemarthrosis to detect osteochondral fractures.
  • Rockwood and Green's Fractures in Adults 10th Ed, p. 3020

Arthroscopy (Selective Use)

  • Not routine; reserved for:
    • Large hemarthrosis with suspected loose osteochondral fragments
    • Failure of conservative management
    • Pre-surgical assessment
    • Concurrent intra-articular pathology

Patient-Reported Outcome Measures

  • Kujala Score (AKPS): Anterior knee pain scale; gold standard for patellofemoral outcomes
  • IKDC (International Knee Documentation Committee)
  • Lysholm score
  • KOOS (Knee Injury and Osteoarthritis Outcome Score)

6. EVALUATION FRAMEWORK (SYSTEMATIC APPROACH)

STEP 1: Establish dislocation vs subluxation vs alternative diagnosis
         ↓
STEP 2: Assess for urgent reduction (if still dislocated)
         ↓
STEP 3: Post-reduction imaging (X-ray: AP + lateral + sunrise)
         ↓
STEP 4: Identify osteochondral fracture / loose bodies
         ↓
STEP 5: MRI if hemarthrosis or suspected osteochondral injury
         ↓
STEP 6: Risk-stratify for recurrence (age, sex, trochlear dysplasia, 
         TT-TG distance, patella alta, prior episodes)
         ↓
STEP 7: If surgical candidate: CT for TT-TG; full morphological assessment
         ↓
STEP 8: Outcome measures + physiotherapy assessment

7. TREATMENT

A. Emergency / Acute Management: Closed Reduction

Technique:
  1. Provide adequate analgesia (oral analgesics usually sufficient; rarely needs sedation)
  2. Patient supine, hip flexed to relax hamstrings
  3. Slowly and gently extend the knee (passively)
  4. Apply inferomedial pressure to the lateral patella as the knee extends
  5. A second provider may assist with gentle downward pressure on the lateral patella to "open" the medial facet
  6. Confirm reduction - a palpable/audible "click" as patella seats into trochlea
Post-reduction:
  • Neurovascular examination
  • Knee immobilizer in full extension
  • Post-reduction radiographs: AP + lateral + sunrise (to confirm centering + exclude fracture)
  • Weight bear as tolerated with crutches
  • Orthopedic follow-up within 1-2 weeks

B. Conservative (Non-Operative) Treatment

Indicated for: First-time dislocations without osteochondral fracture or loose bodies.
Phase 1: Acute (0-2 weeks)
  • Knee immobilizer in extension
  • Cryotherapy + NSAIDs for pain and swelling
  • Isometric quadriceps exercises
Phase 2: Rehabilitation (2-6 weeks)
  • Patella-stabilizing brace
  • Progressive range of motion
  • Closed-chain quadriceps exercises (especially VMO emphasis)
  • Hip abductor/external rotator strengthening
  • Proprioception training
Phase 3: Return to Sport (6-12 weeks)
  • Return when full passive ROM regained, no effusion, quadriceps strength ≥80% of contralateral side
  • Functional sport-specific training
Outcomes:
  • Recurrence rates 15-44% (conservative) to 15-60% in some series
  • Recurrence risk highest in: age <15, trochlear dysplasia, patella alta, elevated TT-TG, female sex
  • Surgically and conservatively managed patients have similar long-term functional outcomes in first-time dislocators without major structural injury

C. Surgical Treatment

Indications for surgery:
  • Osteochondral fracture with free fragment (fix or remove)
  • Large complete MPFL avulsion/midsubstance tear (selected cases)
  • Failure of conservative management / recurrent instability
  • First-time dislocation in high-risk patients (trochlear dysplasia grade B-D, TT-TG >20 mm, high-level athletes)
  • Medial iatrogenic instability (if from prior surgery)
Surgical Procedures:

1. MPFL Reconstruction (Proximal Realignment)

The current gold standard for recurrent patellar instability.
  • Autograft: gracilis or semitendinosus tendon
  • Graft routed from patella (patellar tunnels or suture anchors) to anatomic femoral origin
  • Schottle's point = anatomic femoral attachment of MPFL, identified on lateral radiograph:
    • 1 mm anterior to posterior cortex extension line
    • 2.5 mm distal to posterior origin of medial femoral condyle
    • Proximal to posterior point of Blumensaat line
Schottle's point for MPFL femoral attachment
Schottle's point for anatomic MPFL femoral tunnel placement (A - anatomic diagram, B - lateral X-ray landmarks, C - intraoperative fluoroscopy confirmation). Incorrect tunnel position leads to isometry failure: too proximal = tight in flexion; too distal = loose in flexion.
  • Isolated medial retinacular plication and VMO advancement have largely fallen out of favour due to higher failure rates

2. Tibial Tubercle Osteotomy (Distal Realignment)

  • Anteromedialization (Fulkerson osteotomy): Combined anteromedial transfer of the tibial tubercle
    • Indicated when: TT-TG >20 mm, associated distal patellar chondromalacia (anteriorization offloads the patella)
    • Contraindication: Proximal patellar arthrosis of the medial facet
  • Medial transfer alone (Elmslie-Trillat): For isolated TT-TG elevation without chondral disease
  • Fixed with screws; partial weight-bearing post-op

3. Trochleoplasty

  • Surgical deepening/reshaping of the trochlear groove
  • Indicated for: High-grade trochlear dysplasia (Dejour B/D with supratrochlear bump) when bony containment is insufficient
  • Technically demanding; usually combined with MPFL reconstruction
  • Sulcus-deepening trochleoplasty (Bereiter technique) most common

4. Lateral Release / Lateral Retinacular Lengthening

  • Isolated lateral release should NOT be performed for patellar instability (causes medial iatrogenic instability)
  • Can be added as an adjunct to address excessive lateral tightness alongside other procedures

5. Proximal Femoral Osteotomy

  • For patients with significant femoral anteversion (>25-30°) contributing to instability
  • Derotational femoral osteotomy corrects excessive anteversion

6. Loose Body Removal / Osteochondral Repair

  • Arthroscopic removal of free intra-articular fragments
  • OATS (osteochondral autograft transfer) or autologous chondrocyte implantation for large cartilage defects
Decision algorithm for surgery:
Recurrent instability / first-time with risk factors
         ↓
Assess TT-TG distance (CT)
         ↓
TT-TG <20 mm                    TT-TG >20 mm
     ↓                                ↓
MPFL reconstruction alone       MPFL + tibial tubercle 
                                 anteromedialisation
         ↓
Assess trochlear dysplasia (Dejour)
         ↓
Grade A/C (no bump)            Grade B/D (bump present)
MPFL +/- TTO               MPFL + trochleoplasty +/- TTO

8. SPECIAL CONSIDERATIONS

Pediatric Patients (Open Physes)

  • First-time dislocation remains most common cause of traumatic hemarthrosis in children
  • Bony procedures at tibial tubercle risk physeal damage until skeletal maturity
  • MPFL reconstruction can be performed physeal-sparing
  • Higher recurrence risk in children <15 years

Superior Patellar Dislocation

  • Rare (fewer than 50 cases reported)
  • Presents with: high-riding patella, acute knee pain, inability to perform straight leg raise, skin dimple deformity
  • Often in patients with large osteophytes (knee osteoarthritis) - the osteophyte entraps the patella in hyperextension
  • Requires manual reduction; may need surgical osteophyte removal if recurs

Iatrogenic Medial Dislocation

  • Occurs after overzealous lateral release or distal realignment over-correction
  • Presents with medial apprehension, locking in flexion
  • Requires revision realignment (medial retinacular release + lateral reconstruction)

Habitual Dislocation

  • Occurs predictably at a consistent flexion angle during normal walking
  • Usually congenital/developmental basis
  • Often requires surgical correction combining MPFL reconstruction + trochleoplasty

9. COMPLICATIONS

ComplicationDetail
Osteochondral fractureUp to 95% articular cartilage injury in first-time dislocators
Recurrent dislocation15-60% after conservative management
Medial iatrogenic instabilityExcessive lateral release or over-correction
Post-traumatic osteoarthritisCartilage injury leads to early patellofemoral OA
StiffnessPost-immobilization; especially after tibial tubercle osteotomy
MPFL graft failureIncorrect tunnel placement; isometry failure
InfectionPost-operative
Neurovascular injuryRare; saphenous nerve at risk in medial approaches

Summary Table

ParameterKey Points
Most common typeLateral dislocation
Peak incidenceAge 10-17 years; 29/100,000
Key risk factorsTrochlear dysplasia, patella alta, TT-TG >20 mm, MPFL rupture, female sex
Gold standard imagingCT for TT-TG; MRI for soft tissue/cartilage
First-line treatmentClosed reduction + immobilizer + rehab
Surgery indicationsOsteochondral fracture, recurrence, high-risk anatomy
Best surgical procedureMPFL reconstruction ± TTO ± trochleoplasty
Recurrence risk (conservative)15-60%
TT-TG normal9-13 mm
MPFL tear locationMost commonly patellar insertion

Sources:
  • Miller's Review of Orthopaedics, 9th Ed - pp. 364-366 (patellar instability, Dejour classification, MPFL)
  • Campbell's Operative Orthopaedics, 15th Ed 2026 - p. 1861 (patellar dislocations, treatment)
  • Rockwood and Green's Fractures in Adults, 10th Ed 2025 - pp. 3019-3021 (acute patellar dislocations)
  • Rosen's Emergency Medicine - pp. 720-721 (acute management)
  • Tintinalli's Emergency Medicine - p. 960 (patellar dislocations in children)
  • Patellar instability: current approach (PMC, 2026) - Dejour V3.0 classification, updated surgical algorithms

Patellar instability different type of patella dislocations explain in detail the signs and symptoms and investigations and evaluation treatment explain in detail with appropriate illustrative diagrams and images

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I now have all four Dejour types illustrated, the TT-TG diagram, and the Schottle's point MPFL images. I have comprehensive textbook content from Campbell's, Rockwood & Green's, Miller's, and Rosen's, plus the latest 2024-2026 evidence. Let me now compose the full, detailed, well-illustrated answer.

Patellar Instability and Patellar Dislocations: A Complete Clinical Guide


SECTION 1 — ANATOMY AND STABILIZERS OF THE PATELLOFEMORAL JOINT

The patella sits within the femoral trochlear groove and is stabilized by a combination of bony and soft tissue restraints. Understanding these is essential to understanding why instability occurs.

Bony Stabilizers

  • The lateral trochlear facet is normally taller and more prominent than the medial, creating a "wall" that resists lateral patellar displacement
  • The patella engages the trochlea at approximately 20-30° of knee flexion - below this arc, soft tissues provide primary restraint
  • Any trochlear shallowness (dysplasia) eliminates this bony containment

Soft Tissue Stabilizers

StructureRole
MPFL (Medial Patellofemoral Ligament)Primary medial restraint - provides ~50-60% of resistance to lateral translation. Runs from superomedial patella to medial femoral epicondyle (Schottle's point). Ruptured in >90% of acute dislocations
VMO (Vastus Medialis Obliquus)Active dynamic medial stabilizer; weakness allows lateral subluxation
Lateral retinaculumNormally thicker than medial; excessive tightness contributes to patellar tilt and lateral tracking
Patellotibial and patellomeniscal ligamentsSecondary distal restraints

Key Biomechanical Measurements

MeasurementNormal ValuesSignificance
Q angle<15° (male), <20° (female)Represents lateralizing force vector; increased with femoral anteversion/genu valgum
TT-TG distance9-13 mmLateral tubercle displacement increases lateral pull; >20 mm = high instability risk
Insall-Salvati ratio0.8-1.2>1.2 = patella alta; reduces trochlear engagement
Caton-Deschamps index0.6-1.2>1.2 = patella alta
Trochlear sulcus angle<140° (axial view)>145-150° = trochlear dysplasia

SECTION 2 — CLASSIFICATION OF PATELLAR INSTABILITY

A. By Functional Pattern (Lyon/Dejour Classification)

Three major clinical groups, introduced by H. Dejour:
GroupDefinition
Objective Patellar Instability (OPI)At least one documented dislocation + identifiable anatomical risk factor. Lateral dislocation during knee extension (non-contact) is the most common presentation. Within OPI: acute (first-time), recurrent (>3 events), habitual (occurs at a consistent knee flexion angle every time), permanent (patella stays dislocated throughout full ROM)
Potential Patellar Instability (PPI)Anatomical risk factors present + knee discomfort, but no documented complete dislocation - only subluxation or apprehension
Painful Patellar Syndrome (PPS)Anterior knee pain without risk factors or dislocation - often misdiagnosed as instability

B. By Direction of Dislocation

TypeDescriptionMechanismFrequency
Lateral dislocationPatella displaced laterally - the patella completely exits the trochlea laterallyKnee flexion + valgus stress + external tibial rotation on planted foot; or direct medial blow~95% - by far the most common
Medial dislocationPatella displaced mediallyAlmost exclusively iatrogenic (over-aggressive lateral release surgery or over-correction of realignment); traumatic medial dislocation is exceedingly rareRare
Superior dislocationHigh-riding patella locked on the anterior femoral condyle in extensionHyperextension (often in osteoarthritic knees with large osteophytes that trap the patella)Very rare (<50 cases reported)
Intra-articular / horizontal dislocationPatella rotates 90° inside the jointHigh-energy direct traumaExtremely rare
Habitual dislocationPredictable dislocation occurring at a specific flexion angle during normal gaitCongenital/developmental; tight lateral structures or abnormal muscle insertionsUncommon
Permanent / obligate dislocationPatella remains permanently displaced, irreducible throughout entire ROMSevere congenital dysplasia; neuromuscular conditionsRare

C. Dejour Classification of Trochlear Dysplasia

Trochlear dysplasia is the single strongest predictor of patellar instability. It is classified based on lateral radiograph signs and axial cross-sectional morphology:
Dejour Type A - Shallow Trochlea
Dejour Type A: Crossing sign on lateral X-ray; shallow trochlea with sulcus angle >145°
Type A: The trochlear groove line crosses the anterior femoral condyle outline (crossing sign) on the lateral view. Axially the trochlea is shallower than normal but still concave. Sulcus angle >145°.
Dejour Type B - Flat Trochlea with Supratrochlear Spur
Dejour Type B: Supratrochlear spur; flat or convex trochlea
Type B: Crossing sign PLUS a supratrochlear spur (bony bump protruding proximally). Axially the trochlea is flat or frankly convex - no groove to contain the patella.
Dejour Type C - Lateral Convexity with Medial Hypoplasia
Dejour Type C: Double contour; lateral convexity + medial hypoplasia
Type C: Crossing sign PLUS double contour (a second line below the crossing sign representing the hypoplastic medial condyle). Axially: lateral facet is convex while medial facet is hypoplastic.
Dejour Type D - Most Severe "Cliff Pattern"
Dejour Type D: Crossing sign + spur + double contour; cliff pattern
Type D: All three lateral signs present (crossing sign + supratrochlear spur + double contour). Axially there is an abrupt asymmetry ("cliff") between the two facets - the most unstable bony morphology. Type D requires trochleoplasty.
Dejour TypeLateral X-ray SignsAxial MorphologySurgical Implication
ACrossing sign onlyShallow trochleaMPFL recon ± TTO
BCrossing sign + spurFlat/convex trochleaMPFL recon + trochleoplasty
CCrossing sign + double contourConvex lateral + medial hypoplasiaMPFL recon ± TTO
DCrossing sign + spur + double contourCliff/asymmetricMPFL recon + trochleoplasty + TTO
Updated Dejour V3.0 (2024 - quantitative MRI-based):
  • Type 0 (no dysplasia): Sulcus angle <157°; LTI >14°
  • Type 1 (low-grade): Sulcus angle >157°; LTI <14°; central bump <5 mm
  • Type 2 (moderate): Sulcus angle/LTI unmeasurable; central bump <5 mm
  • Type 3 (high-grade): Bump >5 mm; most severe - requires trochleoplasty

SECTION 3 — RISK FACTORS AND PREDISPOSING ANATOMY

Risk FactorDetail
Trochlear dysplasiaMost powerful single risk factor; eliminates bony containment
Patella altaDelayed engagement into the trochlea; the patella is "free" for longer during flexion
Increased TT-TG distanceLateral pull vector; >20 mm is strongly associated with instability
MPFL deficiencyTraumatic rupture or congenital laxity; >90% ruptured in acute dislocation
VMO weakness / hypoplasiaLoss of dynamic medial stabilization
Ligamentous laxityHypermobility syndromes (Ehlers-Danlos, Down syndrome, Marfan's)
Female sexHigher physiological laxity; broader pelvis increases Q angle
Age 10-17 yearsPeak incidence 29/100,000 in this age group
Femoral anteversionIncreases effective valgus; rotates the trochlea into a less favorable orientation
Genu valgumIncreases Q angle and lateral pull
Positive family historyGenetic predisposition to dysplasia and laxity
"Miserable malalignment syndrome"Femoral anteversion + genu valgum + pronated feet - compounds instability, especially in adolescents
  • Campbell's Operative Orthopaedics 15th Ed, p. 1861
  • Miller's Review of Orthopaedics 9th Ed, p. 365

SECTION 4 — SIGNS AND SYMPTOMS

History

  • Mechanism: Pivoting/twisting on a planted foot (most common - non-contact); external tibial rotation with knee flexed; or direct blow to the medial patella
  • Age 9-14: A pop + swelling after twisting is more likely patellar instability than ACL tear
  • Prior episodes: Recurrence rate is 15-75% after first dislocation
  • Spontaneous reduction: Patella often self-reduces as the knee extends; patient describes seeing or feeling the kneecap "pop out and back in"

Symptoms

SymptomDescription
Acute, severe anterior knee painImmediate, localised to the patella
Swelling / effusionDevelops within hours post-injury; hemarthrosis common especially with osteochondral fracture
Inability to weight bearDifficulty walking and bending the knee
Giving wayEpisodic buckling - hallmark of recurrent instability
Catching / lockingSuggests loose osteochondral bodies
Pop or crackHeard/felt at time of dislocation; should not be confused with ACL rupture
Visible deformityPatella displaced laterally if not yet spontaneously reduced
StiffnessPost-injury; worse in the morning
Subluxation sensationPatella "threatening to shift" without complete dislocation (in chronic instability)
Numbness / paraesthesiaOccasional; from pressure on saphenous nerve or peroneal nerve with significant displacement

Physical Examination

Inspection:
  • Knee held in partial flexion (guarding)
  • Visible lateral displacement if still dislocated
  • Diffuse swelling and ecchymosis (especially medially - from medial retinaculum/MPFL tear)
  • Quadriceps wasting in chronic cases
Palpation:
  • Medial retinacular tenderness - the cardinal sign; along medial patellar border and at MPFL attachment on medial femoral condyle
  • Hemarthrosis / joint effusion - ballottement, fluctuance
  • Palpable defect in the medial retinaculum (acute)
  • Diffuse patellar tenderness
Special Clinical Tests:
TestTechniquePositive FindingSignificance
Patellar apprehension test (Fairbank's sign)Patient supine, knee at 20-30°; examiner pushes patella laterallyFear, quadriceps contraction, patient grabs examiner's handMost specific test for lateral instability; sensitivity ~39%, specificity ~92%
Patellar glide testDivide patella into 4 quadrants mediolaterally; assess excursion>3 quadrants lateral = hypermobility; <1 quadrant medial = lateral retinacular tightnessQuantifies passive restraints
J-signObserve patella tracking during active extension from 90° to 0°Patella jumps laterally ("J" shape) as it exits the trochlea near full extensionSuggests patella alta or trochlear dysplasia
Patellar tilt testExaminer lifts lateral patellar edge with knee extended<0° tilt (lateral edge cannot be elevated to neutral) = lateral retinacular tightnessGuides lateral release decision
Medial apprehension testMedially directed pressure on patellaApprehension = iatrogenic medial instability (post lateral release)
Clarke's testCompress patella into trochlea, patient contracts quadricepsPain under patellaNon-specific; patellofemoral chondromalacia
VMO assessmentPalpate VMO during straight leg raiseAtrophy or absent contractionChronic instability

SECTION 5 — INVESTIGATIONS AND EVALUATION

Step-by-Step Investigation Framework

ACUTE PRESENTATION
        ↓
1. Clinical examination → Confirm dislocation or spontaneous reduction
        ↓
2. Plain radiographs (AP + lateral ± Merchant/sunrise)
        ↓
3. Reduce if still dislocated → Post-reduction films
        ↓
4. Is there hemarthrosis or suspected osteochondral fracture?
   YES → MRI
   NO → Conservative management
        ↓
5. Recurrent/chronic instability: CT for TT-TG measurement
        ↓
6. Risk stratification → Guide surgical planning

Radiography (First-Line in All Cases)

Views Required:
ViewWhat It Shows
AP (weight-bearing)Overall alignment, fractures, loose bodies, patella position
True lateral (30° flexion)Patella alta/baja; trochlear morphology (crossing sign, supratrochlear spur, double contour); Blumensaat line
Axial/Merchant (30-45°)Patellar tilt and subluxation; sulcus angle; congruence angle; typically hard to obtain pre-reduction
Sunrise view (post-reduction)Confirms patella centred in groove; detects residual subluxation; osteochondral fragments
Radiographic findings in acute dislocation:
  • Empty or shallow trochlear groove (AP/axial)
  • Laterally displaced patella
  • Soft tissue swelling/effusion
  • Osteochondral fracture fragment at medial inferior patellar margin (pathognomonic avulsion) or lateral femoral condyle
  • Lipohemarthrosis on horizontal-beam lateral = intra-articular fracture
Trochlear dysplasia signs on lateral X-ray:
  • Crossing sign: Floor of trochlear groove crosses the anterior femoral condyle line = dysplasia present
  • Supratrochlear spur: Bony prominence proximal to trochlea = Type B/D
  • Double contour: Hypoplastic medial condyle line below crossing sign = Type C/D

MRI (Recommended for All Acute Dislocations with Effusion)

The International Patellofemoral Study Group recommends MRI for patients with large hemarthrosis to detect osteochondral fractures. (Rockwood and Green's, p. 3020)
MRI FindingSignificance
"Kissing contusions" - lateral femoral condyle + medial patellar bone bruisePathognomonic of lateral patellar dislocation mechanism
MPFL disruptionPresent in >90% of first-time dislocations; most commonly at patellar insertion
Osteochondral fractureArticular cartilage injury in up to 95% of first-time dislocators; patellar side (26.5%) > femoral side (15%)
Loose bodiesFree chondral/osteochondral fragments requiring arthroscopic removal
VMO injuryTear or haematoma in medial soft tissues
Cartilage mappingGrading of chondral damage on patellar and femoral surfaces
TT-TG measurementPossible on MRI but underestimates vs CT; CT preferred for this measurement

CT Scan (For Surgical Planning - TT-TG Measurement)

TT-TG distance measurement on axial CT
TT-TG distance: measured on superimposed axial CT slices comparing the tibial tubercle (TT) centre to the deepest point of the trochlear groove (TG). This quantifies the lateral displacement of the tibial tubercle relative to the trochlear groove.
TT-TG ValueInterpretation
9-13 mmNormal
15-20 mmBorderline; consider clinical context
>20 mmSignificantly abnormal; indication for tibial tubercle osteotomy
Other CT uses:
  • Precise trochlear depth and sulcus angle measurement
  • Torsional profile (femoral anteversion, tibial torsion)
  • Detect osseous loose bodies
  • Dynamic CT to assess patellar tracking during flexion

Arthroscopy (Selective)

Reserved for:
  • Large hemarthrosis with suspected loose osteochondral fragments
  • Concurrent intra-articular pathology
  • Pre-surgical assessment of cartilage
  • Removal of free fragments

Patient-Reported Outcomes

  • Kujala score (AKPS) - gold standard patellofemoral outcome tool
  • IKDC - International Knee Documentation Committee
  • KOOS - Knee Injury and Osteoarthritis Outcome Score
  • Lysholm score

SECTION 6 — TREATMENT

A. Emergency Management: Closed Reduction

Indications: Patella still dislocated on presentation.
Technique:
  1. Analgesia (oral/IV; conscious sedation usually not needed)
  2. Patient supine, hip flexed to relax hamstrings
  3. Gently and slowly extend the knee while simultaneously applying inferomedially-directed pressure to the patella
  4. If difficult: gentle downward pressure on the lateral patellar border to "open" the medial facet
  5. A "click" is felt/heard as the patella seats into the trochlea
  6. Neurovascular check immediately after reduction
  7. Post-reduction radiographs (AP + lateral + sunrise) to confirm centring and exclude osteochondral fracture
Post-reduction care:
  • Knee immobilizer in full extension
  • Weight bear as tolerated with crutches
  • Orthopedic follow-up within 1-2 weeks
  • Ice + elevation + NSAIDs for pain/swelling

B. Conservative (Non-Operative) Management

Indicated for: First-time dislocation without osteochondral fracture. (Campbell's, p. 1861; Rockwood and Green's, p. 3020)
PhaseTimelineTreatment
Phase 1 - Acute protection0-2 weeksKnee immobilizer in extension; partial weight-bearing; cryotherapy; NSAIDs; isometric quad sets
Phase 2 - Rehabilitation2-6 weeksTransition to patellar-stabilizing brace; progressive ROM; closed-chain quadriceps exercises with VMO emphasis; hip abductor/ER strengthening; patellar taping; proprioception training
Phase 3 - Return to sport6-12 weeksFull ROM, no effusion, quad strength ≥80% of contralateral side; sport-specific training
Outcomes of conservative treatment:
  • Recurrence rate: 15-44% (Rockwood & Green) to 15-75% (Campbell's)
  • No long-term functional difference vs surgery in first-time dislocators without osteochondral injury

C. Surgical Treatment

Indications for surgery:
IndicationEvidence Level
Displaced osteochondral fracture / free loose bodyStrong - most authors agree
Recurrent instability (≥2 dislocations)Strong
Large complete MPFL avulsion on MRIRelative indication
Patellar subluxation on Merchant view post-reductionRelative
First-time dislocation in high-risk anatomy (Dejour B/D, TT-TG >20 mm)Relative - growing consensus
High-level athletes with predisposing anatomyRelative
Persistent pain/laxity after failed conservative managementStrong

Surgical Procedure 1: MPFL Reconstruction (Proximal Realignment)

The current gold standard for recurrent lateral patellar instability.
Principle: Restore the medial soft tissue restraint by reconstructing the MPFL with a tendon graft.
Technique:
  • Graft: gracilis or semitendinosus tendon (autograft or allograft)
  • Patellar attachment: suture anchors or bone tunnels at superomedial patellar border
  • Femoral attachment: Schottle's point (anatomically critical)
Schottle's point - MPFL femoral attachment identification
Schottle's point - the anatomic origin of the MPFL on the medial femoral condyle. (A) Anatomical diagram: 1 mm anterior to the posterior cortex line, 2.5 mm distal to the posterior medial femoral condyle, proximal to Blumensaat line. (B) Lateral radiograph landmarks. (C) Intraoperative fluoroscopy for tunnel confirmation. Incorrect placement causes isometry failure: too proximal = graft tight in flexion; too distal = graft loose in flexion.
  • Miller's Review of Orthopaedics 9th Ed, p. 366
Procedures that have fallen out of favour:
  • Isolated lateral release (contraindicated for instability - causes medial iatrogenic dislocation)
  • VMO advancement (Green procedure)
  • Medial retinacular plication alone

Surgical Procedure 2: Tibial Tubercle Osteotomy (Distal Realignment)

Indicated when: TT-TG >20 mm, or symptomatic distal patellar chondromalacia (anteriorization component offloads the distal pole).
Main techniques:
ProcedureComponentsPrimary Indication
Fulkerson osteotomy (anteromedialization - AMZ)Osteotomy moves TT anteriorly + mediallyTT-TG >20 mm + distal patellar chondromalacia
Elmslie-TrillatPurely medial transfer of TTIsolated TT-TG elevation without chondral disease
MaquetAnterior transfer only (rarely used now)Isolated patellar chondromalacia
Contraindication to medialization: Proximal patellar arthrosis of the medial facet (would impinge the medially transferred patella against medial osteophytes).

Surgical Procedure 3: Trochleoplasty

  • Indicated for: High-grade trochlear dysplasia with supratrochlear bump (Dejour Type B, D, and V3.0 Type 3) where the bony deformity is too severe for soft tissue procedures alone
  • Technique: Bereiter sulcus-deepening trochleoplasty - the supratrochlear bump is resected/levelled by elevating a thin osteochondral flap, deepening the underlying groove, then reseating the cartilage flap
  • Usually combined with MPFL reconstruction
  • Technically demanding; requires significant surgical expertise
  • Indicated when: central bump >5 mm on MRI (V3.0 Type 3)

Current Treatment Algorithm (2024-2026)

ACUTE FIRST-TIME DISLOCATION
         ↓
Osteochondral fracture present?
   YES → Surgery (fix or remove fragment)
   NO → Conservative management
         ↓
RECURRENT INSTABILITY / SURGICAL CANDIDATE
         ↓
Step 1: ALWAYS include MPFL reconstruction (ruptured >90% of cases)
         ↓
Step 2: Evaluate TT-TG distance (CT)
         ↓
      TT-TG <20 mm              TT-TG >20 mm
      MPFL alone                 MPFL + tibial tubercle
                                  osteotomy (medialization)
         ↓
Step 3: Evaluate trochlear dysplasia (Dejour)
         ↓
    Type A/C (no bump)        Type B/D or V3.0 Type 3 (bump)
    No trochleoplasty          Add trochleoplasty
         ↓
Step 4: Evaluate patella height
         ↓
    Patella alta (CDI >1.2)    Target: PHI <1.16 post-op
    Consider tubercle          SPE >38%, TT-TG 8-14 mm post-op
    distalization

D. Specific Management of Rare Dislocation Types

TypeSpecific Management
Medial dislocation (iatrogenic)Medial retinacular release + lateral retinacular reconstruction; address over-corrected anatomy
Superior dislocationManual closed reduction (flex the knee while applying inferior pressure); surgical osteophyte removal if recurs; common in osteoarthritic knees
Habitual dislocationTypically requires surgery: MPFL reconstruction + trochleoplasty; lateral retinacular lengthening if the distal ITB/VL is too tight
Permanent dislocationSurgical: requires complete lateral soft tissue release + reconstruction; high complexity
Congenital dislocationOften associated with syndromes (Down, Larsen, nail-patella syndrome); early surgical correction before gait patterns are established
Intra-articular dislocationMay require surgical reduction + fixation/removal of osteochondral fragment

SECTION 7 — COMPLICATIONS

ComplicationDetail
Osteochondral fractureUp to 95% articular cartilage injury in first-time dislocators
Recurrent instability15-75% after conservative management; highest in young patients with dysplasia
Post-traumatic patellofemoral osteoarthritisLong-term consequence of cartilage damage
Iatrogenic medial instabilityAfter over-aggressive lateral release; disabling
MPFL graft failureIncorrect tunnel position (non-isometric placement); over-tensioning
Stiffness/loss of flexionEspecially after tibial tubercle osteotomy or prolonged immobilization
Osteotomy non-union / hardware issuesAfter tibial tubercle osteotomy
Saphenous nerve injuryAt risk in medial surgical approaches
Patellar fractureComplication of patellar tunnel MPFL technique

SECTION 8 — SUMMARY TABLE

ParameterKey Points
Most common typeLateral dislocation (~95%)
Peak incidenceAge 10-17 years; 29/100,000/year
Most important risk factorTrochlear dysplasia
Strongest instability predictorTT-TG >20 mm
MPFL rupture rate in acute dislocation>90%
First-line imagingAP + lateral + post-reduction sunrise X-ray
MRI indicationsHemarthrosis, suspected osteochondral fracture, effusion present
CT indicationTT-TG measurement for surgical planning
First-line treatmentClosed reduction + immobilizer + physiotherapy
Surgery - first-time dislocationOnly if osteochondral fracture/loose body, or high-risk anatomy
Surgery - recurrent instabilityMPFL reconstruction ± TTO ± trochleoplasty
TT-TG normal9-13 mm
Recurrence rate (conservative)15-75%
Gold standard surgical procedureMPFL reconstruction at Schottle's point

Sources cited:
  • Campbell's Operative Orthopaedics, 15th Ed 2026, p. 1861 - patellar dislocations, treatment
  • Miller's Review of Orthopaedics, 9th Ed, pp. 364-366 - patellar instability, Dejour classification, MPFL, surgical techniques
  • Rockwood and Green's Fractures in Adults, 10th Ed 2025, pp. 3019-3021 - acute patellar dislocations, preferred treatment
  • Rosen's Emergency Medicine, pp. 720-721 - clinical features, closed reduction technique
  • Tintinalli's Emergency Medicine - pediatric patellar dislocations
  • Patellar instability: current approach (PMC 2026) - Dejour V3.0 classification, 2024 updated treatment algorithm
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