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🦵 ALL CLINICALS OF THE KNEE JOINT
(MBBS 1st Year - Anatomy Exam Prep)
1. UNHAPPY TRIAD (O'Donoghue's Triad)
Structures injured: ACL + Tibial Collateral Ligament (MCL) + Medial Meniscus
Mechanism: Valgus + external rotation force on a flexed knee (e.g., football tackle from the side, skiing injury)
What happens step by step:
- Knee forced into valgus + external rotation
- ACL becomes taut (acts as fulcrum)
- MCL gets stressed and tears
- Medial meniscus tears (attached to MCL - so pulled along)
- ACL finally ruptures
Why swelling is delayed (hemarthrosis)?
- ACL is extra-synovial but intracapsular - rich blood supply
- When torn, blood enters joint cavity and irritates synovium
- Swelling develops gradually over hours (not immediate)
Medial meniscus is more commonly torn than lateral because:
- Medial meniscus is firmly attached to joint capsule AND MCL
- Less mobile - cannot "escape" the damaging force
- Lateral meniscus is free from capsule - more mobile, moves out of the way
2. MENISCAL INJURIES
Mechanism: Forceful rotation/twisting of the knee. Can occur even without significant trauma (degenerate tears in older patients).
Symptoms:
- Pain at medial or lateral joint line
- Knee locking or clicking (bucket-handle tear)
- Sensation of knee "giving way"
- Swelling (intermittent, delayed - unlike ligament tears)
Types of tears:
| Type | Description |
|---|
| Bucket-handle tear | Longitudinal tear; displaced fragment into intercondylar notch → causes locking |
| Vertical tear | Perpendicular to tibial plateau |
| Horizontal tear | Parallel to long axis of meniscus |
| Radial tear | Perpendicular to circumferential fibers; disrupts hoop stresses |
| Flap tear | |
| Degenerate tear | Older patients, no specific injury |
Investigation: MRI is gold standard (identifies tear type + associated injuries)
Clinical tests:
- McMurray's test - flex knee fully, then extend with internal/external rotation; click/pain at joint line = positive
- Apley's grind test - prone patient, knee 90°, compress + rotate tibia on femur; pain = positive
- Thessaly test - standing on affected leg, knee 20° flexion, rotate body; joint-line pain = positive
Treatment:
- Tears with mechanical symptoms (locking, giving way) → arthroscopic repair or debridement
- Factors favoring repair: young patient, early presentation, simple tear, vascular outer 1/3 of meniscus, stable knee
- Degenerate tears without mechanical symptoms → conservative first
Why meniscal loss is dangerous: Loss of meniscus removes shock absorption and hoop stress protection → accelerates cartilage degeneration → osteoarthritis
3. ANTERIOR CRUCIATE LIGAMENT (ACL) INJURY
Most common serious knee ligament injury
Mechanism: Twisting/landing injury in pivoting sports (football, basketball, skiing). Audible "pop," immediate swelling, inability to continue activity ("carried off field").
Why females are at higher risk:
- Smaller ligaments
- Smaller femoral notch
- Different landing biomechanics
Clinical Tests:
| Test | How | Positive = |
|---|
| Lachman test | Knee at 30° flexion, pull tibia anteriorly | Excessive anterior translation (most sensitive test) |
| Anterior drawer test | Knee at 90° flexion, pull tibia anteriorly | Anterior translation of tibia |
| Pivot shift test | Extend knee with valgus force + internal rotation | Clunk as tibia relocates (specific for ACL) |
Note: Anterior drawer can be falsely negative if hamstrings are in spasm (tense) or if the knee cannot be bent to 90° due to pain. Lachman test is therefore preferred.
Investigation: MRI - confirms tear, identifies associated meniscal/cartilage injury
Treatment:
- Initial: RICE, brace, physiotherapy, reduce swelling
- Surgical reconstruction: indicated in multiligament injuries, persistent instability, young/high-demand athletes
- Reconstruction uses: patellar ligament graft OR hamstring tendons (semitendinosus/gracilis) - hamstring grafts have less donor site morbidity
- Surgery best when: acute swelling has settled and knee has full range of motion
4. POSTERIOR CRUCIATE LIGAMENT (PCL) INJURY
Much less common than ACL (1:10 ratio)
Mechanism: Dashboard injury (knee hits dashboard in forced flexion - tibia pushed posteriorly); direct fall on flexed knee
Clinical Test:
- Posterior drawer test - knee at 90°, push tibia posteriorly → excessive posterior translation
- Sag sign - with knee at 90°, tibia sags posteriorly compared to other side
Treatment: Often conservative (PCL has better healing capacity than ACL)
5. COLLATERAL LIGAMENT INJURIES
Medial Collateral Ligament (MCL) Injury
Mechanism: Valgus stress (force from lateral side pushing knee medially)
- MCL has superficial and deep layers; deep layer attached to medial meniscus
- Most common collateral ligament injury
- Usually heals with conservative management (brace + physiotherapy)
Test: Valgus stress test at 0° and 30° flexion
Lateral Collateral Ligament (LCL) Injury
Mechanism: Varus stress
- LCL works with popliteus, biceps femoris, popliteofibular ligament, and iliotibial band = posterolateral corner (PLC)
- Less likely to heal conservatively
- Often requires surgical reconstruction
Test: Varus stress test at 0° and 30° flexion
6. BURSITIS AROUND THE KNEE
| Bursa | Location | Condition |
|---|
| Prepatellar bursa | Subcutaneous, anterior to patella | "Housemaid's knee" - repeated kneeling; swelling over patella |
| Superficial infrapatellar bursa | Subcutaneous, over patellar ligament | "Clergyman's knee" - kneeling more upright |
| Deep infrapatellar bursa | Deep to patellar ligament | Overuse/irritation |
| Suprapatellar bursa | Above patella, communicates with joint cavity | Fills with fluid in knee effusion |
| Semimembranosus bursa | Posterior medial | Baker's cyst |
Baker's Cyst (Popliteal Cyst)
- Distension of semimembranosus bursa (or posterior joint capsule herniation)
- Located in popliteal fossa, medial side
- Associated with intraarticular pathology (meniscal tear, OA, RA)
- Presents as posterior knee swelling
- Can rupture → pain/swelling in calf (mimics DVT)
- Treatment: address underlying cause; aspiration/excision if symptomatic
7. PATELLAR DISLOCATION
Direction: Almost always lateral dislocation
Mechanism: Twisting injury; more common in females, knock-kneed (genu valgum), shallow trochlear groove
Predisposing factors:
- Shallow femoral trochlear groove
- Genu valgum (knock knee)
- High-riding patella (patella alta)
- Weak VMO (vastus medialis oblique)
- Abnormal Q-angle (angle between quadriceps force and patellar tendon)
Clinical: Obvious deformity, medial retinaculum tear, medial patellar tenderness, associated osteochondral fracture (cartilage chip)
Apprehension test: Gently push patella laterally - patient becomes anxious/resists = positive
8. EXTENSOR MECHANISM RUPTURE
Structures: Quadriceps tendon OR patellar tendon rupture
Risk factors: Steroid use, steroid injections, chronic kidney disease, previous knee surgery
Presentation: Sudden pain, swelling, inability to actively extend knee, palpable gap in tendon
Treatment: Urgent surgical repair (delay leads to permanent loss of extension)
9. OSTEOCHONDRAL / ARTICULAR CARTILAGE INJURY
Important features:
- Hyaline cartilage = no blood vessels, no lymphatics, no nerves → very limited healing capacity
- Partial-thickness defects: do NOT heal
- Full-thickness defects in young: may form fibrocartilage repair tissue
- Can be associated with ACL tears or patellar dislocations
Treatment options for full-thickness defects:
- Microfracture - drill holes through subchondral bone to release marrow cells
- Microfracture + collagen membrane (enhanced)
- Mosaicplasty - osteochondral plugs transplanted from non-weight-bearing area
- ACI/MACI - autologous chondrocyte implantation (harvest, culture, re-implant)
- Osteochondral allograft
10. OSTEOARTHRITIS (OA) OF KNEE
Most common joint disease - degenerative loss of articular cartilage
Risk factors: Age, obesity, previous injury/meniscectomy, malalignment
Compartments: Medial > lateral > patellofemoral
X-ray findings (4 key signs):
- Loss of joint space (especially medial)
- Subchondral sclerosis (bone density increases under damaged cartilage)
- Osteophytes (bony spurs at joint margins)
- Subchondral cysts
Deformity: Medial OA → varus deformity (bow-legged)
Treatment ladder:
- Conservative: weight loss, physiotherapy, analgesia, bracing
- Injections: corticosteroid, hyaluronic acid
- Surgical: unicompartmental knee replacement (UKR) or total knee replacement (TKR)
TKR details (exam favourite):
- Femoral surface = metal; tibial surface = polyethylene in metal baseplate
- Cemented with PMMA (polymethylmethacrylate)
- CR (cruciate retaining) or PS (PCL sacrificing) designs
- Revision rate < 5% at 15 years
- Goal: correct mechanical axis, balance ligaments
11. GENU VALGUM & GENU VARUM
| Genu Valgum | Genu Varum |
|---|
| Common name | Knock knee | Bow leg |
| Deformity | Knees touch, ankles apart | Ankles touch, knees apart |
| Mechanical axis | Lateral to knee | Medial to knee |
| Compartment OA | Lateral compartment | Medial compartment |
| Normal in children | Up to age 7 years | Up to age 2 years |
12. COMMON FIBULAR NERVE INJURY AT THE KNEE
Most commonly injured nerve at the knee - winds around neck of fibula (very superficial)
Causes: Fracture of fibular neck, tight plaster cast, pressure (crossing legs), knee dislocation
Result: Foot drop
- Loss of dorsiflexion (deep fibular nerve)
- Loss of eversion (superficial fibular nerve)
- High-stepping gait
- Sensory loss over dorsum of foot and lateral leg
13. POPLITEAL ARTERY INJURY
Most vulnerable in posterior knee dislocations (high-energy trauma)
Why dangerous: Popliteal artery is the deepest structure in the popliteal fossa, tightly bound to posterior joint capsule → torn in severe dislocations
Must check: Distal pulses (dorsalis pedis, posterior tibial), capillary refill, ABI (ankle-brachial index)
Complications: Limb ischemia → compartment syndrome → amputation if not urgently revascularized
14. ARTHROSCOPY OF THE KNEE
Entry portals: Anterolateral and anteromedial aspects of the knee
Uses: Diagnosis + treatment of meniscal tears, ACL reconstruction, cartilage procedures, synovial biopsy, removal of loose bodies
Advantage over open surgery: Less trauma, faster recovery, lower infection risk
Quick Clinical Test Summary Table
| Test | Structure tested | Positive finding |
|---|
| Lachman test (30° flex) | ACL | Anterior tibial translation |
| Anterior drawer (90° flex) | ACL | Anterior tibial translation |
| Posterior drawer (90° flex) | PCL | Posterior tibial translation |
| Pivot shift | ACL | Clunk on extension |
| Valgus stress test | MCL | Medial joint opening |
| Varus stress test | LCL | Lateral joint opening |
| McMurray's | Meniscus | Click + joint line pain |
| Apley's grind | Meniscus | Pain on compression + rotation |
| Patellar apprehension | Patella stability | Anxiety + resistance to lateral push |
| Sag sign | PCL | Tibia sags posteriorly at 90° |
| Thompson test | Achilles tendon | No plantarflexion on calf squeeze |
Sources: Gray's Anatomy for Students; Bailey & Love's Short Practice of Surgery 28th Ed; THIEME Atlas of Anatomy - General Anatomy & Musculoskeletal System