Now I have excellent ECG images. Let me compile the comprehensive guide with all 10 conditions, ECG images, and detailed lead-by-lead findings.
10 Common Heart Diseases - ECG Guide (All 12 Leads)
ECG Anatomy & Lead Reference
This master diagram shows which leads correspond to which coronary artery territory - essential for localizing infarctions.
1. Normal Sinus Rhythm (Baseline Reference)
Rate: 60-100 bpm | Rhythm: Regular
| Lead Group | Findings |
|---|
| I, II, III | Upright P waves before every QRS |
| aVR | Negative P wave (normal) |
| aVL, aVF | Small upright P waves |
| V1-V2 | Small r wave, deep S wave |
| V3-V6 | Progressive R wave growth (R wave progression) |
| PR interval | 120-200 ms (3-5 small squares) |
| QRS | <120 ms (narrow) |
| QT | <440 ms in men, <460 ms in women |
2. Inferior STEMI (ST-Elevation Myocardial Infarction)
Culprit vessel: Right Coronary Artery (RCA) in ~80% of cases
Lead-by-lead step-by-step:
| Lead | Finding |
|---|
| II, III, aVF | ST elevation ≥1mm (diagnostic territory) - the classic inferior leads |
| I, aVL | Reciprocal ST depression - mirror image of III; aVL is exact inverse of lead III |
| V1-V3 | May show ST depression (posterior extension); check V4R for RV involvement |
| V4R | ST elevation ≥1mm = right ventricular MI (occurs in 40% of inferior STEMI) |
| V5-V6 | ST elevation here means extensive disease and worse prognosis |
| aVR | Typically ST depression |
Key rule: ST elevation in III > II suggests RCA occlusion. aVL reciprocal change is the most sensitive sign.
3. Anterior STEMI
Culprit vessel: Left Anterior Descending artery (LAD)
Lead-by-lead findings:
| Lead | Finding |
|---|
| V1-V4 | ST elevation (V2-V3 most prominent; criteria: ≥2mm in men, ≥1.5mm in women) |
| V5-V6, I, aVL | May show lateral ST elevation if ostial/proximal LAD occlusion |
| II, III, aVF | Reciprocal ST depression (if large anterior/proximal occlusion) |
| aVR | ST elevation in aVR + diffuse depression = left main coronary artery (LMCA) occlusion |
| V1-V2 | "Tombstone" pattern (massive STE) = proximal LAD, worst prognosis |
Hyperacute T waves (tall, bulky, asymmetric) appear within minutes - earliest ECG sign.
4. Atrial Fibrillation (AF)
The most common sustained arrhythmia
Lead-by-lead findings:
| Lead | Finding |
|---|
| All leads | Irregularly irregular R-R intervals - no two complexes the same distance apart |
| II, V1 | Absent P waves - replaced by chaotic fibrillatory "f" waves (350-600/min) |
| V1 | Fibrillatory waves may be most visible here (coarse vs. fine AF) |
| All leads | QRS complexes are usually narrow (unless aberrant conduction or pre-existing BBB) |
| Rate | Uncontrolled: 100-180 bpm; Controlled: 60-100 bpm |
Rate: Irregularly irregular with no discernible P waves is the diagnostic pattern. Coarse AF shows visible f-waves; fine AF may appear as flat baseline.
5. Complete Heart Block (3rd Degree AV Block)
AV node completely fails to conduct - atria and ventricles beat independently
Lead-by-lead findings:
| Lead | Finding |
|---|
| All leads | P waves present at normal sinus rate (60-100/min) but with NO relationship to QRS |
| II (rhythm strip) | Best to see P-P regularity and R-R regularity - both regular but independent |
| V1-V6 | Wide, bizarre QRS if ventricular escape (>120ms); narrow if junctional escape |
| II, III, aVF | Escape rate: junctional (40-60 bpm, narrow QRS) vs. ventricular (20-40 bpm, wide QRS) |
| Rate | Atrial rate 60-100, ventricular escape rate 20-60 |
Key: P waves "march through" QRS complexes with no fixed PR interval - classic AV dissociation.
6. Wolff-Parkinson-White (WPW) Syndrome
Accessory pathway (Bundle of Kent) bypasses the AV node
Lead-by-lead findings (sinus rhythm WPW):
| Lead | Finding |
|---|
| All leads | Short PR interval <120ms (pre-excitation bypasses AV node delay) |
| I, V5-V6 | Delta wave - slurred upstroke at start of QRS (slow accessory pathway conduction) |
| V1 | Positive delta = Type A WPW (left-sided pathway); Negative delta = Type B (right-sided) |
| V2-V6 | Broad QRS (>120ms) due to fusion of normal + accessory conduction |
| V1-V3 | Pseudo-R wave or pseudo-Q wave mimicking MI |
In WPW + AF (as shown above): irregular, pre-excited wide complex tachycardia - potentially life-threatening (can degenerate to VF). Never give AV nodal blocking agents.
7. Ventricular Tachycardia (VT)
Regular broad-complex tachycardia arising from ventricular ectopic focus
Lead-by-lead findings:
| Lead | Finding |
|---|
| All leads | Wide QRS (>120ms) at rate 100-250 bpm, regular rhythm |
| II (rhythm strip) | AV dissociation: P waves visible but unrelated to QRS (pathognomonic of VT) |
| V1 | LBBB morphology (positive) or RBBB morphology depending on origin site |
| V1-V6 | Concordance (all QRS pointing same direction) strongly suggests VT |
| Capture/fusion beats | Random narrow QRS or hybrid complex during VT - diagnostic of VT |
| Axis | Often extreme left axis deviation ("northwest axis") |
Brugada criteria and Vereckei algorithm use these lead-specific patterns to distinguish VT from SVT with aberrancy.
8. Pericarditis
Saddle-shaped diffuse ST elevation without reciprocal changes (except aVR)
Lead-by-lead findings (Stage 1 - Acute):
| Lead | Finding |
|---|
| I, II, aVF, V2-V6 | Diffuse ST elevation - saddle-shaped (concave upward), nearly all leads |
| aVR (and V1) | Reciprocal ST depression + PR elevation |
| All leads | PR depression - the most specific sign for pericarditis |
| aVR | PR elevation (unique - opposite of all other leads) |
| No | Reciprocal changes in inferior/lateral leads (distinguishes from STEMI) |
4-Stage ECG evolution:
- Stage 1 (hours-days): Diffuse STE + PR depression
- Stage 2 (days): ST normalizes, T waves flatten
- Stage 3 (weeks): T wave inversions
- Stage 4 (weeks-months): ECG normalizes
ST/PR ratio >0.25 in lead II is 94% sensitive for pericarditis.
9. Left Bundle Branch Block (LBBB)
Delayed conduction through the left bundle - important marker of structural disease
Lead-by-lead findings:
| Lead | Finding |
|---|
| V1 | Deep, broad S wave (QS or rS pattern) - "W" shape in V1 |
| V5-V6, I, aVL | Broad, notched R wave (>120ms) - "M" shape in lateral leads |
| V1-V4 | No septal Q waves (normally Q in V5-V6, I, aVL are absent in LBBB) |
| V5-V6, I | Discordant ST-T changes - ST and T point opposite to QRS |
| II, III, aVF | May have broad S wave |
| QRS | Must be ≥120ms to qualify as complete LBBB |
New LBBB + chest pain = STEMI equivalent until proven otherwise. Use Sgarbossa criteria (concordant ST elevation ≥1mm, or discordant ST elevation ≥25% of QRS depth) to detect STEMI in LBBB.
10. Pulmonary Embolism (PE)
Classic ECG shows right heart strain pattern - often subtle
Lead-by-lead findings:
| Lead | Finding |
|---|
| V1-V3 | ST depression + T wave inversion (right heart strain) |
| V1 | New RBBB pattern (rSR') in large PE |
| II (rhythm strip) | Sinus tachycardia (most common ECG finding in PE) |
| III, aVF | S1Q3T3 pattern - S wave in I, Q wave in III, inverted T in III |
| I | New S wave (right axis shift) |
| II, III, aVF | P pulmonale (peaked P waves >2.5mm if severe) |
| aVR | May show ST elevation |
S1Q3T3 is classic but only present in ~20% of PE cases. Sinus tachycardia is the most sensitive but non-specific sign. New RBBB + T inversion V1-V4 in a hypoxic patient is highly suggestive.
ECG Special Patterns Quick Reference
The image above shows advanced STEMI-equivalent (OMI) patterns across leads including Wellens syndrome, De Winter T-waves, posterior OMI, and Modified Sgarbossa criteria.
Quick Lead Localization Summary Table
| Territory | Leads | Artery |
|---|
| Inferior | II, III, aVF | RCA (80%), LCx (20%) |
| Anterior | V1-V4 | LAD |
| Lateral | I, aVL, V5-V6 | LCx / diagonal LAD |
| Posterior | V1-V2 (reciprocal depression) | RCA or LCx |
| Right Ventricle | V4R | Proximal RCA |
| Septal | V1-V2 | LAD (septal branch) |
Step-by-Step ECG Reading Protocol (Every 12-lead)
- Rate - 300/R-R intervals in large squares
- Rhythm - Regular or irregular? P before every QRS?
- Axis - Lead I and aVF: both positive = normal axis
- P wave - Present, upright in II, morphology
- PR interval - 120-200ms (3-5 small squares)
- QRS duration - <120ms (narrow) vs. wide
- ST segment - Elevation or depression vs. TP baseline
- T waves - Upright in I, II, V2-V6; inverted in aVR (normal)
- QT interval - Correct for rate (QTc); abnormal if >440ms men / >460ms women
- Compare to prior ECG - Changes over time are often more informative than any single trace
Sources: LITFL ECG Library - Life in the Fast Lane, ECGlibrary.com, and ECG anatomy diagram via LITFL/Visible Body. All ECG images are from open-access educational resources.