Here are the full detailed answers for Case 10 and Case 20.
CASE 10
A 19-year-old primigravida at 37th week of gestation presents with lower abdominal pain, vaginal bleeding, weakness and giddiness. Ps – 100, BP – 100/60 mmHg. Uterus is firm and tender. FHR – 90, weak.
1. Primary Diagnosis
Abruptio Placentae (Placental Abruption) - Severe (Grade 2-3), with Acute Fetal Distress
Clinical reasoning point by point:
Lower abdominal pain + vaginal bleeding at 37 weeks:
This combination in the third trimester must immediately raise suspicion for abruptio placentae. The hallmark of abruption is painful antepartum bleeding (contrasting with placenta previa where bleeding is classically painless).
Uterus FIRM and TENDER:
This is the single most pathognomonic finding of abruptio placentae. The mechanism is as follows: blood accumulates between the placenta and the uterine wall forming a retroplacental haematoma. This blood infiltrates between the myometrial muscle fibres, causing intense uterine irritability and a state of persistent hypertonic contraction. The uterus does not relax - it remains continuously tense, hard (described as "wooden" or "board-like"), and exquisitely tender to palpation. This is called uterotonic spasm or hypertonus.
In contrast, placenta previa has a SOFT, NON-TENDER uterus - this one distinguishing feature separates the two diagnoses on clinical examination.
Maternal haemodynamic compromise (Pulse 100, BP 100/60):
This indicates significant blood loss. It is important to note that in abruption, the VISIBLE external bleeding often significantly underestimates total blood loss, because blood may be trapped behind the placenta as a concealed retroplacental haematoma. A patient can lose 1500-2000 mL internally with minimal external bleeding. This is the "concealed" type of abruption, and it is more dangerous precisely because the severity is not apparent.
FHR 90/min, weak:
Normal fetal heart rate is 110-160 bpm. A rate of 90 bpm sustained is a late deceleration equivalent / severe bradycardia - this represents profound uteroplacental insufficiency. The retroplacental haematoma compresses the placental intervillous space, cutting off oxygen exchange between maternal and fetal circulations. The fetus is acutely hypoxic and acidotic. A weak fetal heart tone suggests the Doppler or Pinard auscultation signal is poor, consistent with severe fetal compromise.
Weakness and giddiness:
Symptoms of hypovolaemic shock from blood loss - reduced cerebral perfusion.
Classification (Page's Classification - as per DC Dutta's Obstetrics):
| Grade | Description |
|---|
| Grade 0 | Clinically silent; retroplacental clot found after delivery |
| Grade 1 (Mild) | External bleeding, mild uterine tenderness, no maternal or fetal compromise |
| Grade 2 (Moderate) | Moderate bleeding (external + concealed), definite uterine tenderness, fetal distress but fetus alive |
| Grade 3 (Severe) | Massive haemorrhage, board-like uterus, maternal shock, fetal death |
This patient = Grade 2 to Grade 3 (severe Grade 2 at minimum). Fetal distress is present (FHR 90) and the mother is haemodynamically compromised. Whether the fetus is still alive needs urgent USS confirmation.
Types of Abruption:
- Revealed (External): Blood tracks through cervix and is visible vaginally - accounts for ~80% of cases
- Concealed: Blood trapped behind placenta, no external bleeding - accounts for ~20%; the most dangerous type as blood loss is underestimated
- Mixed: Both external and concealed components
2. Plan of Investigations
CRITICAL NOTE: Investigations must run SIMULTANEOUSLY with resuscitation - do not delay treatment while waiting for results.
Immediate Bedside/STAT Investigations:
1. Full Blood Count (CBC):
- Haemoglobin and Haematocrit: assess severity of anaemia
- Platelet count: thrombocytopenia suggests developing DIC (Disseminated Intravascular Coagulation)
- WBC: baseline; elevated with stress response
2. Blood Group and Crossmatch (URGENT):
- Type and screen URGENTLY
- Crossmatch minimum 4-6 units packed red blood cells and 4-6 units Fresh Frozen Plasma (FFP)
- If exsanguinating and crossmatched blood not available: O-negative uncrossmatched blood
3. Coagulation Profile:
- Prothrombin Time (PT) and INR
- Activated Partial Thromboplastin Time (aPTT)
- Serum Fibrinogen - THE most important test; fibrinogen is consumed early in DIC
- Normal in pregnancy: ≥400 mg/dL (pregnancy is a hypercoagulable state with elevated baseline fibrinogen)
- Fibrinogen 200-400 mg/dL: early coagulopathy
- Fibrinogen <200 mg/dL: significant DIC
- Fibrinogen <100 mg/dL: severe DIC, major haemorrhage risk
- D-dimer: Elevated in DIC (fibrin degradation products)
- Thrombin time
4. Bedside Clot Observation Test (Whole Blood Clotting Time):
- Draw 5 mL blood into a plain glass tube; observe
- Normally a firm clot forms within 6 minutes
- If no clot forms in 6 minutes or clot is soft/lysed = significant DIC
- A RAPID, cheap bedside test - available even in resource-limited settings
- Williams Obstetrics and DC Dutta both emphasise this test in managing abruption
5. Serum Electrolytes, Urea, and Creatinine:
- Assess renal function
- Acute Tubular Necrosis (ATN) is a recognised complication of severe abruption - from renal cortical ischaemia due to hypovolaemia + DIC microthrombi in glomerular capillaries
- In severe cases: Bilateral Renal Cortical Necrosis (catastrophic, irreversible)
6. Liver Function Tests (LFTs):
- Exclude HELLP syndrome (haemolysis, elevated liver enzymes, low platelets) as a co-existing condition or mimicker
- Serum uric acid (elevated in preeclampsia)
7. Blood Glucose:
- Hypoglycaemia can complicate shock
8. Urine Output Monitoring:
- Insert Foley catheter immediately
- Target urine output ≥30 mL/hour (0.5 mL/kg/hour)
- Oliguria is the earliest sign of renal compromise
9. Serum Lactate:
- Lactate ≥2 mmol/L = sepsis/tissue hypoperfusion
- Lactate ≥4 mmol/L = septic/haemorrhagic shock with poor tissue perfusion
- Guides resuscitation adequacy
Fetal Assessment:
10. Cardiotocography (CTG) - Continuous Electronic Fetal Monitoring:
- Connect immediately
- FHR of 90 bpm sustained is a Category III (non-reassuring/pathological) pattern
- Look for: late decelerations, variable decelerations, sinusoidal pattern, absent variability
- Confirms degree of fetal compromise and urgency for delivery
11. Ultrasound (USS):
- Confirm fetal cardiac activity - is the fetus alive? (This guides whether vaginal or CS delivery is selected)
- Identify retroplacental clot: echogenic/hypoechoic area behind placenta - though USS has only ~50% sensitivity for acute abruption (fresh blood is isodense with placenta on USS)
- Estimate size of the haematoma
- IMPORTANT: A normal USS does NOT exclude abruption - the diagnosis is CLINICAL
- Confirm fetal presentation, AFI, placental location (to exclude co-existing placenta previa)
- Fetal biometry (estimated fetal weight)
12. Kleihauer-Betke Test:
- If mother is Rh-negative: quantify feto-maternal haemorrhage (FMH) to calculate correct dose of anti-D immunoglobulin
- Qualitative: confirms FMH occurred (maternal blood contains fetal cells)
Additional (if clinically stable):
13. CRP and Procalcitonin: Infection/sepsis screen (usually not needed acutely in abruption unless infection co-exists)
14. Thyroid function, LFTs extended panel: If preeclampsia/HELLP suspected
3. Management
This is a LIFE-THREATENING OBSTETRIC EMERGENCY. The treatment is simultaneous: RESUSCITATE the mother and DELIVER the baby.
The principle in abruptio placentae is: "Delivery is the ONLY definitive treatment." The placenta will not re-attach; ongoing separation will worsen maternal and fetal outcome with every passing minute.
A. IMMEDIATE RESUSCITATION
Airway:
- Ensure patent airway
- Supplemental oxygen via non-rebreather face mask at 10-15 L/min
- Maintain SpO2 >95%
- If unconscious or GCS falling: intubation and ventilation
Breathing:
- Monitor respiratory rate and SpO2
- If pulmonary oedema develops (from massive transfusion or pre-existing pre-eclampsia): positive pressure ventilation
Circulation:
- Two large-bore peripheral IV cannulas (14-16 gauge) - do both simultaneously
- IV Fluid resuscitation: Ringer's Lactate or Normal Saline 1-2 litres rapidly, then reassess vital signs
- Blood transfusion: Start packed red blood cells (pRBCs) as soon as crossmatched blood available; use 1:1:1 ratio (pRBC : FFP : platelets) if massive transfusion protocol activated
- If blood not immediately available and patient exsanguinating: O-negative uncrossmatched blood
- Target: Systolic BP ≥90 mmHg, HR <100, urine output ≥30 mL/hour, Hb ≥8 g/dL
Foley Catheter:
- Insert immediately; strict monitoring of urine output hourly
- Oliguria (<20 mL/hour) = renal compromise; escalate management
Monitoring:
- Continuous pulse oximetry
- Continuous CTG
- BP every 15 minutes
- Urine output hourly
- Repeat CBC, coagulation profile every 2-4 hours until stable
B. MANAGEMENT OF DIC (if present)
DIC complicates severe abruption in approximately 10-15% of cases. It occurs because:
- Massive tissue factor (thromboplastin) release from the damaged decidua and placenta activates the coagulation cascade
- Simultaneous fibrinolysis consumes clotting factors
- Result: paradoxical simultaneous thrombosis (microthrombi) and bleeding (factor depletion)
Treatment of DIC:
- Fresh Frozen Plasma (FFP): 2-4 units IV (each unit contains all clotting factors; 1 unit raises fibrinogen by ~10 mg/dL)
- Cryoprecipitate: 10 units IV (rich in fibrinogen, factor VIII, vWF; 10 units raise fibrinogen by ~150 mg/dL; preferred when fibrinogen <1 g/L)
- Platelet Concentrate: Transfuse if platelets <50,000/μL with active bleeding; aim to keep >80,000 if CS planned
- Tranexamic Acid 1 g IV over 10 minutes (antifibrinolytic; the WOMAN trial demonstrated significantly reduced death from PPH when given within 3 hours of haemorrhage onset)
- Haematologist involvement in massive haemorrhage protocol
- Do NOT give heparin in obstetric DIC (worsens bleeding)
C. DELIVERY - MODE AND TIMING
Delivery must not be delayed. Every contraction in abruption can further separate the placenta, worsening both maternal haemorrhage and fetal hypoxia.
If Fetus is ALIVE with Fetal Distress (this case):
Emergency Caesarean Section (CS) is the treatment of choice.
Rationale:
- Term fetus (37 weeks), alive but in severe distress (FHR 90, weak)
- Vaginal delivery is not imminent (cervical status not stated, but abruption may or may not be in labour; even if some dilatation is present, the fetal distress requires immediate delivery)
- CS delivers the fetus in the shortest possible time
- Grade 2-3 abruption with live fetus = indication for immediate CS in most guidelines
Anaesthesia for CS:
- If haemodynamically stable: Spinal anaesthesia (faster onset than epidural, avoids maternal airway manipulation)
- If haemodynamically unstable or coagulopathy present: General Anaesthesia (GA) - faster to induce; avoids risks of spinal in hypovolaemic patient (profound hypotension) and unsafe if coagulopathy (epidural haematoma risk)
Intraoperative findings to anticipate:
- Couvelaire Uterus (Uteroplacental Apoplexy): Blood infiltrates into the myometrium between muscle fibres, giving the uterus a bruised, purple-blue, mottled appearance. Named after the French obstetrician Alexandre Couvelaire. The Couvelaire uterus is atonic - it does NOT contract well, setting the stage for severe PPH.
- Retroplacental clot: Found on the maternal surface of the placenta; confirms diagnosis
- Uterine atony post-delivery: Uterus feels "boggy" and fails to contract despite oxytocin
Prevention and Treatment of PPH at CS:
- Oxytocin infusion: 20-40 IU in 500 mL NS, run over 4-8 hours after delivery of anterior shoulder
- Ergometrine 0.2-0.5 mg IV (caution in hypertension)
- Carboprost (PGF2α) 0.25 mg IM every 15 minutes (max 8 doses) - avoid in asthma
- Misoprostol 800-1000 mcg PR or SL
- B-Lynch suture: Compression suture around the uterus - first-line surgical option for atony at CS
- Uterine artery ligation (O'Leary stitch)
- Internal iliac artery ligation
- Peripartum hysterectomy: Last resort for uncontrolled haemorrhage - life-saving. Higher risk in Couvelaire uterus.
- Balloon tamponade (Bakri balloon): Can be used at CS if uterus is closing and still oozing
Neonatologist must be in theatre at delivery: The neonate will likely be severely asphyxiated; immediate resuscitation required (bag-mask ventilation, intubation, chest compressions if HR <60, cord blood gases).
If Fetus is DEAD (Grade 3 with absent cardiac activity on USS):
Vaginal delivery is preferred (to avoid surgical risks in a coagulopathic mother):
- Artificial Rupture of Membranes (ARM) if not already done
- Oxytocin augmentation (20-40 IU in 500 mL NS)
- Allow labour to progress with close monitoring
- Most patients deliver within 6-8 hours
- CS reserved for: maternal exsanguination not responding to resuscitation, unfavourable cervix with expected prolonged labour, other obstetric indication (e.g. obstruction)
D. POSTOPERATIVE/POST-DELIVERY CARE
Monitor for complications:
- PPH (uterine atony from Couvelaire uterus, DIC) - most immediate danger
- Acute Kidney Injury / Acute Tubular Necrosis:
- From renal ischaemia (prolonged hypotension) + DIC (glomerular microthrombi)
- In severe cases: Bilateral Renal Cortical Necrosis (irreversible, requires dialysis)
- Monitor urine output strictly; renal replacement therapy if oliguria persists despite adequate resuscitation
- DIC (may worsen or develop post-delivery)
- Pulmonary oedema (from massive transfusion, fluid overload, or pre-eclampsia)
- Sheehan's Syndrome (anterior pituitary necrosis from prolonged hypotension - presents weeks/months later as failure of lactation, amenorrhoea, adrenal/thyroid insufficiency)
- ARDS (Acute Respiratory Distress Syndrome) - from shock, massive transfusion
- Maternal death - severe abruption with DIC and shock is one of the leading causes of direct maternal mortality
ICU/HDU admission post-delivery if:
- Received massive transfusion
- DIC present
- Haemodynamic instability
- Oliguria/AKI
Anti-D immunoglobulin 300 mcg IM if Rh-negative mother (within 72 hours of delivery)
Counselling:
- Explain the diagnosis and what happened to the patient and family
- Recurrence risk in subsequent pregnancy: approximately 10-15x increased risk of abruption in next pregnancy
- Counsel regarding modifiable risk factors: smoking cessation, blood pressure control, aspirin prophylaxis in next pregnancy (if hypertensive disorders co-existed)
- Psychological support if fetal death occurred
CASE 20
Primipara in labour 10 hours, rupture of amniotic sac 4 hours ago. Contractions regular, for 50 sec, every 1 min. Heartbeat of fetus dull, arrhythmic, 105-110/min. On vaginal examination: opening of cervix complete, head of fetus in pelvic floor, sagittal suture in exit plane, small fontanel in front from symphysis.
1. Diagnosis
Acute Intrapartum Fetal Distress (Fetal Asphyxia) in Second Stage of Labour with Direct Occiput Anterior (DOA) presentation at Outlet
Let me analyse every component systematically:
"Small fontanel (posterior fontanelle) in front from symphysis":
The posterior fontanelle is the triangular, smaller fontanelle located at the junction of the sagittal and lambdoid sutures. It represents the occiput. When it is felt ANTERIORLY (in front of/towards the symphysis pubis), it means the occiput is pointing anteriorly - this is Occiput Anterior (OA) position. More specifically, if the posterior fontanelle is directly behind the symphysis (midline anterior), this is Direct Occiput Anterior (DOA) - the ideal position for vaginal delivery.
The large/anterior fontanelle (bregma) is diamond-shaped and would be felt posteriorly in this case (towards the sacrum) - consistent with DOA.
"Sagittal suture in exit plane (outlet plane)":
The sagittal suture runs between the two parietal bones, from anterior to posterior fontanelle. Its orientation in the "exit/outlet plane" means it is aligned with the anteroposterior (AP) diameter of the outlet. In OA, the sagittal suture aligns AP at the outlet - this is correct and expected for DOA delivery.
"Head in the pelvic floor":
The pelvic floor = levator ani muscles = station +3 to +4. The head is at or on the perineum - this is outlet territory. Delivery is imminent.
"Complete opening of cervix, amniotic sac absent":
- Cervix fully dilated (10 cm) = second stage ✓
- Membranes ruptured 4 hours ago ✓
Labour pattern:
- Contractions every 1 minute for 50 seconds = hyperstimulation/very frequent, strong contractions
- This pattern (contractions every 1 minute) is abnormal - normal active labour has contractions every 2-3 minutes. Contractions this frequent reduce the diastolic relaxation phase → reduced uteroplacental blood flow between contractions → fetal hypoxia accumulates
Fetal heart rate 105-110/min, DULL (muffled), ARRHYTHMIC:
This is a Category III (pathological/non-reassuring) fetal heart rate pattern - severe fetal distress:
- FHR 105-110: borderline bradycardia (normal 110-160 bpm)
- Dull/muffled: Reduced amplitude of heart tones = fetal cardiac depression from hypoxia/acidosis
- Arrhythmic: Loss of normal beat-to-beat variability + irregular rhythm = profound hypoxia-acidosis causing cardiac depression; variability loss is the most ominous CTG sign
- Combined: this is a fetus in severe acute asphyxia
Labour duration context:
- 10 hours total labour in a primipara = not prolonged (within normal limits for first stage)
- The issue is not prolonged labour per se - the issue is acute fetal distress with the head already at the outlet
Complete Diagnosis:
Acute Intrapartum Fetal Asphyxia / Fetal Distress in Second Stage of Labour, with Direct Occiput Anterior Presentation at Pelvic Outlet. Immediate Operative Vaginal Delivery Indicated.
2. Tactic (Plan) of Management
This is an obstetric emergency requiring IMMEDIATE delivery. The fetus is at the pelvic floor in a favourable position. Every minute of delay increases hypoxic-ischaemic brain injury.
Step 1 - Call for Emergency Help
Immediately summon:
- Senior obstetrician
- Anaesthetist
- Neonatologist/Paediatrician - essential; this neonate will need immediate resuscitation for birth asphyxia
- Scrub nurse/midwife
- Inform NICU to be ready
Step 2 - Immediate Maternal Management
- High-flow oxygen: 10-15 L/min via non-rebreather mask to maximise oxygen delivery to the fetus via placenta
- Maternal position: Left lateral decubitus (left lateral tilt) - relieves aortocaval compression by the gravid uterus; improves venous return and cardiac output; increases uteroplacental blood flow
- IV access (if not already in situ): 16-18G cannula
- Stop oxytocin (if it was running): Hyperstimulation (contractions every 1 minute) must be stopped immediately. Oxytocin should be discontinued or reduced to allow uterine relaxation between contractions. If uterine hyperstimulation is the cause of fetal distress, this single intervention may temporarily improve FHR.
- Tocolysis for acute fetal resuscitation (if time permits): Terbutaline 0.25 mg SC (beta-2 agonist) causes uterine relaxation, improving uteroplacental blood flow during preparation for delivery. Used as a temporising measure ONLY - not a substitute for delivery.
- Empty the bladder: Foley catheter if not already done. A full bladder obstructs head descent and complicates instrumental delivery.
Step 3 - Position the Patient for Delivery
- Lithotomy position on the delivery table/bed
- Legs in lithotomy stirrups, buttocks slightly off the edge of the table
- Perineum cleansed with antiseptic (Betadine/chlorhexidine)
- Sterile draping
Step 4 - Anaesthesia
Pudendal nerve block (bilateral):
- Landmark: Ischial spine (felt on VE)
- 10 mL of 1% lignocaine on each side, injected posterior to the ischial spine
- The pudendal nerve (S2, S3, S4) supplies sensation to the perineum, vulva, and lower vagina
- Onset: 3-5 minutes; provides adequate analgesia for episiotomy and instrumental delivery
- Plus: perineal infiltration of 5-10 mL 1% lignocaine subcutaneously at the planned episiotomy site
Alternatives if epidural in place:
- Top-up epidural block immediately
If inadequate time: In extreme emergency with head visible at perineum, delivery can be performed with perineal infiltration alone or even without anaesthesia (the fetus must be delivered NOW).
Step 5 - Instrument Selection
OUTLET FORCEPS is the instrument of choice for this case.
Why forceps over vacuum?
| Feature | Forceps | Vacuum |
|---|
| Speed | Traction begins immediately after locking | 2-3 minutes needed to build negative pressure; cup positioning |
| Fetal distress | Preferred - fastest delivery | Relatively slower |
| OA at outlet | Perfect indication for outlet forceps | Vacuum also works at outlet OA |
| Scalp trauma | None | Chignon, cephalhaematoma, subgaleal haematoma risk |
| Failure rate | Low at outlet | Low at outlet |
| Operator skill | Requires experience | Less technically demanding |
| Neonatal asphyxia | Does not worsen (fast delivery) | Slight delay in negative pressure build-up |
Forceps selection for DOA at outlet:
- Wrigley's forceps (outlet forceps) - short handles, short blades; ideal for outlet OA delivery
- Simpson's forceps - longer blades; used for low-outlet deliveries with minimal moulding
- Anderson's forceps - similar to Simpson's
Step 6 - Application of Forceps (Step-by-Step)
Confirm prerequisites one final time:
- Cervix fully dilated ✓ (complete dilatation confirmed on VE)
- Membranes ruptured ✓ (absent 4 hours)
- Head at pelvic floor/outlet (station +3/+4) ✓
- Position KNOWN: Direct Occiput Anterior (posterior fontanelle anterior, sagittal suture AP) ✓
- Bladder empty ✓ (catheterised)
- Anaesthesia administered ✓
- Fetal distress: INDICATION ✓
- Fetus alive ✓
- No CPD (head already at outlet - by definition adequate outlet)
- Experienced operator ✓
- Theatre available if forceps fails ✓
Application:
a. Determine exact position:
Confirm by VE: posterior fontanelle is directly anterior (under symphysis), anterior fontanelle posterior (near sacrum). Sagittal suture runs AP in the midline. This is Direct OA.
b. Left blade first:
- Hold the LEFT handle in the LEFT hand (thumb on top, fingers wrapping the handle - held like a pen, not a fist)
- Insert the RIGHT hand into the vagina as a guiding hand (two fingers between the fetal head and the left lateral vaginal wall, at approximately 4 o'clock position)
- The LEFT blade (which goes to the LEFT side of the maternal pelvis and the LEFT side of the fetal head) is introduced with the handle pointing VERTICALLY upward initially
- The toe of the blade is guided toward the left side of the fetal head, sliding along the guiding right hand
- As the blade advances, the HANDLE IS LOWERED (drops toward the horizontal) - this is the classic teaching: "handle falls as blade rises to the head"
- The blade should lie between the fetal head and the vaginal wall, following the curve of the head
- Remove the right guiding hand; leave the left handle in position
c. Right blade:
- Switch: hold RIGHT handle in RIGHT hand
- Insert LEFT guiding hand into vagina at 8 o'clock (right lateral side)
- The RIGHT blade slides in along the left guiding hand to the right side of the fetal head
- Handle lowered as blade advances
- In DOA, both blades should be introduced symmetrically
d. Lock/articulate the forceps:
- The two handles come together naturally (if correctly placed)
- The SHANKS LOCK at the articulation point (English lock for Wrigley's/Simpson's)
- If the shanks do not come together without force, or one side is higher → the blades are malpositioned → REMOVE both blades and re-examine before re-applying
e. Check application (VERY IMPORTANT before any traction):
- The sagittal suture should lie MIDWAY between the two blades (in the midline) - confirms symmetric application
- The posterior fontanelle should lie ONE FINGER-BREADTH ABOVE the plane of the shanks - confirms flexion/correct cephalic application
- No more than one finger should fit between the fenestration of the blade and the fetal head - confirms blades are not too far lateral or slipping
- Both blades should feel equally applied to the head
f. Episiotomy:
- Right mediolateral episiotomy at the point of crowning/maximal perineal distension
- Incision: from the posterior fourchette, angled 45-60 degrees to the right, 3-4 cm in length
- Use scissors or scalpel; cut at peak of a contraction when tissues are thinned
- This protects the perineum and sphincter, provides room for the forceps blades and fetal head
g. Traction:
Direction of traction follows the pelvic axis (Carus' curve) - the J-shaped curve of the pelvis:
-
Phase 1 (initial traction): Directed POSTERIORLY AND DOWNWARD (toward the floor)
- This follows the outlet of the pelvis: the head must clear the coccyx/sacrum inferiorly first
- Pajot's maneuver: Right hand grasps the handles and applies traction; left hand rests on the shanks and applies downward counter-pressure (this converts horizontal traction on handles into a downward-outward vector following pelvic axis)
-
Phase 2 (as head descends to perineum): Direction becomes HORIZONTAL
-
Phase 3 (as occiput appears at vulva under symphysis): Direction becomes UPWARD (toward ceiling) - the head delivers by EXTENSION over the symphysis
-
Traction is applied with CONTRACTIONS and maternal pushing - "Pull WITH her, not against her"
-
Between contractions: release traction but keep blades in position
-
Traction should be STEADY and CONTROLLED, never jerky or rotational
-
In DOA, NO ROTATION is needed - the head is already in the ideal position
h. Delivery of the head:
- As the occiput clears under the symphysis: handle the forceps gently upward
- The head delivers by extension
- Once the widest diameter of the head (biparietal) passes the vulval ring: RELEASE the handles
- Allow the head to deliver slowly and gently over the perineum (reduces third and fourth degree tear risk)
- Alternatively: some operators remove the blades just as the head crowns and allow the head to deliver naturally - reduces pressure on the perineum
i. Delivery of the body:
- Check for cord around neck immediately
- Delivery of shoulders: gentle downward traction on the head to deliver the anterior shoulder first; then upward to deliver posterior shoulder
- Deliver body by assisted mechanism
Step 7 - Immediate Neonatal Care
The neonatologist takes over at the moment of delivery:
- Apgar score at 1 and 5 minutes (and every 5 minutes if <7 until >7 twice)
- This neonate is expected to have a LOW Apgar score (1-3) given FHR 105, dull, arrhythmic
- Neonatal Resuscitation Protocol (NRP):
- Dry, stimulate, position
- Assess breathing and heart rate at 30 seconds
- If not breathing or gasping, HR <100: Bag-mask ventilation (BMV) with room air (or 21% O2 initially; escalate to 100% O2 if no response)
- If HR <60 despite 30 seconds of effective BMV: Chest compressions (2-thumb technique; 3:1 ratio with ventilations)
- If HR <60 despite CPR: Epinephrine (IV via umbilical venous catheter or endotracheal) 0.01-0.03 mg/kg
- Intubate if prolonged resuscitation or if BMV ineffective
- Cord blood gas sampling (from umbilical artery): Send for pH, pCO2, pO2, base excess
- pH <7.0 and base deficit >12 mmol/L = significant metabolic acidosis = hypoxic-ischaemic insult
- This guides further neonatal management (Therapeutic Hypothermia / Cooling if birth asphyxia confirmed)
- Therapeutic Hypothermia (Cooling Protocol): If neonate meets criteria for hypoxic-ischaemic encephalopathy (HIE):
- Gestational age ≥36 weeks
- Cord pH <7.0 or base deficit ≥16
- Encephalopathy signs (abnormal tone, seizures, depressed consciousness)
- Cooling to 33-34°C core body temperature for 72 hours (reduces neuronal death)
- MUST be started within 6 hours of birth to be effective
- Transfer to NICU level III
Step 8 - Active Management of Third Stage
- Oxytocin 10 IU IM immediately after delivery of the anterior shoulder (or IV 5 IU bolus)
- Controlled cord traction (Brandt-Andrews technique) after signs of placental separation
- Uterine massage after placental delivery
- Examine placenta and membranes for completeness
Step 9 - Inspection and Repair
- Thorough inspection of:
- Episiotomy site: Repair in layers (vaginal mucosa, perineal muscle, skin)
- Vaginal walls: Lateral vaginal lacerations from forceps blades - most common in outlet forceps
- Cervix: Examine with ring forceps (post-instrumental delivery, cervical lacerations are common)
- Perineal body: Degree of tear (first through fourth degree)
- Rectum and anal sphincter: Examine carefully per rectum to exclude occult 3rd/4th degree tear
- Repair all lacerations under adequate anaesthesia
Step 10 - Post-delivery Monitoring
Mother:
- Vital signs every 15-30 minutes for 2 hours (immediate puerperium)
- Monitor for PPH: uterine tone, lochia amount, BP
- Urinary output monitoring (catheter in situ 24 hours post-instrumental delivery)
- Analgesia: diclofenac/ibuprofen + paracetamol regularly; opioids if needed
- Thromboprophylaxis: LMWH 6-12 hours post-delivery (if no bleeding); TED stockings
- Stool softeners (lactulose) to avoid straining against the repair
Neonate:
- NICU admission if Apgar <7 at 5 minutes
- Monitor for HIE, seizures, respiratory distress, hypoglycaemia, hypocalcaemia
- Cooling protocol if HIE criteria met (as above)
- Inform parents: explain what happened, what is being done, prognosis
References:
- Williams Obstetrics, 25th Edition (Cunningham, Leveno, Dashe et al.)
- DC Dutta's Textbook of Obstetrics, 8th Edition (Konar)
- Berek & Novak's Gynecology, 16th Edition
- Creasy & Resnik's Maternal-Fetal Medicine, 8th Edition
- ACOG Practice Bulletin No. 154: Operative Vaginal Delivery
- ACOG Practice Bulletin: Abruptio Placentae
- NRP (Neonatal Resuscitation Program) Guidelines, 8th Edition