Femoral nerve paralysis in cattle sign

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femoral nerve paralysis cattle signs clinical symptoms

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https://www.msdvetmanual.com/musculoskeletal-system/lameness…

Here is a complete summary of Femoral Nerve Paralysis in Cattle - its signs, causes, and key clinical features:

Femoral Nerve Paralysis in Cattle

Anatomy Basis

The femoral nerve (L4-L6) innervates the quadriceps femoris muscle group (which extends the stifle joint) and partially innervates the psoas major (hip flexor). Damage to this nerve disrupts stifle extension and weight-bearing ability.

Etiology / Cause

  • Most common in large, newborn calves (e.g., Charolais, Simmental) after difficult/assisted births with mechanical traction - especially when the calf becomes hip-locked in anterior presentation
  • Excessive pulling force stretches and damages the femoral nerve
  • Rarely seen in adult cows

Clinical Signs

SignDescription
Inability to extend the stifleThe hallmark sign - the calf cannot lock the stifle joint to bear weight
Inability to bear weightAffected limb(s) collapse when weight is placed on them
Dropped/flexed stifleStifle buckles forward, the limb folds under the animal
Lateral patellar luxationReduced quadriceps tone causes the patella to displace laterally; it can be manually reduced easily but recurs
Absent patellar (patella) reflexThe patellar reflex is entirely absent (requires an intact femoral nerve and quadriceps) - diagnostically important
Quadriceps atrophyRapid, obvious atrophy of the quadriceps muscle group within 7-10 days
"Dog-sitting" postureIn bilateral femoral paralysis, calves are unable to stand and sit in a dog-sitting posture with hind limbs extended forward
Pelvic tiltIn unilateral cases, the pelvis tilts noticeably toward the affected side
Hip flexion difficultyPartial psoas major involvement limits hip flexion on the affected side

Unilateral vs. Bilateral

  • Unilateral: Calf can attempt to rise but has great difficulty; pelvis is tilted toward the affected side; prognosis is better
  • Bilateral: Calf is completely unable to stand; adopts a characteristic dog-sitting posture; prognosis is guarded to poor

Differentiating from Other Nerve Paralyses

FeatureFemoralSciaticObturator
Key deficitStifle extensionFetlock knuckling + dropped hockAdduction (limbs splay)
Patellar reflexAbsentProminent (increased)Normal
Quadriceps atrophyYes (rapid)NoNo
PostureDog-sitting (bilateral)Knuckled fetlockSplay-legged ("doing splits")
TimingBirth traumaBirth traumaBirth trauma

Diagnosis

  • Based on history (difficult assisted delivery) + clinical signs
  • Absent patellar reflex is the key diagnostic finding
  • Radiographs recommended to rule out slipped capital femoral physis or proximal femoral fractures (can appear clinically similar before neurogenic atrophy develops)

Treatment & Prognosis

  • Maintain in a well-bedded area (sand stall is ideal) to prevent pressure sores
  • Ensure adequate colostrum intake (calf may be unable to suckle unaided)
  • Anti-inflammatory drugs: dexamethasone (0.05 mg/kg IM q 1-2 days) or flunixin meglumine (1 mg/kg IM q 12h) to reduce perineural swelling
  • Prognosis is fair to good in unilateral cases if managed promptly; guarded to poor in bilateral cases
  • No improvement within 5-7 days indicates poor prognosis

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