Question 19 - Full Interpretation
Clinical Context
- 88-year-old, nursing home, "unwell"
- T 35.8°C (hypothermic), HR 74, BP 101/70 (borderline hypotensive), RR 15, SpO₂ 92% RA
- Troponin positive
ECG Findings
Rhythm/Rate: Sinus rhythm, ~74 bpm, regular
Key Abnormalities Visible:
| Lead Group | Finding |
|---|
| III, aVF | T-wave inversions |
| V1, V2, V3 | T-wave inversions (deep) |
| V4, V5 | T-wave inversions |
| V1-V2 | No significant ST elevation |
| Overall | No ST elevation in any lead |
The T-wave inversions are widespread across the precordial leads (V1-V5) and inferior leads (III), with no ST elevation - this is the defining pattern of NSTEMI.
Answers
a) Diagnosis: Non-ST Elevation Myocardial Infarction (NSTEMI) ✓
The combination of:
- Positive troponin
- T-wave inversions across multiple leads (no ST elevation)
- Clinical presentation (unwell, borderline hemodynamics, low SpO₂)
...confirms NSTEMI. The other options are incorrect because:
- No evidence of junctional rhythm (P waves present, normal rate)
- No wide QRS or RSR' pattern to suggest RBBB
- HR is 74, not bradycardic
b) Leads with changes: T-wave inversion in III, V1, V2, V3, V4, V5 ✓
The handwritten answer in the image is correct. The widespread anterior-inferior T-wave inversions suggest likely LAD territory ischemia (anterior leads V1-V5) with possible additional inferior involvement.
Clinical Significance
This is a high-risk NSTEMI given:
- Age 88 with borderline vitals
- Hypothermia (35.8°C) suggesting poor perfusion/sepsis co-existing possibility
- SpO₂ only 92% - consider pulmonary edema from LV dysfunction
- Widespread ischemic changes on ECG
Immediate management priorities: Dual antiplatelet therapy (aspirin + P2Y12 inhibitor), anticoagulation, cardiology consult for urgent vs. early invasive strategy, oxygen supplementation, continuous monitoring.