Here is the complete continuation - Day 2 through Day 10 - fully detailed with every question and model answer.
DAY 2 - AIRWAY MANAGEMENT (COMPLETE)
Q1. What are the types of airway adjuncts and how do you use them?
OPA - Oropharyngeal Airway (Guedel Airway)
Purpose: Keeps tongue from falling back and blocking airway in unconscious patients.
Indications:
- Unconscious patient with NO gag reflex
- To maintain airway during BVM ventilation
Contraindications:
- Conscious or semi-conscious patient (stimulates gag reflex - causes vomiting and aspiration)
- Intact gag reflex
Sizing:
- Measure from corner of mouth to earlobe OR center of mouth to angle of jaw
- Sizes: 000 (neonate) to 5 (large adult)
- Wrong size = worsens obstruction. Too small pushes tongue back. Too large stimulates larynx.
Insertion technique (adult):
- Select correct size
- Open mouth (cross-finger or jaw-thrust technique)
- Insert OPA upside down (curved tip pointing UP toward palate)
- Advance until resistance felt at hard-soft palate junction
- Rotate 180 degrees so curve follows tongue
- Ensure flange rests on lips
Insertion technique (child/infant):
- Insert with tongue depressor directly (curved tip pointing DOWN) - do NOT rotate, risk of trauma
NPA - Nasopharyngeal Airway (Nasal Trumpet)
Purpose: Provides airway passage through nose, past tongue and soft palate.
Indications:
- Patients who are semiconscious (intact gag reflex) - better tolerated than OPA
- Trismus (clenched teeth - jaw cannot open)
- Seizure patients (teeth clenched)
Contraindications:
- Suspected basal skull fracture (risk of entering cranial vault) - signs: raccoon eyes, Battle's sign, CSF from nose/ears
- Severe nasal trauma or obstruction
- Anticoagulation (relative - risk of epistaxis)
Sizing:
- Length: nostril to earlobe
- Diameter: matches patient's little finger OR largest that fits comfortably
Insertion technique:
- Lubricate generously with water-based gel
- Insert into the right nostril (bevel toward septum) - left nostril if right blocked
- Advance gently along the floor of the nasal passage (not upward!)
- If resistance: do NOT force; try other nostril or smaller size
- Correctly placed: tip in oropharynx behind tongue
Q2. Explain the BVM technique in detail.
BVM = Bag-Valve-Mask - used to provide positive pressure ventilation when patient is not breathing adequately.
Components:
- Self-inflating bag (450 mL pediatric / 1600 mL adult)
- One-way valve
- Mask (transparent, cushioned, sizes 0-5)
- Oxygen reservoir bag attached = delivers close to 100% O2
One-person BVM technique (E-C Clamp):
- C = Thumb and index finger form a C, pressing mask firmly onto face (apex over nose bridge, base between lower lip and chin)
- E = Remaining 3 fingers form an E, hooking under mandible (lower jaw) to perform jaw thrust
- Other hand squeezes bag
- Give 1 breath every 5-6 seconds (10-12/min) for adults
- Each breath: 1 second; visible chest rise; do NOT over-ventilate
Two-person BVM technique (PREFERRED):
- Person 1: Both hands on mask using two-thumb E-C or thenar eminence technique = better seal
- Person 2: Squeezes bag
- Far superior seal, reduces air leaks
Volume:
- Adults: squeeze enough to see chest rise (~500-600 mL)
- Do NOT give excessive volumes - causes gastric insufflation and aspiration
Oxygen flow:
- Without reservoir: ~40-60% O2
- With reservoir bag at 15 L/min flow: ~90-100% O2
Q3. What is the difference between LMA, i-gel, and ETT?
| Feature | LMA (Laryngeal Mask Airway) | i-gel | ETT (Endotracheal Tube) |
|---|
| Type | Supraglottic airway | Supraglottic airway | Definitive airway |
| Cuff | Inflatable cuff | No cuff (gel-filled) | Inflatable cuff |
| Insertion | Blind, over tongue | Blind, simple insertion | Requires laryngoscopy |
| Aspiration protection | Partial | Partial | Full (when cuffed) |
| Skill required | Moderate | Easy | Advanced |
| Use when | ETT difficult/failed | Quick emergency airway | Definitive, prolonged ventilation |
| Gold standard | No | No | YES |
Q4. What is a surgical airway and when is it needed?
Surgical airway is required when ALL other airway methods have failed - the "can't intubate, can't oxygenate" (CICO) scenario.
Needle Cricothyrotomy:
- Insert 14G or 16G IV cannula through cricothyroid membrane (between thyroid and cricoid cartilage)
- Aspirate air to confirm tracheal placement
- Attach O2 tubing (jet ventilation)
- Provides temporary oxygenation (15-45 minutes maximum)
- Allows oxygenation but NOT adequate ventilation (CO2 builds up)
Surgical Cricothyrotomy:
- Identify cricothyroid membrane (landmark: notch below thyroid cartilage)
- Make horizontal stab incision through skin and membrane
- Insert tracheostomy tube or small ETT (size 6.0)
- Inflate cuff, confirm with ETCO2
- This IS a definitive (though temporary) airway
Landmarks: Place finger on Adam's apple (thyroid cartilage notch) - slide finger down to soft depression = cricothyroid membrane
(Source: Washington Manual of Medical Therapeutics - Surgical Airways)
Q5. What are the signs of a difficult airway? (LEMON assessment)
LEMON mnemonic - predict difficult intubation before attempting:
- L - Look externally: beard, short neck, obesity, facial burns, trauma, small mouth
- E - Evaluate 3-3-2 rule:
- 3 fingers between upper and lower teeth (mouth opening)
- 3 fingers between hyoid bone and chin
- 2 fingers between notch of thyroid cartilage and floor of mouth
- M - Mallampati score: Open mouth, protrude tongue, look for uvula/palate visibility
- Class I (all visible) = easy; Class IV (nothing visible) = very difficult
- O - Obstruction: foreign body, hematoma, epiglottitis, abscess
- N - Neck mobility: limited (c-spine injury, ankylosing spondylitis, cervical collar)
If LEMON predicts difficult airway: prepare video laryngoscope, surgical airway kit, and senior help BEFORE attempting intubation
Q6. What oxygen delivery devices are there and what FiO2 do they deliver?
| Device | Flow Rate | FiO2 (%) | Notes |
|---|
| Nasal Cannula | 1-6 L/min | 24-44% | Add 4% FiO2 per L/min above 21% |
| Simple Face Mask | 5-10 L/min | 35-50% | Minimum 5 L/min to flush CO2 |
| Venturi Mask | Fixed flow | 24%, 28%, 35%, 40%, 60% | Most accurate FiO2; use in COPD |
| Non-Rebreather Mask (NRM) | 10-15 L/min | 60-90% | One-way valve; reservoir bag inflated first |
| BVM with O2 reservoir | 15 L/min | ~100% | Positive pressure ventilation |
Key clinical points:
- In COPD: target SpO2 88-92% (risk of hypercapnic respiratory failure with high O2) - use Venturi mask
- In all other emergencies: target SpO2 94-98%
- In CO poisoning, anaphylaxis, cardiac arrest: always use 100% O2 / NRM first
DAY 3 - CARDIAC EMERGENCIES & ECG (EXTENDED)
Q7. How do you read a 12-lead ECG systematically?
Systematic ECG reading (use this every time - RSVP):
Step 1 - Rate
- Count large squares between two R waves: divide into 300
- 1 box = 300, 2 boxes = 150, 3 boxes = 100, 4 boxes = 75, 5 boxes = 60
- Normal: 60-100 bpm
Step 2 - Rhythm
- Regular or irregular?
- P before every QRS? QRS after every P?
- Regular = sinus rhythm
- Irregularly irregular (no pattern) = Atrial Fibrillation
- Regular irregular (pattern to it) = 2nd degree AV block
Step 3 - Axis
- Normal axis: leads I and aVF both positive (upright QRS)
- Left axis deviation: lead I positive, aVF negative (LAD)
- Right axis deviation: lead I negative, aVF positive (RAD)
Step 4 - P wave
- Present? Upright in II? PR interval 0.12-0.20 sec (3-5 small boxes)
- Prolonged PR = 1st degree AV block
- Absent P waves = AF or junctional rhythm
Step 5 - QRS complex
- Duration: <0.12 sec (3 small boxes) = normal
- Wide QRS (>0.12) = LBBB, RBBB, hyperkalemia, ventricular rhythm
- Pathological Q wave: >0.04 sec wide and >25% of R wave height = old MI
Step 6 - ST segment
- ST elevation (STEMI or pericarditis)
- ST depression (ischemia, NSTEMI, digoxin)
- Measure J-point (where S wave meets ST segment)
Step 7 - T wave
- Normal: upright in I, II, V3-V6; inverted in aVR
- Tall peaked T waves = hyperkalemia (first ECG sign)
- Inverted T waves = ischemia, PE, RBBB
- Flat/biphasic T waves = hypokalemia
Step 8 - QTc interval
- Normal: <440ms (men), <460ms (women)
- Prolonged QTc = risk of Torsades de Pointes
- Causes: medications (amiodarone, haloperidol), hypokalemia, hypomagnesemia
Q8. Explain common arrhythmias with their ECG features and management.
1. Sinus Tachycardia
- ECG: Normal P-QRS-T; rate >100 bpm
- Cause: Pain, fever, anxiety, anemia, PE, hypovolemia
- Treatment: Treat the underlying cause (NOT the rhythm itself)
2. Atrial Fibrillation (AF)
- ECG: No P waves; irregularly irregular QRS; fibrillatory baseline
- Symptoms: Palpitations, breathlessness, dizziness, stroke risk
- Treatment:
- Rate control: Metoprolol or Diltiazem IV
- Rhythm control (if <48 hrs onset): Electrical cardioversion (synchronized) or Amiodarone
- Anticoagulation: Warfarin/NOACs to prevent stroke (CHA2DS2-VASc score)
- Unstable AF (BP <90, altered consciousness): immediate synchronized cardioversion
3. SVT - Supraventricular Tachycardia
- ECG: Narrow QRS; rate 150-250; P waves may be buried in QRS
- Treatment (stable):
- Step 1: Vagal maneuvers (Valsalva, carotid massage)
- Step 2: Adenosine 6 mg rapid IV push (flush immediately with 20 mL NS)
- If no response after 1-2 min: Adenosine 12 mg IV (can repeat once)
- Step 3: Verapamil or Diltiazem IV
- Treatment (unstable): Synchronized cardioversion 50-100J
4. Ventricular Tachycardia (VT) with pulse
- ECG: Wide QRS (>0.12 sec); rate >100; regular; AV dissociation
- Treatment (stable):
- Amiodarone 150 mg IV over 10 minutes, then 1 mg/min infusion
- OR Lidocaine 1-1.5 mg/kg IV
- Treatment (unstable): Synchronized cardioversion 100J (biphasic)
- Pulseless VT: = cardiac arrest - immediate defibrillation
5. Complete Heart Block (3rd Degree AV Block)
- ECG: P waves and QRS completely dissociated (no relationship); both regular but independent
- Symptoms: Severe bradycardia, syncope (Stokes-Adams attacks), hypotension
- Treatment: Atropine; Transcutaneous pacing immediately; definitive = permanent pacemaker
6. 1st Degree AV Block
- ECG: PR interval prolonged (>0.20 sec); every P followed by QRS
- Usually benign - no treatment needed; monitor
7. 2nd Degree AV Block - Mobitz Type I (Wenckebach)
- ECG: Progressively lengthening PR interval until a P wave is NOT followed by QRS; then cycle repeats
- Usually benign; may need pacing if symptomatic
8. 2nd Degree AV Block - Mobitz Type II
- ECG: PR interval constant; some P waves suddenly not followed by QRS (2:1, 3:1 ratio)
- Dangerous - can progress to complete heart block
- Treatment: Pacemaker
Q9. What is the full STEMI management protocol?
Immediate Assessment (within 10 minutes of arrival):
- 12-lead ECG (door-to-ECG within 10 minutes)
- IV access x2; bloods: Troponin, CK-MB, FBC, U&E, coagulation
- O2 if SpO2 <94%
- Continuous monitoring (BP, ECG, SpO2)
Immediate Drugs (MONA + P2Y12):
- Aspirin 300 mg chewable (unless contraindicated)
- P2Y12 inhibitor: Ticagrelor 180 mg OR Clopidogrel 300-600 mg
- Heparin (LMWH or UFH)
- Nitrates (GTN) if BP >90 systolic (NOT in right ventricular MI - causes severe hypotension)
- Morphine 2-4 mg IV for pain (use cautiously)
Reperfusion Decision:
- Primary PCI (angioplasty) - if cathlab available within 90 minutes of first medical contact - GOLD STANDARD
- Thrombolysis - if PCI not available within 120 minutes from diagnosis:
- Streptokinase 1.5 million units IV over 60 min, OR
- Alteplase (tPA) 15 mg bolus + 0.75 mg/kg over 30 min + 0.5 mg/kg over 60 min
Contraindications to Thrombolysis:
- Absolute: Prior intracranial hemorrhage, current stroke, aortic dissection, active bleeding, recent head/facial trauma
- Relative: Severe uncontrolled hypertension (>180/110), recent major surgery (<3 weeks), pregnancy, oral anticoagulants
DAY 4 - TRAUMA MANAGEMENT (EXTENDED)
Q10. What is the PHTLS (Pre-Hospital Trauma Life Support) approach?
PHTLS is designed specifically for pre-hospital emergency care. It teaches the same CABCDE framework but focuses on:
-
Scene safety and mechanism of injury (MOI)
- What happened? (MVC, fall height, penetrating vs blunt)
- High energy = high index of suspicion for serious injury
- Consider spinal injury if: fall >3x patient height, high-speed MVC, diving accident, axial loading
-
Primary survey with simultaneous treatment (not sequential)
- In the field: C-A-B-C-D-E performed rapidly (2-3 min goal)
-
Scoop and run vs stay and play:
- Critical trauma patient: LOAD AND GO - stabilize basics only; get to hospital fast (goal <10 min on scene for penetrating trauma)
- Only minor injuries: may take time on scene to fully treat
-
Spinal Motion Restriction (SMR):
- Full SMR: C-collar + long spine board/vacuum mattress
- Indications: Altered mental status + trauma, spine pain/tenderness, neurological deficits, high-energy mechanism
- In conscious, alert, sober patients with no spine pain and normal motor/sensory = can clear clinically (NEXUS criteria)
Q11. What are the NEXUS criteria for clearing cervical spine without X-ray?
A patient can have cervical spine injury excluded clinically (no X-ray needed) if ALL 5 criteria are met:
- No midline cervical tenderness (press along posterior spinous processes - no pain)
- No focal neurological deficit
- Normal alertness (GCS 15, no drugs/alcohol)
- No intoxication
- No painful distracting injury (e.g., no femur fracture causing distraction)
If ANY criterion fails: C-collar on; CT cervical spine required.
Q12. What are the types of head injuries an EMT must know?
1. Concussion (Mild TBI - GCS 13-15):
- Temporary loss of consciousness or confusion after head injury
- Headache, nausea, amnesia
- No structural damage on CT
- Rest and observation
2. Epidural Hematoma:
- Lucid interval followed by rapid deterioration = CLASSIC presentation
- Cause: Temporal bone fracture + middle meningeal artery rupture
- CT: Biconvex (lens-shaped) hyperdense lesion
- Crosses midline: NO | Crosses suture lines: NO
- Treatment: Emergency craniotomy
3. Subdural Hematoma:
- Acute: Bridging vein rupture; rapid deterioration; crescent-shaped bleed on CT
- Chronic: Elderly patients on anticoagulants; weeks after minor fall; headache, confusion
- CT: Crescent-shaped (concave) lesion
- Crosses suture lines: YES
- Treatment: Craniotomy or burr holes
4. Subarachnoid Hemorrhage (SAH):
- "Worst headache of my life" - sudden onset thunderclap headache
- Cause: Ruptured cerebral aneurysm
- CT: Star-shaped blood in subarachnoid space
- Treatment: Neurosurgery; nimodipine (prevents vasospasm)
5. Intracerebral Hemorrhage:
- Bleeding within brain tissue; hypertension most common cause
- Altered consciousness, focal deficits
- CT: Hyperdense lesion within brain parenchyma
EMT priorities in ALL head injuries:
- Protect airway (intubate if GCS ≤8)
- Maintain SpO2 >94% and ETCO2 35-40 mmHg (avoid hypoxia AND hypocapnia)
- Maintain MAP ≥80 mmHg (cerebral perfusion pressure)
- Do NOT give excessive fluids
- Head of bed 30 degrees
- Urgent CT head
Q13. What is the management of burns?
Classification:
| Depth | Old name | Appearance | Pain | Treatment |
|---|
| Superficial | 1st degree | Red, dry, no blisters | Very painful | Cool water, analgesia |
| Superficial partial thickness | 2nd degree superficial | Red, wet, blisters | Very painful | Dressing, may need skin graft |
| Deep partial thickness | 2nd degree deep | Pale, less wet | Less painful (nerve damage) | Skin graft |
| Full thickness | 3rd degree | Leathery, white/brown/black | Painless (nerves destroyed) | Skin graft |
| Sub-dermal | 4th degree | Charred, involves bone | Painless | Amputation, complex reconstruction |
Rule of Nines (adult BSA estimation):
- Head and neck: 9%
- Each arm: 9% (total 18%)
- Chest: 9% | Abdomen: 9% (Anterior trunk = 18%)
- Upper back: 9% | Lower back: 9% (Posterior trunk = 18%)
- Each thigh: 9% | Each lower leg: 9% (each leg = 18%; total = 36%)
- Genitalia: 1%
- Lund-Browder chart is more accurate for children (head larger)
Parkland Formula (fluid resuscitation for burns >20% BSA):
4 mL x Weight (kg) x %BSA burned = total IV fluid (Lactated Ringer's) in first 24 hours
- Give half in first 8 hours from time of burn (not from hospital arrival)
- Give remaining half over next 16 hours
Initial EMT management:
- Stop burning (remove clothing, cool with room temperature water 10-20 min - NOT ice)
- Airway priority: singed eyebrows/nasal hair, hoarseness, carbonaceous sputum = airway burn = intubate EARLY
- IV access x2; fluid resuscitation (Parkland formula)
- Pain management (IV morphine/ketamine)
- Cover with clean dry dressing; keep warm
- Catheter (urine output target: 0.5-1 mL/kg/hr adults; 1 mL/kg/hr children)
DAY 5 - SHOCK MANAGEMENT (EXTENDED)
Q14. What is the Surviving Sepsis Campaign bundle for septic shock?
Definition of Septic Shock (Sepsis-3):
- Life-threatening organ dysfunction due to dysregulated host response to infection
- Septic shock = sepsis + vasopressor needed to maintain MAP ≥65 mmHg + serum lactate >2 mmol/L despite adequate fluid resuscitation
1-Hour Bundle (all within 1 hour of diagnosis):
- Measure lactate (repeat if >2 mmol/L)
- Blood cultures x2 (before antibiotics - peripheral + central line)
- Broad-spectrum antibiotics IV within 1 hour (do NOT delay for cultures)
- IV Crystalloid 30 mL/kg for hypotension or lactate ≥4 mmol/L
- Norepinephrine if hypotensive during or after fluid resuscitation
Vasopressors in Septic Shock:
- 1st line: Norepinephrine (balanced alpha + beta; prevents tachycardia)
- 2nd line: Vasopressin 0.01-0.03 units/min (to reduce norepinephrine dose)
- Dobutamine if cardiac dysfunction (low cardiac output component)
- Goal: MAP ≥65 mmHg, urine output ≥0.5 mL/kg/hr, normalizing lactate
(Source: Miller's Anesthesia - Distributive Shock)
Q15. What is cardiac tamponade and how is it treated?
Pathophysiology:
Fluid (blood, pus, exudate) accumulates in pericardial sac - normally contains only 15-50 mL. As volume increases, pericardial pressure rises, compressing the heart, reducing diastolic filling, and decreasing cardiac output.
Signs - Beck's Triad:
- Hypotension (low BP)
- Raised JVP (distended neck veins - elevated CVP)
- Muffled heart sounds (fluid dampens sounds)
Additional signs:
- Pulsus paradoxus: BP drops >10 mmHg during inspiration (classic for tamponade)
- Tachycardia
- Electrical alternans on ECG (alternating QRS height)
Causes:
- Penetrating chest trauma (stab wound) - most common in young trauma patients
- Post-cardiac surgery
- Malignancy (slow-filling)
- Pericarditis, TB, uraemia
Treatment:
- Pericardiocentesis - needle aspiration
- Patient sitting up at 45 degrees
- Insert needle at subxiphoid angle, aim toward left shoulder at 45 degrees
- Even 10-15 mL removal dramatically improves cardiac output
- Ideally ultrasound-guided
- Definitive: Surgical pericardial window or pericardiotomy in theatre
Q16. What is massive transfusion protocol (MTP)?
When activated: Blood product requirement >10 units pRBCs in 24 hrs, or ongoing hemorrhage with hemodynamic instability.
Damage Control Resuscitation (DCR) principles:
- Permissive hypotension: Target systolic BP 80-90 mmHg (NOT 120) in penetrating trauma until surgical hemorrhage control - prevents "popping the clot"
- Balanced resuscitation: Ratio of pRBC : FFP : Platelets = 1:1:1
- Avoid crystalloid - dilutes clotting factors and worsens coagulopathy
- Tranexamic acid (TXA): Give within 3 hours of injury - 1g IV over 10 min + 1g over 8 hours
- Correct the "lethal triad": Hypothermia + Acidosis + Coagulopathy
(Source: Sabiston Textbook of Surgery; Rosen's Emergency Medicine)
DAY 6 - NEUROLOGICAL EMERGENCIES (EXTENDED)
Q17. What are the types of strokes and how do you differentiate them?
| Feature | Ischemic Stroke (87%) | Hemorrhagic Stroke (13%) |
|---|
| Mechanism | Thrombotic or embolic vessel occlusion | Vessel rupture - ICH or SAH |
| Onset | Gradual or sudden | Sudden, often with severe headache |
| Headache | Mild or absent | SEVERE ("thunderclap") in SAH |
| CT scan | Normal initially (first 4-6 hrs); later hypodense area | Hyperdense (bright white) blood immediately |
| Vomiting | Uncommon | Common |
| Treatment | tPA if eligible; thrombectomy | NO tPA; BP control; neurosurgery |
Why CT scan BEFORE tPA is mandatory:
- Hemorrhagic stroke looks IDENTICAL clinically to ischemic stroke
- tPA in a hemorrhagic stroke = catastrophic - massively worsens the bleed
- CT scan immediately differentiates: blood appears WHITE on non-contrast CT
Q18. What is increased intracranial pressure (ICP) and how do you manage it?
Normal ICP: 5-15 mmHg
Raised ICP: >20 mmHg - brain is being compressed
Cushing's Triad (late, severe sign - impending brain herniation):
- Hypertension (rising BP - body trying to perfuse brain)
- Bradycardia (reflex)
- Irregular respirations (Cheyne-Stokes)
Treatment of raised ICP:
- Head of bed 30-45 degrees
- Avoid hyponatremia - may use hypertonic saline
- Mannitol 0.25-1 g/kg IV (osmotic diuretic - draws water out of brain)
- Controlled hyperventilation (ETCO2 30-35 mmHg) - temporary bridge only (causes cerebral vasoconstriction)
- Avoid hypotension (maintain MAP ≥80 mmHg for cerebral perfusion)
- Surgical decompression (decompressive craniectomy) for refractory cases
Q19. What is spinal cord injury and what is neurogenic shock?
Spinal cord injury levels and function:
- C3-C5: Diaphragm (C3,4,5 keeps the diaphragm alive) - above this level = cannot breathe
- C5-C6: Biceps, wrist extension
- C7: Triceps, wrist flexion
- T1-T12: Intercostals, abdominal muscles
- L1-L2: Hip flexors
- L3-L4: Quadriceps, knee extension
- L4-L5: Ankle dorsiflexion (foot drop if injured)
- S2-S4: Bladder, bowel, erectile function
Neurogenic Shock (from cervical or high thoracic cord injury):
- Loss of sympathetic tone below lesion
- Result: Vasodilation + bradycardia
- UNIQUE FEATURE: Bradycardia with hypotension (all other shocks have tachycardia)
- Warm, dry, flushed skin (vasodilated)
- Treatment: Atropine for bradycardia; Vasopressors (norepinephrine/phenylephrine) for hypotension; careful fluids (risk of pulmonary edema)
- Maintain spinal immobilization
Q20. What is meningitis and how do you recognise it?
Definition: Inflammation of meninges (lining of brain and spinal cord) - bacterial meningitis is a life-threatening emergency.
Classic triad:
- Fever
- Neck stiffness (nuchal rigidity)
- Photophobia (sensitivity to light)
Additional signs:
- Kernig's sign: Cannot extend knee when hip flexed at 90 degrees (pain)
- Brudzinski's sign: Passive neck flexion causes involuntary hip and knee flexion
- Purpuric/petechial rash (non-blanching) = meningococcal disease - EMERGENCY
- Altered consciousness, seizures
EMT action:
- Immediate assessment and IV access
- Do NOT delay antibiotics for CT scan or LP if patient deteriorating
- IV Benzylpenicillin (if pre-hospital, in UK: 1.2g for adults before hospital transfer)
- In hospital: Ceftriaxone 2g IV + Dexamethasone 0.15 mg/kg IV
- Isolation precautions (droplet)
DAY 7 - TOXICOLOGY (EXTENDED)
Q21. What are the major toxidromes (poisoning syndromes)?
A toxidrome is a group of symptoms that together point to a specific class of poison:
1. Cholinergic Toxidrome (too much acetylcholine)
- Causes: Organophosphates, carbamates, nerve agents, pilocarpine
- DUMBBELS: Diarrhea, Urination, Miosis, Bradycardia, Bronchospasm/Bronchorrhea, Emesis, Lacrimation, Salivation
- Nicotinic effects: Muscle fasciculations, weakness, paralysis (can't breathe)
- Treatment: Atropine (large doses) + Pralidoxime
2. Anticholinergic Toxidrome (blocked acetylcholine)
- Causes: Atropine, antihistamines, TCA antidepressants, jimsonweed
- Mnemonic: "Hot as a hare, Blind as a bat, Dry as a bone, Red as a beet, Mad as a hatter"
- Hyperthermia, Mydriasis (dilated pupils), Dry skin/mouth, Flushing, Confusion/hallucinations
- Tachycardia, urinary retention, decreased bowel sounds
- Treatment: Physostigmine (specific antidote); supportive care; benzodiazepines for agitation
3. Opioid Toxidrome (too much opioid)
- Causes: Morphine, heroin, codeine, fentanyl, tramadol
- Classic triad: Miosis (pinpoint pupils) + Respiratory depression + Decreased consciousness
- Treatment: Naloxone 0.4-2 mg IV/IM/IN; repeat every 2-3 min
4. Sympathomimetic Toxidrome (too much adrenaline)
- Causes: Cocaine, amphetamines, MDMA (ecstasy), caffeine overdose
- Signs: Mydriasis (dilated pupils), tachycardia, hypertension, hyperthermia, diaphoresis, agitation
- Treatment: Benzodiazepines (calm hyperadrenergic state); cooling; avoid beta-blockers (causes unopposed alpha = worse hypertension)
5. Sedative-Hypnotic Toxidrome
- Causes: Benzodiazepines, barbiturates, alcohol
- Signs: CNS depression, respiratory depression, slurred speech, ataxia; NORMAL pupils
- Treatment: Airway support; flumazenil for benzodiazepines (use with caution)
Q22. What is the management of paracetamol (acetaminophen) overdose?
Mechanism of toxicity:
- Normal dose: paracetamol conjugated safely in liver
- Overdose: NAPQI (toxic metabolite) accumulates, depletes glutathione, causes liver necrosis
- Critical period: 72-96 hours after ingestion = maximum liver damage
Stages:
- 0-24 hr: Nausea, vomiting, malaise (may appear well)
- 24-72 hr: Right upper quadrant pain, rising LFTs
- 72-96 hr: Liver failure, jaundice, coagulopathy, encephalopathy
-
96 hr: Recovery OR death from liver failure
Rumack-Matthew nomogram: Plot paracetamol level on graph at known time of ingestion to determine treatment threshold
Treatment:
- If <1-2 hours: Activated charcoal 50g orally (absorbs paracetamol in gut)
- N-Acetylcysteine (NAC) = definitive antidote:
- Replenishes glutathione stores
- IV regime: 150 mg/kg in 200 mL 5% Dextrose over 60 min → 50 mg/kg over 4 hours → 100 mg/kg over 16 hours
- Start within 8 hours for maximum benefit (still give up to 24 hours)
- Monitor LFTs, INR, creatinine, blood glucose
- Liver transplant assessment if fulminant liver failure develops
Q23. What is carbon monoxide (CO) poisoning?
Mechanism: CO has 240x higher affinity for hemoglobin than oxygen → forms carboxyhemoglobin (COHb) → oxygen cannot be carried → tissue hypoxia despite normal PaO2
Important: Pulse oximeter reads NORMAL SpO2 in CO poisoning (it cannot distinguish COHb from OxyHb) - always suspect in enclosed space fire/smoke inhalation
Signs (COHb levels):
| COHb Level | Symptoms |
|---|
| 10-20% | Headache, nausea |
| 20-40% | Severe headache, confusion, syncope |
| 40-60% | Seizures, coma, cardiac arrhythmias |
| >60% | Death |
| Classic "cherry red skin" | Unreliable; usually seen only at post-mortem |
Treatment:
- Remove from exposure; scene safety
- 100% O2 via non-rebreather mask - reduces COHb half-life from 5 hours to 90 minutes
- Hyperbaric oxygen (HBO) if: COHb >25%, unconscious, cardiac arrhythmias, neurological signs, pregnancy - reduces half-life to 20-30 min
- Supportive care; ECG monitoring (CO causes arrhythmias)
DAY 8 - PAEDIATRIC & OBSTETRIC EMERGENCIES (EXTENDED)
Q24. What are the key differences in paediatric emergency assessment?
Paediatric Assessment Triangle (PAT) - rapid 30-second visual assessment:
-
Appearance (TICLS):
- Tone: Is child active, limp, or floppy?
- Interactiveness: Engaged, curious, or unresponsive?
- Consolability: Can parent comfort them?
- Look/Gaze: Direct eye contact or glazed?
- Speech/Cry: Strong cry or weak/absent?
-
Work of Breathing:
- Nasal flaring, grunting, retractions (subcostal, intercostal, sternal)
- Head bobbing (severe respiratory distress in infants)
- Audible sounds: stridor, wheeze, grunting
-
Circulation (skin color):
- Pallor (anemia, shock), cyanosis (hypoxia), mottling (impaired perfusion), flushing
Abnormal PAT findings = sick child - act immediately
Q25. How do you manage croup vs epiglottitis?
| Feature | Croup (Laryngotracheobronchitis) | Epiglottitis |
|---|
| Age | 6 months - 3 years | Any (classically 2-5 years, now adults) |
| Cause | Parainfluenza virus | Haemophilus influenzae type B (now rare due to Hib vaccine) |
| Onset | Gradual over days | Rapid (hours) |
| Sound | Barking/seal-like cough | Muffled voice, no cough |
| Drooling | No | YES (cannot swallow - painful) |
| Position | Any | Tripod position (leaning forward) |
| Fever | Low grade | High fever |
| X-ray | Steeple sign (subglottic narrowing on AP) | Thumbprint sign (swollen epiglottis on lateral) |
| Treatment | Dexamethasone + nebulized adrenaline (epinephrine) | Do NOT examine throat; immediate intubation in theatre by ENT + anaesthetist + surgeon present |
NEVER use tongue depressor to examine throat in suspected epiglottitis - can cause complete obstruction
Q26. What is the Paediatric Early Warning Score (PEWS)?
PEWS triggers earlier escalation in deteriorating children based on:
- Behaviour (normal/sleeping/irritable/lethargic/confused)
- Cardiovascular (colour/capillary refill/HR)
- Respiratory (RR/work of breathing/SpO2)
Each parameter scored 0-3. Total >3 = escalate immediately.
Q27. What are the neonatal emergencies an EMT must recognise?
Neonatal Resuscitation:
- At birth: Dry, stimulate, assess tone, breathing, HR
- HR <100 + not breathing adequately → BVM ventilation with air (21% O2) at 40-60 breaths/min
- HR <60 despite 30 sec effective ventilation → start CPR: 3:1 ratio (3 compressions : 1 breath), rate 120 events/min
- Vascular access: umbilical vein catheter
Hypoglycaemia in neonate:
- Blood glucose <2.6 mmol/L in neonate = emergency
- Treatment: 2 mL/kg 10% Dextrose IV
Respiratory Distress Syndrome (RDS):
- Premature baby (lungs not mature, lack surfactant)
- Signs: grunting, nasal flaring, subcostal retractions, cyanosis
- Treatment: surfactant replacement therapy, CPAP
Q28. Obstetric emergencies - full details
Pre-eclampsia vs Eclampsia:
| Feature | Pre-eclampsia | Eclampsia |
|---|
| BP | Hypertension (>140/90) | Severe hypertension |
| Proteinuria | Yes | Yes |
| Seizures | No | YES |
| Timing | >20 weeks gestation | >20 weeks (can be postnatal) |
| Treatment | Antihypertensives; monitor | MgSO4 + antihypertensives + delivery |
MgSO4 protocol:
- Loading dose: 4g IV in 100 mL NS over 10-15 min
- Maintenance: 1g/hr IV infusion
- Toxicity signs (in order): Loss of DTRs → Respiratory depression → Cardiac arrest
- Antidote for MgSO4 toxicity: Calcium gluconate 10 mL of 10% IV
- Therapeutic level: 2-3.5 mmol/L
- Check: patellar reflexes, urine output, respiratory rate before each dose
HELLP Syndrome (severe pre-eclampsia variant):
- Hemolysis + Elevated Liver enzymes + Low Platelets
- Treatment: Urgent delivery; transfuse if platelets <20,000
Ectopic Pregnancy:
- Fertilised egg implants outside uterus (usually fallopian tube)
- Risk factors: PID, previous ectopic, IUD
- Signs: Amenorrhoea + lower abdominal pain + vaginal bleeding
- Can rupture → massive intraperitoneal hemorrhage → shock
- Positive pregnancy test + hemodynamic instability = ruptured ectopic until proven otherwise
- Treatment: Emergency surgery (salpingectomy)
Placenta Praevia vs Placental Abruption:
| Feature | Placenta Praevia | Placental Abruption |
|---|
| Bleeding | Painless, bright red, PV | Painful, dark blood, tender uterus |
| Uterus | Soft | Rigid/board-like |
| Cause | Placenta covers cervix | Premature separation of placenta |
| Fetal distress | Variable | Common (fetus deprived) |
| Management | Do NOT do vaginal exam; C-section | Immediate delivery; fluid resuscitation |
DAY 9 - MEDICAL EMERGENCIES (EXTENDED)
Q29. What is acute pulmonary oedema and how do you manage it?
Definition: Fluid accumulation in lung alveoli and interstitium, causing severe dyspnoea.
Most common cause: Left ventricular failure (LVF) - backed up pressure from failing LV pushes fluid into lungs
Presentation:
- Severe breathlessness (orthopnoea - cannot lie flat)
- Pink frothy sputum
- Bilateral crackles (basal, moving upward as severity worsens)
- SpO2 dropping despite O2
- Tachycardia; raised JVP; gallop rhythm (S3)
CXR findings (ABCDE):
- Alveolar oedema (bat wing opacification)
- B-lines / Kerley B lines (interstitial oedema)
- Cardiomegaly
- Diversion (upper lobe blood diversion)
- Effusions (pleural)
Management:
- Sit patient upright
- High-flow O2 (target SpO2 94-98%)
- CPAP (Continuous Positive Airway Pressure) - if available; improves gas exchange significantly
- IV Furosemide (Frusemide) 40-80 mg IV (diuresis - removes fluid)
- GTN (Glyceryl Trinitrate) sublingual/infusion (vasodilation - reduces preload/afterload; only if BP >90)
- Morphine 2.5-5 mg IV (reduces anxiety and venodilation - use cautiously)
- Treat underlying cause: AF (rate control), MI (reperfusion), hypertensive crisis (antihypertensives)
- Intubation if respiratory failure despite CPAP
Q30. What is COPD exacerbation and how do you manage it?
COPD exacerbation: Acute worsening of chronic breathlessness, cough, sputum beyond normal day-to-day variation
Triggers: Respiratory infection (viral/bacterial), air pollution, medication non-compliance
Signs:
- Barrel chest, pursed lip breathing, use of accessory muscles
- Prolonged expiratory phase, wheeze
- Cyanosis, confusion (hypercapnia)
- SpO2 low but may be chronically low
Management:
- Controlled O2: target SpO2 88-92% (NOT 100% - hypercapnic drive)
- Use Venturi mask 24-28% first; titrate up slowly
- Nebulized Salbutamol 2.5-5 mg + Ipratropium 0.5 mg (bronchodilators)
- Oral Prednisolone 30-40 mg (or hydrocortisone 200 mg IV if cannot swallow)
- Antibiotics if purulent sputum/signs of infection (amoxicillin or doxycycline)
- NIV (BiPAP) for hypercapnic respiratory failure (pH <7.35 + PaCO2 >6 kPa)
- Intubation only if NIV fails (high mortality in COPD)
Q31. What is acute kidney injury (AKI) and what are the causes?
Definition: Sudden decrease in kidney function over hours to days, causing build-up of waste products (creatinine, urea).
RIFLE/KDIGO criteria: Rising creatinine >1.5x baseline or decreased urine output <0.5 mL/kg/hr for >6 hours
Causes (Pre-renal, Renal, Post-renal):
| Category | Causes | Key feature |
|---|
| Pre-renal (most common) | Hypovolemia, sepsis, cardiac failure, NSAIDs | BUN:Cr ratio >20; responds to fluids |
| Intrinsic Renal | ATN (ischemia/nephrotoxins), glomerulonephritis, contrast nephropathy | BUN:Cr ratio <20; does NOT respond to fluids alone |
| Post-renal (obstructive) | BPH, kidney stones, pelvic tumour | Oliguria or anuria; ultrasound shows hydronephrosis |
Treatment:
- Treat underlying cause
- Stop nephrotoxins (NSAIDs, aminoglycosides, contrast)
- Fluid challenge for pre-renal AKI (500 mL crystalloid bolus)
- Monitor electrolytes - especially hyperkalemia (cardiac arrhythmias)
- Strict fluid balance and urine output measurement
- Dialysis if: severe hyperkalemia, metabolic acidosis, pulmonary edema, uremia
Q32. How do you manage hyperkalemia?
Dangerous potassium levels: K+ >6.0 mmol/L = life-threatening cardiac arrhythmias
ECG changes (in order of worsening):
- Peaked (tall, tented) T waves - FIRST sign
- Prolonged PR interval
- Widening QRS
- Sine wave pattern
- VF/Asystole
Emergency treatment:
- Calcium gluconate 10 mL 10% IV over 2-5 min (stabilizes cardiac membrane - FIRST LINE; works in 1-3 min; does NOT reduce K+)
- Insulin 10 units + 50 mL 50% Dextrose IV (shifts K+ into cells; works in 15-30 min)
- Salbutamol 10-20 mg nebulized (shifts K+ into cells; adjunct)
- Sodium bicarbonate (if metabolic acidosis)
- Calcium resonium (oral/PR) or Patiromer (binds K+ in gut - slow)
- Dialysis for severe/refractory hyperkalemia
DAY 10 - HOSPITAL PROTOCOLS, COMMUNICATION & FINAL EXAM PREP
Q33. What is the hospital emergency response for Code Blue?
Code Blue = Adult cardiac/respiratory arrest in hospital
Response Protocol:
-
Person who finds patient: Calls "Code Blue" to switchboard (or presses emergency button), starts CPR
-
Code Blue team responds within 3 minutes (typically: senior doctor, anaesthetist, nurse, runner)
-
Role allocation on arrival:
- Team leader: Most senior clinician; directs team; rhythm analysis decisions
- Compressor: CPR (rotated every 2 min to prevent fatigue)
- Airway person: BVM → intubation
- IV/IO access + drug administration
- Recorder: Documents time of events, drugs, rhythm changes
- Runner: Gets drugs, equipment
-
DNAR (Do Not Attempt Resuscitation): Always check before starting resuscitation; valid DNAR order overrides team response
-
Post-resuscitation care (ROSC):
- Target SpO2 94-98%; ETCO2 35-40 mmHg
- Target MAP ≥65 mmHg; avoid hypotension
- 12-lead ECG (check for STEMI → PCI)
- Targeted Temperature Management (TTM): 32-36°C for 24 hours if still unconscious
- Urgent CT brain if cause unclear
Q34. What are medication safety practices in emergency settings?
"5 Rights" of drug administration:
- Right Patient (check ID band + ask name + DOB)
- Right Drug (read label twice)
- Right Dose (calculate correctly; double-check with colleague for high-risk drugs)
- Right Route (IV, IM, oral, nebulized)
- Right Time (give at correct interval)
High-alert medications in emergency (double-check ALWAYS):
- Insulin (type + dose)
- Concentrated electrolytes (KCl, NaCl 3%, MgSO4)
- Opioids
- Anticoagulants (heparin, warfarin)
- Neuromuscular blockers (succinylcholine, rocuronium)
- Concentrated adrenaline (1:1000 vs 1:10,000)
Never do:
- Give a verbal order without read-back confirmation
- Pre-draw and label multiple syringes without immediately using them
- Give any drug without checking allergy status first
Q35. What are infection prevention and control (IPC) fundamentals?
Standard Precautions (apply to ALL patients):
- Hand hygiene (5 moments)
- PPE: Gloves, apron, mask, eye protection (based on risk)
- Safe sharps disposal
- Safe handling of body fluids
- Environmental cleaning
Transmission-Based Precautions:
| Type | Route | Conditions | PPE |
|---|
| Contact | Direct/indirect contact | MRSA, VRE, Norovirus, C. diff | Gloves + apron; single room |
| Droplet | Large droplets (>5 microns) | Meningitis, influenza, mumps | Surgical mask within 1 meter |
| Airborne | Small particles (<5 microns) | TB, measles, chickenpox | N95/FFP2 mask; negative pressure room |
N95 vs surgical mask:
- Surgical mask: Protects OTHERS from the wearer (large droplets only)
- N95/FFP2/FFP3: Protects the WEARER from airborne particles (TB, aerosol-generating procedures)
Q36. What are the key documentation principles in emergency care?
MIST Handover (used for trauma/emergency handover):
- M - Mechanism of injury
- I - Injuries identified (and suspected)
- S - Signs and vital signs (initial and current)
- T - Treatment given (drugs, fluids, interventions) and time given
Legal documentation rules:
- Write legibly in BLACK ink
- Date, time, sign every entry
- Never use correction fluid/Tipp-Ex - strike through with single line and initial
- Record: Time of events, vital signs, assessments, interventions, responses, discussions with relatives
- Document any refusal of treatment (patient must sign)
- Document DNAR discussions
Electronic records:
- Every login is auditable - never use another person's login
- Document immediately or within the same shift
Q37. What are common interview MCQ scenario questions with answers?
Scenario 1:
A 60-year-old male collapses in the hospital corridor. He is unresponsive and not breathing. An ECG monitor shows VF. What is your IMMEDIATE next action?
Answer: Immediate defibrillation (do not start CPR first if a defibrillator is immediately available and VF is confirmed on monitor).
- Call for help
- Deliver shock: 200J biphasic (or 360J monophasic)
- Immediately resume CPR for 2 min post-shock
- Continue ACLS algorithm
Scenario 2:
A 25-year-old male is brought in by his friend after being found unconscious at a party. RR 6/min, pinpoint pupils, SpO2 78%.
Answer: Opioid overdose.
- Airway first: BVM ventilation with 100% O2
- IV/IM Naloxone 0.4-2 mg immediately
- Repeat every 2-3 minutes if no response (up to 10 mg total)
- Watch for re-sedation - observe for minimum 4 hours after last naloxone dose
Scenario 3:
A 30-year-old female, 32 weeks pregnant, has a tonic-clonic seizure in the antenatal ward. BP 170/110. No history of epilepsy.
Answer: Eclampsia.
- Airway, left lateral tilt, O2
- Diazepam 10 mg IV if actively seizing
- MgSO4 4g IV over 15 min (loading dose) + 1g/hr infusion
- Labetalol 20 mg IV or Nifedipine orally for BP
- Urgent obstetric review for delivery
Scenario 4:
A patient comes in with severe central chest pain radiating to back, BP right arm 180/100, left arm 140/80. What do you suspect?
Answer: Aortic dissection (BP difference >20 mmHg between arms is a key sign).
- Do NOT give thrombolysis (catastrophic)
- Urgent CT aortogram
- Urgent cardiothoracic surgery review
- Control BP: IV Labetalol or Esmolol (target systolic 100-120 mmHg)
Scenario 5:
A 70-year-old male with known COPD is brought in confused, RR 28, SpO2 82% on air. A junior nurse gives him 15 L O2 via NRM. What should you do?
Answer: Change to Venturi mask 24-28%, target SpO2 88-92%.
- In COPD: chronically high CO2; respiratory drive is hypoxic (not hypercapnic)
- High-flow O2 suppresses this drive → respiratory arrest
- Titrate O2 slowly; prepare for NIV (BiPAP) if hypercapnic
Q38. What are the most likely written exam MCQ topics for Apollo/CMC/Kavery?
Guaranteed Topics (appear in almost every hospital exam):
- CPR ratios and compression depths
- Defibrillation energy settings and shockable vs non-shockable rhythms
- ACLS drugs (epinephrine dose, amiodarone dose)
- 4H 4T reversible causes
- Anaphylaxis management (epinephrine dose, route, concentration)
- GCS scoring and intubation threshold
- Oxygen delivery devices and FiO2
- Types of shock and differentiating features
- Triage (START system - Red/Yellow/Green/Black)
- ECG rhythm recognition (AF, VT, VF, SVT, Complete heart block)
- STEMI location on ECG (leads)
- Stroke FAST assessment and tPA criteria
- Organophosphate poisoning signs (DUMBBELS) and antidotes
- Burns: Rule of Nines and Parkland formula
- Needle decompression site (tension pneumothorax)
- WHO 5 moments of hand hygiene
- Hospital emergency codes (Code Blue, Red, Pink, etc.)
- Normal vital signs by age group
- Pediatric CPR differences
- MgSO4 for eclampsia - dose and toxicity signs
COMPLETE RAPID REVISION CARD
Cut this out and read every morning:
| Topic | Key Number/Fact |
|---|
| Adult CPR compression rate | 100-120/min |
| Adult CPR depth | 5-6 cm |
| Adult CPR ratio (any number rescuers) | 30:2 |
| Child/Infant CPR ratio (2 rescuers) | 15:2 |
| VF/VT defibrillation (biphasic) | 200 J |
| VF/VT defibrillation (monophasic) | 360 J |
| Epinephrine cardiac arrest dose | 1 mg IV q3-5 min |
| Amiodarone VF/VT (1st dose) | 300 mg IV |
| Atropine bradycardia | 0.5 mg IV, max 3 mg |
| Adenosine SVT | 6 mg rapid IV |
| Intubate if GCS | ≤ 8 |
| Anaphylaxis drug, dose, route | Epinephrine 0.3 mg IM (1:1000) |
| Needle decompression site | 2nd ICS, MCL |
| Stroke tPA window | 4.5 hours |
| MgSO4 eclampsia load | 4 g IV over 15 min |
| Oxytocin PPH | 10 IU IM |
| Normal O2 target (general) | SpO2 94-98% |
| O2 target in COPD | SpO2 88-92% |
| Burns rule of nines (head) | 9% |
| Parkland formula | 4 mL x kg x % BSA |
| CO poisoning - give | 100% O2 via NRM |
| Opioid antidote | Naloxone 0.4-2 mg IV/IM |
| Organophosphate antidote | Atropine + Pralidoxime |
| Paracetamol antidote | N-Acetylcysteine (NAC) |
| Code Blue | Adult cardiac arrest |
| Code Pink | Paediatric cardiac arrest |
| Code Red | Fire |
Sources used: Tintinalli's Emergency Medicine | Rosen's Emergency Medicine | Miller's Anesthesia | Roberts and Hedges' Clinical Procedures in Emergency Medicine | The Washington Manual of Medical Therapeutics | Sabiston Textbook of Surgery | Goldman-Cecil Medicine | Lippincott's Pharmacology | Goodman & Gilman's Pharmacology | AHA 2020 Guidelines
You are now fully prepared for Days 2-10. Study one day per day, speak your answers out loud, and practice scenarios with a colleague. All the best for Apollo, CMC Vellore, Kavery and your multi-speciality hospital interviews!