Respiratory system examination comprehensive notes

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Respiratory System Examination - Comprehensive Notes

Based primarily on Murray & Nadel's Textbook of Respiratory Medicine and Goldman-Cecil Medicine

1. OVERVIEW

Physical examination of the chest uses four classic techniques performed in this sequence:
  1. Inspection
  2. Palpation
  3. Percussion
  4. Auscultation
Optimal conditions: privacy, warmth, good lighting, and a quiet room. The patient should be adequately undressed to expose the entire thorax, typically sitting upright.

2. GENERAL INSPECTION (from a distance)

Begin the moment you first see the patient. Note:

a) General Appearance

  • Level of distress, use of accessory muscles
  • Ability to speak in full sentences
  • Posture (tripod position in severe obstruction)
  • Nutritional status and cachexia (suggests malignancy or COPD)
  • Mental status changes (CO2 retention - "CO2 narcosis")

b) Chest Shape and Symmetry

DeformityAssociation
Barrel chestCOPD, emphysema (AP:lateral ratio approaches 1:1)
KyphoscoliosisRestrictive lung disease, cor pulmonale
Pectus excavatum (funnel chest)Usually cosmetic; severe cases restrict lung expansion
Pectus carinatum (pigeon chest)Chronic childhood asthma, Marfan syndrome
Harrison's sulcusHorizontal groove at level of diaphragm attachment; childhood respiratory disease

c) Breathing Pattern

Several classic patterns of ventilation:
PatternDescriptionCause
EupneaNormal (12-20 breaths/min)-
Tachypnoea>20 breaths/minHypoxia, fever, pain, anxiety
Bradypnoea<12 breaths/minOpiates, CNS depression
Cheyne-StokesWaxing-waning with apnoeic pausesHeart failure, CNS lesions, high altitude
Biot's (ataxic)Irregular without patternMedullary brainstem damage
KussmaulDeep, rapid, laboredMetabolic acidosis (DKA)
ApneusticProlonged inspiratory crampPontine lesion

d) Use of Accessory Muscles

  • Sternocleidomastoid, scalene, trapezius: activated in obstructive disease
  • Intercostal retraction: indicates increased work of breathing
  • Paradoxical chest movement (chest sinks inward on inspiration): diaphragm paralysis or flail chest

e) Peripheral Signs (Inspect the Hands, Face, Neck)

Hands:
  • Clubbing - loss of the normal angle between the nail and nail bed (Schamroth's sign); seen in bronchogenic carcinoma, chronic suppurative lung disease (bronchiectasis, abscess), cyanotic heart disease, fibrosing alveolitis, mesothelioma
  • Peripheral cyanosis - bluish discoloration of fingers/nail beds
  • Tar staining - tobacco leaf stains on teeth, lips, fingers
  • Asterixis (flapping tremor) - CO2 retention
  • Wasting of intrinsic hand muscles - Pancoast tumour (T1 involvement)
Face and Lips:
  • Central cyanosis - bluish discoloration of tongue and mucous membranes; indicates SaO2 <85%
  • Anaemia - pallor of conjunctivae
  • Horner's syndrome (ptosis, miosis, anhidrosis) - Pancoast tumour, mediastinal mass
Neck:
  • Elevated JVP - right heart failure, COPD with cor pulmonale, SVC obstruction
  • Lymphadenopathy - malignancy, sarcoidosis, infection
  • Tracheal position (see Palpation below)

3. PALPATION

a) Trachea

  • Palpate with a single finger in the suprasternal notch
  • Normally midline
  • Displaced toward the lesion: collapse, lung fibrosis, pneumonectomy
  • Displaced away from the lesion: large pleural effusion, tension pneumothorax, large mass
  • Fixed/immobile trachea: mediastinal fibrosis or malignant infiltration

b) Chest Expansion

  • Place both hands flat on the posterior chest with thumbs meeting at the midline (or anterior chest)
  • Ask the patient to take a deep breath
  • Normally symmetric; thumbs should move 5-6 cm apart
  • Reduced unilaterally: consolidation, effusion, pneumothorax, collapse on that side
  • Reduced bilaterally: COPD, bilateral fibrosis, neuromuscular disease

c) Vocal (Tactile) Fremitus

  • Place the ulnar aspect of both hands (or palms) on the chest wall
  • Ask the patient to say "one-two-three" or "ninety-nine"
  • Feel for transmitted vibration and compare symmetrically
FindingCause
Increased fremitusConsolidation (pneumonia) - enhanced sound transmission through solid tissue
Decreased fremitusPleural effusion, pneumothorax, emphysema, obesity (impaired transmission)
Absent fremitusLarge effusion, complete collapse with blocked airway

d) Other Palpation Findings

  • Subcutaneous emphysema: crepitant crackling sensation under the fingers (air in subcutaneous tissue - pneumothorax, tracheobronchial injury)
  • Bony tenderness: rib fractures, malignant deposits
  • Cervical rib: palpable supraclavicular mass
  • Fluctuant area: empyema necessitans (pus tracking through chest wall)
  • Apical impulse / heaves: cor pulmonale; in severe COPD, best felt subxiphoid

4. PERCUSSION

Technique

  • Place the middle finger of the non-dominant hand (pleximeter) firmly on the chest wall, parallel to ribs
  • Strike the middle phalanx sharply with the middle finger of the dominant hand (plexor)
  • Free, easy, uniform stroke - predominantly felt, not just heard
  • Compare symmetrical areas systematically

Percussion Notes

NoteQualityNormal LocationPathological Cause
ResonantHollow, low-pitched, longNormal lung-
HyperresonantBooming, louder than normalNormal lung in childrenEmphysema, pneumothorax
TympaniticDrum-like, higher frequencyStomach/bowelTension pneumothorax, large cavity
DullShort, high-pitched, thud-likeLiver, heartConsolidation, pleural effusion (upper zone), collapse
Stony dull (flat)Very dull, no resonanceNo normal locationMassive pleural effusion (below the fluid level)
Key Clinical Correlation: With a large pleural effusion, three zones are detected on percussion:
  • Above the fluid: normal resonance
  • At the fluid level: dullness
  • Below (at base): stony flat

Useful Percussion Tests

  • Liver dullness: normally begins at right 5th intercostal space mid-clavicular line; if absent - emphysema (hyperinflation pushes liver down)
  • Cardiac dullness: if absent - emphysema
  • Traube's space (LUQ): tympanic normally; dullness suggests splenomegaly or left-sided effusion

5. AUSCULTATION

Use the diaphragm of the stethoscope (high-pitched sounds). Listen at multiple symmetrical locations systematically - apex to base, anterior and posterior.

Normal Breath Sounds

SoundLocationCharacter
Bronchial (tubular)Over trachea and main bronchiLoud, high-pitched, hollow; expiration longer than or equal to inspiration; gap between I and E
BronchovesicularOver main bronchi; 1st and 2nd intercostal spacesIntermediate quality; I = E in duration
VesicularOver peripheral lungSoft, low-pitched, rustling; inspiration longer than expiration (I:E = 3:1); no gap
Bronchial breathing heard peripherally = pathological, suggests consolidation or collapse with patent airway

Added (Adventitious) Sounds

Crackles (formerly "rales")

  • Fine crackles (late inspiratory): brief, high-pitched, discontinuous sounds like rubbing hair near the ear; caused by sudden opening of small airways
    • Causes: pulmonary fibrosis (cryptogenic fibrosing alveolitis), pulmonary oedema (early), pneumonia
  • Coarse crackles (early inspiratory or expiratory): lower-pitched, bubbling, gurgling sounds; caused by secretions in larger airways
    • Causes: bronchiectasis, COPD with secretions, pulmonary oedema (later)

Wheezes (formerly "rhonchi")

  • Polyphonic (multiple pitches): diffuse airway obstruction; characteristic of asthma during an attack
  • Monophonic (single pitch): fixed obstruction of a single airway; suggests carcinoma, foreign body, or mucus plug
  • Inspiratory wheeze (stridor): high-pitched, audible at the neck; indicates upper airway/extrathoracic obstruction (laryngeal oedema, croup, foreign body)

Pleural Friction Rub

  • Creaking, leathery sound during both inspiration and expiration (sometimes only one phase)
  • Caused by roughened, inflamed pleural surfaces rubbing together
  • Heard in: pleurisy, pulmonary embolism with infarction, mesothelioma
  • Does NOT disappear with coughing (distinguishes from crackles)
  • May be heard even in the presence of pleural effusion (shape change of thickened pleura)

Voice-Transmitted Sounds

TestTechniqueNormalAbnormal (Consolidation)
Vocal resonancePatient says "ninety-nine"; listenMuffled, indistinctIncreased, clearer - "bronchophony"
Whispering pectoriloquyPatient whispers "one-two-three"; listenBarely audible, indistinctWhispered words heard clearly and distinctly
EgophonyPatient says "eeeee"; listen"eeeee" soundSounds like "aaaay" (E-to-A change)
All three reflect increased sound transmission through consolidated lung (as in lobar pneumonia). Egophony is also heard at the top of a pleural effusion where the lung is compressed.

6. CLINICAL PROFILES OF COMMON CONDITIONS

ConditionTracheaExpansionFremitusPercussionBreath SoundsAdded Sounds
Consolidation (Pneumonia)CentralReduced ipsilateralIncreasedDullBronchial breathingFine crackles, bronchophony, egophony
Pleural EffusionCentral (small); pushed away (large)Reduced ipsilateralAbsent/reducedStony dullReduced/absent (bronchial at top of effusion)Pleural rub (if pleurisy); egophony at top
PneumothoraxCentral (small); pushed away (tension)Reduced ipsilateralAbsent/reducedHyperresonantAbsent/reducedNone
Lobar Collapse (with blocked bronchus)Pulled towardReduced ipsilateralAbsentDullAbsentNone
Lobar Collapse (with patent bronchus)Pulled towardReducedIncreasedDullBronchialNone
Emphysema/COPDCentralReduced bilaterallyReducedHyperresonant, loss of liver/cardiac dullnessReduced vesicular, prolonged expirationWheeze, coarse crackles
Pulmonary FibrosisCentralReduced bilaterallyNormal/increasedNormalNormal, or bronchialFine, late inspiratory crackles (basal)
Asthma (attack)CentralReduced bilaterallyNormal/reducedNormal or hyperresonantProlonged expirationPolyphonic wheeze
BronchiectasisCentralNormal or reducedNormalNormalNormalCoarse crackles, bronchophony

7. EXTRAPULMONARY SOUNDS

  • Stridor: high-pitched continuous sound from turbulent flow in the extrathoracic (upper) airway; louder during inspiration; causes include epiglottitis, croup, foreign body, laryngeal oedema, tracheal stenosis - potentially life-threatening
  • Hamman's sign (mediastinal crunch): crunching or clicking sound with each heartbeat; caused by pneumomediastinum
  • Succussion splash: splashing sound with patient movement; suggests hydropneumothorax

8. STRUCTURED APPROACH SUMMARY

A systematic bedside approach:
  1. Wash hands, introduce, position (sitting upright)
  2. General inspection from end of bed (distress, colour, breathing pattern, body habitus, equipment like inhalers, O2, sputum pots)
  3. Hands (clubbing, cyanosis, tar staining, asterixis, wasting)
  4. Arms (pulse rate and rhythm, pulsus paradoxus - >10 mmHg drop in systolic BP during inspiration, suggests severe asthma or cardiac tamponade)
  5. Face (central cyanosis, anaemia, Horner's syndrome, pursed lip breathing)
  6. Neck (JVP, tracheal position, lymphadenopathy, accessory muscle use)
  7. Chest - Inspection (shape, deformities, scars, chest movement symmetry, intercostal recession, chest drains)
  8. Chest - Palpation (confirm trachea, apex beat, expansion, vocal fremitus)
  9. Chest - Percussion (all zones, anterior and posterior, compare sides)
  10. Chest - Auscultation (breath sounds quality, added sounds, vocal resonance)
  11. Complete (check for ankle oedema, peak flow, oxygen saturations, request CXR and spirometry)

9. IMPORTANT MNEMONICS

IPPA = Inspection, Palpation, Percussion, Auscultation
Causes of DECREASED chest expansion (unilateral):
  • Pneumothorax
  • Pleural effusion
  • Pneumonia/consolidation
  • Post-lobectomy/pneumonectomy
  • Pulmonary fibrosis (unilateral)
Causes of CLUBBING in respiratory:
  • BLASTC: Bronchiectasis, Lung abscess, Asbestosis/fibrosing alveolitis, Suppurative lung disease (empyema), Tumour (bronchogenic carcinoma), Cyanotic heart disease (not strictly respiratory but often tested)
SHIFT of trachea:
  • Pushed away: effusion, tension pneumothorax
  • Pulled toward: collapse, fibrosis, pneumonectomy

Sources: Murray & Nadel's Textbook of Respiratory Medicine, 2-Volume; Goldman-Cecil Medicine; Barash Clinical Anesthesia, 9e
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