Reproductive health kmtc notes detailed exam oriented

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These notes cover the 10 core units of KMTC Reproductive Health. Here's what's included:
UnitTopic
1Introduction & Definition
2Family Planning & Contraception (FAB methods, OCPs, IUDs, medical abortion)
3Antenatal Care (Naegele's rule, visit schedule, GBS, GDM screening)
4STIs (table, screening, neonatal risks)
5Hypertensive disorders (Preeclampsia, Eclampsia, HELLP, MgSO₄)
6Postpartum Haemorrhage (4 T's, uterotonic drugs)
7Teratogens & drugs in pregnancy
8Adolescent reproductive health
9Infertility
10Cervical cancer & HPV
Exam hot spots: The 4 T's of PPH, HELLP criteria, GBS screening timing (35–37 weeks), GDM screening (26–28 weeks), Naegele's rule, and contraceptive failure rates are the most frequently tested areas. Let me know if you want any unit expanded further!

Obstetrics and gynae notes detailed exam oriented

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Excellent — I now have comprehensive content from multiple authoritative textbooks. Let me compile the full, exam-oriented O&G notes.

🏥 OBSTETRICS & GYNAECOLOGY — KMTC EXAM-ORIENTED NOTES


UNIT 1: NORMAL LABOUR & DELIVERY

1.1 Definition of Labour

Regular uterine contractions producing progressive cervical change (effacement + dilation).
True vs False Labour:
FeatureTrue LabourFalse Labour (Braxton Hicks)
ContractionsRegular, increasing in frequency & intensityIrregular, no progression
Cervical changePresentAbsent
Effect of walkingIntensifies contractionsNo effect or diminishes
Pain locationBack → abdomenAbdomen only

1.2 Stages of Labour (HIGH YIELD — always in exams)

StageOnsetEnd
First stage — Latent + ActiveOnset of true labourFull cervical dilation (10 cm)
Second stageFull dilationDelivery of baby
Third stageDelivery of babyDelivery of placenta
Fourth stageDelivery of placentaContracted uterus (first 1–2 hours)

First Stage — Latent Phase

  • Variable length
  • Nullipara: <20 hours; Multipara: <14 hours
  • Little cervical dilation; mainly cervical preparation (softening, effacement, anterior positioning)
  • Conduction anaesthesia in this phase may prolong or arrest progress
  • Prolonged latent phase → treat with morphine in hospital setting

First Stage — Active Phase

  • Begins at approximately 4–5 cm cervical dilation
  • Rapid dilation
  • Normal dilation rate:
    • Nullipara: ≥1.2 cm/hr
    • Multipara: ≥1.5 cm/hr
  • Progress depends on: uterine contraction strength/frequency, fetal head size/position/attitude, and pelvic shape

1.3 Cardinal Movements of Labour (7 — memorise in order)

  1. Engagement — fetal head enters pelvic inlet
  2. Descent — downward movement
  3. Flexion — chin to chest; reduces AP diameter
  4. Internal rotation — occiput rotates anteriorly
  5. Extension — head extends under symphysis pubis
  6. External rotation (restitution) — head aligns with shoulders
  7. Expulsion — delivery of body
Mnemonic: "Every Decent Family Is Exceedingly Extraordinary"

1.4 Fetal Heart Rate (FHR) Monitoring

Normal baseline FHR: 110–160 beats/min
Reactive NST (Non-Stress Test): ≥2 accelerations of ≥15 bpm above baseline lasting ≥15 seconds in 20 minutes
Types of Decelerations:
TypeCauseShapeRelation to ContractionSignificance
EarlyFetal head compression → vagalSmooth mirror imageBegins with contraction, nadir at peakBenign/Reassuring
VariableUmbilical cord compression"V", "U", or "W" shapeVariable relationshipMost common (50–80% of deliveries); concerning if severe
LateUteroplacental insufficiencyGradual; begins AFTER contraction peakBegins late, returns after contraction endsOminous — Category III
SinusoidalFetal anaemia / severe hypoxiaSmooth sine-wave, 2–5 cycles/minOminous — Category III
FHR Tachycardia (>160 bpm) causes:
  • Chorioamnionitis, maternal fever
  • Sympathomimetic drugs (terbutaline)
  • Atropine, fetal tachyarrhythmia
  • Fetal hypoxia, prematurity
FHR Bradycardia causes:
  • Cord prolapse/compression
  • Tetanic contractions
  • Paracervical/epidural block
  • Maternal seizure
Late deceleration management:
  1. Turn patient on side (lateral position) → ↑ cardiac output & uterine blood flow
  2. IV fluids to correct hypotension
  3. Stop oxytocin infusion
  4. Administer oxygen
Tachysystole: >5 contractions in 10 minutes (averaged over 30 minutes)

1.5 NICHHD FHR Classification

CategoryInterpretationAction
I (Normal)Normal pH, fetal well-beingContinue current management
II (Indeterminate)UncertainFurther evaluation
III (Abnormal)Fetal hypoxia/acidosisImmediate intervention; expedited delivery

UNIT 2: COMPLICATIONS OF PREGNANCY — ANTEPARTUM HAEMORRHAGE

2.1 Causes of Antepartum Haemorrhage (APH)

Complicates 3–5% of pregnancies; leading cause of maternal and perinatal mortality
Main causes:
  1. Placenta praevia (painless)
  2. Abruptio placentae (painful)
  3. Vasa praevia
  4. Cervical/vaginal lesions

2.2 Placenta Praevia

Definition: Placenta implanted in the lower uterine segment, covering or near the internal cervical os
Classification (3 types):
TypeDescription
Complete/TotalPlacenta completely covers the cervical os
PartialPlacenta partially covers the os
MarginalPlacenta extends to the edge of the os
Incidence: 1 in 200–250 pregnancies
Classic presentation:
  • Painless, bright-red vaginal bleeding in the 3rd trimester
  • Average first bleed: 27–32 weeks
  • Abnormal lie (transverse/breech) — suspicious
  • Initial bleed is usually not fatal and stops spontaneously, then recurs
Risk factors:
  • Advanced maternal age
  • Increased parity (multiparity)
  • Previous uterine surgery (C-section scar)
  • Prior placenta praevia
  • Multiple gestation
  • Uterine abnormalities
  • Smoking
Diagnosis: Transabdominal ultrasound (93–98% accurate); use transvaginal US if position unclear — do NOT do vaginal examination (risk of massive haemorrhage)
Management:
  • If actively bleeding → IV access, crossmatch blood, IV fluids, oxygen
  • If fetus viable but compromised → urgent caesarean section
  • Rh-negative mothers → D immune globulin (RhoGAM)

2.3 Abruptio Placentae (Placental Abruption)

Definition: Premature separation of a normally situated placenta before delivery of the fetus
Incidence: 1 in 129 births (~1–2% of pregnancies)
Classic presentation:
  • Painful vaginal bleeding — hallmark
  • Uterine tenderness (board-like/woody uterus)
  • Frequent contractions
  • Fetal distress
  • Blood is characteristically dark red
Note: Vaginal bleeding occurs in 70% of cases; in 30%, bleeding is concealed (retroplacental)
Grades:
GradeFeatures
Grade I (Mild)<150 mL blood loss; mother/fetus stable
Grade II (Moderate)Fetal distress; maternal compromise
Grade III (Severe)Fetal death; DIC possible; 25% of cases
Complications:
  • DIC (disseminated intravascular coagulation)
  • Hypovolaemic shock
  • Renal failure
  • Couvelaire uterus (blood penetrates uterine wall)
Key investigations:
  • CBC, coagulation studies (PT/PTT, fibrinogen, D-dimer)
  • Kleihauer-Betke test (feto-maternal haemorrhage)
  • Blood type and crossmatch
Management:
  • Aggressive IV fluids/resuscitation
  • Packed RBCs on standby
  • Continuous fetal monitoring
  • Rh-negative mothers → D immune globulin
  • Urgent C-section if fetus viable but compromised

2.4 Placenta Praevia vs Abruptio — EXAM COMPARISON TABLE

FeaturePlacenta PraeviaAbruptio Placentae
PainPainlessPainful
Blood colourBright redDark red
Uterine toneSoftRigid/tender ("woody")
Onset3rd trimester; 27–32 wksAny time
ShockProportional to visible bloodOut of proportion to visible blood
Fetal lieOften abnormalUsually normal
DiagnosisUltrasoundClinical (US may be false-negative)
Vaginal examinationCONTRAINDICATEDCan be done

UNIT 3: HYPERTENSIVE DISORDERS OF PREGNANCY

3.1 Classification

ConditionBPProteinuriaOnset
Gestational hypertension≥140/90 mmHgAbsent>20 weeks
Preeclampsia≥140/90 mmHgPresent (≥300 mg/24h)>20 weeks
Severe preeclampsia≥160/110 mmHgPresent + end-organ damage>20 weeks
EclampsiaAnyPresentSeizures in preeclamptic
Chronic hypertension≥140/90 mmHgVariable<20 weeks or pre-pregnancy

3.2 Preeclampsia

Signs/symptoms of severe preeclampsia:
  • Severe headache
  • Visual disturbances (photopsia, scotoma)
  • Epigastric/RUQ pain (liver capsule stretch)
  • Pulmonary oedema
  • Oliguria
HELLP Syndrome:
  • H — Haemolysis (microangiopathic)
  • EL — Elevated Liver enzymes (↑AST, ↑ALT, ↑LDH)
  • LP — Low Platelets (<100,000/µL)
Suspect HELLP in a pregnant/postpartum woman with abdominal pain
Labs in HELLP: Peripheral blood smear → microangiopathic haemolytic anaemia; ↑transaminases; ↓platelets

3.3 Eclampsia

Definition: Seizures in a woman with pre-eclampsia; can occur up to 12 weeks postpartum
Incidence: 0.2% of pregnancies; terminates 1 in 1000 pregnancies
Major cause of maternal death: Intracranial haemorrhage
Perinatal mortality: 2–8.6%

3.4 Management of Severe Preeclampsia/Eclampsia

Magnesium Sulphate Protocol (MgSO₄ · 7H₂O):
  • Loading dose: 4 g (range 2–6 g) in 500 mL 5% dextrose over 15 minutes IV
  • Maintenance: 1–3 g/hr continuous infusion
  • Continue for 24 hours after delivery or last seizure
  • Keep calcium gluconate at bedside as antidote
Monitoring on MgSO₄:
  • Deep tendon reflexes (first sign of toxicity = loss of DTRs)
  • Respiratory rate (>12/min)
  • Urine output (>25 mL/hr)
  • Plasma Mg levels
Antihypertensives for acute severe HTN:
  • Labetalol IV (first-line)
  • Hydralazine IV
  • Nifedipine oral
Definitive treatment: DELIVERY of the baby
Gestational hypertension resolves within 10 days postpartum

UNIT 4: PRETERM LABOUR

Definition: Labour occurring between 20–37 weeks of gestation
Risk factors:
  • Prior preterm labour (most important risk factor)
  • Low socioeconomic status
  • Multiple gestation
  • Uterine anomalies
  • Bacterial vaginosis / UTI
  • Placenta praevia/abruption
  • Polyhydramnios
  • Low maternal weight (<50 kg)
  • Smoking, cocaine use
  • Poor prenatal care
Management of preterm labour:
  1. Tocolytics (delay delivery 48 hrs to allow corticosteroids to work):
    • Nifedipine (calcium channel blocker — preferred)
    • Terbutaline (β₂-agonist)
    • Indomethacin (PG inhibitor; <32 weeks)
    • MgSO₄ (also neuroprotective)
  2. Corticosteroids (fetal lung maturity):
    • Betamethasone 12 mg IM × 2 doses, 24 hours apart OR
    • Dexamethasone 6 mg IM × 4 doses, 12 hours apart
    • Give between 24–34 weeks
  3. Antibiotics if GBS positive or PROM

UNIT 5: PREMATURE RUPTURE OF MEMBRANES (PROM)

Definition: Rupture of membranes before onset of labour
Types:
TypeDefinition
PROMAt term (≥37 weeks)
PPROMPreterm (<37 weeks)
Diagnosis:
  • Pooling of fluid in vagina
  • Ferning (amniotic fluid crystallises in fern pattern on glass slide)
  • Nitrazine test — amniotic fluid turns paper blue (alkaline)
  • Ultrasound — oligohydramnios
Complications: Cord prolapse, infection (chorioamnionitis), preterm labour, fetal distress
Management:
  • ≥37 weeks: Induce labour (oxytocin)
  • <37 weeks: Expectant management, antibiotics (ampicillin/erythromycin), corticosteroids if <34 weeks

UNIT 6: ECTOPIC PREGNANCY

Definition: Implantation of the fertilised ovum outside the uterine cavity
Sites (order of frequency):
  1. Ampulla of fallopian tube (most common — ~70%)
  2. Isthmus
  3. Fimbria
  4. Ovary
  5. Cervix
  6. Abdominal (rarest)
Risk factors:
  • Previous ectopic pregnancy (greatest risk)
  • Previous tubal surgery
  • PID/salpingitis
  • IUD use
  • Infertility treatment (IVF)
  • Smoking
  • Prior pelvic surgery
Clinical features:
  • Amenorrhoea (missed period)
  • Unilateral pelvic/lower abdominal pain
  • Vaginal bleeding (brown, irregular)
  • Ruptured ectopic: Sudden severe pain + haemoperitoneum → shoulder tip pain (diaphragmatic irritation) → shock
Investigations:
  • Serum β-hCG (positive even at low levels; single low level does NOT exclude ectopic)
  • Transvaginal ultrasound — empty uterus + adnexal mass; free fluid in Pouch of Douglas
  • Serial β-hCG: In normal IUP, hCG doubles every 48–72 hours; slower rise or plateau → suspect ectopic
  • Falling hCG — most common indicator for spontaneous resolution of ectopic
Management:
OptionCriteriaDetails
ExpectantFalling hCG, small ectopic, stable patientClose monitoring
Medical — MethotrexateUnruptured, hCG <5000 IU/L, no cardiac activity, stableFolate antagonist; inhibits trophoblast cells; 70–95% success
Surgical — LaparoscopyRuptured/unstable OR medical failedSalpingostomy (preserve tube for future fertility); Salpingectomy (not wanting future fertility)
Methotrexate contraindications: Hepatic/renal disease, active infection, breastfeeding, immunodeficiency

UNIT 7: ABORTION (MISCARRIAGE)

Definition: Expulsion of products of conception before 28 weeks (or fetal weight <1000 g)
Incidence: ~15% of clinically recognised pregnancies; sporadic chromosomal aneuploidy accounts for ~60% of losses

Types of Abortion (HIGH YIELD)

TypeCervical OsBleedingProducts PassedPregnancy Viable?Management
ThreatenedClosedMildNoneYes (Doppler confirms)Reassure, bed rest, avoid coitus
InevitableOpenProfuseNone yetNoD&C
IncompleteOpenHeavyPartialNoD&C
CompleteClosedMinimalAll passedNoConfirm with USS; observe
Missed (Silent)ClosedAbsent or brownNone (retained >4 wks)NoD&C or misoprostol
SepticOpenFoul dischargePresentNoIV antibiotics + D&C
Recurrent≥3 losses; investigate cause
Rh-negative management:
  • <13 weeks: 50 µg anti-D (Rh immunoglobulin) IM
  • 13 weeks: 300 µg anti-D IM

Causes of Recurrent Miscarriage (≥3 losses):

  • Chromosomal (parental/fetal aneuploidy) — most common
  • Antiphospholipid syndrome
  • Uterine anomalies (septum, fibroids)
  • Cervical incompetence
  • Luteal phase defect
  • Thyroid disease

UNIT 8: MOLAR PREGNANCY (GESTATIONAL TROPHOBLASTIC DISEASE)

Definition: Abnormal fertilisation resulting in proliferating trophoblastic tissue
Types:
FeatureComplete MoleIncomplete Mole
Fetal partsAbsentPresent (usually abnormal)
Karyotype46XX (diploid — all paternal)Triploid (69XXY)
Villous changesAll villi hydropicSome villi hydropic
Risk of malignancy (GTD)15–20%5–10%
Uterine sizeLarge for datesSmall or normal
hCGVery highMildly elevated
Ultrasound (complete mole): "Snowstorm" appearance — distended cavity with numerous anechoic cysts
Presentation:
  • Vaginal bleeding
  • Uterus large for dates
  • Hyperemesis
  • Early preeclampsia (<20 weeks — pathognomonic!)
  • Very high hCG → bilateral theca lutein cysts → bilateral ovarian enlargement
Management:
  1. Suction curettage (D&C)
  2. Serial β-hCG monitoring until 3 consecutive normal levels
  3. Contraception for 1 year (to allow hCG normalisation and detect malignant transformation)
  4. Chest X-ray (metastases)
Indications for chemotherapy: Rising or plateauing hCG after evacuation → gestational trophoblastic neoplasia (GTN) → Methotrexate ± Actinomycin D

UNIT 9: PREECLAMPSIA IN LABOUR — MgSO₄ PROTOCOL (STRUCTURED)

StepAction
PositionLateral recumbent
DietNothing by mouth (NBM); clear liquids if muscle tone adequate
IV accessLactated Ringer's 5% dextrose
Loading doseMgSO₄ 4g IV over 15 min
MaintenanceMgSO₄ 1–3 g/hr IV infusion
Antidote at bedsideCalcium gluconate
MonitorDTRs, urine output (I&O), lung bases (pulmonary oedema), plasma Mg levels
DurationContinue 24 hours post-delivery

UNIT 10: GYNAECOLOGY — KEY TOPICS

10.1 Pelvic Inflammatory Disease (PID)

Definition: Ascending infection from the cervix/vagina → uterus, fallopian tubes, ovaries
Causative organisms:
  • Chlamydia trachomatis (most common in developed countries)
  • Neisseria gonorrhoeae
  • Anaerobes, Gram-negative rods
Clinical features (Chandelier sign):
  • Lower abdominal pain (bilateral)
  • Cervical motion tenderness (CMT) — "chandelier sign"
  • Adnexal tenderness
  • Fever
  • Vaginal discharge (mucopurulent)
  • ↑WBC, ↑ESR, ↑CRP
Complications:
  • Tubo-ovarian abscess (TOA)
  • Infertility (blocked tubes)
  • Ectopic pregnancy risk ↑
  • Fitz-Hugh-Curtis syndrome (perihepatitis — RUQ pain + perihepatic adhesions)
  • Chronic pelvic pain
Treatment:
  • Outpatient: Ceftriaxone IM + Doxycycline + Metronidazole
  • Inpatient: IV Cefoxitin + Doxycycline OR Clindamycin + Gentamicin

10.2 Ovarian Cysts & Torsion

Common types:
CystFeatures
Follicular cystMost common; resolves spontaneously in 4–8 weeks
Corpus luteum cystCan rupture → haemoperitoneum
PCOS (polycystic ovaries)Multiple small follicles; anovulation, hyperandrogenism
Dermoid (teratoma)Most common benign tumour in women <30; contains hair/teeth/fat
Endometrioma ("chocolate cyst")Endometriosis; filled with old blood
Ovarian torsion:
  • Sudden severe unilateral pelvic pain
  • Nausea and vomiting
  • Tender adnexal mass on exam
  • Doppler USS: absent/reduced blood flow
  • Management: Surgical untwisting (detorsion) → salpingo-oophorectomy if necrotic

10.3 Uterine Fibroids (Leiomyomata)

Most common benign tumour of the uterus; oestrogen-dependent; regress after menopause
Types by location:
  • Intramural (most common)
  • Subserosal
  • Submucosal (most likely to cause heavy bleeding and infertility)
  • Pedunculated
Symptoms: Heavy menstrual bleeding (menorrhagia), pressure symptoms, infertility, recurrent miscarriage
Management:
  • Medical: GnRH analogues (shrink fibroid pre-surgery), tranexamic acid (bleeding), NSAIDs
  • Surgical: Myomectomy (fertility-sparing), hysterectomy (definitive)

10.4 Endometriosis

Definition: Presence of endometrial tissue outside the uterus
Sites: Ovaries (most common), peritoneum, Pouch of Douglas, bladder, bowel
Symptoms (3 Ds):
  • Dysmenorrhoea (painful periods — cyclical)
  • Dyspareunia (painful intercourse)
  • Dyschezia (painful defaecation)
Gold standard diagnosis: Laparoscopy + biopsy
Treatment: NSAIDs, OCPs (cycle suppression), GnRH analogues, laparoscopic ablation, hysterectomy

10.5 Cervical Cancer

Cause: HPV types 16 and 18 (responsible for ~70% of cases)
Risk factors:
  • Early sexual debut
  • Multiple sexual partners
  • Multiparity
  • Immunosuppression
  • Smoking
  • STIs
Screening: Pap smear (cervical cytology)
  • Start: Age 21 or 3 years after sexual debut
  • Frequency: Every 3 years if normal (cytology alone)
HPV vaccine: Ideally age 9–14 (before sexual debut); can be given up to age 26 routinely
Staging (FIGO):
StageDescription
IConfined to cervix
IIExtends beyond cervix but not pelvic wall
IIIExtends to pelvic wall / lower third of vagina
IVBladder/rectum or distant metastases
Treatment: Stage I → surgery (Wertheim's hysterectomy); Stage II–IV → chemoradiotherapy

10.6 Menstrual Disorders

TermDefinition
AmenorrhoeaAbsence of periods
Primary amenorrhoeaNo period by age 16 (with secondary sex characteristics) or age 14 (without)
Secondary amenorrhoeaNo period for ≥6 months in previously menstruating woman
MenorrhagiaHeavy periods (>80 mL/cycle)
MetrorrhagiaIrregular intermenstrual bleeding
MenometrorrhagiaHeavy + irregular bleeding
DysmenorrhoeaPainful periods
OligomenorrhoeaCycles >35 days
PolymenorrhoeaCycles <21 days

UNIT 11: POSTPARTUM HAEMORRHAGE (PPH)

Definition: Blood loss:
  • Vaginal delivery: >500 mL
  • C-section: >1000 mL
Primary PPH: Within 24 hours of delivery Secondary PPH: 24 hours–6 weeks postpartum

The 4 T's of PPH (EXAM GOLD)

TCauseFrequency
ToneUterine atony~80% (most common)
TissueRetained placenta/products~10%
TraumaGenital tract lacerations~7%
ThrombinCoagulopathy (DIC, von Willebrand)~3%
Risk factors for uterine atony:
  • Overdistended uterus (macrosomia, multiple gestation, polyhydramnios)
  • Prolonged labour
  • Grand multiparity
  • General anaesthesia
  • Oxytocin use

Management of PPH (stepwise)

  1. Uterine massage (bimanual compression)
  2. Uterotonic drugs:
    • Oxytocin (1st line): 10 IU IM or IV infusion
    • Ergometrine/Methylergonovine (Syntometrine): 0.2 mg IM (CI in hypertension)
    • Misoprostol: 600–1000 µg rectal/sublingual (useful in low-resource settings)
    • Carboprost (PGF2α): 0.25 mg IM q15 min (CI in asthma)
  3. Balloon tamponade (Bakri balloon)
  4. Surgical:
    • B-Lynch compression suture
    • Uterine artery ligation
    • Internal iliac artery ligation
    • Hysterectomy (last resort)
  5. Blood products: Fresh frozen plasma (FFP), packed RBCs, cryoprecipitate for DIC

UNIT 12: PUERPERAL SEPSIS

Definition: Fever ≥38°C on ≥2 occasions, >24 hours apart, within the first 10 days postpartum (excluding first 24 hours)
Sources of infection:
  • Uterus (endometritis — most common)
  • Urinary tract
  • Breast (mastitis)
  • Wound (episiotomy, C-section)
Causative organisms: Group A Streptococcus, Staphylococcus aureus, E. coli, anaerobes
Features of endometritis:
  • Fever
  • Uterine tenderness
  • Foul-smelling lochia
  • Subinvolution of uterus
Treatment: IV Clindamycin + Gentamicin

QUICK REVISION SUMMARY TABLES

Antihypertensive Drugs in Pregnancy — Safety

DrugSafe?Notes
Methyldopa✅ Safe (1st line)
Labetalol✅ SafeAlso used IV for acute HTN
Nifedipine✅ SafeOral; 2nd line
Hydralazine✅ SafeIV for acute HTN
ACE inhibitors (enalapril etc.)❌ ContraindicatedRenal dysgenesis (2nd/3rd trimester)
ARBs❌ ContraindicatedSame as ACE inhibitors

Types of Caesarean Section

TypeIndication
ElectivePlanned; no emergency
Emergency Category 1Immediate threat to mother/fetus; <30 min
Emergency Category 2Maternal/fetal compromise; not immediate
Emergency Category 3Needs early delivery; no compromise

HIGH-FREQUENCY EXAM QUESTIONS

Q: Painless APH in the 3rd trimester — diagnosis? A: Placenta praevia. Do NOT do vaginal examination. First-line investigation: ultrasound
Q: Painful APH with rigid uterus — diagnosis? A: Abruptio placentae. Clinical diagnosis; ultrasound has high false-negative rate
Q: Seizures in a patient with BP 170/110 at 34 weeks — diagnosis & management? A: Eclampsia. Give MgSO₄ (loading dose 4g IV over 15 min), stabilise, deliver baby. Antidote = calcium gluconate
Q: Empty uterus on TVS + positive hCG + unilateral pain in a woman with history of PID — diagnosis? A: Ectopic pregnancy. Manage with methotrexate if unruptured and stable; laparoscopy if ruptured
Q: Type of abortion where cervical os is closed, no products passed, but the fetus is dead on ultrasound? A: Missed abortion
Q: What is the most important risk factor for preterm labour? A: Previous preterm labour
Q: What is the 7th cardinal movement of labour? A: Expulsion
Q: At what cervical dilation does the active phase of labour begin? A: 4–5 cm
Q: What type of FHR deceleration is associated with umbilical cord compression? A: Variable deceleration
Q: What is the most common benign ovarian tumour in women under 30? A: Dermoid cyst (mature cystic teratoma)
Q: What is the gold standard for diagnosing endometriosis? A: Laparoscopy + biopsy

Sources: Textbook of Family Medicine 9e; Goldman-Cecil Medicine; Morgan & Mikhail's Clinical Anesthesiology 7e; Creasy & Resnik's Maternal-Fetal Medicine; Swanson's Family Medicine Review; Berek & Novak's Gynecology; Tintinalli's Emergency Medicine; Rosen's Emergency Medicine

These notes cover 12 units of core O&G content. Summary of topics:
#Topic
1Normal Labour — stages, cardinal movements, FHR monitoring
2Antepartum Haemorrhage — placenta praevia vs abruption
3Hypertensive disorders — preeclampsia, eclampsia, HELLP
4Preterm labour — tocolytics, steroids
5PROM
6Ectopic pregnancy
7Abortion types (6 types with complete comparison table)
8Molar pregnancy/GTD
9MgSO₄ protocol structured table
10Gynaecology — PID, fibroids, endometriosis, cervical cancer, menstrual disorders
11PPH — 4 T's + stepwise management
12Puerperal sepsis
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