Excellent — I now have comprehensive content from multiple authoritative textbooks. Let me compile the full, exam-oriented O&G notes.
🏥 OBSTETRICS & GYNAECOLOGY — KMTC EXAM-ORIENTED NOTES
UNIT 1: NORMAL LABOUR & DELIVERY
1.1 Definition of Labour
Regular uterine contractions producing progressive cervical change (effacement + dilation).
True vs False Labour:
| Feature | True Labour | False Labour (Braxton Hicks) |
|---|
| Contractions | Regular, increasing in frequency & intensity | Irregular, no progression |
| Cervical change | Present | Absent |
| Effect of walking | Intensifies contractions | No effect or diminishes |
| Pain location | Back → abdomen | Abdomen only |
1.2 Stages of Labour (HIGH YIELD — always in exams)
| Stage | Onset | End |
|---|
| First stage — Latent + Active | Onset of true labour | Full cervical dilation (10 cm) |
| Second stage | Full dilation | Delivery of baby |
| Third stage | Delivery of baby | Delivery of placenta |
| Fourth stage | Delivery of placenta | Contracted uterus (first 1–2 hours) |
First Stage — Latent Phase
- Variable length
- Nullipara: <20 hours; Multipara: <14 hours
- Little cervical dilation; mainly cervical preparation (softening, effacement, anterior positioning)
- Conduction anaesthesia in this phase may prolong or arrest progress
- Prolonged latent phase → treat with morphine in hospital setting
First Stage — Active Phase
- Begins at approximately 4–5 cm cervical dilation
- Rapid dilation
- Normal dilation rate:
- Nullipara: ≥1.2 cm/hr
- Multipara: ≥1.5 cm/hr
- Progress depends on: uterine contraction strength/frequency, fetal head size/position/attitude, and pelvic shape
1.3 Cardinal Movements of Labour (7 — memorise in order)
- Engagement — fetal head enters pelvic inlet
- Descent — downward movement
- Flexion — chin to chest; reduces AP diameter
- Internal rotation — occiput rotates anteriorly
- Extension — head extends under symphysis pubis
- External rotation (restitution) — head aligns with shoulders
- Expulsion — delivery of body
Mnemonic: "Every Decent Family Is Exceedingly Extraordinary"
1.4 Fetal Heart Rate (FHR) Monitoring
Normal baseline FHR: 110–160 beats/min
Reactive NST (Non-Stress Test): ≥2 accelerations of ≥15 bpm above baseline lasting ≥15 seconds in 20 minutes
Types of Decelerations:
| Type | Cause | Shape | Relation to Contraction | Significance |
|---|
| Early | Fetal head compression → vagal | Smooth mirror image | Begins with contraction, nadir at peak | Benign/Reassuring |
| Variable | Umbilical cord compression | "V", "U", or "W" shape | Variable relationship | Most common (50–80% of deliveries); concerning if severe |
| Late | Uteroplacental insufficiency | Gradual; begins AFTER contraction peak | Begins late, returns after contraction ends | Ominous — Category III |
| Sinusoidal | Fetal anaemia / severe hypoxia | Smooth sine-wave, 2–5 cycles/min | — | Ominous — Category III |
FHR Tachycardia (>160 bpm) causes:
- Chorioamnionitis, maternal fever
- Sympathomimetic drugs (terbutaline)
- Atropine, fetal tachyarrhythmia
- Fetal hypoxia, prematurity
FHR Bradycardia causes:
- Cord prolapse/compression
- Tetanic contractions
- Paracervical/epidural block
- Maternal seizure
Late deceleration management:
- Turn patient on side (lateral position) → ↑ cardiac output & uterine blood flow
- IV fluids to correct hypotension
- Stop oxytocin infusion
- Administer oxygen
Tachysystole: >5 contractions in 10 minutes (averaged over 30 minutes)
1.5 NICHHD FHR Classification
| Category | Interpretation | Action |
|---|
| I (Normal) | Normal pH, fetal well-being | Continue current management |
| II (Indeterminate) | Uncertain | Further evaluation |
| III (Abnormal) | Fetal hypoxia/acidosis | Immediate intervention; expedited delivery |
UNIT 2: COMPLICATIONS OF PREGNANCY — ANTEPARTUM HAEMORRHAGE
2.1 Causes of Antepartum Haemorrhage (APH)
Complicates 3–5% of pregnancies; leading cause of maternal and perinatal mortality
Main causes:
- Placenta praevia (painless)
- Abruptio placentae (painful)
- Vasa praevia
- Cervical/vaginal lesions
2.2 Placenta Praevia
Definition: Placenta implanted in the lower uterine segment, covering or near the internal cervical os
Classification (3 types):
| Type | Description |
|---|
| Complete/Total | Placenta completely covers the cervical os |
| Partial | Placenta partially covers the os |
| Marginal | Placenta extends to the edge of the os |
Incidence: 1 in 200–250 pregnancies
Classic presentation:
- Painless, bright-red vaginal bleeding in the 3rd trimester
- Average first bleed: 27–32 weeks
- Abnormal lie (transverse/breech) — suspicious
- Initial bleed is usually not fatal and stops spontaneously, then recurs
Risk factors:
- Advanced maternal age
- Increased parity (multiparity)
- Previous uterine surgery (C-section scar)
- Prior placenta praevia
- Multiple gestation
- Uterine abnormalities
- Smoking
Diagnosis: Transabdominal ultrasound (93–98% accurate); use transvaginal US if position unclear — do NOT do vaginal examination (risk of massive haemorrhage)
Management:
- If actively bleeding → IV access, crossmatch blood, IV fluids, oxygen
- If fetus viable but compromised → urgent caesarean section
- Rh-negative mothers → D immune globulin (RhoGAM)
2.3 Abruptio Placentae (Placental Abruption)
Definition: Premature separation of a normally situated placenta before delivery of the fetus
Incidence: 1 in 129 births (~1–2% of pregnancies)
Classic presentation:
- Painful vaginal bleeding — hallmark
- Uterine tenderness (board-like/woody uterus)
- Frequent contractions
- Fetal distress
- Blood is characteristically dark red
Note: Vaginal bleeding occurs in 70% of cases; in 30%, bleeding is concealed (retroplacental)
Grades:
| Grade | Features |
|---|
| Grade I (Mild) | <150 mL blood loss; mother/fetus stable |
| Grade II (Moderate) | Fetal distress; maternal compromise |
| Grade III (Severe) | Fetal death; DIC possible; 25% of cases |
Complications:
- DIC (disseminated intravascular coagulation)
- Hypovolaemic shock
- Renal failure
- Couvelaire uterus (blood penetrates uterine wall)
Key investigations:
- CBC, coagulation studies (PT/PTT, fibrinogen, D-dimer)
- Kleihauer-Betke test (feto-maternal haemorrhage)
- Blood type and crossmatch
Management:
- Aggressive IV fluids/resuscitation
- Packed RBCs on standby
- Continuous fetal monitoring
- Rh-negative mothers → D immune globulin
- Urgent C-section if fetus viable but compromised
2.4 Placenta Praevia vs Abruptio — EXAM COMPARISON TABLE
| Feature | Placenta Praevia | Abruptio Placentae |
|---|
| Pain | Painless | Painful |
| Blood colour | Bright red | Dark red |
| Uterine tone | Soft | Rigid/tender ("woody") |
| Onset | 3rd trimester; 27–32 wks | Any time |
| Shock | Proportional to visible blood | Out of proportion to visible blood |
| Fetal lie | Often abnormal | Usually normal |
| Diagnosis | Ultrasound | Clinical (US may be false-negative) |
| Vaginal examination | CONTRAINDICATED | Can be done |
UNIT 3: HYPERTENSIVE DISORDERS OF PREGNANCY
3.1 Classification
| Condition | BP | Proteinuria | Onset |
|---|
| Gestational hypertension | ≥140/90 mmHg | Absent | >20 weeks |
| Preeclampsia | ≥140/90 mmHg | Present (≥300 mg/24h) | >20 weeks |
| Severe preeclampsia | ≥160/110 mmHg | Present + end-organ damage | >20 weeks |
| Eclampsia | Any | Present | Seizures in preeclamptic |
| Chronic hypertension | ≥140/90 mmHg | Variable | <20 weeks or pre-pregnancy |
3.2 Preeclampsia
Signs/symptoms of severe preeclampsia:
- Severe headache
- Visual disturbances (photopsia, scotoma)
- Epigastric/RUQ pain (liver capsule stretch)
- Pulmonary oedema
- Oliguria
HELLP Syndrome:
- H — Haemolysis (microangiopathic)
- EL — Elevated Liver enzymes (↑AST, ↑ALT, ↑LDH)
- LP — Low Platelets (<100,000/µL)
Suspect HELLP in a pregnant/postpartum woman with abdominal pain
Labs in HELLP: Peripheral blood smear → microangiopathic haemolytic anaemia; ↑transaminases; ↓platelets
3.3 Eclampsia
Definition: Seizures in a woman with pre-eclampsia; can occur up to 12 weeks postpartum
Incidence: 0.2% of pregnancies; terminates 1 in 1000 pregnancies
Major cause of maternal death: Intracranial haemorrhage
Perinatal mortality: 2–8.6%
3.4 Management of Severe Preeclampsia/Eclampsia
Magnesium Sulphate Protocol (MgSO₄ · 7H₂O):
- Loading dose: 4 g (range 2–6 g) in 500 mL 5% dextrose over 15 minutes IV
- Maintenance: 1–3 g/hr continuous infusion
- Continue for 24 hours after delivery or last seizure
- Keep calcium gluconate at bedside as antidote
Monitoring on MgSO₄:
- Deep tendon reflexes (first sign of toxicity = loss of DTRs)
- Respiratory rate (>12/min)
- Urine output (>25 mL/hr)
- Plasma Mg levels
Antihypertensives for acute severe HTN:
- Labetalol IV (first-line)
- Hydralazine IV
- Nifedipine oral
Definitive treatment: DELIVERY of the baby
Gestational hypertension resolves within 10 days postpartum
UNIT 4: PRETERM LABOUR
Definition: Labour occurring between 20–37 weeks of gestation
Risk factors:
- Prior preterm labour (most important risk factor)
- Low socioeconomic status
- Multiple gestation
- Uterine anomalies
- Bacterial vaginosis / UTI
- Placenta praevia/abruption
- Polyhydramnios
- Low maternal weight (<50 kg)
- Smoking, cocaine use
- Poor prenatal care
Management of preterm labour:
- Tocolytics (delay delivery 48 hrs to allow corticosteroids to work):
- Nifedipine (calcium channel blocker — preferred)
- Terbutaline (β₂-agonist)
- Indomethacin (PG inhibitor; <32 weeks)
- MgSO₄ (also neuroprotective)
- Corticosteroids (fetal lung maturity):
- Betamethasone 12 mg IM × 2 doses, 24 hours apart OR
- Dexamethasone 6 mg IM × 4 doses, 12 hours apart
- Give between 24–34 weeks
- Antibiotics if GBS positive or PROM
UNIT 5: PREMATURE RUPTURE OF MEMBRANES (PROM)
Definition: Rupture of membranes before onset of labour
Types:
| Type | Definition |
|---|
| PROM | At term (≥37 weeks) |
| PPROM | Preterm (<37 weeks) |
Diagnosis:
- Pooling of fluid in vagina
- Ferning (amniotic fluid crystallises in fern pattern on glass slide)
- Nitrazine test — amniotic fluid turns paper blue (alkaline)
- Ultrasound — oligohydramnios
Complications: Cord prolapse, infection (chorioamnionitis), preterm labour, fetal distress
Management:
- ≥37 weeks: Induce labour (oxytocin)
- <37 weeks: Expectant management, antibiotics (ampicillin/erythromycin), corticosteroids if <34 weeks
UNIT 6: ECTOPIC PREGNANCY
Definition: Implantation of the fertilised ovum outside the uterine cavity
Sites (order of frequency):
- Ampulla of fallopian tube (most common — ~70%)
- Isthmus
- Fimbria
- Ovary
- Cervix
- Abdominal (rarest)
Risk factors:
- Previous ectopic pregnancy (greatest risk)
- Previous tubal surgery
- PID/salpingitis
- IUD use
- Infertility treatment (IVF)
- Smoking
- Prior pelvic surgery
Clinical features:
- Amenorrhoea (missed period)
- Unilateral pelvic/lower abdominal pain
- Vaginal bleeding (brown, irregular)
- Ruptured ectopic: Sudden severe pain + haemoperitoneum → shoulder tip pain (diaphragmatic irritation) → shock
Investigations:
- Serum β-hCG (positive even at low levels; single low level does NOT exclude ectopic)
- Transvaginal ultrasound — empty uterus + adnexal mass; free fluid in Pouch of Douglas
- Serial β-hCG: In normal IUP, hCG doubles every 48–72 hours; slower rise or plateau → suspect ectopic
- Falling hCG — most common indicator for spontaneous resolution of ectopic
Management:
| Option | Criteria | Details |
|---|
| Expectant | Falling hCG, small ectopic, stable patient | Close monitoring |
| Medical — Methotrexate | Unruptured, hCG <5000 IU/L, no cardiac activity, stable | Folate antagonist; inhibits trophoblast cells; 70–95% success |
| Surgical — Laparoscopy | Ruptured/unstable OR medical failed | Salpingostomy (preserve tube for future fertility); Salpingectomy (not wanting future fertility) |
Methotrexate contraindications: Hepatic/renal disease, active infection, breastfeeding, immunodeficiency
UNIT 7: ABORTION (MISCARRIAGE)
Definition: Expulsion of products of conception before 28 weeks (or fetal weight <1000 g)
Incidence: ~15% of clinically recognised pregnancies; sporadic chromosomal aneuploidy accounts for ~60% of losses
Types of Abortion (HIGH YIELD)
| Type | Cervical Os | Bleeding | Products Passed | Pregnancy Viable? | Management |
|---|
| Threatened | Closed | Mild | None | Yes (Doppler confirms) | Reassure, bed rest, avoid coitus |
| Inevitable | Open | Profuse | None yet | No | D&C |
| Incomplete | Open | Heavy | Partial | No | D&C |
| Complete | Closed | Minimal | All passed | No | Confirm with USS; observe |
| Missed (Silent) | Closed | Absent or brown | None (retained >4 wks) | No | D&C or misoprostol |
| Septic | Open | Foul discharge | Present | No | IV antibiotics + D&C |
| Recurrent | — | — | — | — | ≥3 losses; investigate cause |
Rh-negative management:
- <13 weeks: 50 µg anti-D (Rh immunoglobulin) IM
-
13 weeks: 300 µg anti-D IM
Causes of Recurrent Miscarriage (≥3 losses):
- Chromosomal (parental/fetal aneuploidy) — most common
- Antiphospholipid syndrome
- Uterine anomalies (septum, fibroids)
- Cervical incompetence
- Luteal phase defect
- Thyroid disease
UNIT 8: MOLAR PREGNANCY (GESTATIONAL TROPHOBLASTIC DISEASE)
Definition: Abnormal fertilisation resulting in proliferating trophoblastic tissue
Types:
| Feature | Complete Mole | Incomplete Mole |
|---|
| Fetal parts | Absent | Present (usually abnormal) |
| Karyotype | 46XX (diploid — all paternal) | Triploid (69XXY) |
| Villous changes | All villi hydropic | Some villi hydropic |
| Risk of malignancy (GTD) | 15–20% | 5–10% |
| Uterine size | Large for dates | Small or normal |
| hCG | Very high | Mildly elevated |
Ultrasound (complete mole): "Snowstorm" appearance — distended cavity with numerous anechoic cysts
Presentation:
- Vaginal bleeding
- Uterus large for dates
- Hyperemesis
- Early preeclampsia (<20 weeks — pathognomonic!)
- Very high hCG → bilateral theca lutein cysts → bilateral ovarian enlargement
Management:
- Suction curettage (D&C)
- Serial β-hCG monitoring until 3 consecutive normal levels
- Contraception for 1 year (to allow hCG normalisation and detect malignant transformation)
- Chest X-ray (metastases)
Indications for chemotherapy: Rising or plateauing hCG after evacuation → gestational trophoblastic neoplasia (GTN) → Methotrexate ± Actinomycin D
UNIT 9: PREECLAMPSIA IN LABOUR — MgSO₄ PROTOCOL (STRUCTURED)
| Step | Action |
|---|
| Position | Lateral recumbent |
| Diet | Nothing by mouth (NBM); clear liquids if muscle tone adequate |
| IV access | Lactated Ringer's 5% dextrose |
| Loading dose | MgSO₄ 4g IV over 15 min |
| Maintenance | MgSO₄ 1–3 g/hr IV infusion |
| Antidote at bedside | Calcium gluconate |
| Monitor | DTRs, urine output (I&O), lung bases (pulmonary oedema), plasma Mg levels |
| Duration | Continue 24 hours post-delivery |
UNIT 10: GYNAECOLOGY — KEY TOPICS
10.1 Pelvic Inflammatory Disease (PID)
Definition: Ascending infection from the cervix/vagina → uterus, fallopian tubes, ovaries
Causative organisms:
- Chlamydia trachomatis (most common in developed countries)
- Neisseria gonorrhoeae
- Anaerobes, Gram-negative rods
Clinical features (Chandelier sign):
- Lower abdominal pain (bilateral)
- Cervical motion tenderness (CMT) — "chandelier sign"
- Adnexal tenderness
- Fever
- Vaginal discharge (mucopurulent)
- ↑WBC, ↑ESR, ↑CRP
Complications:
- Tubo-ovarian abscess (TOA)
- Infertility (blocked tubes)
- Ectopic pregnancy risk ↑
- Fitz-Hugh-Curtis syndrome (perihepatitis — RUQ pain + perihepatic adhesions)
- Chronic pelvic pain
Treatment:
- Outpatient: Ceftriaxone IM + Doxycycline + Metronidazole
- Inpatient: IV Cefoxitin + Doxycycline OR Clindamycin + Gentamicin
10.2 Ovarian Cysts & Torsion
Common types:
| Cyst | Features |
|---|
| Follicular cyst | Most common; resolves spontaneously in 4–8 weeks |
| Corpus luteum cyst | Can rupture → haemoperitoneum |
| PCOS (polycystic ovaries) | Multiple small follicles; anovulation, hyperandrogenism |
| Dermoid (teratoma) | Most common benign tumour in women <30; contains hair/teeth/fat |
| Endometrioma ("chocolate cyst") | Endometriosis; filled with old blood |
Ovarian torsion:
- Sudden severe unilateral pelvic pain
- Nausea and vomiting
- Tender adnexal mass on exam
- Doppler USS: absent/reduced blood flow
- Management: Surgical untwisting (detorsion) → salpingo-oophorectomy if necrotic
10.3 Uterine Fibroids (Leiomyomata)
Most common benign tumour of the uterus; oestrogen-dependent; regress after menopause
Types by location:
- Intramural (most common)
- Subserosal
- Submucosal (most likely to cause heavy bleeding and infertility)
- Pedunculated
Symptoms: Heavy menstrual bleeding (menorrhagia), pressure symptoms, infertility, recurrent miscarriage
Management:
- Medical: GnRH analogues (shrink fibroid pre-surgery), tranexamic acid (bleeding), NSAIDs
- Surgical: Myomectomy (fertility-sparing), hysterectomy (definitive)
10.4 Endometriosis
Definition: Presence of endometrial tissue outside the uterus
Sites: Ovaries (most common), peritoneum, Pouch of Douglas, bladder, bowel
Symptoms (3 Ds):
- Dysmenorrhoea (painful periods — cyclical)
- Dyspareunia (painful intercourse)
- Dyschezia (painful defaecation)
Gold standard diagnosis: Laparoscopy + biopsy
Treatment: NSAIDs, OCPs (cycle suppression), GnRH analogues, laparoscopic ablation, hysterectomy
10.5 Cervical Cancer
Cause: HPV types 16 and 18 (responsible for ~70% of cases)
Risk factors:
- Early sexual debut
- Multiple sexual partners
- Multiparity
- Immunosuppression
- Smoking
- STIs
Screening: Pap smear (cervical cytology)
- Start: Age 21 or 3 years after sexual debut
- Frequency: Every 3 years if normal (cytology alone)
HPV vaccine: Ideally age 9–14 (before sexual debut); can be given up to age 26 routinely
Staging (FIGO):
| Stage | Description |
|---|
| I | Confined to cervix |
| II | Extends beyond cervix but not pelvic wall |
| III | Extends to pelvic wall / lower third of vagina |
| IV | Bladder/rectum or distant metastases |
Treatment: Stage I → surgery (Wertheim's hysterectomy); Stage II–IV → chemoradiotherapy
10.6 Menstrual Disorders
| Term | Definition |
|---|
| Amenorrhoea | Absence of periods |
| Primary amenorrhoea | No period by age 16 (with secondary sex characteristics) or age 14 (without) |
| Secondary amenorrhoea | No period for ≥6 months in previously menstruating woman |
| Menorrhagia | Heavy periods (>80 mL/cycle) |
| Metrorrhagia | Irregular intermenstrual bleeding |
| Menometrorrhagia | Heavy + irregular bleeding |
| Dysmenorrhoea | Painful periods |
| Oligomenorrhoea | Cycles >35 days |
| Polymenorrhoea | Cycles <21 days |
UNIT 11: POSTPARTUM HAEMORRHAGE (PPH)
Definition: Blood loss:
- Vaginal delivery: >500 mL
- C-section: >1000 mL
Primary PPH: Within 24 hours of delivery
Secondary PPH: 24 hours–6 weeks postpartum
The 4 T's of PPH (EXAM GOLD)
| T | Cause | Frequency |
|---|
| Tone | Uterine atony | ~80% (most common) |
| Tissue | Retained placenta/products | ~10% |
| Trauma | Genital tract lacerations | ~7% |
| Thrombin | Coagulopathy (DIC, von Willebrand) | ~3% |
Risk factors for uterine atony:
- Overdistended uterus (macrosomia, multiple gestation, polyhydramnios)
- Prolonged labour
- Grand multiparity
- General anaesthesia
- Oxytocin use
Management of PPH (stepwise)
- Uterine massage (bimanual compression)
- Uterotonic drugs:
- Oxytocin (1st line): 10 IU IM or IV infusion
- Ergometrine/Methylergonovine (Syntometrine): 0.2 mg IM (CI in hypertension)
- Misoprostol: 600–1000 µg rectal/sublingual (useful in low-resource settings)
- Carboprost (PGF2α): 0.25 mg IM q15 min (CI in asthma)
- Balloon tamponade (Bakri balloon)
- Surgical:
- B-Lynch compression suture
- Uterine artery ligation
- Internal iliac artery ligation
- Hysterectomy (last resort)
- Blood products: Fresh frozen plasma (FFP), packed RBCs, cryoprecipitate for DIC
UNIT 12: PUERPERAL SEPSIS
Definition: Fever ≥38°C on ≥2 occasions, >24 hours apart, within the first 10 days postpartum (excluding first 24 hours)
Sources of infection:
- Uterus (endometritis — most common)
- Urinary tract
- Breast (mastitis)
- Wound (episiotomy, C-section)
Causative organisms: Group A Streptococcus, Staphylococcus aureus, E. coli, anaerobes
Features of endometritis:
- Fever
- Uterine tenderness
- Foul-smelling lochia
- Subinvolution of uterus
Treatment: IV Clindamycin + Gentamicin
QUICK REVISION SUMMARY TABLES
Antihypertensive Drugs in Pregnancy — Safety
| Drug | Safe? | Notes |
|---|
| Methyldopa | ✅ Safe (1st line) | — |
| Labetalol | ✅ Safe | Also used IV for acute HTN |
| Nifedipine | ✅ Safe | Oral; 2nd line |
| Hydralazine | ✅ Safe | IV for acute HTN |
| ACE inhibitors (enalapril etc.) | ❌ Contraindicated | Renal dysgenesis (2nd/3rd trimester) |
| ARBs | ❌ Contraindicated | Same as ACE inhibitors |
Types of Caesarean Section
| Type | Indication |
|---|
| Elective | Planned; no emergency |
| Emergency Category 1 | Immediate threat to mother/fetus; <30 min |
| Emergency Category 2 | Maternal/fetal compromise; not immediate |
| Emergency Category 3 | Needs early delivery; no compromise |
HIGH-FREQUENCY EXAM QUESTIONS
Q: Painless APH in the 3rd trimester — diagnosis?
A: Placenta praevia. Do NOT do vaginal examination. First-line investigation: ultrasound
Q: Painful APH with rigid uterus — diagnosis?
A: Abruptio placentae. Clinical diagnosis; ultrasound has high false-negative rate
Q: Seizures in a patient with BP 170/110 at 34 weeks — diagnosis & management?
A: Eclampsia. Give MgSO₄ (loading dose 4g IV over 15 min), stabilise, deliver baby. Antidote = calcium gluconate
Q: Empty uterus on TVS + positive hCG + unilateral pain in a woman with history of PID — diagnosis?
A: Ectopic pregnancy. Manage with methotrexate if unruptured and stable; laparoscopy if ruptured
Q: Type of abortion where cervical os is closed, no products passed, but the fetus is dead on ultrasound?
A: Missed abortion
Q: What is the most important risk factor for preterm labour?
A: Previous preterm labour
Q: What is the 7th cardinal movement of labour?
A: Expulsion
Q: At what cervical dilation does the active phase of labour begin?
A: 4–5 cm
Q: What type of FHR deceleration is associated with umbilical cord compression?
A: Variable deceleration
Q: What is the most common benign ovarian tumour in women under 30?
A: Dermoid cyst (mature cystic teratoma)
Q: What is the gold standard for diagnosing endometriosis?
A: Laparoscopy + biopsy
Sources: Textbook of Family Medicine 9e; Goldman-Cecil Medicine; Morgan & Mikhail's Clinical Anesthesiology 7e; Creasy & Resnik's Maternal-Fetal Medicine; Swanson's Family Medicine Review; Berek & Novak's Gynecology; Tintinalli's Emergency Medicine; Rosen's Emergency Medicine
These notes cover 12 units of core O&G content. Summary of topics:
| # | Topic |
|---|
| 1 | Normal Labour — stages, cardinal movements, FHR monitoring |
| 2 | Antepartum Haemorrhage — placenta praevia vs abruption |
| 3 | Hypertensive disorders — preeclampsia, eclampsia, HELLP |
| 4 | Preterm labour — tocolytics, steroids |
| 5 | PROM |
| 6 | Ectopic pregnancy |
| 7 | Abortion types (6 types with complete comparison table) |
| 8 | Molar pregnancy/GTD |
| 9 | MgSO₄ protocol structured table |
| 10 | Gynaecology — PID, fibroids, endometriosis, cervical cancer, menstrual disorders |
| 11 | PPH — 4 T's + stepwise management |
| 12 | Puerperal sepsis |
Let me know if you want any unit expanded further or want MCQ practice questions!