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Diagnosis

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ECG Diagnosis - Sabitri, Female, 55 years


Primary Diagnosis: Narrow-Complex Tachycardia (SVT) with Demand Ischemia


Key Findings

ParameterValueSignificance
Heart Rate206 bpmExtreme tachycardia
PR Interval363 msUnreliable at this rate
QRS Duration76 msNarrow - supraventricular origin
QTc656 msSeverely prolonged (critical)
P/QRS/T Axis-96.1 / 26.2 / -146.4°Abnormal P axis

Rhythm Interpretation

Most likely: Supraventricular Tachycardia (SVT) at 206 bpm - narrow complex, regular rhythm. The differential includes:
  1. Atrial Flutter with 1:1 conduction - the undulating baseline in inferior leads (II, III, aVF) and the extreme rate of ~206 bpm are consistent. Classic flutter rate ~300 bpm with 1:1 conduction can appear at ~150-220 bpm.
  2. AVNRT (AV Nodal Reentrant Tachycardia) - most common SVT, narrow complex, P waves typically hidden in or just after QRS.
  3. AVRT (AV Reentrant Tachycardia) - accessory pathway mediated.
The machine-listed PR of 363 ms and P duration of 339 ms are almost certainly artifact/misidentification at 206 bpm, as a true PR of 363 ms would be impossible to sustain at this rate.

ST-T Changes: Diffuse Inferolateral Demand Ischemia

  • ST depression in II, III, aVF (inferior), V4, V5, V6 (anterolateral) - severe, downsloping
  • ST elevation in aVR - reciprocal change, classic pattern of diffuse subendocardial ischemia
  • Abnormal T waves in II, aVF, V4, V5, V6
At 206 bpm, diffuse ST depression + aVR elevation most likely represents rate-related demand ischemia (Type 2 MI pattern). However, primary ACS must be ruled out once rate is controlled.

Additional Machine Conclusions

  • Left Atrial Hypertrophy (P duration 339 ms - markedly prolonged; likely artifact at this rate, but underlying LA enlargement possible)
  • First-degree AV block (363 ms PR - likely misidentified; needs reassessment post-conversion)
  • QTc 656 ms - critically prolonged, high risk of torsades de pointes

Urgent Clinical Action Required

  1. Stabilize the patient - assess hemodynamic status immediately
  2. Vagal maneuvers - if hemodynamically stable (Valsalva, carotid sinus massage)
  3. IV Adenosine 6 mg rapid push - diagnostic and therapeutic for SVT
  4. Synchronized DC cardioversion - if hemodynamically unstable
  5. Repeat ECG after rate control - reassess ST changes, true PR interval, and P wave morphology
  6. Troponin I/T serial testing - to rule out ACS as primary cause
  7. Electrolytes (K+, Mg2+) - correct if abnormal given prolonged QTc

Disclaimer: This ECG interpretation is for informational support only and must be confirmed by the treating physician. The report itself states: "Report needs physician confirmation."
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