Explain me 10 common heart disease with ecg changes and ecg images of those disease

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ECG electrocardiogram myocardial infarction ST elevation STEMI

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ECG atrial fibrillation irregular rhythm absent P waves

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ECG heart block third degree complete AV block

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ECG hypertrophic cardiomyopathy left ventricular hypertrophy

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ECG Wolff-Parkinson-White WPW delta wave pre-excitation

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ECG pulmonary embolism S1Q3T3 right heart strain

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ECG ventricular tachycardia wide complex tachycardia

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ECG pericarditis diffuse ST elevation saddle shape PR depression

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ECG left bundle branch block LBBB wide QRS

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ECG hyperkalemia peaked T waves sine wave pattern

Here is a comprehensive guide to 10 common heart diseases with their ECG changes and representative ECG images:

10 Common Heart Diseases: ECG Changes & Images


1. πŸ«€ ST-Elevation Myocardial Infarction (STEMI)

ECG Changes

PhaseFinding
HyperacuteTall, peaked (hyperacute) T-waves
AcuteST-segment elevation β‰₯1 mm (β‰₯2 mm in V1–V4), convex "tombstone" morphology
EvolvingPathological Q waves (>40 ms wide, >25% of R-wave height)
ReciprocalST depression in leads opposite the infarct zone
ChronicQ waves persist; T-wave inversion
Territory localization:
  • Anterior (LAD): ST elevation in V1–V4
  • Inferior (RCA): ST elevation in II, III, aVF; reciprocal depression in I, aVL
  • Lateral (LCx): ST elevation in I, aVL, V5–V6
  • Posterior: ST depression in V1–V3 + tall R waves

ECG Images

Anterior STEMI – tombstoning in V2–V5, LAD occlusion
Anterior STEMI: pronounced "tombstone" ST elevation in V2–V5, evolving QS waves, consistent with proximal LAD occlusion
Anterolateral STEMI with reciprocal changes in inferior leads
Anterolateral STEMI: ST elevation V1–V6 + leads I and aVL with reciprocal ST depression in II, III, aVF
Inferolateral STEMI with annotated ST elevation and reciprocal changes
Inferolateral STEMI: ST elevation in II, III, aVF, V4–V6 (green circles) with reciprocal depressions in I, aVL, V2–V3 (yellow arrows)

2. ❀️‍πŸ”₯ Atrial Fibrillation (AF)

ECG Changes

FeatureFinding
P wavesAbsent β€” replaced by chaotic fibrillatory (f) waves at 350–600/min
RhythmIrregularly irregular R-R intervals (hallmark)
RateVariable; uncontrolled: 100–160 bpm
QRSNarrow (unless aberrant conduction or pre-excitation)
BaselineFine or coarse fibrillatory activity, best seen in V1

ECG Image

Atrial fibrillation – absent P waves, irregularly irregular rhythm with RVR
AF with rapid ventricular response (~134 bpm): absent P waves, chaotic baseline, irregularly irregular QRS complexes with narrow morphology

3. 🚫 Complete (Third-Degree) AV Block

ECG Changes

FeatureFinding
AV dissociationComplete β€” P waves and QRS bear NO relationship
P wavesRegular at faster atrial rate (e.g., 75 bpm)
QRS rateSlow escape rhythm (20–45 bpm if ventricular; 40–60 bpm if junctional)
QRS widthWide (>120 ms) if ventricular escape; narrow if junctional
PR intervalNo fixed PR interval β€” P waves "march through" QRS complexes

ECG Images

Complete AV block – 12-lead with wide-complex ventricular escape
Third-degree heart block: wide-complex escape rhythm, P waves dissociated from QRS complexes, postoperative tricuspid valve replacement
Complete heart block – narrow-complex junctional escape, slow rate ~36 bpm
Third-degree AV block with narrow-complex junctional escape at ~36 bpm β€” pacemaker implantation indicated

4. πŸ‹οΈ Hypertrophic Cardiomyopathy (HCM)

ECG Changes

FeatureFinding
LVHSokolow-Lyon criteria: S in V1 + R in V5/V6 >35 mm
ST-T changesDiffuse ST depression + deep T-wave inversions (strain pattern)
Apical HCMGiant T-wave inversions in V3–V5 (Yamaguchi syndrome) β€” β‰₯10 mm deep
Septal Q wavesAbsent in I, aVL, V5–V6 (due to abnormal septal depolarization)
QRS axisLeft axis deviation common
ArrhythmiasAF, ventricular ectopy, non-sustained VT

ECG Images

HCM comparison – concentric, septal, and apical variants with CMR correlation
HCM variants: (A) Concentric β€” high-voltage R waves + strain; (B) Septal β€” moderate LVH; (C) Apical β€” giant T-wave inversions (12 mm) in lateral leads, with CMR correlation
Apical HCM – giant symmetric T-wave inversions V2–V5 (Yamaguchi syndrome)
Apical HCM (Yamaguchi syndrome): high-voltage QRS + dramatic deep symmetric T-wave inversions in V2–V5

5. ⚑ Wolff-Parkinson-White (WPW) Syndrome

ECG Changes

FeatureFinding
PR intervalShort (<120 ms) β€” bypass tract bypasses AV node delay
Delta waveSlurred upstroke of the QRS β€” hallmark of pre-excitation
QRS widthWidened (>120 ms)
ST-T changesDiscordant (opposite to QRS vector) β€” secondary repolarization changes
Pseudo-infarctNegative delta waves in II, III, aVF can mimic inferior Q waves
Arrhythmia riskAVRT, AF with rapid ventricular response (potentially fatal)
Pathway localization: Delta wave polarity in V1 and inferior leads identifies pathway location (left lateral, posteroseptal, anteroseptal, etc.)

ECG Images

WPW with red arrows marking delta waves in leads II and III – anteroseptal pathway
WPW syndrome: short PR, widened QRS with delta waves (red arrows in II and III), regular tachycardia β€” anteroseptal accessory pathway
WPW – classic delta waves, short PR, secondary ST-T changes
WPW: Short PR (<120 ms), widened QRS with prominent delta waves across precordial and lateral limb leads, mid-septal pathway location

6. 🫁 Pulmonary Embolism (PE)

ECG Changes

FeatureFinding
Sinus tachycardiaMost common ECG finding (~40% of PE)
S1Q3T3 patternDeep S in lead I + Q wave in III + T-wave inversion in III (McGinn-White sign)
Right axis deviationReflecting acute RV strain
RBBBNew complete or incomplete right bundle branch block
T-wave inversionsV1–V4 (right precordial) indicating RV strain
P pulmonaleTall, peaked P waves in II if severe pulmonary hypertension
⚠️ ECG is non-specific; normal ECG does not rule out PE. Always correlate with clinical probability and CT-PA.

ECG Images

PE – S1Q3T3 pattern with annotated arrows
Acute PE: S1Q3T3 pattern with annotated black circles β€” deep S in I, Q in III, T-wave inversion in III; sinus tachycardia
PE with S1Q3T3 + incomplete RBBB + T-wave inversions V1–V3
Acute PE: S1Q3T3 pattern + incomplete RBBB (QRS 110 ms) + T-wave inversions in V1–V3 and aVF β€” consistent with acute right ventricular strain

7. πŸ’₯ Ventricular Tachycardia (VT)

ECG Changes

FeatureFinding
Rate100–250 bpm
QRSWide (>120 ms), bizarre morphology
RhythmRegular (monomorphic VT) or irregular (polymorphic/torsades)
P wavesAbsent or dissociated from QRS (AV dissociation)
Fusion beatsNarrow QRS "capture" or hybrid beats = pathognomonic for VT
ConcordanceAll precordial leads positive or negative = strongly suggests VT
Brugada criteriaRS absent in precordials, RS >100 ms, AV dissociation, morphology criteria

ECG Image

Monomorphic VT – wide complex tachycardia, no P waves, positive concordance
Monomorphic VT: rapid wide-complex tachycardia, positive precordial concordance, no discernible P waves β€” associated with structural heart disease
Left fascicular VT – RBBB morphology + left axis deviation (Belhassen VT)
Left fascicular VT (Belhassen): RBBB morphology + left axis deviation β€” idiopathic, verapamil-sensitive form; typically younger patients without structural disease

8. πŸ”₯ Acute Pericarditis

ECG Changes (4 Stages)

StageFinding
Stage I (hours–days)Diffuse concave ("saddle-shaped") ST elevation in all leads except aVR and V1; PR depression in II + PR elevation in aVR
Stage II (1–3 weeks)ST normalizes; T waves flatten
Stage IIIDiffuse T-wave inversion
Stage IVECG normalizes
Key differentiators from STEMI:
  • Diffuse (not territory-specific) ST elevation
  • Concave (not convex/tombstone) morphology
  • PR segment depression
  • Spodick's sign: downsloping TP segment
  • No reciprocal changes (except aVR)

ECG Images

Pericarditis – diffuse saddle-shaped ST elevation + PR depression in multiple leads
Acute pericarditis: concave ST elevation in I, II, III, aVF, V2–V6; PR depression in II; reciprocal ST depression + PR elevation in aVR; Spodick's sign present
Pericarditis – diffuse concave ST elevation + PR segment changes annotated
Pericarditis Stage I: saddle-shaped ST elevation across multiple territories, PR depression in II, III, aVF, V4–V6, reciprocal PR elevation in aVR β€” no Q waves

9. πŸ”€ Left Bundle Branch Block (LBBB)

ECG Changes

FeatureFinding
QRS durationβ‰₯120 ms (complete LBBB)
V1Deep broad QS or rS pattern
I, aVL, V5–V6Broad, notched M-shaped (bifid) R waves β€” no Q waves
ST-T discordanceST/T-wave changes opposite to QRS direction
Lateral T-wavesT-wave inversion in I, aVL, V5–V6
No septal Q wavesAbsent in lateral leads
Clinical significanceNew LBBB in ACS = treat as STEMI equivalent (Sgarbossa criteria)

ECG Images

Classic LBBB – broad M-shaped R in lateral leads, deep QS in V1, discordant T waves
Classic LBBB: wide QRS >120 ms, notched M-shaped R waves in I, aVL, V5–V6; deep broad S in V1–V3; T-wave discordance (upright in V1–V3, inverted laterally)
LBBB with Sgarbossa criteria – discordant STE in V1–V3, helps detect underlying STEMI
LBBB with discordant ST elevation in V1–V3 and ST depression in I, aVL, V6 β€” Sgarbossa criterion: discordant STE β‰₯1 mm is expected, but concordant STE suggests underlying ischemia

10. βš—οΈ Hyperkalemia

ECG Changes (Progressive with rising K⁺)

Serum K⁺ECG Finding
5.5–6.5 mEq/LTall, narrow-based "tented" peaked T waves (earliest sign, best in V2–V4)
6.5–7.5 mEq/LProlonged PR interval, flattening/loss of P waves
7.5–8.0 mEq/LWidened QRS (intraventricular conduction delay)
>8.0 mEq/LSine-wave pattern (fusion of QRS and T), ventricular fibrillation, asystole
⚠️ Hyperkalemia is a medical emergency. Give IV calcium gluconate immediately when ECG changes are present to stabilize the myocardial membrane.

ECG Images

Early hyperkalemia – tall peaked tented T waves in V2–V4 with K⁺ 7.3 mEq/L
Hyperkalemia (K⁺ = 7.3 mEq/L): classic tall, narrow-based peaked T waves in V2–V4 (blue arrows) β€” earliest ECG sign, no QRS widening yet at this stage
Severe hyperkalemia – QRS widening, peaked T waves, approaching sine-wave pattern
Severe hyperkalemia: broad-complex tachycardia at ~126 bpm with sine-wave QRS morphology, peaked T waves in V2–V4, absent P waves β€” pre-arrest state

Quick Reference Summary Table

#ConditionKey ECG Finding
1STEMIST elevation + Q waves in territory; reciprocal depression
2Atrial FibrillationAbsent P waves + irregularly irregular rhythm
3Complete AV BlockP-QRS dissociation + slow escape rhythm
4HCMLVH voltage + giant T-wave inversions (apical)
5WPWShort PR + delta wave + widened QRS
6Pulmonary EmbolismS1Q3T3 + sinus tachycardia + right heart strain
7Ventricular TachycardiaWide QRS tachycardia + AV dissociation
8PericarditisDiffuse saddle-shaped ST elevation + PR depression
9LBBBWide QRS + notched R in I/V5–V6 + ST discordance
10HyperkalemiaPeaked T waves β†’ QRS widening β†’ sine wave

Sources: Harrison's Principles of Internal Medicine 22E; Tintinalli's Emergency Medicine; Ganong's Review of Medical Physiology; ROSEN's Emergency Medicine β€” ECG images from peer-reviewed PMC literature.
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