Give common heart disease with ECG with ECG graph

Reading File
Finding Sources
Searching the Web

common heart diseases ECG findings patterns images diagrams

Finding Sources
Finding Sources
Loading Image
Finding Sources
Searching the Web

common heart disease ECG patterns STEMI atrial fibrillation heart block complete ECG strip images

Reading File
Reading Web Page

common heart disease ECG findings patterns images

Loading Image
Loading Image
Searching the Web

STEMI ST elevation myocardial infarction ECG graph image site:litfl.com OR site:ecglibrary.com

Loading Image
Loading Image
Generating Image
Here is a thorough guide to the most common heart diseases and their ECG patterns, with real ECG graphs.

Common Heart Diseases and Their ECG Findings

ECG Quick Reference - Generated Diagram

Common Heart Disease ECG Patterns

ECG Pattern Reference Chart (from Life in the Fast Lane ECG Library)

Killer ECG Patterns - LITFL

Common Arrhythmias - ECG Strips

The image below shows real ECG tracings for 5 major arrhythmias:
Common Arrhythmia ECG strips - 2nd degree block, AF, VT, VF, 3rd degree block

1. Normal Sinus Rhythm (Baseline)

Rate: 60-100 bpm | Rhythm: Regular
  • Regular P wave before every QRS
  • PR interval: 0.12-0.20 sec
  • QRS duration: <0.12 sec
  • Normal T wave following QRS

2. ST-Elevation Myocardial Infarction (STEMI) - Heart Attack

The most critical ECG emergency
12-lead STEMI ECG
Key ECG Features:
  • ST elevation ≥1mm in ≥2 contiguous limb leads, or ≥2mm in precordial leads
  • "Tombstone" or "hyperacute" T wave appearance in early phase
  • Pathological Q waves develop within hours (>40ms wide, >25% of R wave height)
  • Reciprocal ST depression in opposite leads
Localisation by leads:
TerritoryLeads with ST elevationArtery
AnteriorV1-V4LAD
InferiorII, III, aVFRCA
LateralI, aVL, V5-V6LCx
PosteriorV7-V9 (ST depression V1-V3)RCA/LCx

3. Atrial Fibrillation (AF)

Most common sustained arrhythmia - affects ~1% of population
Key ECG Features:
  • Absent P waves - replaced by chaotic fibrillatory baseline (f-waves)
  • Irregularly irregular RR intervals (no pattern at all)
  • Ventricular rate 100-160 bpm if uncontrolled
  • Narrow QRS complexes (unless aberrant conduction)
Clinical significance: Risk of stroke (thrombus formation in left atrial appendage), needs anticoagulation assessment using CHA₂DS₂-VASc score.

4. Ventricular Tachycardia (VT)

Life-threatening - requires immediate treatment
Key ECG Features:
  • Wide QRS complexes (>0.12 sec, usually >0.14 sec) - bizarre morphology
  • Rate 150-250 bpm
  • AV dissociation - P waves march through independently (seen in ~50% of cases)
  • Fusion beats and capture beats (pathognomonic of VT)
  • Concordance in precordial leads (all positive or all negative)

5. Ventricular Fibrillation (VF)

Cardiac arrest - no cardiac output
Key ECG Features:
  • Completely chaotic electrical activity - no recognizable P, QRS, or T waves
  • Rapid irregular undulations of varying amplitude
  • No organized rhythm whatsoever
  • Requires immediate defibrillation

6. Complete (Third-Degree) AV Block

Key ECG Features:
  • Complete AV dissociation - P waves and QRS complexes bear NO relationship to each other
  • P waves march at their own rate (usually 60-100 bpm)
  • QRS complexes march at their own slower escape rate (20-40 bpm if ventricular, 40-60 if junctional)
  • Broad QRS if escape rhythm is ventricular in origin
  • Patient is severely bradycardic - requires pacing

7. Second-Degree AV Block

Two types:
  • Mobitz I (Wenckebach): PR interval progressively lengthens until a QRS is dropped, then resets
  • Mobitz II: Constant PR interval with sudden non-conducted P waves (more dangerous, often requires pacing)

8. Left Bundle Branch Block (LBBB)

Key ECG Features:
  • Broad QRS >0.12 sec (usually >0.14 sec)
  • "WiLLiaM" pattern: W shape in V1, M shape in V5/V6
  • Broad, notched R waves in I, aVL, V5-V6
  • Deep S waves in V1-V3
  • No septal Q waves in lateral leads
  • New LBBB in the context of chest pain = treat as STEMI equivalent

9. Wolff-Parkinson-White (WPW) Syndrome

Pre-excitation syndrome
Key ECG Features:
  • Short PR interval (<0.12 sec) - due to bypassing the AV node
  • Delta wave - slurred upstroke at the beginning of QRS
  • Broad QRS complex
  • Can cause supraventricular tachycardia (SVT) or dangerous AF with rapid conduction

10. Brugada Syndrome

Channelopathy - cause of sudden cardiac death in young people
Key ECG Features (V1-V3):
  • Coved-type ST elevation >2mm in V1-V3 (Type 1 - diagnostic)
  • Followed by a negative T wave
  • Can be unmasked by fever, flecainide, or sodium channel blockers
  • Risk of VF and sudden death - managed with ICD implantation

11. Left Ventricular Hypertrophy (LVH)

Key ECG Features:
  • High voltage QRS - Sokolov-Lyon criterion: S in V1 + R in V5/V6 >35mm
  • Left axis deviation
  • ST depression and T wave inversion in lateral leads (strain pattern)
  • Seen in long-standing hypertension, aortic stenosis, HCM

12. Pericarditis

Key ECG Features (evolve in stages):
  • Stage 1: Diffuse saddle-shaped ST elevation in almost all leads + PR depression
  • Stage 2: ST returns to baseline, T waves flatten
  • Stage 3: T wave inversion
  • Stage 4: Normalization
  • Key: Widespread ST elevation (not in a coronary territory) distinguishes it from STEMI

Quick Summary Table

ConditionRateP wavesQRSHallmark feature
Normal60-100Present, regularNarrowBaseline
STEMIVariablePresentNarrowST elevation + Q waves
Atrial Fibrillation100-160Absent (f-waves)NarrowIrregularly irregular
VT150-250DissociatedWideAV dissociation
VF-NoneNoneChaotic waveform
3rd degree block20-60Present, regularWide/NarrowP-QRS dissociation
LBBBNormalPresentWideWiLLiaM pattern
WPWNormalPresentWideDelta wave + short PR
BrugadaNormalPresentNormalCoved ST V1-V3
LVHNormalPresentTallHigh voltage + strain
PericarditisNormalPresentNarrowDiffuse saddle ST elevation

Sources: Braunwald's Heart Disease (Textbook of Cardiovascular Medicine), Life in the Fast Lane ECG Library, Geeky Medics ECG Guide. ECG interpretation should always be correlated with the clinical picture.
This is a shared conversation. Sign in to Orris to start your own chat.