ECG Interpretation

• Patient ID: 4001070
• Date/Time: 23-04-2026, 03:58:08
• Speed: 25 mm/s | Gain: 10 mm/mV
• Filters: 0.5–100 Hz, LP50
• System: SEMIR V1.7

Rhythm & Rate

• Rhythm: Sinus rhythm (P waves visible preceding QRS complexes)
• Heart rate: Approximately 60–80 bpm based on RR intervals at 25 mm/s paper speed

P Waves

• Present and upright in leads I, II, aVF
• Inverted/negative in aVR (normal)
• Axis is normal, suggesting normal sinus origin

PR Interval

• Appears within normal range (~120–200 ms) — no overt first-degree AV block visible

QRS Complex

• Duration: Appears narrow (< 120 ms) — no bundle branch block pattern
• Axis: Appears normal to leftward
• Voltage: The precordial leads (especially V4–V6) show notably tall R waves, raising consideration of Left Ventricular Hypertrophy (LVH)
  • Sokolow-Lyon criterion: S in V1 + R in V5 or V6 ≥ 35 mm — appears potentially met
• Deep S waves in V1–V2, tall R in V5–V6 morphology consistent with LVH pattern

ST Segment & T Waves

• V1–V3: Possible early repolarization or ST changes in the right precordial leads
• V4–V6: The ST segments appear to show downsloping ST depression or strain pattern, consistent with LV strain (a repolarization abnormality associated with LVH)
• Inferior leads (II, III, aVF): ST-T changes visible; need careful assessment for ischemia
• aVR: Reciprocal changes (ST elevation in aVR can indicate left main or proximal LAD disease in the right clinical context)
• T-wave inversions appear present in lateral leads (I, aVL, V5, V6)

Key Findings Summary

Impression

Sinus rhythm with findings consistent with Left Ventricular Hypertrophy (LVH) and associated strain pattern. The lateral ST-T changes (V4–V6, I, aVL) may represent classic LVH repolarization abnormality but cannot exclude subendocardial ischemia without clinical correlation.

Clinical Correlation Recommended

• Check BP — LVH is most commonly due to longstanding hypertension
• Symptoms: Chest pain, dyspnea, or syncope would raise suspicion for acute ischemia (NSTEMI) superimposed on LVH
• Troponins if ischemia is suspected
• Echocardiography to confirm LVH and assess EF
• Compare with prior ECGs if available

⚠️ Disclaimer: This is a visual interpretation of a photograph of an ECG and is intended for educational/supportive purposes only. Definitive clinical ECG interpretation must be performed by a qualified physician with access to the original tracing and full patient history.