Laser haemorrhoidoplasty (LHP): the surgeon’s explanation
Laser haemorrhoidoplasty is a tissue-preserving operation for internal haemorrhoids. It does not cut out the haemorrhoidal cushions as in a conventional haemorrhoidectomy. Instead, a radial diode-laser fibre is placed into each haemorrhoidal cushion and delivers controlled thermal energy. This coagulates small vessels and causes collagen contraction, so the cushion shrinks, scars internally, and is pulled upward over the following weeks.
The aim is simple: reduce bleeding and prolapse while avoiding a large painful anoderm wound.
What problem is it designed to treat?
Haemorrhoids are normal vascular cushions in the anal canal. They become disease when they bleed, prolapse, soil, itch, thrombose, or cause troublesome symptoms. The key distinction is:
- Internal haemorrhoids are above the dentate line and usually cause painless bleeding or prolapse.
- External haemorrhoids/skin tags are below the dentate line and are sensitive.
LHP principally treats the internal vascular and submucosal component. It does not reliably remove a bulky external component, large fibrotic skin tags, or advanced fixed prolapse. Anoscopy is central to assessing the internal cushions and their relation to the dentate line. Sleisenger and Fordtran's Gastrointestinal and Liver Disease, p. 2670.
Best candidates
In experienced hands, LHP is most reasonable for:
- Symptomatic grade II or selected grade III internal haemorrhoids
- Bleeding with modest-to-moderate prolapse
- Patients who value less postoperative pain and a faster return to activity
- Those in whom preservation of anoderm and mucosa is desirable
It is less attractive when there is:
- A large external haemorrhoidal component or significant skin tags
- Grade IV, irreducible, strangulated, or markedly fibrotic prolapse
- Coexistent anal pathology needing excision, such as a fissure, fistula, suspicious lesion, or major external disease
- Unexplained rectal bleeding, iron-deficiency anaemia, altered bowel habit, or cancer risk without appropriate colorectal evaluation
A recent colorectal-surgery consensus describes LHP as an option for grade II and III disease, but advises added caution in grade IV disease and notes that external disease may still need excision.
Taiwan consensus
How the operation is performed
Exact settings and instruments vary by platform and surgeon. The principles matter more than a fixed recipe.
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Assessment under anaesthesia
The surgeon performs examination and anoscopy to identify the main cushions, commonly left lateral, right anterior, and right posterior. Any external component and degree of mucosal prolapse are assessed.
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Access above the sensitive anoderm
A small puncture or incision is made at the base of the internal haemorrhoidal cushion, maintaining a safe position relative to the dentate line.
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Submucosal fibre placement
A laser fibre, usually with a radial-emitting tip, is advanced within the submucosal plane of the haemorrhoidal cushion. It should not be in the sphincter muscle and should not be perforating mucosa.
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Controlled intrahaemorrhoidal energy delivery
The fibre is activated in a controlled, staged manner while being withdrawn. The intended endpoint is tissue contraction and blanching, not charring or a deep crater. The laser energy seals small vessels and denatures collagen, initiating later fibrosis and volume reduction.
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Address prolapse if needed
Where prolapse is a major component, some surgeons add a mucopexy, using sutures to lift and fix the prolapsing mucosa. Laser alone shrinks the cushion but may not provide enough mechanical lift for substantial prolapse.
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Haemostasis and recovery
Because there is little or no open wound, bleeding is usually limited. The procedure is commonly ambulatory, under local anaesthesia with sedation, regional, or general anaesthesia depending on patient, extent of disease, and local practice.
The operation is conceptually different from laser-assisted excision. In a true LHP, the haemorrhoidal cushion is ablated from within and preserved, not dissected off the internal sphincter.
Why patients often recover more comfortably
Conventional excisional haemorrhoidectomy creates wounds in or near highly sensitive anoderm. That is why it remains very effective but can hurt substantially. LHP seeks to avoid that wound burden.
A 2024 meta-analysis of 17 trials involving 1,196 patients found that, compared with Milligan-Morgan or Ferguson haemorrhoidectomy, LHP was associated with:
- Less operative blood loss
- Less postoperative bleeding
- Shorter operating time
- Lower pain on postoperative day 1
- Lower anal-stenosis rates
It did
not show a statistically significant difference in recurrence, urinary retention, or continence outcomes overall. See the
2024 meta-analysis.
That is the appealing trade-off: less early pain and quicker recovery, but not necessarily a more durable answer for every pattern of disease.
Limitations and the recurrence question
LHP is not a magic substitute for excision.
The fundamental limitation is that it shrinks vascular cushions but does not remove redundant tissue to the same degree as an excisional operation. If the dominant pathology is severe mechanical prolapse, large external disease, or scarred high-grade haemorrhoids, a conventional excisional haemorrhoidectomy can be the more definitive operation.
The evidence base also has limitations:
- Techniques, laser wavelengths, energy protocols, and patient selection differ between studies.
- Follow-up is often shorter than is ideal for judging long-term recurrence.
- The 2024 wavelength review found lower pain and complications with LHP compared with open surgery, but reported a signal of higher recurrence with 1470-nm LHP versus conventional haemorrhoidectomy. Systematic review
Practical surgical message: choose LHP for the right anatomy, not simply because it is called “laser” or “painless.”
Risks and complications
Even a minimally invasive anorectal operation has real risks:
- Postoperative bleeding, including delayed bleeding
- Pain, tenesmus, spasm, or transient difficulty passing stool
- Urinary retention
- Thrombosis of residual external haemorrhoids
- Infection, ulceration, or tissue necrosis if energy is excessive or misplaced
- Persistent prolapse, recurrent bleeding, or need for subsequent banding, mucopexy, or excisional surgery
- Rarely, mucosal injury, fistulation, anal stenosis, or continence disturbance
Avoiding deep thermal injury to the internal sphincter and avoiding excessive energy near the mucosa are technical safety priorities.
How it compares
| Procedure | Main strength | Main limitation |
|---|
| Rubber-band ligation | Office procedure, inexpensive, effective for many grade I-II and selected III internal haemorrhoids | May need repeat treatment; not for external disease |
| LHP | Less wound-related pain, minimal tissue removal, faster early recovery | Cost, variable technique, may be less definitive for major prolapse/external disease |
| Doppler-guided artery ligation with mucopexy | Treats arterial inflow and adds lift for prolapse | Requires equipment and suturing; recurrence can occur |
| Excisional haemorrhoidectomy | Most definitive for large mixed grade III-IV disease, external component, and major prolapse | More painful recovery and wound morbidity |
Surgical texts emphasize that no single operation fits every patient, and that mixed internal-external disease or advanced grade III-IV disease often warrants definitive surgical assessment. Sleisenger and Fordtran's Gastrointestinal and Liver Disease, pp. 2670-2673.
Bottom line
LHP is a minimally invasive, submucosal shrinkage operation, not a laser “removal” of piles. For bleeding grade II or selected grade III internal haemorrhoids, it can offer materially less early pain and less wound morbidity than conventional excision. For bulky external disease, fixed grade IV prolapse, or a need for the most durable anatomical correction, conventional excisional haemorrhoidectomy may be the better operation.
For any rectal bleeding, diagnosis should be established before choosing a haemorrhoid procedure, particularly with anaemia, weight loss, bowel-habit change, family history of colorectal cancer, or bleeding that does not fit a typical haemorrhoid pattern.