UMN FACIAL PALSY

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facial palsy

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upper motor neuron facial palsy forehead sparing clinical examination

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central facial palsy forehead sparing bilateral cortical innervation stroke reliable source

UMN Facial Palsy (Central facial palsy)

A UMN facial palsy results from a lesion above the facial nerve nucleus in the corticobulbar pathway, commonly a contralateral cerebral hemisphere stroke.

Clinical features

  • Weakness of the contralateral lower face
    • Flattened nasolabial fold
    • Drooping of angle of mouth
    • Difficulty showing teeth or puffing out cheek
  • Forehead is relatively spared
    • Can wrinkle forehead
    • Can raise eyebrows
    • Can usually close the eye tightly

Why is the forehead spared?

The facial nucleus has:
  • Upper part supplying forehead and eye-closing muscles, with bilateral cortical innervation
  • Lower part supplying lower facial muscles, with mainly contralateral cortical innervation
Therefore, a unilateral UMN lesion causes weakness mainly of the opposite lower half of the face, while the unaffected hemisphere still drives the upper facial muscles.

Site and side

LesionFacial weakness
Right corticobulbar tract / right cerebral hemisphereLeft lower facial weakness
Left corticobulbar tract / left cerebral hemisphereRight lower facial weakness

Associated findings

Because the lesion is usually cerebral, look for other UMN/cortical signs:
  • Contralateral arm and/or leg weakness
  • Hyperreflexia and extensor plantar response
  • Dysarthria
  • Aphasia, if dominant hemisphere involved
  • Hemisensory loss
  • Visual-field defect

UMN versus LMN facial palsy

FeatureUMN facial palsyLMN facial palsy
SiteCortex or corticobulbar tractFacial nucleus or facial nerve
Facial involvementContralateral lower face predominantlyIpsilateral upper and lower face
Forehead wrinklingPreserved/relatively preservedLost
Eye closureUsually preservedWeak or incomplete
Common causeStrokeBell palsy, Ramsay Hunt syndrome, trauma
Important caution: forehead sparing is a helpful localization sign but not absolute. Some central strokes can produce upper facial weakness, so sudden facial weakness with limb weakness, speech disturbance, imbalance, visual symptoms, or severe headache should be assessed urgently as possible stroke. A recent review discusses this limitation of the traditional rule in central facial palsy after stroke.
One-line exam answer:
UMN facial palsy causes contralateral weakness of the lower face with sparing of the forehead, because the upper facial nucleus receives bilateral corticobulbar innervation, whereas the lower facial nucleus receives predominantly contralateral input.
Source: Localization in Clinical Neurology, 8e, “Anatomy of the Motor System”; Scott-Brown’s Otorhinolaryngology Head & Neck Surgery, “Assessment of the Patient.”

Treatment for umn facial palsy

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acute ischemic stroke treatment

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AHA ASA acute ischemic stroke guideline thrombolysis thrombectomy 2026

UMN facial palsy has no separate facial-nerve treatment. Management is directed at the underlying central lesion, most often an acute stroke.

1. Treat as acute stroke until proven otherwise

New-onset facial asymmetry, especially with limb weakness, speech difficulty, visual symptoms, ataxia, or sensory loss, needs urgent emergency assessment.
  • Establish time last known well, check glucose, perform neurological examination.
  • Urgent non-contrast CT head to exclude hemorrhage, with vascular and perfusion imaging when indicated.
  • Admit to a stroke unit and identify the cause: ischemic stroke, hemorrhage, tumor, demyelination, etc.

2. Reperfusion treatment for eligible ischemic stroke

Under specialist stroke-team assessment:
  • IV thrombolysis for eligible patients within the appropriate time window, including selected extended-window cases guided by imaging.
  • Mechanical thrombectomy for eligible large-vessel occlusion, with selection based on vessel imaging and time/imaging criteria.
Current AHA/ASA guidance covers expanded thrombolysis and thrombectomy eligibility in acute ischemic stroke guidance.

3. Secondary prevention after ischemic stroke

Depending on the cause and bleeding risk:
  • Antiplatelet therapy or anticoagulation for cardioembolic disease such as atrial fibrillation
  • Control hypertension, diabetes, and lipids
  • High-intensity statin when indicated
  • Smoking cessation, physical activity, healthy diet
  • Carotid evaluation and carotid revascularization in selected symptomatic carotid stenosis

4. Rehabilitation

  • Early physiotherapy, occupational therapy, and speech/swallow assessment if deficits coexist
  • Facial exercises and functional retraining may be used when facial weakness persists
  • Treat communication, feeding, mood, and social effects of stroke

Important distinction

Do not routinely give corticosteroids or antivirals for UMN facial palsy. Those are treatments considered for Bell palsy, which is a LMN facial palsy involving the forehead and eye closure.
Exam answer:
Treatment of UMN facial palsy is treatment of its cause, usually acute stroke management with rapid imaging, reperfusion therapy when eligible, stroke-unit care, secondary prevention, and rehabilitation. Steroids are not indicated unless there is another specific diagnosis.

bells palsy treatment

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Bell palsy treatment corticosteroids within 72 hours eye care clinical guideline

Bell palsy treatment

Bell palsy is an acute lower motor neuron facial palsy. Treatment should begin promptly after excluding stroke and alternative diagnoses.

1. Oral corticosteroids: first-line

Start within 72 hours of onset, unless contraindicated.
Common adult regimen:
  • Prednisolone 50-60 mg orally once daily for 5 days, then taper over 5 days
    or
  • Prednisolone 50 mg daily for 10 days
Steroids improve the chance and speed of complete recovery. Prescribers should consider diabetes, active infection, peptic-ulcer risk, psychiatric history, and other steroid contraindications. The AAO-HNS guideline recommends oral steroids within 72 hours in people aged 16 years or older, and NICE guidance also supports considering prednisolone in this window.

2. Eye protection: essential if eye closure is incomplete

Prevent corneal injury:
  • Artificial tears/lubricating drops frequently during the day
  • Lubricating eye ointment at night
  • Tape the eyelid closed or use an eye patch during sleep
  • Protective glasses outdoors
Urgent ophthalmology review is needed for eye pain, redness, blurred vision, corneal exposure, or persistent inability to close the eye.

3. Antivirals: not routine monotherapy

  • Do not use aciclovir/valaciclovir alone instead of corticosteroids.
  • Antiviral therapy may be considered with steroids in severe or complete Bell palsy, though the extra benefit is modest and evidence is less certain.
  • If there is a painful vesicular rash in or around the ear, consider Ramsay Hunt syndrome, where combined antiviral plus steroid treatment is generally used urgently.

4. Supportive care and follow-up

  • Analgesia for ear or facial pain, for example paracetamol or an NSAID if appropriate.
  • Facial physiotherapy may be helpful in persistent weakness, especially chronic cases or synkinesis, but it is not a substitute for early steroids.
  • Facial nerve decompression surgery is not routinely recommended.

Refer or reassess urgently if

  • Forehead is spared, or there are limb weakness, speech disturbance, severe headache, diplopia, or ataxia: possible central stroke
  • Facial weakness is progressive beyond 3 weeks
  • No improvement by about 3 weeks, or incomplete recovery by 3 months
  • Recurrent or bilateral palsy
  • Hearing loss, marked vertigo, ear vesicles, systemic illness, trauma, or a parotid mass
Exam answer:
Treat Bell palsy with oral prednisolone within 72 hours, eye lubrication and protection, analgesia, and follow-up. Add an antiviral only selectively, particularly in severe palsy or suspected Ramsay Hunt syndrome.
Recent evidence note: systematic reviews continue to examine whether antiviral-plus-steroid treatment improves outcomes over steroids alone, including PMID 39724817, but corticosteroids remain the key proven treatment.
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