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DM Comprehensive Medical Record

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The record synthesises all four of your PDFs (Goldman-Cecil, Harrison's 22e, Tintinalli's, Firestein & Kelley's Rheumatology) into 17 fully formatted sections:
SectionContent
1 — BiodataFull patient registration table with occupational-risk note
2 — Chief ComplaintScripted opening questions; verbatim recording space
3 — History of Presenting IllnessSOCRATES framework + all "poly" symptoms (polyuria with ant-attraction question, polydipsia, polyphagia, weight, fatigue, blurred vision, infections) + DKA / HHS / hypoglycaemia acute decompensation screening
4 — Complications Screening8 organ systems: Eyes, Kidneys, Peripheral Neuropathy, Autonomic Neuropathy, CVD, Feet, Musculoskeletal (prayer sign, Dupuytren's, trigger finger, CTS, frozen shoulder, Charcot — from Firestein), Skin
5 — Glycaemic Control HistoryHbA1c trend, SMBG, CGM, hypoglycaemia unawareness table
6 — Past Medical History20+ comorbidities including PCOS, LADA, pancreatitis, autoimmune co-conditions
7 — Drug HistoryAll DM drug classes by name + dose table; drugs that WORSEN glycaemia (steroids, antipsychotics, thiazides, beta-blockers); allergies
8 — Family HistoryDM type, CVD, CKD, obesity, MODY flag
9 — Social HistorySmoking (pack-years formula), alcohol (hypoglycaemia risk), 24-hour diet recall, exercise targets, adherence capacity
10 — DM ClassificationT1/T2/MODY/Secondary/GDM/LADA comparison table with clinician assessment column
11 — Physical ExaminationGeneral, Vitals (with orthostatic BP), Eyes/ENT, CVS (all 4 foot pulses + ABI), Abdomen, Neuro (monofilament, tuning fork, pin-prick, ankle jerks, proprioception), Foot exam, MSK signs, Skin signs
12 — InvestigationsADA diagnostic criteria alert + full results table (HbA1c, OGTT, C-peptide, ACR, eGFR, lipids, ECG, autoantibodies, B12, TSH)
13 — Risk Stratification10-factor table linking each risk factor to clinical implication
14 — Problem ListBlank numbered table
15 — Management PlanEducation, lifestyle, pharmacotherapy step-up (with trial names — LEADER, SUSTAIN-6, CREDENCE), comorbidities, complication-specific treatments, follow-up schedule
16 — Bedside ChecklistPrint-and-tick format for history, examination, and investigations
17 — SignaturesStudent, supervisor, consultant, diagnosis, HbA1c target, next review

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COMPREHENSIVE HISTORY-TAKING MEDICAL RECORD

Diabetes Mellitus & Associated Diseases

Based on: Goldman-Cecil Medicine | Harrison's 22e | Tintinalli's Emergency Medicine | Firestein & Kelley's Rheumatology

Patient: ______________________________ Date: ______________ Hospital No: ______________
Ward/Clinic: ______________ Clerk/Student: ______________________________ Supervisor: ______________________________

SECTION 1: BIODATA

Always record biodata FIRST before asking any history. It anchors the clinical picture.
FieldPatient Entry
Full Name
Age
SexMale / Female / Other
Date of Birth
Occupation
Marital StatusSingle / Married / Divorced / Widowed
Address / Living Situation
Religion / Ethnicity
Date of Admission / Consultation
Referral SourceSelf / GP / Specialist / Emergency
Informant
Reliability of HistoryGood / Fair / Poor — Reason: __________
CLINICAL NOTE — Occupation matters in DM: Shift workers have disrupted glycaemic patterns; sedentary workers have worse insulin resistance; professional drivers face hypoglycaemia risk (licensing implications).

SECTION 2: CHIEF COMPLAINT (CC)

Ask in the patient's own words — do not suggest answers or use medical terminology.
Opening questions:
  • "What brought you here today?"
  • "What is the main problem that is bothering you?"
Write verbatim — record exactly one or two main complaints with duration:
CC: ____________________________________________________________________________
Duration: ______________________________________________________________________

SECTION 3: HISTORY OF PRESENTING ILLNESS (HPI)

Use SOCRATES for EACH complaint. For DM, always probe for hyperglycaemic symptoms, acute decompensation, and complication clues.

3A. SOCRATES Framework

LetterStands ForQuestion to Ask
SSite"Where exactly is the problem?"
OOnset"When did it start? Was it sudden (hours–days) or gradual (weeks–months)?"
CCharacter"What does it feel like? Burning? Tingling? Pressure?"
RRadiation"Does it spread or go anywhere else?"
AAssociated Symptoms"Is there anything else that comes with it?"
TTime Course"Is it constant or does it come and go? Is it getting better or worse?"
EExacerbating / Relieving"What makes it better? What makes it worse?"
SSeverity"On a scale of 0–10, how bad is it? Does it affect daily life?"

3B. Cardinal Symptoms of Hyperglycaemia — Ask ALL

Polyuria (Excessive Urination)

Mechanism: Osmotic diuresis from glucosuria when plasma glucose exceeds renal threshold (~10 mmol/L / 180 mg/dL)
  • "How many times do you urinate during the day?"
  • "Do you wake up at night to urinate? How many times?" (Nocturia)
  • "How much urine do you pass each time — a small amount or a lot?"
  • "Is your urine pale, dark, or does it look foamy?"
  • "Have you noticed ants being attracted to where you urinate?" (Glycosuria — classical bedside clue)
Findings: _____________________________________________________________________

Polydipsia (Excessive Thirst)

Mechanism: Compensatory response to dehydration and hyperosmolarity from glucosuria
  • "Do you feel very thirsty more than usual?"
  • "How much water or fluids do you drink in a day?"
  • "Is the thirst there all the time, or does drinking relieve it only briefly?"
Findings: _____________________________________________________________________

Polyphagia (Excessive Hunger)

Mechanism: Cellular starvation despite hyperglycaemia — especially prominent in Type 1 DM
  • "Is your appetite increased, decreased, or normal?"
  • "Do you feel hungry soon after eating a full meal?"
Findings: _____________________________________________________________________

Weight Changes

PatternMechanism
T1DM — Weight lossCatabolism of fat and muscle
T2DM — Often weight gainObesity-driven; weight loss only in late uncontrolled T2DM
  • "Have you lost or gained weight recently without trying?"
  • "How much weight have you lost/gained, and over what period of time?"
Findings: _____________________________________________________________________

Fatigue and Weakness

  • "Do you feel tired all the time?"
  • "Does the tiredness come on even without activity, or only after effort?"
  • "Do you feel weak in your arms or legs?"
Findings: _____________________________________________________________________

Blurred Vision

Mechanism: Lens osmotic swelling from sorbitol accumulation during hyperglycaemia
  • "Has your vision changed recently?"
  • "Is the blurring in one eye or both?"
  • "Is it constant or does it fluctuate with your blood sugar levels?"
  • "Have you seen floaters, flashes of light, or had sudden loss of vision?"
Findings: _____________________________________________________________________

Recurrent Infections and Poor Wound Healing

Mechanism: Phagocyte dysfunction + glucosuria + impaired vascular and neuropathic tissue repair
  • "Do you get infections frequently — skin, genital, or urinary?"
  • "Have you noticed itching around the genitals or white discharge?" (Candidiasis)
  • "Do wounds, cuts, or sores take a long time to heal?"
  • "Do you have any sores or ulcers not healing, especially on the feet?"
Findings: _____________________________________________________________________

3C. Onset and Type Classification Clues

Onset PatternClinical Implication
Acute onset (days–weeks)Suggests Type 1 DM / DKA → ask about nausea, vomiting, abdominal pain, fruity breath
Insidious onset (months–years)Suggests Type 2 DM → often found incidentally or when complication presents first
During pregnancyGestational diabetes → screen with OGTT
Post-illness / surgery / steroidsSecondary / drug-induced DM → review precipitants

3D. Acute Decompensation — Screen for ALL Three

CRITICAL ALERT — Always ask these questions directly. Missing DKA or HHS is a clinical emergency.
EmergencySymptoms to Screen
DKA (T1DM, occasionally T2DM)Nausea, vomiting, abdominal pain, fruity/acetone breath odour, rapid breathing (Kussmaul), decreased consciousness
HHS — Hyperosmolar Hyperglycaemic State (T2DM)Extreme thirst, confusion, neurological changes (focal deficits, seizures), profoundly elevated blood sugar (>33 mmol/L), marked dehydration, NO significant acidosis/ketonuria
HypoglycaemiaSweating, palpitations, tremor, confusion, aggression, unconsciousness — especially on insulin or sulfonylurea
  • "Have you had nausea, vomiting, or abdominal pain with very high blood sugar?"
  • "Have you had episodes of sweating, shakiness, heart racing, or confusion — especially if you missed a meal?"
  • "Have you ever been hospitalised for very high blood sugar or a diabetic coma?"

SECTION 4: COMPLICATIONS SCREENING — SYSTEMATIC REVIEW BY ORGAN

Tell the patient: "I am now going to ask about different parts of your body to make sure everything is being checked carefully."

4A. EYES — Diabetic Retinopathy

  • "Has a doctor ever looked at the back of your eyes with a special instrument?" (Fundoscopy / dilated eye exam)
  • "Have you had any changes in your vision — blurring, dark spots, or sudden loss of sight?"
  • "Have you seen floaters, cobweb-like shapes, or flashes of light?"
  • "Have you had any eye injections (anti-VEGF) or laser treatment for your eyes?"
  • "Have you ever been told you have diabetic eye disease or glaucoma?"
Eyes findings: ________________________________________________________________

4B. KIDNEYS — Diabetic Nephropathy

  • "Have you noticed your urine becoming foamy or frothy?" (Proteinuria)
  • "Do you have swelling in your legs, ankles, or around your eyes in the morning?" (Nephrotic oedema)
  • "Have you ever been told your kidneys are not working properly?"
  • "Do you know your kidney function test results — creatinine or eGFR?"
  • "Have you ever been on dialysis or been referred to a kidney specialist?"
Kidney findings: ______________________________________________________________

4C. PERIPHERAL NERVOUS SYSTEM — Sensorimotor Neuropathy

  • "Do you have numbness, tingling, or a burning sensation in your feet or hands?"
  • "Does it feel like you are walking on cotton wool or sand?"
  • "Is the pain or tingling worse at night?"
  • "Have you lost feeling in your feet — for example, unable to feel hot or cold water?"
  • "Have you had falls because of loss of balance or unsteadiness?"
  • "Do you have any foot ulcers, wounds, or sores that are not healing?"
  • "Have you ever had an amputation?"
Neuropathy findings: __________________________________________________________

4D. AUTONOMIC NERVOUS SYSTEM — Autonomic Neuropathy

  • "Do you feel dizzy or lightheaded when you stand up quickly?" (Postural / orthostatic hypotension)
  • "Do you feel full very quickly after eating only a small meal?" (Gastroparesis)
  • "Do you have nausea, vomiting, or bloating after eating?"
  • "Do you have problems with your bowels — constipation or diarrhoea that comes and goes?" (Autonomic gut dysmotility)
  • "Do you sweat abnormally — too much or not at all?"
  • "Do you have difficulty controlling your bladder or do you need to strain to pass urine?" (Neurogenic bladder)
  • "[Male patients — ask sensitively and privately]: Do you have difficulty getting or maintaining an erection?" (Erectile dysfunction — earliest autonomic symptom in men)
Autonomic findings: ___________________________________________________________

4E. HEART AND LARGE BLOOD VESSELS — Macrovascular Disease

NOTE: Diabetic patients may have SILENT myocardial infarction — chest pain may be absent due to cardiac autonomic neuropathy. Always ask about atypical symptoms.
  • "Do you have chest pain or tightness — especially on walking, climbing stairs, or at rest?"
  • "Do you feel short of breath with activity or when lying flat?" (Orthopnoea — heart failure)
  • "Do you have swelling in both ankles?" (Biventricular failure)
  • "Have you had a heart attack or been told your heart arteries are blocked?"
  • "Have you had any stroke or sudden weakness / numbness on one side of your body?"
  • "Do you get pain in your calves when walking that goes away with rest?" (Intermittent claudication = Peripheral Arterial Disease)
  • "Do you have palpitations or feel your heart racing or skipping?"
Cardiovascular findings: _______________________________________________________

4F. FEET — Diabetic Foot Disease

  • "Do you check your feet every day?"
  • "What type of footwear do you use? Do they fit well?"
  • "Do you have any calluses, corns, or hard skin on your feet?"
  • "Have you had any cuts, blisters, or sores on your feet that you did not feel?" (Loss of protective sensation)
  • "Have you been told your foot bones have changed shape?" (Charcot's arthropathy)
Foot findings: ________________________________________________________________

4G. MUSCULOSKELETAL — Diabetic Rheumatological Conditions

From Firestein & Kelley's Rheumatology: Diabetic cheiroarthropathy, Dupuytren's, trigger finger, carpal tunnel, adhesive capsulitis, and Charcot arthropathy are all strongly DM-associated.
ConditionKey Features
Cheiroarthropathy (Limited Joint Mobility)Prayer sign — cannot fully oppose palmar surfaces; Tabletop sign
Dupuytren's Contracture>20% in T2DM; palm nodules/cords; ring and little finger fixed flexion
Trigger Finger (Flexor Tenosynovitis)A1 pulley nodule; snapping/locking; multiple fingers in DM
Carpal Tunnel SyndromeMedian nerve; Tinel's, Phalen's, Durkan's tests; thenar wasting
Adhesive Capsulitis (Frozen Shoulder)Limited shoulder ROM; insidious onset; bilateral in DM
Charcot Arthropathy (Neuroarthropathy)Painless swollen warm foot/ankle; bone destruction on X-ray
Diabetic AmyotrophySevere unilateral thigh pain, weakness, quadriceps wasting; L2–L4
  • "Do you have stiffness in your fingers or cannot fully close/open your hands?"
  • "Do you have difficulty getting your palm flat on a table?"
  • "Do any of your fingers catch, snap, or lock when you bend them?"
  • "Do you have pain or tingling in your hands at night?" (Carpal Tunnel)
  • "Do you have pain or stiffness in your shoulder that limits lifting your arm?"
MSK findings: _________________________________________________________________

4H. SKIN

  • "Do you have dry, itchy, or discoloured skin?"
  • "Do you have any non-healing wounds or sores?"
  • "Have you noticed darker velvety skin at the back of your neck or in the armpits?" (Acanthosis nigricans — insulin resistance)
Skin findings: ________________________________________________________________

SECTION 5: GLYCAEMIC CONTROL HISTORY

This section establishes current control, monitoring practice, and hypoglycaemia risk — essential for every DM encounter.
  • "When were you first told you have diabetes? Who made the diagnosis?"
  • "What type of diabetes do you have — Type 1, Type 2, or were you not told?"
  • "What was your most recent HbA1c result? When was it done? Is it improving or worsening?"
  • "Do you check your blood sugar at home with a glucometer? How often?"
  • "What are your typical fasting blood sugar readings in the morning?"
  • "What are your blood sugar readings after meals?"
  • "Do you use a continuous glucose monitor (CGM)? What is your time-in-range?"

Hypoglycaemia Assessment

  • "Have you had episodes of low blood sugar (hypoglycaemia)?"
  • "How often do these episodes happen?"
  • "What are your warning signs — sweating, shaking, heart racing, confusion?"
  • "Have you lost your warning signs for low blood sugar?" (Hypoglycaemia unawareness — HIGH RISK)
  • "Have you ever needed help from another person, or called emergency services?"
ParameterResult / Record
HbA1c Last Result & Date
Fasting BG Range (mmol/L)
Post-meal BG Range (mmol/L)
Hypoglycaemia FrequencyNever / Occasional / Weekly / Daily
Hypoglycaemia UnawarenessYes / No
Severe Hypoglycaemia (needed help)Yes / No — Date: _________
DKA / HHS Hospital AdmissionsNumber: ___ Last date: _________
Self-Monitoring PracticeYes / No — Frequency: ___________
CGM UseYes / No — Device: _______________

SECTION 6: PAST MEDICAL HISTORY (PMH)

Ask systematically — these comorbidities are both DM complications AND DM accelerators.
ConditionStatus / Details
HypertensionYes / No — Since: ___ Treated: Yes / No
Dyslipidaemia (High Cholesterol / Triglycerides)Yes / No
Coronary Artery Disease / Angina / MIYes / No — Date: _______
Congestive Heart FailureYes / No — NYHA Class: ___
Stroke or TIAYes / No — Date: _______ Side: L / R
Peripheral Arterial DiseaseYes / No — ABI known: ___
Diabetic RetinopathyYes / No — Grade: NPDR / PDR / Maculopathy
Diabetic Nephropathy / CKDYes / No — eGFR: ___ ACR: ___ Stage: ___
Diabetic Neuropathy (peripheral / autonomic)Yes / No — Type: ______________
Diabetic Foot Ulcer / AmputationYes / No — Level: ________________
Charcot ArthropathyYes / No
Non-alcoholic Fatty Liver Disease (NAFLD/NASH)Yes / No
Sleep ApnoeaYes / No — On CPAP: Yes / No
Polycystic Ovary Syndrome (PCOS)Yes / No (women)
Gestational DiabetesYes / No (women) — Year: ___
Pancreatitis or Pancreatic DiseaseYes / No
Thyroid DiseaseYes / No — Type: ________________
Other Autoimmune Conditions (T1DM)Coeliac / Adrenal insufficiency / Vitiligo / Other
Previous DKA / HHS EpisodesYes / No — Dates / Number: __________
Previous Surgeries
Psychiatric ConditionsDepression / Anxiety / Eating disorder

SECTION 7: DRUG HISTORY & ALLERGIES

For each drug: Name → Dose → Frequency → Route → Compliance → Duration.

7A. Diabetes Medications

Drug ClassDetails
MetforminDose: ___ Freq: ___ Compliance: Good / Poor
Sulfonylurea (gliclazide, glibenclamide, glipizide)Dose: ___ Freq: ___ Compliance: Good / Poor
SGLT2 Inhibitor (empagliflozin, dapagliflozin, canagliflozin)Dose: ___ Freq: ___ Compliance: Good / Poor
GLP-1 Receptor Agonist (semaglutide, liraglutide, dulaglutide)Dose: ___ Freq: ___ Route: SC / Oral
DPP-4 Inhibitor (sitagliptin, linagliptin, saxagliptin)Dose: ___ Freq: ___ Compliance: Good / Poor
Thiazolidinedione (pioglitazone)Dose: ___ Freq: ___
Insulin — Basal (glargine / detemir / degludec)Dose: ___ Timing: ___ Injection site: ___
Insulin — Rapid-Acting (aspart / lispro / glulisine)Dose: ___ Timing: ___ Technique: ___
Insulin — PremixedType: ___ Dose: ___ Timing: ___
Insulin Pump (CSII)Yes / No — Basal rate: ___ Bolus: ___
  • "Do you take your diabetes medications every day? Do you ever miss doses?"
  • "For insulin: Do you rotate injection sites? Are there any lumpy areas?" (Lipohypertrophy)

7B. Other Medications (Comorbidities)

DrugName / Dose / Frequency
ACE Inhibitor / ARB (renoprotective)
Statin (atorvastatin, rosuvastatin)
Aspirin or Antiplatelet
Antihypertensive (amlodipine, bisoprolol, doxazosin)
Diuretic (furosemide, spironolactone, HCTZ)

7C. Drugs That WORSEN Glycaemic Control

Always check for these — they are a common and frequently overlooked cause of poor glycaemic control.
DrugStatus
Corticosteroids (prednisolone, dexamethasone)Currently taking: Yes / No
Thiazide Diuretics (hydrochlorothiazide, indapamide)Currently taking: Yes / No
Atypical Antipsychotics (olanzapine, clozapine)Currently taking: Yes / No
Beta-Blockers (propranolol, atenolol)Currently taking: Yes / No — masks hypoglycaemia symptoms
Calcineurin Inhibitors (tacrolimus, cyclosporin)Currently taking: Yes / No
Protease Inhibitors (HIV treatment)Currently taking: Yes / No
Niacin / Nicotinic acidCurrently taking: Yes / No
Herbal / Traditional MedicinesDetails: __________________________

7D. Allergies

Drug NameReaction (e.g. rash, anaphylaxis, GI intolerance)

SECTION 8: FAMILY HISTORY (FH)

A strong family history of T2DM confers 2–4× increased risk. First-degree relatives with T1DM increase risk ~15-fold.
Family ConditionDetails
Diabetes (Type 1 or 2) — first-degree relativesYes / No — Who: Father / Mother / Sibling — Type: ___
HypertensionYes / No — Who: ______________________
Coronary Artery Disease / Early MI (<55 years)Yes / No — Who: ______________________
StrokeYes / No — Who: ______________________
Chronic Kidney DiseaseYes / No — Who: ______________________
ObesityYes / No — Who: ______________________
Thyroid DiseaseYes / No — Who: ______________________
MODY (DM in 3 generations, young, non-obese, no antibodies)Yes / No — Suggest genetic testing if suspected
Autoimmune ConditionsYes / No — Type: ____________________

SECTION 9: SOCIAL HISTORY (SH)

9A. Smoking

  • "Do you smoke or have you ever smoked?"
  • "How many cigarettes per day? For how many years?"
  • Pack-years = (cigarettes/day ÷ 20) × years smoked
  • "If stopped — when did you stop?"
ParameterDetails
Smoking StatusNever / Current / Ex-smoker
Cigarettes/day
Duration (years)
Pack-years
Year stopped (if ex)
Smoking DOUBLES cardiovascular risk in DM. It also impairs wound healing, worsens neuropathy, and accelerates nephropathy. Smoking cessation is a core DM management goal.

9B. Alcohol

  • "Do you drink alcohol? What type? How much per week?"
  • "Do you ever drink on an empty stomach?"
Alcohol can mask hypoglycaemia symptoms AND cause delayed hypoglycaemia up to 24 hours after drinking — especially on insulin or sulfonylurea. Educate all patients.
ParameterDetails
Alcohol StatusNever / Social / Regular / Dependent
Units per week
Type (beer, spirits, wine)
Binge drinking (>6 units/session)Yes / No

9C. Diet

  • "What do you usually eat in a day from morning to night?" (24-hour dietary recall)
  • "Do you eat regular meals, or do you skip meals?"
  • "How much rice, bread, sugar, or sweet drinks do you consume daily?"
  • "Are you following a special diabetic diet?"
  • "Do you count carbohydrates or follow a meal plan?"
ParameterDetails
Meal RegularityRegular / Irregular / Skips meals
Carbohydrate IntakeHigh / Moderate / Low / Unknown
Sweet Drinks (soda, juice, energy drinks)Frequency: ___________________
Diabetic Diet AdherenceYes / No / Partial

9D. Physical Activity

  • "How active are you on a daily basis?"
  • "Do you do any structured exercise? What type, how often, and for how long?"
Target: 150 min/week moderate aerobic exercise + resistance training 2–3×/week. Exercise reduces HbA1c by ~0.6% independently.
ParameterDetails
Activity LevelSedentary / Lightly active / Moderately active / Very active
Exercise Type
Exercise Frequency / Duration

9E. Socioeconomic Status & Adherence Capacity

  • "Do you have any difficulty affording your medications or test strips?"
  • "Who cooks your meals at home?"
  • "Do you live alone or with family?"
  • "Do you understand how to use your glucometer and insulin device?"
ParameterDetails
Lives Alone / With Family
Medication AffordabilityNo difficulty / Moderate / Significant difficulty
Health LiteracyGood / Fair / Poor
Occupational Risk for HypoglycaemiaDriving / Machinery / Heights: Yes / No

SECTION 10: DIABETES CLASSIFICATION — CLINICIAN ASSESSMENT

TypeKey Features
Type 1 DMUsually <30 yrs, lean, acute onset, prone to DKA, autoimmune markers (anti-GAD, anti-islet, anti-IA2, anti-ZnT8), C-peptide low/undetectable, requires insulin from outset
Type 2 DMUsually >35 yrs, overweight/obese, insidious onset, strong family history, associated with metabolic syndrome, C-peptide elevated
MODYYoung, non-obese, autosomal dominant FH over 3 generations, no autoantibodies — refer for genetic testing (GCK, HNF1A mutations)
Secondary DMPancreatitis, pancreatectomy, haemochromatosis, Cushing's syndrome, acromegaly, drug-induced — underlying cause drives management
Gestational DMDiagnosed during pregnancy, resolves post-partum — but 50% develop T2DM within 10 years; screen with OGTT
LADAAge >30, initially resembles T2DM, anti-GAD positive, progressive insulin deficiency — often misclassified as T2DM
Clinical DM Classification: _____________________________________________________

SECTION 11: PHYSICAL EXAMINATION — GUIDE & FINDINGS

11A. General Assessment

ParameterFinding
Height (cm) / Weight (kg) / BMI___ / ___ / ___ (Overweight: >25; Obese: >30)
Waist Circumference (cm)___ (At risk: women >80 cm; men >90 cm Asian / >102 cm Western)
General AppearanceWell / Unwell / Ill — Cushingoid features: Yes / No
Hydration StatusNormal / Dehydrated — Skin turgor / dry mucous membranes
Acanthosis NigricansPresent / Absent — Site: posterior neck / axillae / groin

11B. Vital Signs

ParameterFinding
Blood Pressure (Right arm)/ mmHg — Target <130/80 in DM
Blood Pressure (Left arm)/ mmHg
Orthostatic BPSupine: / | Standing 1 min: / | Standing 3 min: / — Drop ≥20 mmHg systolic = autonomic neuropathy
Heart Rate___ bpm — Regular / Irregular — Fixed resting tachycardia = autonomic neuropathy
Respiratory Rate___ /min — Kussmaul breathing (deep sighing) = DKA
Temperature___ °C — Fever suggests infection (foot cellulitis / osteomyelitis)
SpO2___ %
Random Capillary Blood Glucose___ mmol/L

11C. Eyes, ENT, Oral Cavity

ExaminationFinding
Visual Acuity (Snellen)Right: ___ Left: ___
Rubeosis IridisPresent / Absent (neovascularisation of iris = advanced retinopathy)
FundoscopyMicroaneurysms / Haemorrhages / Cotton-wool spots / Hard exudates / Neovascularisation / Normal
CN III PalsyPresent / Absent (painless, pupil-sparing = diabetic mononeuropathy)
Oral CandidiasisPresent / Absent — White plaques on mucosa
Periodontal DiseasePresent / Absent
ThyroidNormal / Goitre / Nodule

11D. Cardiovascular

ExaminationFinding
Carotid BruitsRight: Present / Absent | Left: Present / Absent
Apex Beat
Heart SoundsS1+S2+___ (S3 = Heart Failure; S4 = Diabetic cardiomyopathy)
Pedal OedemaPresent / Absent — Grade: ___ Bilateral / Unilateral
Femoral PulseRight: Present / Absent | Left: Present / Absent
Popliteal PulseRight: Present / Absent | Left: Present / Absent
Posterior Tibial PulseRight: Present / Absent | Left: Present / Absent
Dorsalis Pedis PulseRight: Present / Absent | Left: Present / Absent
Ankle-Brachial Index (ABI)Right: ___ Left: ___ (Normal ≥0.9; PAD <0.9)

11E. Abdomen

ExaminationFinding
Hepatomegaly (NAFLD)Present / Absent — Size: ___ cm below costal margin
Renal Angle TendernessRight: Yes / No | Left: Yes / No
Insulin Injection SitesAbdomen / Flanks / Thighs — Lipohypertrophy: Yes / No — Site: ___
Renal Artery BruitPresent / Absent

11F. Neurological — Peripheral Neuropathy Screening

Diabetic neuropathy is a diagnosis of exclusion — always rule out B12 deficiency, hypothyroidism, uraemia, CIDP, and vasculitic neuropathy.
TestFinding
10g Semmes-Weinstein Monofilament (10 plantar sites per foot)Right: ___/10 normal | Left: ___/10 normal
128-Hz Tuning Fork (hallux → medial malleolus)Right: Normal / Reduced / Absent | Left: Normal / Reduced / Absent
Pin-prick (dorsal foot — small fibre)Right: Normal / Reduced | Left: Normal / Reduced
Temperature Discrimination (cool vs warm)Right: Normal / Impaired | Left: Normal / Impaired
Ankle Jerk ReflexRight: Present / Absent / Diminished | Left: Present / Absent / Diminished
Patellar ReflexRight: Present / Absent | Left: Present / Absent
Proprioception (hallux up/down with eyes closed)Right: Normal / Impaired | Left: Normal / Impaired
Romberg's TestPositive / Negative
Gait AssessmentNormal / Wide-based / Antalgic

11G. Foot Examination — 5-Minute Minimum

ALWAYS remove shoes and socks. Check the feet at EVERY DM visit. Foot disease is the most preventable major complication of DM.
FeatureFinding — Right Foot / Left Foot
Skin IntegrityUlcers: Yes / No — Location: ___ Size: ___ Depth: ___ Wagner grade: ___
Calluses / CornsPresent / Absent — Location: _______________
DeformitiesHallux valgus / Hammertoe / Charcot foot / Normal
Interdigital SpacesMaceration / Tinea pedis / Normal
NailsOnychomycosis / Ingrowing / Normal
Skin Colour / TemperatureWarm / Cool / Discoloured
Callus Over Pressure PointsPresent / Absent
Footwear InspectionAppropriate / Ill-fitting / Absent

11H. Musculoskeletal — Diabetic-Specific Signs

SignHow to PerformPositive Finding
Prayer Sign (Cheiroarthropathy)Both palms pressed together, wrists dorsiflexed, hold 5 secondsGap remains between palmar surfaces — cannot fully oppose
Tabletop SignPalm flat on table, fingers extendedFingers remain partially flexed — visible gap
Dupuytren's ContracturePalpate entire palmar fascia; palpate for nodules, cords; passive extension testPalpable cord + fixed flexion deformity — ring and little fingers
Trigger FingerPalpate A1 pulley at palmar crease; ask patient to close and open fistCatch, snap, or locking during flexion/extension
Tinel's SignTap over carpal tunnel at palmar wrist crease 2–3 timesElectric shock/tingling into thumb, index, middle fingers
Phalen's TestBoth wrists in maximum passive flexion for 60 secondsNumbness/tingling in median nerve distribution within 60 seconds
Durkan's CompressionFirm thumb pressure over carpal tunnel for 30 secondsParaesthesia in median nerve distribution
Shoulder Abduction ROMActive and passive rangeNormal >180°; reduced = frozen shoulder (adhesive capsulitis)
Charcot FootInspect and palpate mid-foot and ankleWarm, swollen, deformed, painless foot — bone destruction on X-ray
Examination FindingResult
Prayer SignPositive / Negative
Tabletop SignPositive / Negative
Dupuytren's ContracturePresent / Absent — Fingers: ___ Flexion deficit: ___ degrees
Trigger FingerPresent / Absent — Fingers affected: ___
Tinel's Sign at wristPositive / Negative — Right / Left
Phalen's TestPositive / Negative — Latency: ___ seconds
Thenar Wasting (advanced CTS)Present / Absent
Shoulder Abduction RangeRight: ___° Left: ___°
Charcot FootPresent / Absent

11I. Skin Examination

Skin FindingResult
Acanthosis NigricansPresent / Absent — Location: _______________
Necrobiosis LipoidicaPresent / Absent (anterior tibiae — strongly associated with T1DM)
Diabetic Dermopathy (shin spots)Present / Absent — most common cutaneous finding; marker of duration
Eruptive XanthomasPresent / Absent (severe hypertriglyceridaemia)
Tinea Pedis / OnychomycosisPresent / Absent
Lipohypertrophy at Injection SitesPresent / Absent — Site: _______________
Lipoatrophy (rare)Present / Absent
VitiligoPresent / Absent (T1DM autoimmune marker)

SECTION 12: INVESTIGATIONS — RESULTS RECORD

ADA Diagnostic Criteria — Any ONE of the following:
  • FPG ≥7.0 mmol/L
  • 2-hr OGTT ≥11.1 mmol/L
  • Random PG ≥11.1 mmol/L + symptoms
  • HbA1c ≥48 mmol/mol (≥6.5%)
Asymptomatic patients require TWO abnormal tests on DIFFERENT days to confirm diagnosis.

12A. Glycaemic

TestResult / Reference Range
Fasting Plasma Glucose (FPG)___ mmol/L (DM: ≥7.0 | Pre-DM: 5.6–6.9 | Normal: <5.6)
2-hour OGTT (75g glucose)___ mmol/L (DM: ≥11.1 | IGT: 7.8–11.0)
HbA1c___ mmol/mol (___ %) (DM: ≥48/≥6.5% | Pre-DM: 39–47/5.7–6.4%)
Random Plasma Glucose + Symptoms___ mmol/L (DM: ≥11.1 with symptoms)
C-Peptide___ pmol/L (T1DM: low/undetectable; T2DM: normal/high)
Fasting Insulin___ mU/L (elevated with normal/high FPG = insulin resistance)
HOMA-IR___ (insulin resistance index)

12B. Autoimmune (Type 1 / LADA Classification)

TestResult
Anti-GAD AntibodiesPositive / Negative / Not done
Islet Cell Antibodies (ICA)Positive / Negative / Not done
Anti-IA2 AntibodiesPositive / Negative / Not done
Anti-ZnT8 AntibodiesPositive / Negative / Not done

12C. Metabolic Panel

TestResult
Full Blood Count (FBC)Hb: ___ WBC: ___ Platelets: ___ (Anaemia affects HbA1c interpretation)
Urea / BUN___ mmol/L
Creatinine___ micromol/L
eGFR (CKD-EPI)___ mL/min/1.73m² (CKD Stage: _____)
Urine Albumin-to-Creatinine Ratio (ACR)___ mg/mmol (Microalbuminuria: 3–30; Macroalbuminuria: >30)
Urine DipstickGlucose: ___ Protein: ___ Ketones: ___ Nitrites: ___ Blood: ___
Total Cholesterol___ mmol/L
LDL Cholesterol___ mmol/L (Target <1.8 mmol/L if high CV risk)
HDL Cholesterol___ mmol/L
Triglycerides___ mmol/L (>5.6 = hypertriglyceridaemia with pancreatitis risk)
Liver Function Tests (ALT, AST, ALP, GGT)ALT: ___ AST: ___ ALP: ___ GGT: ___ (NAFLD monitoring)
TSH / Free T4TSH: ___ FT4: ___ (Annual in T1DM; consider in T2DM)
Uric Acid___ mmol/L (elevated in insulin resistance / metabolic syndrome)
Serum B12___ pmol/L (deficiency from long-term metformin)

12D. Cardiovascular

TestResult
12-Lead ECGNormal / LVH / Q waves (silent MI) / AF / Conduction defect: ___
EchocardiogramEF: ___ Diastolic dysfunction: Yes / No (if clinical HF)
Ankle-Brachial Index (ABI)Right: ___ Left: ___ (PAD <0.9)
Chest X-RayNormal / Cardiomegaly / Pulmonary oedema / Other: ___

12E. Ophthalmology

ScreeningResult
Dilated Fundus Exam / Retinal PhotographyDate: ___ Finding: NPDR / PDR / Maculopathy / Normal
FrequencyAt diagnosis (T2DM) | Within 5 years (T1DM) | Then annually

12F. Coexisting Autoimmune (T1DM Screening)

TestResult
Anti-TPO and Anti-Thyroglobulin AntibodiesPositive / Negative / Not done
Anti-tTG IgA (Coeliac Disease)Positive / Negative / Not done
Adrenal Antibodies (21-hydroxylase) if Addison's suspectedPositive / Negative / Not done

SECTION 13: RISK STRATIFICATION SUMMARY

Risk FactorPatient StatusClinical Implication
Disease Duration (years)>10 years = significantly increased microvascular risk
HbA1c>75 mmol/mol (>9%) = very high risk
Blood PressureUncontrolled BP accelerates nephropathy and retinopathy
DyslipidaemiaDrives macrovascular disease
SmokingDoubles CVD risk; impairs wound healing
ObesityWorsens insulin resistance and CV risk
Existing Organ Damage (retinopathy / nephropathy / neuropathy)Already present = highest risk tier
Hypoglycaemia UnawarenessRelax targets; refer to specialist
PregnancyTight control; target HbA1c <48 mmol/mol pre-conceptionally
Established Cardiovascular DiseaseSGLT2i or GLP-1 RA preferred; high-intensity statin; antiplatelet
Overall Risk Stratification: ____________________________________________________
HbA1c Target for this patient: _________________________________________________

SECTION 14: ACTIVE PROBLEM LIST

#Problem
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SECTION 15: MANAGEMENT PLAN FRAMEWORK

15A. Patient Education

  • Disease understanding — what is DM and what causes complications
  • Sick-day rules — never stop insulin when ill
  • Hypoglycaemia recognition and treatment — carry fast-acting glucose (15g rule)
  • Foot care — daily inspection, proper footwear, nail care, when to seek help
  • SMBG technique
  • Medication adherence counselling

15B. Lifestyle Modification

  • Diet: Reduced refined carbohydrates; Mediterranean or DASH diet pattern; caloric restriction if obese
  • Exercise: 150 min/week moderate aerobic activity + resistance training 2–3×/week (reduces HbA1c ~0.6%)
  • Weight loss: 5–10% body weight reduction significantly improves glycaemic control, BP, and lipid profile
  • Smoking cessation — pharmacotherapy (varenicline) if needed
  • Alcohol reduction counselling

15C. Pharmacotherapy — T2DM Step-Up Approach

StepDrugKey Points
1st LineMetforminReduces hepatic glucose output; weight-neutral; renally dosed (reduce if eGFR 30–45; stop if <30)
Add-on (CVD/CKD)SGLT2 Inhibitor (empagliflozin, dapagliflozin)Renal and cardioprotective; reduce HbA1c, BP, weight; risk of genital mycotic infections
Add-on (CVD/Obesity)GLP-1 RA (semaglutide, liraglutide)Weight loss; CV benefit (LEADER trial, SUSTAIN-6); injectable or oral
Add-onDPP-4 Inhibitor (sitagliptin)Weight-neutral; well-tolerated; oral
Add-onSulfonylurea (gliclazide)Inexpensive; hypoglycaemia risk; weight gain
EscalationBasal Insulin (glargine / detemir)When oral agents fail; start 0.1–0.2 U/kg/day
EscalationBasal-Bolus InsulinMost physiological; essential in T1DM from outset
T1DM: Insulin is mandatory from diagnosis. Use basal insulin (glargine/detemir/degludec) + rapid-acting at mealtimes (aspart/lispro/glulisine). Consider SGLT2i or GLP-1 RA as adjuncts in poorly controlled T1DM.

15D. Treating Comorbidities

ComorbidityManagement
HypertensionACE inhibitor or ARB as first choice (renoprotective + anti-proteinuric); target <130/80 with CKD/CVD
DyslipidaemiaHigh-intensity statin for most DM patients with CVD risk; fenofibrate if severe hypertriglyceridaemia
Antiplatelet TherapyLow-dose aspirin for ESTABLISHED CVD; not routine for primary prevention
ObesityGLP-1 RA (semaglutide 2.4 mg); consider bariatric surgery if BMI >35 with inadequate control

15E. Complication-Specific Treatment

ComplicationKey Treatment
Retinopathy (NPDR)Tight glycaemic + BP control; annual ophthalmology follow-up
Retinopathy (PDR)Laser photocoagulation or anti-VEGF (ranibizumab)
Nephropathy (microalbuminuria)ACEi or ARB; SGLT2i reduces CKD progression (CREDENCE, DAPA-CKD trials); protein restriction
Painful NeuropathyDuloxetine (1st line); pregabalin; gabapentin; amitriptyline; topical capsaicin
GastroparesisMetoclopramide; domperidone; small frequent meals; low-fat diet
Erectile DysfunctionPDE5 inhibitors (sildenafil, tadalafil); refer to urology
Foot UlcerOffloading (total contact cast); wound debridement; culture-guided antibiotics; vascular surgery if PAD
Charcot Foot (active)Total contact casting; no weight-bearing; urgent orthopaedic / diabetic foot team referral
Cheiroarthropathy / Trigger FingerPhysiotherapy; corticosteroid injection (less effective in DM); surgical release if severe

15F. Follow-Up Schedule

ParameterFrequency
HbA1cEvery 3 months if poorly controlled; every 6 months if stable
BP, Weight, Waist CircumferenceEvery clinic visit
Urine ACR + eGFRAnnually
Fasting Lipid ProfileAnnually
Dilated FundoscopyAnnually after initial screen
Comprehensive Foot ExamAnnually; every visit if high-risk foot
Thyroid Function (TSH)Annually (T1DM); as clinically indicated (T2DM)
B12 Level (on metformin)Every 2 years; annually if low intake or symptoms
Dental ReviewEvery 6–12 months (periodontal disease worsens glycaemic control)
Influenza VaccineAnnually
Pneumococcal VaccineAs per local guidelines

SECTION 16: BEDSIDE QUICK-REFERENCE CHECKLIST

History Checklist

  • Biodata complete (name, age, sex, occupation, address, informant)
  • Chief complaint in patient's own words with duration
  • SOCRATES applied to each complaint
  • Polyuria, polydipsia, polyphagia, weight loss asked
  • Blurred vision, recurrent infections, poor wound healing asked
  • DKA symptoms (nausea/vomiting/fruity breath) screened
  • HHS symptoms (confusion/extreme thirst/very high BG) screened
  • Hypoglycaemia episodes and unawareness assessed
  • Eyes complications screened (retinopathy, laser treatment)
  • Kidney complications screened (foamy urine, oedema, eGFR)
  • Peripheral neuropathy screened (numbness, burning, cotton-wool sensation)
  • Autonomic neuropathy screened (postural dizziness, gastroparesis, ED, neurogenic bladder)
  • Cardiovascular history (chest pain, dyspnoea, claudication, palpitations)
  • Foot history (ulcers, amputation, footwear)
  • MSK history (stiff fingers, trigger finger, shoulder pain)
  • Glycaemic control history (HbA1c trend, SMBG, hypoglycaemia)
  • Past medical history (HTN, dyslipidaemia, CVD, CKD, thyroid, PCOS)
  • All diabetes medications recorded with dose, frequency, compliance
  • Drugs that worsen glucose checked (steroids, antipsychotics, thiazides, beta-blockers)
  • Allergies recorded
  • Family history (DM, CVD, CKD, obesity, MODY)
  • Smoking status and pack-years calculated
  • Alcohol history with hypoglycaemia education noted
  • Diet (24-hour recall), exercise, adherence capacity assessed

Examination Checklist

  • Height, weight, BMI, waist circumference recorded
  • Blood pressure both arms; orthostatic BP measured
  • Capillary blood glucose measured
  • Fundoscopy attempted or referral arranged
  • Oral cavity (Candidiasis) and thyroid examined
  • All peripheral pulses palpated (femoral, popliteal, posterior tibial, dorsalis pedis)
  • Abdomen: liver size, renal angle, injection sites
  • Neurological: monofilament, tuning fork, pin-prick, ankle jerks, proprioception, Romberg's
  • Shoes and socks removed; feet fully inspected
  • MSK: prayer sign, tabletop, Dupuytren's, trigger finger, Tinel's, Phalen's, shoulder ROM
  • Skin: acanthosis nigricans, necrobiosis lipoidica, dermopathy, xanthomas, vitiligo

Investigations Checklist

  • HbA1c requested / result recorded
  • FPG or random BG with result
  • Urine ACR (nephropathy screening)
  • eGFR / creatinine / urea
  • Fasting lipid profile
  • FBC (rules out anaemia affecting HbA1c)
  • LFTs (NAFLD screening, medication monitoring)
  • TSH
  • B12 (on metformin)
  • ECG
  • Anti-GAD / C-peptide (if type classification unclear)
  • Dilated fundoscopy referral made if not done within past year

SECTION 17: CLINICAL ASSESSMENT & SIGNATURE

FieldEntry
Date / Time of Clerking
Medical Student / Clerk Name
Signature
Supervising Doctor Name
Supervisor Signature
Provisional Diagnosis (Student)
Confirmed Diagnosis (Supervisor)
HbA1c Target Set
Next Review Date

Sources: Goldman-Cecil Medicine (26th ed.) | Harrison's Principles of Internal Medicine (22nd ed.) | Tintinalli's Emergency Medicine (9th ed.) | Firestein & Kelley's Textbook of Rheumatology (11th ed.) | ADA Standards of Medical Care in Diabetes 2024
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