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COMPREHENSIVE HISTORY-TAKING MEDICAL RECORD
Diabetes Mellitus & Associated Diseases
Based on: Goldman-Cecil Medicine | Harrison's 22e | Tintinalli's Emergency Medicine | Firestein & Kelley's Rheumatology
Patient: ______________________________ Date: ______________ Hospital No: ______________
Ward/Clinic: ______________ Clerk/Student: ______________________________ Supervisor: ______________________________
SECTION 1: BIODATA
Always record biodata FIRST before asking any history. It anchors the clinical picture.
Field Patient Entry Full Name Age Sex Male / Female / Other Date of Birth Occupation Marital Status Single / Married / Divorced / Widowed Address / Living Situation Religion / Ethnicity Date of Admission / Consultation Referral Source Self / GP / Specialist / Emergency Informant Reliability of History Good / Fair / Poor — Reason: __________
CLINICAL NOTE — Occupation matters in DM: Shift workers have disrupted glycaemic patterns; sedentary workers have worse insulin resistance; professional drivers face hypoglycaemia risk (licensing implications).
SECTION 2: CHIEF COMPLAINT (CC)
Ask in the patient's own words — do not suggest answers or use medical terminology.
Opening questions:
"What brought you here today?"
"What is the main problem that is bothering you?"
Write verbatim — record exactly one or two main complaints with duration:
CC: ____________________________________________________________________________
Duration: ______________________________________________________________________
SECTION 3: HISTORY OF PRESENTING ILLNESS (HPI)
Use SOCRATES for EACH complaint. For DM, always probe for hyperglycaemic symptoms, acute decompensation, and complication clues.
3A. SOCRATES Framework
Letter Stands For Question to Ask S Site "Where exactly is the problem?" O Onset "When did it start? Was it sudden (hours–days) or gradual (weeks–months)?" C Character "What does it feel like? Burning? Tingling? Pressure?" R Radiation "Does it spread or go anywhere else?" A Associated Symptoms "Is there anything else that comes with it?" T Time Course "Is it constant or does it come and go? Is it getting better or worse?" E Exacerbating / Relieving "What makes it better? What makes it worse?" S Severity "On a scale of 0–10, how bad is it? Does it affect daily life?"
3B. Cardinal Symptoms of Hyperglycaemia — Ask ALL
Polyuria (Excessive Urination)
Mechanism: Osmotic diuresis from glucosuria when plasma glucose exceeds renal threshold (~10 mmol/L / 180 mg/dL)
"How many times do you urinate during the day?"
"Do you wake up at night to urinate? How many times?" (Nocturia)
"How much urine do you pass each time — a small amount or a lot?"
"Is your urine pale, dark, or does it look foamy?"
"Have you noticed ants being attracted to where you urinate?" (Glycosuria — classical bedside clue)
Findings: _____________________________________________________________________
Polydipsia (Excessive Thirst)
Mechanism: Compensatory response to dehydration and hyperosmolarity from glucosuria
"Do you feel very thirsty more than usual?"
"How much water or fluids do you drink in a day?"
"Is the thirst there all the time, or does drinking relieve it only briefly?"
Findings: _____________________________________________________________________
Polyphagia (Excessive Hunger)
Mechanism: Cellular starvation despite hyperglycaemia — especially prominent in Type 1 DM
"Is your appetite increased, decreased, or normal?"
"Do you feel hungry soon after eating a full meal?"
Findings: _____________________________________________________________________
Weight Changes
Pattern Mechanism T1DM — Weight loss Catabolism of fat and muscle T2DM — Often weight gain Obesity-driven; weight loss only in late uncontrolled T2DM
"Have you lost or gained weight recently without trying?"
"How much weight have you lost/gained, and over what period of time?"
Findings: _____________________________________________________________________
Fatigue and Weakness
"Do you feel tired all the time?"
"Does the tiredness come on even without activity, or only after effort?"
"Do you feel weak in your arms or legs?"
Findings: _____________________________________________________________________
Blurred Vision
Mechanism: Lens osmotic swelling from sorbitol accumulation during hyperglycaemia
"Has your vision changed recently?"
"Is the blurring in one eye or both?"
"Is it constant or does it fluctuate with your blood sugar levels?"
"Have you seen floaters, flashes of light, or had sudden loss of vision?"
Findings: _____________________________________________________________________
Recurrent Infections and Poor Wound Healing
Mechanism: Phagocyte dysfunction + glucosuria + impaired vascular and neuropathic tissue repair
"Do you get infections frequently — skin, genital, or urinary?"
"Have you noticed itching around the genitals or white discharge?" (Candidiasis)
"Do wounds, cuts, or sores take a long time to heal?"
"Do you have any sores or ulcers not healing, especially on the feet?"
Findings: _____________________________________________________________________
3C. Onset and Type Classification Clues
Onset Pattern Clinical Implication Acute onset (days–weeks) Suggests Type 1 DM / DKA → ask about nausea, vomiting, abdominal pain, fruity breath Insidious onset (months–years) Suggests Type 2 DM → often found incidentally or when complication presents first During pregnancy Gestational diabetes → screen with OGTT Post-illness / surgery / steroids Secondary / drug-induced DM → review precipitants
3D. Acute Decompensation — Screen for ALL Three
CRITICAL ALERT — Always ask these questions directly. Missing DKA or HHS is a clinical emergency.
Emergency Symptoms to Screen DKA (T1DM, occasionally T2DM)Nausea, vomiting, abdominal pain, fruity/acetone breath odour, rapid breathing (Kussmaul), decreased consciousness HHS — Hyperosmolar Hyperglycaemic State (T2DM)Extreme thirst, confusion, neurological changes (focal deficits, seizures), profoundly elevated blood sugar (>33 mmol/L), marked dehydration, NO significant acidosis/ketonuria Hypoglycaemia Sweating, palpitations, tremor, confusion, aggression, unconsciousness — especially on insulin or sulfonylurea
"Have you had nausea, vomiting, or abdominal pain with very high blood sugar?"
"Have you had episodes of sweating, shakiness, heart racing, or confusion — especially if you missed a meal?"
"Have you ever been hospitalised for very high blood sugar or a diabetic coma?"
SECTION 4: COMPLICATIONS SCREENING — SYSTEMATIC REVIEW BY ORGAN
Tell the patient: "I am now going to ask about different parts of your body to make sure everything is being checked carefully."
4A. EYES — Diabetic Retinopathy
"Has a doctor ever looked at the back of your eyes with a special instrument?" (Fundoscopy / dilated eye exam)
"Have you had any changes in your vision — blurring, dark spots, or sudden loss of sight?"
"Have you seen floaters, cobweb-like shapes, or flashes of light?"
"Have you had any eye injections (anti-VEGF) or laser treatment for your eyes?"
"Have you ever been told you have diabetic eye disease or glaucoma?"
Eyes findings: ________________________________________________________________
4B. KIDNEYS — Diabetic Nephropathy
"Have you noticed your urine becoming foamy or frothy?" (Proteinuria)
"Do you have swelling in your legs, ankles, or around your eyes in the morning?" (Nephrotic oedema)
"Have you ever been told your kidneys are not working properly?"
"Do you know your kidney function test results — creatinine or eGFR?"
"Have you ever been on dialysis or been referred to a kidney specialist?"
Kidney findings: ______________________________________________________________
4C. PERIPHERAL NERVOUS SYSTEM — Sensorimotor Neuropathy
"Do you have numbness, tingling, or a burning sensation in your feet or hands?"
"Does it feel like you are walking on cotton wool or sand?"
"Is the pain or tingling worse at night?"
"Have you lost feeling in your feet — for example, unable to feel hot or cold water?"
"Have you had falls because of loss of balance or unsteadiness?"
"Do you have any foot ulcers, wounds, or sores that are not healing?"
"Have you ever had an amputation?"
Neuropathy findings: __________________________________________________________
4D. AUTONOMIC NERVOUS SYSTEM — Autonomic Neuropathy
"Do you feel dizzy or lightheaded when you stand up quickly?" (Postural / orthostatic hypotension)
"Do you feel full very quickly after eating only a small meal?" (Gastroparesis)
"Do you have nausea, vomiting, or bloating after eating?"
"Do you have problems with your bowels — constipation or diarrhoea that comes and goes?" (Autonomic gut dysmotility)
"Do you sweat abnormally — too much or not at all?"
"Do you have difficulty controlling your bladder or do you need to strain to pass urine?" (Neurogenic bladder)
"[Male patients — ask sensitively and privately]: Do you have difficulty getting or maintaining an erection?" (Erectile dysfunction — earliest autonomic symptom in men)
Autonomic findings: ___________________________________________________________
4E. HEART AND LARGE BLOOD VESSELS — Macrovascular Disease
NOTE: Diabetic patients may have SILENT myocardial infarction — chest pain may be absent due to cardiac autonomic neuropathy. Always ask about atypical symptoms.
"Do you have chest pain or tightness — especially on walking, climbing stairs, or at rest?"
"Do you feel short of breath with activity or when lying flat?" (Orthopnoea — heart failure)
"Do you have swelling in both ankles?" (Biventricular failure)
"Have you had a heart attack or been told your heart arteries are blocked?"
"Have you had any stroke or sudden weakness / numbness on one side of your body?"
"Do you get pain in your calves when walking that goes away with rest?" (Intermittent claudication = Peripheral Arterial Disease)
"Do you have palpitations or feel your heart racing or skipping?"
Cardiovascular findings: _______________________________________________________
4F. FEET — Diabetic Foot Disease
"Do you check your feet every day?"
"What type of footwear do you use? Do they fit well?"
"Do you have any calluses, corns, or hard skin on your feet?"
"Have you had any cuts, blisters, or sores on your feet that you did not feel?" (Loss of protective sensation)
"Have you been told your foot bones have changed shape?" (Charcot's arthropathy)
Foot findings: ________________________________________________________________
4G. MUSCULOSKELETAL — Diabetic Rheumatological Conditions
From Firestein & Kelley's Rheumatology: Diabetic cheiroarthropathy, Dupuytren's, trigger finger, carpal tunnel, adhesive capsulitis, and Charcot arthropathy are all strongly DM-associated.
Condition Key Features Cheiroarthropathy (Limited Joint Mobility) Prayer sign — cannot fully oppose palmar surfaces; Tabletop sign Dupuytren's Contracture >20% in T2DM; palm nodules/cords; ring and little finger fixed flexion Trigger Finger (Flexor Tenosynovitis) A1 pulley nodule; snapping/locking; multiple fingers in DM Carpal Tunnel Syndrome Median nerve; Tinel's, Phalen's, Durkan's tests; thenar wasting Adhesive Capsulitis (Frozen Shoulder) Limited shoulder ROM; insidious onset; bilateral in DM Charcot Arthropathy (Neuroarthropathy) Painless swollen warm foot/ankle; bone destruction on X-ray Diabetic Amyotrophy Severe unilateral thigh pain, weakness, quadriceps wasting; L2–L4
"Do you have stiffness in your fingers or cannot fully close/open your hands?"
"Do you have difficulty getting your palm flat on a table?"
"Do any of your fingers catch, snap, or lock when you bend them?"
"Do you have pain or tingling in your hands at night?" (Carpal Tunnel)
"Do you have pain or stiffness in your shoulder that limits lifting your arm?"
MSK findings: _________________________________________________________________
4H. SKIN
"Do you have dry, itchy, or discoloured skin?"
"Do you have any non-healing wounds or sores?"
"Have you noticed darker velvety skin at the back of your neck or in the armpits?" (Acanthosis nigricans — insulin resistance)
Skin findings: ________________________________________________________________
SECTION 5: GLYCAEMIC CONTROL HISTORY
This section establishes current control, monitoring practice, and hypoglycaemia risk — essential for every DM encounter.
"When were you first told you have diabetes? Who made the diagnosis?"
"What type of diabetes do you have — Type 1, Type 2, or were you not told?"
"What was your most recent HbA1c result? When was it done? Is it improving or worsening?"
"Do you check your blood sugar at home with a glucometer? How often?"
"What are your typical fasting blood sugar readings in the morning?"
"What are your blood sugar readings after meals?"
"Do you use a continuous glucose monitor (CGM)? What is your time-in-range?"
Hypoglycaemia Assessment
"Have you had episodes of low blood sugar (hypoglycaemia)?"
"How often do these episodes happen?"
"What are your warning signs — sweating, shaking, heart racing, confusion?"
"Have you lost your warning signs for low blood sugar?" (Hypoglycaemia unawareness — HIGH RISK)
"Have you ever needed help from another person, or called emergency services?"
Parameter Result / Record HbA1c Last Result & Date Fasting BG Range (mmol/L) Post-meal BG Range (mmol/L) Hypoglycaemia Frequency Never / Occasional / Weekly / Daily Hypoglycaemia Unawareness Yes / No Severe Hypoglycaemia (needed help) Yes / No — Date: _________ DKA / HHS Hospital Admissions Number: ___ Last date: _________ Self-Monitoring Practice Yes / No — Frequency: ___________ CGM Use Yes / No — Device: _______________
SECTION 6: PAST MEDICAL HISTORY (PMH)
Ask systematically — these comorbidities are both DM complications AND DM accelerators.
Condition Status / Details Hypertension Yes / No — Since: ___ Treated: Yes / No Dyslipidaemia (High Cholesterol / Triglycerides) Yes / No Coronary Artery Disease / Angina / MI Yes / No — Date: _______ Congestive Heart Failure Yes / No — NYHA Class: ___ Stroke or TIA Yes / No — Date: _______ Side: L / R Peripheral Arterial Disease Yes / No — ABI known: ___ Diabetic Retinopathy Yes / No — Grade: NPDR / PDR / Maculopathy Diabetic Nephropathy / CKD Yes / No — eGFR: ___ ACR: ___ Stage: ___ Diabetic Neuropathy (peripheral / autonomic) Yes / No — Type: ______________ Diabetic Foot Ulcer / Amputation Yes / No — Level: ________________ Charcot Arthropathy Yes / No Non-alcoholic Fatty Liver Disease (NAFLD/NASH) Yes / No Sleep Apnoea Yes / No — On CPAP: Yes / No Polycystic Ovary Syndrome (PCOS) Yes / No (women) Gestational Diabetes Yes / No (women) — Year: ___ Pancreatitis or Pancreatic Disease Yes / No Thyroid Disease Yes / No — Type: ________________ Other Autoimmune Conditions (T1DM) Coeliac / Adrenal insufficiency / Vitiligo / Other Previous DKA / HHS Episodes Yes / No — Dates / Number: __________ Previous Surgeries Psychiatric Conditions Depression / Anxiety / Eating disorder
SECTION 7: DRUG HISTORY & ALLERGIES
For each drug: Name → Dose → Frequency → Route → Compliance → Duration.
7A. Diabetes Medications
Drug Class Details Metformin Dose: ___ Freq: ___ Compliance: Good / Poor Sulfonylurea (gliclazide, glibenclamide, glipizide) Dose: ___ Freq: ___ Compliance: Good / Poor SGLT2 Inhibitor (empagliflozin, dapagliflozin, canagliflozin) Dose: ___ Freq: ___ Compliance: Good / Poor GLP-1 Receptor Agonist (semaglutide, liraglutide, dulaglutide) Dose: ___ Freq: ___ Route: SC / Oral DPP-4 Inhibitor (sitagliptin, linagliptin, saxagliptin) Dose: ___ Freq: ___ Compliance: Good / Poor Thiazolidinedione (pioglitazone) Dose: ___ Freq: ___ Insulin — Basal (glargine / detemir / degludec) Dose: ___ Timing: ___ Injection site: ___ Insulin — Rapid-Acting (aspart / lispro / glulisine) Dose: ___ Timing: ___ Technique: ___ Insulin — Premixed Type: ___ Dose: ___ Timing: ___ Insulin Pump (CSII) Yes / No — Basal rate: ___ Bolus: ___
"Do you take your diabetes medications every day? Do you ever miss doses?"
"For insulin: Do you rotate injection sites? Are there any lumpy areas?" (Lipohypertrophy)
7B. Other Medications (Comorbidities)
Drug Name / Dose / Frequency ACE Inhibitor / ARB (renoprotective) Statin (atorvastatin, rosuvastatin) Aspirin or Antiplatelet Antihypertensive (amlodipine, bisoprolol, doxazosin) Diuretic (furosemide, spironolactone, HCTZ)
7C. Drugs That WORSEN Glycaemic Control
Always check for these — they are a common and frequently overlooked cause of poor glycaemic control.
Drug Status Corticosteroids (prednisolone, dexamethasone) Currently taking: Yes / No Thiazide Diuretics (hydrochlorothiazide, indapamide) Currently taking: Yes / No Atypical Antipsychotics (olanzapine, clozapine) Currently taking: Yes / No Beta-Blockers (propranolol, atenolol) Currently taking: Yes / No — masks hypoglycaemia symptoms Calcineurin Inhibitors (tacrolimus, cyclosporin) Currently taking: Yes / No Protease Inhibitors (HIV treatment) Currently taking: Yes / No Niacin / Nicotinic acid Currently taking: Yes / No Herbal / Traditional Medicines Details: __________________________
7D. Allergies
Drug Name Reaction (e.g. rash, anaphylaxis, GI intolerance)
SECTION 8: FAMILY HISTORY (FH)
A strong family history of T2DM confers 2–4× increased risk. First-degree relatives with T1DM increase risk ~15-fold.
Family Condition Details Diabetes (Type 1 or 2) — first-degree relatives Yes / No — Who: Father / Mother / Sibling — Type: ___ Hypertension Yes / No — Who: ______________________ Coronary Artery Disease / Early MI (<55 years) Yes / No — Who: ______________________ Stroke Yes / No — Who: ______________________ Chronic Kidney Disease Yes / No — Who: ______________________ Obesity Yes / No — Who: ______________________ Thyroid Disease Yes / No — Who: ______________________ MODY (DM in 3 generations, young, non-obese, no antibodies) Yes / No — Suggest genetic testing if suspected Autoimmune Conditions Yes / No — Type: ____________________
SECTION 9: SOCIAL HISTORY (SH)
9A. Smoking
"Do you smoke or have you ever smoked?"
"How many cigarettes per day? For how many years?"
Pack-years = (cigarettes/day ÷ 20) × years smoked
"If stopped — when did you stop?"
Parameter Details Smoking Status Never / Current / Ex-smoker Cigarettes/day Duration (years) Pack-years Year stopped (if ex)
Smoking DOUBLES cardiovascular risk in DM. It also impairs wound healing, worsens neuropathy, and accelerates nephropathy. Smoking cessation is a core DM management goal.
9B. Alcohol
"Do you drink alcohol? What type? How much per week?"
"Do you ever drink on an empty stomach?"
Alcohol can mask hypoglycaemia symptoms AND cause delayed hypoglycaemia up to 24 hours after drinking — especially on insulin or sulfonylurea. Educate all patients.
Parameter Details Alcohol Status Never / Social / Regular / Dependent Units per week Type (beer, spirits, wine) Binge drinking (>6 units/session) Yes / No
9C. Diet
"What do you usually eat in a day from morning to night?" (24-hour dietary recall)
"Do you eat regular meals, or do you skip meals?"
"How much rice, bread, sugar, or sweet drinks do you consume daily?"
"Are you following a special diabetic diet?"
"Do you count carbohydrates or follow a meal plan?"
Parameter Details Meal Regularity Regular / Irregular / Skips meals Carbohydrate Intake High / Moderate / Low / Unknown Sweet Drinks (soda, juice, energy drinks) Frequency: ___________________ Diabetic Diet Adherence Yes / No / Partial
9D. Physical Activity
"How active are you on a daily basis?"
"Do you do any structured exercise? What type, how often, and for how long?"
Target: 150 min/week moderate aerobic exercise + resistance training 2–3×/week. Exercise reduces HbA1c by ~0.6% independently.
Parameter Details Activity Level Sedentary / Lightly active / Moderately active / Very active Exercise Type Exercise Frequency / Duration
9E. Socioeconomic Status & Adherence Capacity
"Do you have any difficulty affording your medications or test strips?"
"Who cooks your meals at home?"
"Do you live alone or with family?"
"Do you understand how to use your glucometer and insulin device?"
Parameter Details Lives Alone / With Family Medication Affordability No difficulty / Moderate / Significant difficulty Health Literacy Good / Fair / Poor Occupational Risk for Hypoglycaemia Driving / Machinery / Heights: Yes / No
SECTION 10: DIABETES CLASSIFICATION — CLINICIAN ASSESSMENT
Type Key Features Type 1 DM Usually <30 yrs, lean, acute onset, prone to DKA, autoimmune markers (anti-GAD, anti-islet, anti-IA2, anti-ZnT8), C-peptide low/undetectable, requires insulin from outset Type 2 DM Usually >35 yrs, overweight/obese, insidious onset, strong family history, associated with metabolic syndrome, C-peptide elevated MODY Young, non-obese, autosomal dominant FH over 3 generations, no autoantibodies — refer for genetic testing (GCK, HNF1A mutations) Secondary DM Pancreatitis, pancreatectomy, haemochromatosis, Cushing's syndrome, acromegaly, drug-induced — underlying cause drives management Gestational DM Diagnosed during pregnancy, resolves post-partum — but 50% develop T2DM within 10 years; screen with OGTT LADA Age >30, initially resembles T2DM, anti-GAD positive, progressive insulin deficiency — often misclassified as T2DM
Clinical DM Classification: _____________________________________________________
SECTION 11: PHYSICAL EXAMINATION — GUIDE & FINDINGS
11A. General Assessment
Parameter Finding Height (cm) / Weight (kg) / BMI ___ / ___ / ___ (Overweight: >25; Obese: >30) Waist Circumference (cm) ___ (At risk: women >80 cm; men >90 cm Asian / >102 cm Western) General Appearance Well / Unwell / Ill — Cushingoid features: Yes / No Hydration Status Normal / Dehydrated — Skin turgor / dry mucous membranes Acanthosis Nigricans Present / Absent — Site: posterior neck / axillae / groin
11B. Vital Signs
Parameter Finding Blood Pressure (Right arm) / mmHg — Target <130/80 in DMBlood Pressure (Left arm) / mmHgOrthostatic BP Supine: / | Standing 1 min: / | Standing 3 min: / — Drop ≥20 mmHg systolic = autonomic neuropathy Heart Rate ___ bpm — Regular / Irregular — Fixed resting tachycardia = autonomic neuropathy Respiratory Rate ___ /min — Kussmaul breathing (deep sighing) = DKA Temperature ___ °C — Fever suggests infection (foot cellulitis / osteomyelitis) SpO2 ___ % Random Capillary Blood Glucose ___ mmol/L
11C. Eyes, ENT, Oral Cavity
Examination Finding Visual Acuity (Snellen) Right: ___ Left: ___ Rubeosis Iridis Present / Absent (neovascularisation of iris = advanced retinopathy) Fundoscopy Microaneurysms / Haemorrhages / Cotton-wool spots / Hard exudates / Neovascularisation / Normal CN III Palsy Present / Absent (painless, pupil-sparing = diabetic mononeuropathy) Oral Candidiasis Present / Absent — White plaques on mucosa Periodontal Disease Present / Absent Thyroid Normal / Goitre / Nodule
11D. Cardiovascular
Examination Finding Carotid Bruits Right: Present / Absent | Left: Present / Absent Apex Beat Heart Sounds S1+S2+___ (S3 = Heart Failure; S4 = Diabetic cardiomyopathy) Pedal Oedema Present / Absent — Grade: ___ Bilateral / Unilateral Femoral Pulse Right: Present / Absent | Left: Present / Absent Popliteal Pulse Right: Present / Absent | Left: Present / Absent Posterior Tibial Pulse Right: Present / Absent | Left: Present / Absent Dorsalis Pedis Pulse Right: Present / Absent | Left: Present / Absent Ankle-Brachial Index (ABI) Right: ___ Left: ___ (Normal ≥0.9; PAD <0.9)
11E. Abdomen
Examination Finding Hepatomegaly (NAFLD) Present / Absent — Size: ___ cm below costal margin Renal Angle Tenderness Right: Yes / No | Left: Yes / No Insulin Injection Sites Abdomen / Flanks / Thighs — Lipohypertrophy: Yes / No — Site: ___ Renal Artery Bruit Present / Absent
11F. Neurological — Peripheral Neuropathy Screening
Diabetic neuropathy is a diagnosis of exclusion — always rule out B12 deficiency, hypothyroidism, uraemia, CIDP, and vasculitic neuropathy.
Test Finding 10g Semmes-Weinstein Monofilament (10 plantar sites per foot) Right: ___/10 normal | Left: ___/10 normal 128-Hz Tuning Fork (hallux → medial malleolus) Right: Normal / Reduced / Absent | Left: Normal / Reduced / Absent Pin-prick (dorsal foot — small fibre) Right: Normal / Reduced | Left: Normal / Reduced Temperature Discrimination (cool vs warm) Right: Normal / Impaired | Left: Normal / Impaired Ankle Jerk Reflex Right: Present / Absent / Diminished | Left: Present / Absent / Diminished Patellar Reflex Right: Present / Absent | Left: Present / Absent Proprioception (hallux up/down with eyes closed) Right: Normal / Impaired | Left: Normal / Impaired Romberg's Test Positive / Negative Gait Assessment Normal / Wide-based / Antalgic
11G. Foot Examination — 5-Minute Minimum
ALWAYS remove shoes and socks. Check the feet at EVERY DM visit. Foot disease is the most preventable major complication of DM.
Feature Finding — Right Foot / Left Foot Skin Integrity Ulcers: Yes / No — Location: ___ Size: ___ Depth: ___ Wagner grade: ___ Calluses / Corns Present / Absent — Location: _______________ Deformities Hallux valgus / Hammertoe / Charcot foot / Normal Interdigital Spaces Maceration / Tinea pedis / Normal Nails Onychomycosis / Ingrowing / Normal Skin Colour / Temperature Warm / Cool / Discoloured Callus Over Pressure Points Present / Absent Footwear Inspection Appropriate / Ill-fitting / Absent
11H. Musculoskeletal — Diabetic-Specific Signs
Sign How to Perform Positive Finding Prayer Sign (Cheiroarthropathy)Both palms pressed together, wrists dorsiflexed, hold 5 seconds Gap remains between palmar surfaces — cannot fully oppose Tabletop Sign Palm flat on table, fingers extended Fingers remain partially flexed — visible gap Dupuytren's Contracture Palpate entire palmar fascia; palpate for nodules, cords; passive extension test Palpable cord + fixed flexion deformity — ring and little fingers Trigger Finger Palpate A1 pulley at palmar crease; ask patient to close and open fist Catch, snap, or locking during flexion/extension Tinel's Sign Tap over carpal tunnel at palmar wrist crease 2–3 times Electric shock/tingling into thumb, index, middle fingers Phalen's Test Both wrists in maximum passive flexion for 60 seconds Numbness/tingling in median nerve distribution within 60 seconds Durkan's Compression Firm thumb pressure over carpal tunnel for 30 seconds Paraesthesia in median nerve distribution Shoulder Abduction ROM Active and passive range Normal >180°; reduced = frozen shoulder (adhesive capsulitis) Charcot Foot Inspect and palpate mid-foot and ankle Warm, swollen, deformed, painless foot — bone destruction on X-ray
Examination Finding Result Prayer Sign Positive / Negative Tabletop Sign Positive / Negative Dupuytren's Contracture Present / Absent — Fingers: ___ Flexion deficit: ___ degrees Trigger Finger Present / Absent — Fingers affected: ___ Tinel's Sign at wrist Positive / Negative — Right / Left Phalen's Test Positive / Negative — Latency: ___ seconds Thenar Wasting (advanced CTS) Present / Absent Shoulder Abduction Range Right: ___° Left: ___° Charcot Foot Present / Absent
11I. Skin Examination
Skin Finding Result Acanthosis Nigricans Present / Absent — Location: _______________ Necrobiosis Lipoidica Present / Absent (anterior tibiae — strongly associated with T1DM) Diabetic Dermopathy (shin spots) Present / Absent — most common cutaneous finding; marker of duration Eruptive Xanthomas Present / Absent (severe hypertriglyceridaemia) Tinea Pedis / Onychomycosis Present / Absent Lipohypertrophy at Injection Sites Present / Absent — Site: _______________ Lipoatrophy (rare) Present / Absent Vitiligo Present / Absent (T1DM autoimmune marker)
SECTION 12: INVESTIGATIONS — RESULTS RECORD
ADA Diagnostic Criteria — Any ONE of the following:
FPG ≥7.0 mmol/L
2-hr OGTT ≥11.1 mmol/L
Random PG ≥11.1 mmol/L + symptoms
HbA1c ≥48 mmol/mol (≥6.5%)
Asymptomatic patients require TWO abnormal tests on DIFFERENT days to confirm diagnosis.
12A. Glycaemic
Test Result / Reference Range Fasting Plasma Glucose (FPG) ___ mmol/L (DM: ≥7.0 | Pre-DM: 5.6–6.9 | Normal: <5.6) 2-hour OGTT (75g glucose) ___ mmol/L (DM: ≥11.1 | IGT: 7.8–11.0) HbA1c ___ mmol/mol (___ %) (DM: ≥48/≥6.5% | Pre-DM: 39–47/5.7–6.4%) Random Plasma Glucose + Symptoms ___ mmol/L (DM: ≥11.1 with symptoms) C-Peptide ___ pmol/L (T1DM: low/undetectable; T2DM: normal/high) Fasting Insulin ___ mU/L (elevated with normal/high FPG = insulin resistance) HOMA-IR ___ (insulin resistance index)
12B. Autoimmune (Type 1 / LADA Classification)
Test Result Anti-GAD Antibodies Positive / Negative / Not done Islet Cell Antibodies (ICA) Positive / Negative / Not done Anti-IA2 Antibodies Positive / Negative / Not done Anti-ZnT8 Antibodies Positive / Negative / Not done
12C. Metabolic Panel
Test Result Full Blood Count (FBC) Hb: ___ WBC: ___ Platelets: ___ (Anaemia affects HbA1c interpretation) Urea / BUN ___ mmol/L Creatinine ___ micromol/L eGFR (CKD-EPI) ___ mL/min/1.73m² (CKD Stage: _____) Urine Albumin-to-Creatinine Ratio (ACR) ___ mg/mmol (Microalbuminuria: 3–30; Macroalbuminuria: >30) Urine Dipstick Glucose: ___ Protein: ___ Ketones: ___ Nitrites: ___ Blood: ___ Total Cholesterol ___ mmol/L LDL Cholesterol ___ mmol/L (Target <1.8 mmol/L if high CV risk) HDL Cholesterol ___ mmol/L Triglycerides ___ mmol/L (>5.6 = hypertriglyceridaemia with pancreatitis risk) Liver Function Tests (ALT, AST, ALP, GGT) ALT: ___ AST: ___ ALP: ___ GGT: ___ (NAFLD monitoring) TSH / Free T4 TSH: ___ FT4: ___ (Annual in T1DM; consider in T2DM) Uric Acid ___ mmol/L (elevated in insulin resistance / metabolic syndrome) Serum B12 ___ pmol/L (deficiency from long-term metformin)
12D. Cardiovascular
Test Result 12-Lead ECG Normal / LVH / Q waves (silent MI) / AF / Conduction defect: ___ Echocardiogram EF: ___ Diastolic dysfunction: Yes / No (if clinical HF) Ankle-Brachial Index (ABI) Right: ___ Left: ___ (PAD <0.9) Chest X-Ray Normal / Cardiomegaly / Pulmonary oedema / Other: ___
12E. Ophthalmology
Screening Result Dilated Fundus Exam / Retinal Photography Date: ___ Finding: NPDR / PDR / Maculopathy / Normal Frequency At diagnosis (T2DM) | Within 5 years (T1DM) | Then annually
12F. Coexisting Autoimmune (T1DM Screening)
Test Result Anti-TPO and Anti-Thyroglobulin Antibodies Positive / Negative / Not done Anti-tTG IgA (Coeliac Disease) Positive / Negative / Not done Adrenal Antibodies (21-hydroxylase) if Addison's suspected Positive / Negative / Not done
SECTION 13: RISK STRATIFICATION SUMMARY
Risk Factor Patient Status Clinical Implication Disease Duration (years) >10 years = significantly increased microvascular risk HbA1c >75 mmol/mol (>9%) = very high risk Blood Pressure Uncontrolled BP accelerates nephropathy and retinopathy Dyslipidaemia Drives macrovascular disease Smoking Doubles CVD risk; impairs wound healing Obesity Worsens insulin resistance and CV risk Existing Organ Damage (retinopathy / nephropathy / neuropathy) Already present = highest risk tier Hypoglycaemia Unawareness Relax targets; refer to specialist Pregnancy Tight control; target HbA1c <48 mmol/mol pre-conceptionally Established Cardiovascular Disease SGLT2i or GLP-1 RA preferred; high-intensity statin; antiplatelet
Overall Risk Stratification: ____________________________________________________
HbA1c Target for this patient: _________________________________________________
SECTION 14: ACTIVE PROBLEM LIST
SECTION 15: MANAGEMENT PLAN FRAMEWORK
15A. Patient Education
15B. Lifestyle Modification
15C. Pharmacotherapy — T2DM Step-Up Approach
Step Drug Key Points 1st Line Metformin Reduces hepatic glucose output; weight-neutral; renally dosed (reduce if eGFR 30–45; stop if <30) Add-on (CVD/CKD) SGLT2 Inhibitor (empagliflozin, dapagliflozin)Renal and cardioprotective; reduce HbA1c, BP, weight; risk of genital mycotic infections Add-on (CVD/Obesity) GLP-1 RA (semaglutide, liraglutide)Weight loss; CV benefit (LEADER trial, SUSTAIN-6); injectable or oral Add-on DPP-4 Inhibitor (sitagliptin)Weight-neutral; well-tolerated; oral Add-on Sulfonylurea (gliclazide)Inexpensive; hypoglycaemia risk; weight gain Escalation Basal Insulin (glargine / detemir)When oral agents fail; start 0.1–0.2 U/kg/day Escalation Basal-Bolus Insulin Most physiological; essential in T1DM from outset
T1DM: Insulin is mandatory from diagnosis. Use basal insulin (glargine/detemir/degludec) + rapid-acting at mealtimes (aspart/lispro/glulisine). Consider SGLT2i or GLP-1 RA as adjuncts in poorly controlled T1DM.
15D. Treating Comorbidities
Comorbidity Management Hypertension ACE inhibitor or ARB as first choice (renoprotective + anti-proteinuric); target <130/80 with CKD/CVD Dyslipidaemia High-intensity statin for most DM patients with CVD risk; fenofibrate if severe hypertriglyceridaemia Antiplatelet Therapy Low-dose aspirin for ESTABLISHED CVD; not routine for primary prevention Obesity GLP-1 RA (semaglutide 2.4 mg); consider bariatric surgery if BMI >35 with inadequate control
15E. Complication-Specific Treatment
Complication Key Treatment Retinopathy (NPDR) Tight glycaemic + BP control; annual ophthalmology follow-up Retinopathy (PDR) Laser photocoagulation or anti-VEGF (ranibizumab) Nephropathy (microalbuminuria) ACEi or ARB; SGLT2i reduces CKD progression (CREDENCE, DAPA-CKD trials); protein restriction Painful Neuropathy Duloxetine (1st line); pregabalin; gabapentin; amitriptyline; topical capsaicin Gastroparesis Metoclopramide; domperidone; small frequent meals; low-fat diet Erectile Dysfunction PDE5 inhibitors (sildenafil, tadalafil); refer to urology Foot Ulcer Offloading (total contact cast); wound debridement; culture-guided antibiotics; vascular surgery if PAD Charcot Foot (active) Total contact casting; no weight-bearing; urgent orthopaedic / diabetic foot team referral Cheiroarthropathy / Trigger Finger Physiotherapy; corticosteroid injection (less effective in DM); surgical release if severe
15F. Follow-Up Schedule
Parameter Frequency HbA1c Every 3 months if poorly controlled; every 6 months if stable BP, Weight, Waist Circumference Every clinic visit Urine ACR + eGFR Annually Fasting Lipid Profile Annually Dilated Fundoscopy Annually after initial screen Comprehensive Foot Exam Annually; every visit if high-risk foot Thyroid Function (TSH) Annually (T1DM); as clinically indicated (T2DM) B12 Level (on metformin) Every 2 years; annually if low intake or symptoms Dental Review Every 6–12 months (periodontal disease worsens glycaemic control) Influenza Vaccine Annually Pneumococcal Vaccine As per local guidelines
SECTION 16: BEDSIDE QUICK-REFERENCE CHECKLIST
History Checklist
Biodata complete (name, age, sex, occupation, address, informant)
Chief complaint in patient's own words with duration
SOCRATES applied to each complaint
Polyuria, polydipsia, polyphagia, weight loss asked
Blurred vision, recurrent infections, poor wound healing asked
DKA symptoms (nausea/vomiting/fruity breath) screened
HHS symptoms (confusion/extreme thirst/very high BG) screened
Hypoglycaemia episodes and unawareness assessed
Eyes complications screened (retinopathy, laser treatment)
Kidney complications screened (foamy urine, oedema, eGFR)
Peripheral neuropathy screened (numbness, burning, cotton-wool sensation)
Autonomic neuropathy screened (postural dizziness, gastroparesis, ED, neurogenic bladder)
Cardiovascular history (chest pain, dyspnoea, claudication, palpitations)
Foot history (ulcers, amputation, footwear)
MSK history (stiff fingers, trigger finger, shoulder pain)
Glycaemic control history (HbA1c trend, SMBG, hypoglycaemia)
Past medical history (HTN, dyslipidaemia, CVD, CKD, thyroid, PCOS)
All diabetes medications recorded with dose, frequency, compliance
Drugs that worsen glucose checked (steroids, antipsychotics, thiazides, beta-blockers)
Allergies recorded
Family history (DM, CVD, CKD, obesity, MODY)
Smoking status and pack-years calculated
Alcohol history with hypoglycaemia education noted
Diet (24-hour recall), exercise, adherence capacity assessed
Examination Checklist
Height, weight, BMI, waist circumference recorded
Blood pressure both arms; orthostatic BP measured
Capillary blood glucose measured
Fundoscopy attempted or referral arranged
Oral cavity (Candidiasis) and thyroid examined
All peripheral pulses palpated (femoral, popliteal, posterior tibial, dorsalis pedis)
Abdomen: liver size, renal angle, injection sites
Neurological: monofilament, tuning fork, pin-prick, ankle jerks, proprioception, Romberg's
Shoes and socks removed; feet fully inspected
MSK: prayer sign, tabletop, Dupuytren's, trigger finger, Tinel's, Phalen's, shoulder ROM
Skin: acanthosis nigricans, necrobiosis lipoidica, dermopathy, xanthomas, vitiligo
Investigations Checklist
SECTION 17: CLINICAL ASSESSMENT & SIGNATURE
Field Entry Date / Time of Clerking Medical Student / Clerk Name Signature Supervising Doctor Name Supervisor Signature Provisional Diagnosis (Student) Confirmed Diagnosis (Supervisor) HbA1c Target Set Next Review Date
Sources: Goldman-Cecil Medicine (26th ed.) | Harrison's Principles of Internal Medicine (22nd ed.) | Tintinalli's Emergency Medicine (9th ed.) | Firestein & Kelley's Textbook of Rheumatology (11th ed.) | ADA Standards of Medical Care in Diabetes 2024