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Frozen Shoulder (Adhesive Capsulitis) - SOAP Format Assessment & Management
BACKGROUND
Frozen shoulder (adhesive capsulitis) is an idiopathic condition involving synovial inflammation followed by reactive capsular fibrosis. It affects approximately 2% of the general population, predominantly females aged 40-70, with 70% of patients being women. Associated conditions include diabetes mellitus, thyroid disease, and cardiac disease. - Campbell's Operative Orthopaedics 15th Ed, p.2839
S - SUBJECTIVE
Chief Complaint
- Gradual onset of diffuse shoulder pain and progressive stiffness, usually unilateral
- Often spontaneous onset; patient may recall minor trauma of unknown relevance
History of Present Illness
Obtain:
- Onset & Duration: Typically weeks to months; course lasts 1-2 years (longer in diabetics)
- Pain character: Dull ache at rest, sharp at end-range; classically described as diffuse, difficult to localize
- Aggravating factors: Overhead reach, lying on the affected side, sudden movements, reaching behind the back
- Relieving factors: Rest, heat, analgesics
- Night pain: Common and often chief complaint early in course
- Functional limitations: Difficulty dressing (fastening bra, putting on coat), combing hair, reaching overhead shelves, washing opposite axilla
- Phase-specific symptoms:
- Phase 1 - Painful (Freezing): Severe pain, gradual loss of motion; lasts 2-9 months
- Phase 2 - Stiffening (Frozen): Pain lessens but severe stiffness persists; lasts 4-12 months
- Phase 3 - Thawing: Gradual return of motion; pain diminishes; lasts weeks to months
Past Medical History
- Diabetes mellitus (bilateral disease in 50% of diabetics)
- Thyroid disease, cardiac disease
- Contralateral frozen shoulder (20-30% develop opposite shoulder)
- Prior shoulder injury, surgery, or prolonged immobility
- Depression/anxiety (adversely affect symptoms and function)
Medications
- Current analgesics, NSAIDs, corticosteroids, anti-diabetic drugs
- Previous injection therapy
Social History
- Occupation (overhead work, sedentary desk job - note posture)
- Dominant hand involvement (predictive of better outcome)
- Activity limitations at home and work
Patient Goals
- Pain reduction, return of motion, independence in ADLs
O - OBJECTIVE
Observation / Posture
- Guarding of the affected shoulder; arm held adducted and internally rotated
- Muscle atrophy (deltoid, supraspinatus) in prolonged cases
- Scapular compensatory elevation during arm elevation attempts
Palpation
- Tenderness over the glenohumeral joint, anterior capsule, and coracohumeral ligament
- No redness or warmth (helps differentiate from septic arthritis)
- Palpable stiffness of the shoulder girdle
Range of Motion (Active & Passive)
Loss is global (all planes), both active and passive - this is the hallmark finding:
| Movement | Normal | Typical Frozen Shoulder Finding |
|---|
| Flexion | 180° | Reduced (often <90° in frozen phase) |
| Abduction | 180° | Reduced (often <90°) |
| External Rotation | 60-90° | Most restricted - pathognomonic sign |
| Internal Rotation | 70-90° | Reduced |
| Extension | 60° | Reduced |
Key finding: Global loss of both active AND passive motion, especially external rotation. - Bailey & Love's Surgery 28th Ed, p.552
Strength Testing
- May appear weak due to pain inhibition; true rotator cuff strength relatively preserved
- Supraspinatus (empty can / Jobe's test), infraspinatus, subscapularis testing to rule out cuff tear
Special Tests
| Test | Finding |
|---|
| Painful arc test | Often absent (unlike impingement) |
| Neer's / Hawkins-Kennedy | May be mildly positive due to pain at end-range |
| External rotation restriction | Positive - restricted in both active & passive |
| Glenohumeral rhythm | Reduced GH contribution; increased scapulothoracic compensation |
Outcome Measures
- VAS or NPRS: Pain at rest and with movement (0-10)
- DASH / QuickDASH: Upper limb disability
- ASES (American Shoulder and Elbow Surgeons) Score
- Oxford Shoulder Score
- SPADI (Shoulder Pain and Disability Index)
Imaging (if available)
- X-ray: Normal - used to rule out OA, fracture, calcific tendinitis
- MRI: Not routinely needed; may show capsular thickening, enhancing synovium
- Arthrography: Reduced joint capsule volume (confirms diagnosis but rarely needed clinically) - Goldman-Cecil Medicine, p.2777
A - ASSESSMENT
Diagnosis
Adhesive Capsulitis (Frozen Shoulder) - Right/Left shoulder, Stage ___ (Painful / Stiffening / Thawing)
Clinical Reasoning
- Global restriction of active AND passive GH motion in all planes, with disproportionate loss of external rotation
- Absent signs of infection (no redness, warmth), normal X-ray ruling out OA/fracture
- Associated risk factors (e.g., diabetes, age 40-70, female sex)
- Pathological process: synovial inflammation → capsular fibrosis → contracture of axillary fold and coracohumeral ligament
Differential Diagnoses to Exclude
- Rotator cuff tear (passive motion relatively preserved; positive drop arm, weakness in ER/abduction)
- Osteoarthritis (X-ray changes, older age, crepitus)
- Calcific tendinitis (X-ray shows calcium deposits, acute severe pain)
- Septic arthritis (redness, warmth, fever, systemic signs)
- Glenohumeral OA
- Cervical radiculopathy (neck pain, dermatomal sensory loss, radicular symptoms)
Functional Impact
- Limited independence in ADLs: dressing, grooming, reaching, driving
- Sleep disturbance due to night pain
- Work and recreational limitations
Prognosis
- Predominantly self-limiting; traditional timeline 12-18 months, though ~10% have long-term problems
- Diabetic patients: longer course, lower rates of full ROM recovery (~71% vs 90% non-diabetic)
- Earlier presentation correlates with faster recovery
- Dominant shoulder involvement: associated with better outcomes - Campbell's Operative Orthopaedics 15th Ed, p.2840
P - PLAN
Phase 1 - Painful Phase (Priority: Pain Control)
Physiotherapy Interventions:
- Gentle pendulum (Codman's) exercises - gravity-assisted passive motion
- Passive ROM within pain-free range, no forced stretching
- Heat (prior to exercise) and ice (post-exercise) for pain modulation
- TENS (transcutaneous electrical nerve stimulation) for pain relief
- Ultrasound therapy for tissue extensibility
- Patient education: nature of condition, expected timeline, importance of active participation
Medical:
- NSAIDs (oral): ibuprofen, naproxen for inflammation and pain
- Corticosteroid injection: 1 mL methylprednisolone 40 mg/mL into glenohumeral joint and/or subacromial bursa - most beneficial in early/painful phase
- Oral corticosteroids: improve pain and ROM short-term
Phase 2 - Stiffening / Frozen Phase (Priority: Restore ROM)
Physiotherapy Interventions:
- Progressive passive and active-assisted ROM exercises
- Capsular stretching: posterior capsule stretch, cross-body adduction stretch, sleeper stretch
- Glenohumeral joint mobilization (Maitland Grade III-IV): anterior, inferior, and posterior glides
- Muscle energy techniques (METs) for ER and shoulder flexion
- Pulley exercises and wall-climbing finger exercises
- Scapular stabilization exercises
- Thoracic spine mobilization (accessory mobility)
- Progressive active ROM and strengthening as tolerated
Medical:
- Hydrodistension / distension arthrography: 20-30 mL of local anaesthetic + corticosteroid injected into joint capsule; facilitates ROM exercises - Bailey & Love's Surgery 28th Ed, p.552
- Continue NSAIDs and/or corticosteroid injections
Phase 3 - Thawing Phase (Priority: Restore Strength & Function)
Physiotherapy:
- Progressive active ROM - full arc training
- Rotator cuff strengthening (ER, IR, abduction)
- Scapular stabilizer strengthening (serratus anterior, lower/middle trapezius)
- Proprioception and neuromuscular re-education
- Functional task training (ADL reintegration, overhead work)
- Gradual return to sport/work activities
Operative Management (Reserved for Prolonged/Refractory Cases)
Indicated when conservative management fails over several months:
- Manipulation under anaesthesia (MUA): Closed manipulation; good results; failure usually due to inability to maintain postoperative motion
- Arthroscopic capsular release: Recommended when MUA fails; releases tight capsule with emphasis on coracohumeral ligament
- Open surgical release: Reserved for failure of arthroscopic release; emphasis on restoring rotator interval
- Campbell's Operative Orthopaedics 15th Ed, p.2841 notes MUA, arthroscopic release, and structured physiotherapy were equivalently effective in RCT, but arthroscopic release carried highest risk.
Patient Education
- Explain 3-phase self-limiting course and realistic timeline (1-2 years)
- Reinforce active exercise participation even during pain phase
- Educate on diabetes control (worsens prognosis and course)
- Address sleep positioning (avoid lying on affected side; use pillow support)
- Explain red flags requiring reassessment (fever, acute swelling, neurological symptoms)
Goal Setting (Short & Long Term)
- Short-term: Reduce pain (VAS <3/10), improve passive ER by 15-20°, sleep improvement
- Long-term: Restore functional ROM, independent ADLs, return to work/sport
Follow-up
- Reassess every 2-4 weeks
- Reassess and modify intervention based on phase progression
- Refer to orthopaedic/rheumatology if no improvement after 3 months conservative care
Summary
| SOAP Section | Key Points |
|---|
| S | Gradual diffuse shoulder pain, night pain, progressive stiffness, restricted ADLs, phase-specific symptoms |
| O | Global restriction active+passive ROM, pathognomonic ER loss, normal X-ray, tenderness without warmth |
| A | Adhesive capsulitis, stage identified; DDx excluded; functional impact documented |
| P | Phase-appropriate physio (pendulum → stretching → strengthening), corticosteroid injection, hydrodistension; MUA/arthroscopy if refractory |
Recent Evidence Note: A 2023
meta-analysis (PMID 36861780) confirms manual therapy combined with exercise significantly improves pain and ROM. A 2024
systematic review (PMID 39420459) supports muscle energy techniques and joint mobilization for ROM improvement in frozen shoulder. A 2024
clinical practice guidelines review (PMID 37832814) across shoulder disorders recommends phase-specific physiotherapy and corticosteroid injections as first-line management.