ECG Interpretation - Second Recording
Patient: elangeshwri | ID: 0260719055938 | Facility: Health Haven | Doctor: Bijin
Settings: 25 mm/s | 10 mm/mV | 0.5-35 Hz | AC: 50 Hz
Comparison Note
This appears to be a second ECG of the same patient (elangeshwri, Health Haven), recorded shortly after or alongside the first. The ID suffix differs (055938 vs 055637), suggesting these are two sequential recordings.
Technical Quality
- Computer-generated interval measurements are partially visible at the bottom of the precordial strip (QRS, PR, QT/QTc, RVS/SVT, QTof)
- Some baseline artifact present, but overall quality is better than the first recording
- Calibration pulses (x1mv) are clearly visible at the start of each channel
Rhythm & Rate
| Parameter | Finding |
|---|
| Rhythm | Irregularly irregular |
| P waves | Absent - no consistent P waves before QRS complexes |
| Rate | ~60-75 bpm (ventricular response) |
| Fibrillatory baseline | Present in limb leads |
Primary rhythm: Atrial Fibrillation - consistent with the first ECG. Irregularly irregular rhythm with absent P waves confirmed in leads I, II, III, aVR, aVL, aVF.
Cardiac Axis
| Lead | QRS Polarity |
|---|
| Lead I | Predominantly positive |
| aVF | Predominantly positive |
| Axis | Normal (0° to +90°) |
QRS Morphology - Precordial Leads
| Lead | Morphology |
|---|
| V1 | Small r or rS complex - predominantly negative |
| V2 | rS - predominantly negative with deeper S |
| V3 | rS - transitional |
| V4 | Transitional zone - small R beginning to emerge |
| V5 | Taller R wave - positive deflection |
| V6 | Positive, upright R wave |
R-wave progression: Poor in V1-V3, with transition occurring around V4-V5.
The pattern is consistent with what was seen in the first ECG: QS/rS in right precordial leads, upright in lateral leads - again raising the differential of:
- Right ventricular apical (RVA) pacing with LBBB-like morphology
- Poor R-wave progression from anterior ischemia or old anterior MI
Limb Lead Analysis
- Lead I: Low amplitude, relatively flat - small biphasic or positive QRS
- Lead II: Variable R-R intervals confirming AF; QRS upright
- Lead III: Predominantly negative/biphasic QRS morphology with noisy baseline
- aVR: Negative QRS (expected in normal axis)
- aVL: Low amplitude or flat QRS (isoelectric - consistent with near-perpendicular axis)
- aVF: Positive QRS - confirms normal/inferior axis
ST Segments & T Waves
- No obvious ST elevation in any lead
- T-wave morphology is difficult to assess definitively due to artifact and AF
- No hyperacute changes identified
Computer Measurements (partially legible)
The printed measurements at the bottom include QRS duration, PR interval (not measurable in AF), QT/QTc, and RVS/SVT notation - the specific values are partially cut off, but these auto-measurements should be reviewed against clinical context.
Summary Comparing Both ECGs
| Feature | ECG 1 (ID ...637) | ECG 2 (ID ...938) |
|---|
| Rhythm | AF, controlled (~60 bpm) | AF, controlled (~65-75 bpm) |
| Axis | Normal | Normal |
| R-wave progression | Poor V1-V3 | Poor V1-V3 |
| ST changes | Not clearly seen | Not clearly seen |
| Overall | Same diagnosis | Confirms findings |
Overall Clinical Impression (Both ECGs Combined)
Primary Diagnosis:
Atrial Fibrillation with controlled ventricular rate
Key Associated Finding:
Persistent poor R-wave progression V1-V3 across both recordings - this is a stable, reproducible finding, making it more likely to represent either:
- Old anterior MI (loss of septal/anterior forces)
- RV pacing pattern
- LV hypertrophy with clockwise rotation
- Vertical heart in a thin patient
Recommended Next Steps:
- Confirm pacemaker presence / check device records
- If no pacemaker: 2D echocardiogram to assess anterior wall motion and LV function
- Troponin levels if anterior ischemia is clinically suspected
- CHA₂DS₂-VASc score for stroke risk - anticoagulation decision
- Rate control assessment - currently well-controlled; review medications
- Thyroid function tests (TFTs) - mandatory in new or undertreated AF
⚠️ Disclaimer: ECG interpretation provided for clinical decision-support only. Final diagnosis and management must be made by a qualified physician with full clinical context.