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Analyse this ecg and tell the pathology and axis deviation

analyse_media

ECG Analysis - Male, 57 years (23 Jan 2024)


Axis Deviation

Left Axis Deviation (LAD) is present, approximately -30° to -45°:
  • Lead I: predominantly positive (upright R)
  • Lead aVF: predominantly negative (rS pattern)
  • Lead II: predominantly negative (rS pattern)
This pattern in the context of narrow QRS complexes is consistent with Left Anterior Fascicular Block (LAFB) - the left anterior fascicle of the bundle of His is not conducting normally, causing the electrical axis to shift superiorly and to the left.

Primary Pathology: Atrial Fibrillation (AF) with Controlled Ventricular Response

Evidence:
  • No P waves in any lead - replaced by fine, irregular fibrillatory (f) waves, best seen in V1-V2
  • Irregularly irregular R-R intervals (hallmark of AF)
  • Ventricular rate: ~50-60 bpm - this is a "controlled" or rate-controlled AF (may suggest the patient is on rate-controlling drugs: beta-blockers, diltiazem, verapamil, or digoxin)

Additional Findings

FeatureFinding
QRS durationNarrow (<100 ms) - normal intraventricular conduction
ST segmentsIsoelectric - no acute ischemia/injury pattern
T wavesFlat to mildly inverted in II, III, aVF (secondary to LAFB)
Precordial transitionNormal (V3-V4)
V5-V6Tall R waves, small/absent Q waves

Summary of Diagnoses

  1. Atrial Fibrillation with controlled ventricular rate (~50-60 bpm)
  2. Left Anterior Fascicular Block (LAFB) causing Left Axis Deviation (-30° to -45°)
  3. No acute ST-elevation or STEMI pattern
  4. No right bundle branch block or left bundle branch block (QRS is narrow)

Clinical Note

The combination of AF + LAFB raises consideration of:
  • Hypertensive heart disease (most common cause of both)
  • Dilated cardiomyopathy
  • Ischemic heart disease
  • Valvular disease (especially mitral)
Rate control appears adequate, but anticoagulation status (CHA₂DS₂-VASc score) should be assessed to reduce stroke risk. Correlation with clinical history, echocardiogram, and thyroid function is recommended.
Disclaimer: This analysis is for educational purposes. Clinical decisions must be made by a licensed physician with access to the full clinical picture.
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Analysis of this ecg tell pathology heart rate axis deviation

analyse_media

ECG Analysis - 12-Lead (30 Dec 1972, 07:00:40)


Printed Machine Measurements

ParameterValue
Heart Rate~110 bpm (RR = 546 ms)
PR Interval136 ms
QRS Duration190 ms (severely prolonged)
QT / QTc336 ms / 455 ms (QTc borderline prolonged)
QRS Axis-75° (machine) / -63° (handwritten)
T Axis65°
Paper speed25 mm/s, 10 mm/mV

Axis Deviation

Extreme Left Axis Deviation (LAD): -63° to -75°
  • Lead I: broad, tall, positive monophasic R wave
  • Lead aVF: deep, wide QS pattern (negative)
  • Lead II: deep, wide rS/QS pattern (negative)
This places the axis firmly in the upper-left quadrant, well beyond the -30° threshold for LAD. The degree of leftward shift (-75°) in the presence of LBBB indicates co-existing Left Anterior Fascicular Block (LAFB).

Primary Pathology: Left Bundle Branch Block (LBBB)

The QRS of 190 ms (normal <120 ms) is grossly prolonged. Lead-by-lead morphology confirms LBBB:
LeadFinding
I, aVL, V5, V6Broad, slurred, notched monophasic R waves (no septal Q)
V1, V2, V3Deep, wide QS complexes
V4Wide QS/rS pattern
aVRBroad positive r/R wave
This is the classic "M pattern" in V5-V6 and W pattern in V1 of LBBB.

Rhythm

Tachycardia at ~110 bpm - the baseline shows significant artifact/tremor. Whether this is sinus tachycardia with obscured P waves or Atrial Fibrillation with rapid ventricular response requires clinical correlation. The RR intervals are difficult to assess for regularity due to baseline noise.

ST-T Changes

All ST-T changes are discordant (opposite to the terminal QRS direction) - this is expected in LBBB and is secondary/physiological:
  • V1-V4: ST elevation with upright T waves (opposite to the negative QRS) - secondary to LBBB
  • I, aVL, V5-V6: ST depression + T-wave inversion (opposite to the positive QRS)
Important: The Sgarbossa criteria should be applied if acute MI is clinically suspected:
  • ST elevation concordant with QRS (same direction) would suggest ischemia
  • ST elevation >5 mm discordant may also be suspicious in the right clinical context

Summary of Diagnoses

  1. Left Bundle Branch Block (LBBB) - QRS 190 ms - new or old?
  2. Extreme Left Axis Deviation (-75°) - likely LBBB + Left Anterior Fascicular Block (LAFB) (bifascicular block pattern)
  3. Tachycardia ~110 bpm - sinus tachycardia vs. AF with rapid ventricular response
  4. QTc 455 ms - borderline prolonged (partly due to LBBB itself)
  5. Secondary ST-T changes consistent with LBBB (clinically rule out ACS if new LBBB)

Clinical Significance

New LBBB with tachycardia is a STEMI-equivalent until proven otherwise. If this is a new LBBB in a symptomatic patient, it warrants urgent evaluation for acute anterior MI. Correlation with prior ECGs, cardiac troponins, and clinical symptoms is mandatory.
Disclaimer: This is an educational ECG interpretation. All clinical decisions must be made by a licensed physician with full access to the patient's clinical context.
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Analysis of this ecg tell pathology heart rate axis deviation

analyse_media

ECG Analysis - Furmistrov G.V., Male, 66 years (30 Dec 1972)


Printed Machine Measurements

ParameterValue
Heart Rate~110 bpm (RR = 546 ms)
PR Interval190 ms (normal)
QRS Duration136 ms (prolonged)
P Duration134 ms (prolonged)
QT / QTcB336 ms / 455 ms
P / QRS / T Axis65° / -75° / 60°
Handwritten axis-63°
Paper speed25 mm/s, 10 mm/mV

Axis Deviation

Extreme Left Axis Deviation (LAD): -63° to -75°
LeadFindingSignificance
Lead ITall broad qRPositive - left-directed
Lead aVLTall broad RPositive - superior
Lead IIDeep rSNegative - confirms LAD
Lead IIIDeep rSNegative
Lead aVFDeep rSNegative
Axis in the range of -63° to -75° is well past the LAD threshold (-30°), indicating marked leftward and superior deviation.

Primary Pathology: Left Bundle Branch Block (LBBB)

QRS duration = 136 ms (diagnostic threshold >120 ms). Classic LBBB morphology:
Lead GroupFinding
V1-V4Deep, wide QS complexes with absent R-wave progression
V5, V6Broad, slurred, notched R waves without septal Q waves
I, aVLTall, broad monophasic R waves (qR pattern)
aVRDeep QS/rS complex
This is the hallmark "W" in V1 and "M" in V5-V6 pattern of complete LBBB.

Rhythm

Sinus Tachycardia at ~110 bpm
  • P waves are present and precede every QRS (unlike the previous ECG)
  • PR interval = 190 ms (normal range)
  • RR interval regular at ~546 ms

Co-existing Left Anterior Fascicular Block (LAFB)

The extreme LAD of -75° in the setting of LBBB indicates the left anterior fascicle is also involved:
  • rS in II, III, aVF + qR in I, aVL = LAFB pattern
  • Combined with LBBB = Bifascicular block (left bundle branch + left anterior fascicle - though technically LBBB already implies bifascicular disease anatomically)

ST-T Changes

All discordant (expected with LBBB - secondary changes):
LeadsST ChangeReason
V1-V4ST elevation (discordant to deep negative QRS)Secondary to LBBB
I, aVL, V5-V6ST depression + T inversion (discordant to positive QRS)Secondary to LBBB

Prolonged P Duration (134 ms)

P duration >120 ms suggests left atrial enlargement or intra-atrial conduction delay (also known as P mitrale). This may indicate underlying hypertensive or valvular heart disease.

Summary of Diagnoses

  1. Sinus Tachycardia - ~110 bpm
  2. Complete Left Bundle Branch Block (LBBB) - QRS 136 ms
  3. Extreme Left Axis Deviation (-75°) with features of Left Anterior Fascicular Block (LAFB) - bifascicular conduction disease
  4. Left Atrial Enlargement - P duration 134 ms
  5. Secondary ST-T changes consistent with LBBB (rule out acute ACS if new onset)
  6. QTc 455 ms - borderline prolonged (partly attributable to LBBB)

Clinical Significance

This ECG in a 66-year-old male with LBBB + extreme LAD + left atrial enlargement + tachycardia strongly suggests underlying hypertensive heart disease, dilated cardiomyopathy, or ischemic cardiomyopathy. If this LBBB is new in a symptomatic patient, it should be treated as a STEMI-equivalent and urgent evaluation is required.
Disclaimer: This is an educational ECG interpretation. All clinical decisions must be made by a licensed physician with full clinical context.
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