Provide a proper "Assessment of ENT" diagnosed with Allergic Rhinitis. Give Biographic data, chief complaints, present illness, past illness, physical Examination , lab investigations, medicine, special investigations and NCP of the patient.

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allergic rhinitis nasal turbinate pale edematous mucosa physical examination

This clinical comparison chart features two endoscopic images (A and B) of the left nasal cavity, demonstrating the physical changes in allergic rhinitis before and after treatment. Image A displays the pre-treatment state, characterized by a pale, edematous nasal mucosa and a severely swollen inferior turbinate (IT) that completely obstructs the view of the middle turbinate (MT). The nasal septum (NS) is visible to the left. Image B shows the same anatomical region 10 minutes post-fire needle acupuncture treatment. There is a marked reduction in the swelling and edema of the inferior turbinate, which significantly increases the patency of the nasal passage. This reduction in volume allows for the clear visualization of the middle turbinate (MT) in the background. The comparison highlights the immediate physical effect of the intervention on turbinate hypertrophy and mucosal congestion, serving as an educational resource for rhinological examination and non-pharmacological treatment monitoring.

This clinical comparison chart features two endoscopic images (A and B) of the left nasal cavity, demonstrating the physical changes in allergic rhinitis before and after treatment. Image A displays the pre-treatment state, characterized by a pale, edematous nasal mucosa and a severely swollen inferior turbinate (IT) that completely obstructs the view of the middle turbinate (MT). The nasal septum (NS) is visible to the left. Image B shows the same anatomical region 10 minutes post-fire needle acupuncture treatment. There is a marked reduction in the swelling and edema of the inferior turbinate, which significantly increases the patency of the nasal passage. This reduction in volume allows for the clear visualization of the middle turbinate (MT) in the background. The comparison highlights the immediate physical effect of the intervention on turbinate hypertrophy and mucosal congestion, serving as an educational resource for rhinological examination and non-pharmacological treatment monitoring.

Three clinical endoscopic views of the internal nasal cavity in a patient with allergic rhinitis and epistaxis. The images demonstrate classic mucosal changes including generalized hypervascularity, vascular dilatation, and tortuous superficial blood vessels along the nasal septum and turbinates. The nasal mucosa appears erythematous and edematous, with areas of significant pale swelling (cobblestone appearance) characteristic of allergic inflammation. The inferior turbinates show signs of hypertrophy, causing narrowing of the nasal passage. Visible signs of recent or potential epistaxis are indicated by the prominent, engorged vessels in Kiesselbach’s plexus area and along the anterior septal wall. These endoscopic findings illustrate the chronic inflammatory state of the nasal airway in pediatric or adult allergic patients, highlighting how mucosal fragility and vascular congestion contribute to frequent nosebleeds.

Three clinical endoscopic views of the internal nasal cavity in a patient with allergic rhinitis and epistaxis. The images demonstrate classic mucosal changes including generalized hypervascularity, vascular dilatation, and tortuous superficial blood vessels along the nasal septum and turbinates. The nasal mucosa appears erythematous and edematous, with areas of significant pale swelling (cobblestone appearance) characteristic of allergic inflammation. The inferior turbinates show signs of hypertrophy, causing narrowing of the nasal passage. Visible signs of recent or potential epistaxis are indicated by the prominent, engorged vessels in Kiesselbach’s plexus area and along the anterior septal wall. These endoscopic findings illustrate the chronic inflammatory state of the nasal airway in pediatric or adult allergic patients, highlighting how mucosal fragility and vascular congestion contribute to frequent nosebleeds.

Two side-by-side endoscopic images provide a clinical comparison of the right (R) and left (L) nasal cavities following the treatment of severe allergic rhinitis. The images feature the nasal septum (S) and the inferior turbinate (IT). In the right nasal cavity, the inferior turbinate is moderately enlarged with a smooth, pale mucosal surface, though a patent airway is visible between the turbinate and the septum. In the left nasal cavity, the nasal septum appears deviated toward the left, narrowing the available space. The left inferior turbinate shows mild translucency and less pallor than the right side. The nasal mucosa across both cavities demonstrates reduced congestion and edema compared to typical pre-treatment allergic states. This visualization is used in otorhinolaryngology to assess the efficacy of biological therapies (e.g., omalizumab) in reducing mucosal inflammation and relieving mucosal contact points associated with rhinogenic contact point headache (RCPH) in pediatric patients with refractory seasonal allergic rhinitis.

Two side-by-side endoscopic images provide a clinical comparison of the right (R) and left (L) nasal cavities following the treatment of severe allergic rhinitis. The images feature the nasal septum (S) and the inferior turbinate (IT). In the right nasal cavity, the inferior turbinate is moderately enlarged with a smooth, pale mucosal surface, though a patent airway is visible between the turbinate and the septum. In the left nasal cavity, the nasal septum appears deviated toward the left, narrowing the available space. The left inferior turbinate shows mild translucency and less pallor than the right side. The nasal mucosa across both cavities demonstrates reduced congestion and edema compared to typical pre-treatment allergic states. This visualization is used in otorhinolaryngology to assess the efficacy of biological therapies (e.g., omalizumab) in reducing mucosal inflammation and relieving mucosal contact points associated with rhinogenic contact point headache (RCPH) in pediatric patients with refractory seasonal allergic rhinitis.

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ENT Assessment: Allergic Rhinitis


I. BIOGRAPHIC DATA

FieldDetails
Patient NameMr. Rajan Mehta
Age28 years
SexMale
Date of BirthJuly 15, 1997
Marital StatusSingle
OccupationSoftware Engineer (works indoors, air-conditioned office)
ReligionHindu
NationalityIndian
Address14/B, Green Park Colony, Hyderabad, Telangana
Date of AdmissionJuly 25, 2026
Ward / RoomENT OPD - Room No. 3
Attending PhysicianDr. Suresh Anand, ENT Specialist
InformantPatient himself (reliable)
Source of ReferralGeneral Practitioner
Mode of AdmissionOutpatient (walk-in)

II. CHIEF COMPLAINTS

The patient presents with the following complaints for the past 6 weeks, worsening over the last 2 weeks:
  1. Persistent sneezing (paroxysmal, especially in the morning)
  2. Watery, clear nasal discharge (rhinorrhea)
  3. Nasal congestion / stuffiness (bilateral)
  4. Nasal itching and itching of the palate
  5. Eye itching, redness, and tearing (bilateral)
  6. Headache (mild, frontal region)
  7. Fatigue and disturbed sleep due to nasal congestion

III. HISTORY OF PRESENT ILLNESS

Mr. Rajan Mehta, a 28-year-old male, presents to the ENT OPD with a 6-week history of recurrent sneezing, bilateral nasal obstruction, and profuse clear watery rhinorrhea. Symptoms began insidiously in early June and have progressively worsened. He reports that symptoms are worst in the morning upon waking and during periods of high dust exposure. He works in a building undergoing renovation near his office, which he identifies as a possible trigger.
He also complains of bilateral eye itching, redness, and lacrimation consistent with allergic conjunctivitis. He reports frequent headaches in the frontal region, which he attributes to nasal congestion. He has disturbed sleep due to difficulty breathing through the nose and snores at night.
He denies fever, facial pain, purulent nasal discharge, loss of smell, or blood in the nasal discharge. He does not report any wheezing, chest tightness, or difficulty breathing.
Aggravating Factors: Dust, strong odors, cold air, early morning exposure, proximity to flowers/plants
Relieving Factors: Over-the-counter antihistamine tablets (taken occasionally, partial relief)
Duration: 6 weeks (persistent/perennial pattern with seasonal exacerbation)
As noted in Goldman-Cecil Medicine, "The natural history of allergic rhinitis is for symptoms to worsen inexorably during several weeks in the presence of ongoing allergen exposure. Symptoms often do not peak until well after the peak in pollen counts." - Goldman-Cecil Medicine, p. 2388

IV. PAST MEDICAL HISTORY

CategoryDetails
Previous similar episodesYes - patient had similar sneezing and runny nose episodes every year since age 15
Childhood illnessesRecurrent episodes of "cold" in childhood, eczema at age 7 (resolved)
HospitalizationsNone
SurgeriesNil
Trauma / injuryNil
Known allergiesDust mites (suspected), pollen
AsthmaNo diagnosed asthma; no wheezing reported
Medications (past/current)OTC Cetirizine 10 mg occasionally (partial relief); no topical nasal sprays used previously
ImmunizationsUp to date; no influenza vaccine this year
Family HistoryFather has bronchial asthma; mother has seasonal allergic rhinitis; elder sister has atopic dermatitis
Note: The strong family history of atopy (asthma, eczema, allergic rhinitis in first-degree relatives) is consistent with the hereditary predisposition of atopic disease, as highlighted in K.J. Lee's Essential Otolaryngology: "Family history of allergic rhinitis, conjunctivitis, asthma" is a key component of allergy history-taking.

V. PHYSICAL EXAMINATION

General Survey

  • Patient is a well-nourished young adult male, alert and oriented
  • Appears mildly distressed due to nasal congestion and frequent sneezing during examination
  • Mouth breathing noted
  • No pallor, icterus, cyanosis, clubbing, or lymphadenopathy
  • Vital signs:
    • BP: 118/76 mmHg
    • HR: 78 bpm, regular
    • RR: 17 breaths/min
    • Temperature: 37.1°C (afebrile - fever is NOT a feature of allergic rhinitis)
    • SpO2: 99% on room air
As noted in Goldman-Cecil Medicine: "Fever is not a feature of allergic rhinitis, even though the lay term for this condition is hay fever." - Goldman-Cecil Medicine, p. 2390

Head and Face

  • Allergic salute sign: Transverse crease across the bridge of the nose from habitual upward rubbing
  • Allergic shiners: Dark, bluish-gray discoloration under both eyes (suborbital venous congestion)
  • Dennie-Morgan lines: Infraorbital skin folds present bilaterally
  • No sinus tenderness on palpation over frontal or maxillary sinuses

Eyes (Ocular Examination)

  • Bilateral conjunctival injection (redness)
  • Mild conjunctival edema and excessive lacrimation
  • No purulent discharge
  • Pupils equal, round, reactive to light (PERRL)
  • No photophobia

Nose (Anterior Rhinoscopy)

  • Inferior turbinates: Swollen and hypertrophied bilaterally
  • Nasal mucosa: Pale, bluish-grey/edematous appearance (classic allergic appearance)
  • Nasal discharge: Clear, watery, profuse
  • Nasal septum: Midline, no deviation noted
  • Nasal polyps: Absent
  • No purulent or blood-stained discharge
Tintinalli's Emergency Medicine describes: "Patients with allergic rhinitis report symptoms of paroxysmal sneezing, nasal pruritus, rhinorrhea, oropharyngeal pruritus, hyperemia, and ocular pruritus. On physical examination, there may be hypertrophy and edema of the nasal turbinates with associated pale, bluish hue or pallor." - Tintinalli's, p. 894
Nasal endoscopy showing pale, edematous inferior turbinate characteristic of allergic rhinitis

Throat / Oropharynx

  • Mildly injected posterior pharyngeal wall
  • Mucoid postnasal drip visible along posterior pharyngeal wall
  • Tonsils: Grade I, non-inflamed
  • No exudate

Ears (Otoscopic Examination)

  • Bilateral tympanic membranes intact, pearly gray, light reflex present
  • No middle ear effusion noted
  • Hearing: Normal by finger rubbing test

Neck

  • No cervical lymphadenopathy
  • Thyroid: Not enlarged

Chest / Respiratory

  • Chest: Clear to auscultation bilaterally
  • No wheeze, crackles, or added sounds
  • Normal vesicular breath sounds

VI. LABORATORY INVESTIGATIONS

InvestigationOrdered Value / FindingClinical Relevance
Complete Blood Count (CBC)WBC: 8,200/µL; Eosinophils: 6% (0.49 × 10⁹/L) - mildly elevated; RBC, Hb, Platelets: NormalPeripheral eosinophilia is a supportive finding in allergic rhinitis
Absolute Eosinophil Count (AEC)490 cells/µL (slightly elevated; normal <450)Supports allergic/atopic state
Total Serum IgE320 IU/mL (elevated; normal <100 IU/mL)Elevated total IgE indicates atopic state; not diagnostic alone
Nasal Smear for Eosinophils (NSE)Abundant eosinophils on nasal cytologyHighly specific for allergic rhinitis when positive
Blood Glucose (RBS)96 mg/dLNormal; baseline before steroid prescription
Liver Function Test (LFT)Within normal limitsBaseline prior to medication
Kidney Function Test (KFT)Within normal limitsBaseline
Harrison's Principles of Internal Medicine states: "The nasal secretions of allergic patients can be rich in eosinophils, and a modest peripheral eosinophilia can be observed... Total serum IgE may be moderately elevated." - Harrison's, p. 2853

VII. SPECIAL INVESTIGATIONS

1. Skin Prick Test (SPT) - GOLD STANDARD

  • Purpose: Identifies specific allergens responsible for symptoms
  • Method: Extracts of common aeroallergens (house dust mite, cockroach, grass pollen, tree pollen, cat dander, mold spores, fungal spores) applied on the forearm with a lancet prick
  • Result (expected/typical): Positive wheal-and-flare reaction (>3mm) to house dust mite (Dermatophagoides pteronyssinus) and grass pollen
  • Interpretation: A positive intracutaneous skin test with 1:10-1:20 weight/volume extract has high predictive value
Goldman-Cecil Medicine: "Prick skin testing is safe, specific, and rapid, and it is the diagnostic test of choice for identifying relevant allergens." - Goldman-Cecil Medicine, p. 2495

2. Serum Specific IgE (RAST / ImmunoCAP Assay)

  • ELISA-based measurement of serum allergen-specific IgE
  • Ordered for: House dust mite, grass pollen, cockroach, pet dander
  • Result: Elevated specific IgE to dust mite (Class III: 3.5-17.4 kUA/L)
  • Indicated when skin testing cannot be performed (e.g., dermographism, patient on antihistamines)

3. Nasal Endoscopy

  • Flexible fiberoptic nasal endoscopy performed to rule out nasal polyps, sinusitis, or anatomical abnormality
  • Findings: Pale, edematous inferior turbinates bilaterally; clear mucus; septum midline; no polyps; no purulent discharge

4. CT Scan of Paranasal Sinuses (if indicated)

  • Not immediately required in uncomplicated allergic rhinitis
  • Indicated if: Patient shows signs of chronic rhinosinusitis, nasal polyps, or failure to respond to initial therapy
  • Would reveal mucosal thickening or opacification of sinuses if sinusitis present

5. Peak Flow / Spirometry

  • Ordered given strong family history of asthma
  • Rationale: "Patients with persistent allergic rhinitis should be screened for asthma, and patients with asthma should be evaluated for allergic rhinitis." - Murray & Nadel's Textbook of Respiratory Medicine

VIII. MEDICATIONS PRESCRIBED

DrugClassDose & RouteFrequencyDurationPurpose
Cetirizine 10 mg2nd gen. oral H1-antihistamine10 mg POOnce daily (evening)4 weeksReduces sneezing, itching, rhinorrhea, and ocular symptoms
Fluticasone Furoate Nasal SprayIntranasal glucocorticoid2 sprays per nostrilOnce daily (morning)4-6 weeksMost effective drug for nasal congestion; up to 70% overall symptom relief
Normal Saline Nasal Irrigation (Jal Neti / NaSal wash)Saline (non-pharmacologic)240 mL per nostrilTwice dailyOngoingMechanical clearance of allergens and secretions
Olopatadine 0.1% Eye DropsTopical antihistamine (ocular)1 drop each eyeTwice daily2-4 weeksRelief of allergic conjunctivitis (itching, redness)
Montelukast 10 mgLeukotriene receptor antagonist10 mg POOnce daily (night)4 weeks (if incomplete response)Additional relief of nasal congestion; approved for both seasonal and perennial AR
Pseudoephedrine 60 mg (PRN)Oral alpha-adrenergic decongestant60 mg POAs needed (max 3x/day)Short course only (3-5 days)Rapid relief of severe nasal congestion (avoid in hypertension)
Harrison's Principles of Internal Medicine: "Intranasal high-potency glucocorticoids are the most effective drugs available for the relief of persistent rhinitis, seasonal or perennial, and are effective in relieving nasal congestion as well as ocular symptoms." - Harrison's, p. 304
"Oral long-acting H1 antihistamines, such as fexofenadine, loratadine, desloratadine, cetirizine, and levocetirizine, are effective for nasopharyngeal itching, sneezing, and watery rhinorrhea." - Harrison's, p. 304
Allergen Immunotherapy (future consideration): If symptoms are inadequately controlled with pharmacotherapy or if the patient wishes to reduce long-term medication dependence, subcutaneous immunotherapy (SCIT) or sublingual immunotherapy (SLIT) for dust mite and grass pollen allergens may be considered.

IX. NURSING CARE PLAN (NCP)


NCP 1: Ineffective Airway Clearance

ComponentDetails
Nursing DiagnosisIneffective Airway Clearance related to nasal mucosal edema, hypersecretion, and turbinate hypertrophy secondary to allergic rhinitis, as evidenced by nasal congestion, mouth breathing, and disturbed sleep
GoalPatient will demonstrate improved nasal airway patency and report decreased nasal congestion within 72 hours of treatment initiation
Nursing Interventions1. Elevate head of bed to 30-45° to facilitate nasal drainage and reduce mucosal congestion 2. Teach and demonstrate proper nasal saline irrigation (NaSal wash) technique twice daily 3. Administer intranasal fluticasone furoate as prescribed; instruct patient on correct spray technique (avoid spraying directly on septum) 4. Encourage adequate oral hydration (2-3 L/day) to thin secretions 5. Monitor respiratory rate and oxygen saturation 6. Advise patient to avoid known triggers (dust, strong odors, cold air)
RationaleElevated positioning reduces venous pooling and mucosal edema. Saline irrigation mechanically removes allergens and thick mucus. Topical steroids reduce mucosal inflammation, the primary cause of congestion. Hydration thins secretions and aids mucociliary clearance.
EvaluationPatient reports subjective improvement in nasal breathing; bilateral nasal passages are more patent on rhinoscopy; snoring and mouth breathing reduced

NCP 2: Disturbed Sleep Pattern

ComponentDetails
Nursing DiagnosisDisturbed Sleep Pattern related to nasal congestion, sneezing, and post-nasal drip secondary to allergic rhinitis, as evidenced by patient's report of difficulty breathing at night and fatigue
GoalPatient will report improved sleep quality and reduced nocturnal awakenings within 1 week
Nursing Interventions1. Administer cetirizine in the evening as prescribed (minimizes drowsiness interference with daytime activities; reduces nocturnal symptoms) 2. Educate patient on allergen reduction measures in the bedroom: use dust-mite-proof pillow and mattress covers, wash bed linen in hot water (>60°C) weekly, keep pets out of the bedroom 3. Instruct patient to perform nasal saline irrigation before bedtime 4. Keep bedroom temperature moderate (not too warm or cold) and well ventilated 5. Assess and document sleep quality daily using a simple sleep diary 6. Avoid sedating antihistamines (first-gen) that can disrupt REM sleep architecture
RationaleNighttime antihistamine use reduces nocturnal symptom burden. Allergen reduction in the sleeping environment decreases overnight allergen exposure, which directly reduces symptom intensity. Saline wash before bed clears accumulated secretions.
EvaluationPatient reports sleeping without frequent awakening; fatigue levels decreased; no mouth breathing at rest

NCP 3: Acute Pain (Headache)

ComponentDetails
Nursing DiagnosisAcute Pain related to frontal sinus pressure secondary to nasal mucosal congestion, as evidenced by patient's complaint of frontal headache (rated 4/10 on NRS)
GoalPatient will report pain reduction to ≤2/10 within 48 hours
Nursing Interventions1. Assess pain: location, intensity (NRS 0-10), quality, duration, and aggravating/relieving factors 2. Apply warm compresses over the frontal and maxillary sinus areas for 5-10 minutes, 3x/day 3. Encourage use of prescribed nasal saline rinse to decompress nasal passages and relieve sinus pressure 4. Administer analgesics as ordered (e.g., Paracetamol 500-1000 mg PO if needed) 5. Instruct patient to avoid bending forward or straining, which increases sinus pressure 6. Monitor for worsening headache, fever, or purulent discharge which could indicate sinusitis requiring escalation
RationaleNasal congestion obstructs the ostiomeatal complex, causing sinus pressure buildup and headache. Warm compresses promote local vasodilation and sinus drainage. Saline rinse reduces mucosal edema, relieving blockage. Positioning reduces gravity-dependent pressure.
EvaluationPatient reports headache intensity decreased; no new signs of acute sinusitis; facial tenderness on palpation resolved

NCP 4: Deficient Knowledge

ComponentDetails
Nursing DiagnosisDeficient Knowledge regarding allergic rhinitis, allergen avoidance, and medication use, as evidenced by patient's irregular use of OTC antihistamines and inability to identify specific triggers
GoalPatient will verbalize understanding of allergic rhinitis, demonstrate correct use of nasal spray and saline rinse, and identify at least 3 allergen-avoidance strategies before discharge
Nursing Interventions1. Explain the pathophysiology of allergic rhinitis in simple terms (immune overreaction to harmless substances like dust mites or pollen) 2. Teach proper technique for intranasal steroid spray (tilt head slightly forward, aim spray toward outer eye, avoid septum) 3. Demonstrate nasal saline irrigation technique; have patient return-demonstrate 4. Provide written allergy action plan: what to do when symptoms worsen 5. Educate on allergen reduction: dust-mite covers, HEPA air filters, limiting outdoor exposure during high pollen seasons, avoiding tobacco smoke and strong perfumes 6. Emphasize adherence to intranasal steroid (takes 1-2 weeks for full effect; do not stop when symptoms improve) 7. Explain the side effect profile of each medication and when to seek emergency care
RationaleAllergen avoidance is the most cost-effective strategy for managing allergic rhinitis. Correct spray technique maximizes drug deposition and minimizes septal side effects. Written action plans improve self-management. Medication adherence education reduces treatment failure.
EvaluationPatient correctly demonstrates nasal spray technique and saline rinse; lists specific triggers and 3 avoidance strategies; verbalizes when to return to clinic

NCP 5: Risk for Ineffective Self-Health Management

ComponentDetails
Nursing DiagnosisRisk for Ineffective Self-Health Management related to chronic nature of allergic rhinitis, complexity of multi-drug regimen, and risk of developing complications (sinusitis, asthma)
GoalPatient will adhere to treatment regimen and attend follow-up appointment in 4 weeks
Nursing Interventions1. Simplify medication regimen: provide a written schedule for each drug 2. Counsel regarding the chronic, recurring nature of allergic rhinitis and the importance of maintenance therapy 3. Screen for asthma: provide peak flow meter if available; instruct on its use (given strong family history of asthma) 4. Refer to allergist/immunologist for formal allergy testing and discussion of immunotherapy options 5. Schedule follow-up in 4 weeks to assess treatment response, medication tolerance, and need for step-up therapy 6. Provide contact details and instruct patient to report: development of fever, purulent nasal discharge, new-onset wheezing, loss of smell, or severe facial pain
RationalePersistent allergic rhinitis is a significant risk factor for development of asthma and chronic rhinosinusitis. "Patients with persistent allergic rhinitis should be screened for asthma, and patients with asthma should be evaluated for allergic rhinitis." (Murray & Nadel's). Immunotherapy offers a disease-modifying option that reduces medication burden over time.
EvaluationPatient attends follow-up; no new complications; adherent to medication schedule; understands asthma warning signs

Summary of Diagnosis

ARIA Classification (Allergic Rhinitis and its Impact on Asthma):
  • Persistent (symptoms >4 days/week AND >4 weeks duration)
  • Moderate-Severe (sleep disturbance, impairment of daily activities, fatigue present)
  • Comorbidity: Allergic conjunctivitis
Likely Sensitizing Allergens: House dust mite (primary), grass pollen (secondary), possibly workplace construction dust
Management Plan:
  1. Intranasal glucocorticoids (first-line, most effective for nasal congestion and overall symptoms)
  2. Second-generation oral antihistamine (for itching, sneezing, rhinorrhea, ocular symptoms)
  3. Allergen avoidance measures
  4. Allergen-specific IgE testing / Skin prick test to confirm sensitizers
  5. Asthma screening (spirometry/peak flow) given family history
  6. Consider allergen immunotherapy if pharmacotherapy inadequate

Sources: Cummings Otolaryngology Head and Neck Surgery; Harrison's Principles of Internal Medicine 22E; Goldman-Cecil Medicine; K.J. Lee's Essential Otolaryngology; Murray & Nadel's Textbook of Respiratory Medicine; Tintinalli's Emergency Medicine; Scott-Brown's Otorhinolaryngology; Robbins Pathologic Basis of Disease
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