Pic with circle is 27th June, another is today
plantar wart verruca plantaris sole foot

Clinical photography of a plantar foot displaying a verruca plantaris (plantar wart). Modality is a high‑quality gross image used in dermatology documentation. Precise location is the plantar surface of the foot (sole), with a focal, endophytic lesion that is rough and hyperkeratotic. Visual features include a circular to oval papule with a central crater or indent, irregular surface texture, and surrounding callus‑like thickening. Punctate black dots representing thrombosed capillaries may be seen within the lesion, while surrounding epidermis shows white scaling and fissuring. The lesion is typically mildly elevated above the surrounding skin and may be tender with pressure; it often coexists with other warts on the same sole. The clinical impression is verruca plantaris caused by human papillomavirus infection, with characteristic epidermal hyperplasia, papillomatosis, and hyperkeratosis evident on gross inspection. Differential diagnoses include callus/corn, plantar keratoderma, and foreign body reaction; histology would show koilocytosis and viral cytopathic effects if examined. This image supports clinical decision making, differential diagnosis, and treatment planning (topical salicylic acid, cryotherapy, or laser), and serves as an educational reference for students, residents, and clinicians evaluating plantar lesions. Clinical correlation includes assessing for additional lesions and patient history of HPV exposure; follow-up imaging is not required.

This is a macroscopic clinical photograph of the plantar aspect of the foot, captured for dermatology/foot-health assessment. Imaging modality: Clinical photography; technique: high-resolution color image of the sole, direct view, with standard diffuse lighting and no magnification. Anatomical location: Plantar surface of the hindfoot and midfoot, primarily the heel region, extending to the medial plantar arch. Visual features: a red-orange to pink-tan hyperkeratotic plaque with a thick, rough surface and fissuring; surrounding hypochromic to xerotic skin; margins are relatively ill-defined but the lesion remains clearly localized to the plantar skin. The lesion appears as a diffuse keratotic patch rather than a single discrete nodule, with variable thickness across the plantar surface. Pathology (clinically): evidence of epidermal hyperplasia with thickened stratum corneum (hyperkeratosis) and surface fissures, typical of friction-induced plantar keratopathy; no signs of vesiculation, purulence, or acute inflammation evident. Diagnostic significance: characteristic of friction-related plantar hyperkeratosis or callus; differential considerations include plantar corn, plantar wart (verruca plantaris), and diffuse plantar keratoderma; correlate with symptoms and history. Clinical correlation: evaluate footwear, gait, pressure points, activity level; screen for diabetes risk; may respond to keratolytics, debridement, padding, and orthotic optimization; useful for monitoring response to therapy and for educational and research material.

Clinical photography of the plantar surface of the foot demonstrating a classic hard corn (clavus) due to repetitive pressure. The lesion is a small, well‑circumscribed, hyperkeratotic papule with a central keratin plug, surrounded by peripheral epidermal thickening (callus) on the weight‑bearing sole. The surrounding skin is erythematous to slightly inflamed under pressure. The core appears pale yellow to white and slightly translucent; the lesion projects above the adjacent skin, producing a painful sensation with direct compression. This appearance distinguishes clavus from a plantar wart or simple callus by the presence of a dense keratinized core and a more pointed cone shape toward the dermis. The lesion is typically located at pressure points near the metatarsal heads or along the distal toe margins, often due to ill‑fitting footwear or abnormal gait. Etiology is chronic friction and pressure; pathophysiology involves localized epidermal hyperplasia and keratinocyte hypertrophy. Management focuses on debulking the hyperkeratosis (paring), application of keratolytics (salicylic acid), protective footwear, and orthotic correction; addressing footwear fit reduces recurrence risk. Differential diagnoses include plantar callus, plantar wart (verruca plantaris), and foreign body granuloma. Clinically correlates with pain on palpation and history of footwear‑related irritation. This image is educational for dermatology, podiatry, primary care, and medical education in skin pathology.
| Feature | June 27 | August 9 |
|---|---|---|
| Size/spread | Larger, broader reddened zone | Smaller, more focal/discrete |
| Appearance | Diffuse flush with multiple black dots | Compact, hardened dome with central core |
| Inflammation | More inflamed/red | Less surrounding inflammation |
| Keratosis | Less defined | More defined, thicker |

Disclaimer: This is an AI-assisted visual assessment for informational purposes only - not a medical diagnosis. Please consult a healthcare professional for proper evaluation and treatment.
No when I take a bath in hot shower this fully turn white... And one small bup white arises may be right now the colour because I walk bare foot in my house no pain while sitting only pain when it gets pressed while walking
plantar wart white maceration after water soaking
| Symptom | Significance |
|---|---|
| Turns fully white in hot shower | Classic "maceration" sign - thickened keratin (dead skin) over a wart absorbs water and turns white. This happens more dramatically with warts than with normal skin because the keratin layer is abnormally thick. |
| Small white bump visible | The wart's core - the hyperkeratotic centre becomes very visible when wet |
| No pain at rest/sitting | Typical of plantar warts - they are endophytic (grow inward), so compression is needed to cause pain |
| Pain only when pressed/walking | Direct pressure on the wart's body is what causes the pain - exactly how plantar warts behave |
| No pain barefoot at rest | If it were a corn (heloma), you'd typically have more constant tenderness around it |
This is informational guidance only - not a clinical diagnosis. Please see a podiatrist or dermatologist for confirmation.
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| Week | What you'll see |
|---|---|
| Week 1-2 | Wart turns more white/soft, skin peeling |
| Week 3-4 | Top layer starts coming off in layers |
| Week 6-8 | Wart shrinks, black dots disappear |
| Week 8-12 | Wart fully gone and normal skin replaces it |
This is informational guidance only. If you have diabetes or poor circulation, consult a doctor before using any salicylic acid product.