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Here is a thorough, position-by-position analysis of the entire VNG (Videonystagmography) report for Sunita Gupta (66F, ID: CCDA00279), dated 09-Jun-2026.

VNG Report - Complete Position-by-Position Analysis


1. SACCADES

Horizontal Saccades (0.3 Hz)

ParameterRight EyeLeft EyeNormal Range
Velocity374.34°/s450.73°/s150-700°/s
Precision87.7867.67>80%
Latency300.00 ms272.94 ms<250 ms
Findings: Left eye precision is below normal (67.67 < 80). Latency in both eyes is mildly prolonged (normal < 250 ms). Right eye precision is borderline acceptable.

Horizontal Saccades (0.45 Hz)

ParameterRight EyeLeft Eye
Velocity413.59°/s585.92°/s
Precision82.5673.72
Latency350.48 ms321.90 ms
Findings: Left eye precision again reduced (73.72). Latencies are notably prolonged at higher frequency - suggesting difficulty with rapid, accurate eye movements (saccadic dysmetria/inaccuracy).

Vertical Saccades (0.3 Hz)

ParameterRight EyeLeft Eye
Velocity292.71°/s319.64°/s
Precision85.1881.87
Latency353.85 ms356.92 ms
Findings: Precision within normal limits bilaterally. Latency is significantly prolonged bilaterally (>250 ms), suggesting central pathway slowing affecting vertical saccades.

Vertical Saccades (0.45 Hz)

ParameterRight EyeLeft Eye
Velocity292.56°/s301.91°/s
Precision81.1878.32
Latency206.67 ms200.00 ms
Findings: Latency improved at 0.45 Hz. Precision slightly reduced in left eye.
Saccade Summary: Bilateral prolonged latency and reduced precision (especially left eye) - indicative of central dysfunction possibly at the level of the brainstem/cerebellum or frontal eye fields.

2. SMOOTH PURSUIT

Horizontal Smooth Pursuit (0.2 Hz)

DirectionRight EyeLeft EyeNormal
Rightward Gain0.320.34>0.7
Leftward Gain0.350.36>0.7

Horizontal Smooth Pursuit (0.4 Hz)

DirectionRight EyeLeft Eye
Rightward Gain0.330.28
Leftward Gain0.260.25

Vertical Smooth Pursuit (0.2 Hz)

DirectionRight EyeLeft Eye
Upward Gain0.300.36
Downward Gain0.290.38

Vertical Smooth Pursuit (0.4 Hz)

DirectionRight EyeLeft Eye
Upward Gain0.180.19
Downward Gain0.130.18
Findings: All smooth pursuit gains are severely reduced in all directions (H and V, both frequencies, both eyes). Normal gain should be >0.7; values here range from 0.13-0.36. This is a classic sign of central (cerebellar/brainstem) pathology. Age-related decline alone would not cause such a drastic bilateral reduction across all directions.

3. OPTOKINETIC TEST (OKN)

ConditionRight Eye GainLeft Eye GainNormal
Left-to-Right 10°0.930.91~1.0
Right-to-Left 10°0.980.91~1.0
Top-to-Bottom 10°0.981.11~1.0
Bottom-to-Top 10°1.031.02~1.0
Fast Phase Direction: None recorded in any direction (all "-").
Findings: OKN gains are symmetric and essentially normal (0.91-1.11) in all four directions. No directional asymmetry. This is a reassuring finding and argues against a complete unilateral cortical or peripheral lesion.

4. NYSTAGMUS TESTS

Spontaneous Nystagmus - In Light

  • Horizontal SPV: Absent bilaterally
  • Vertical SPV: Absent bilaterally
  • No fast phase, no amplitude recorded
Finding: No spontaneous nystagmus in light. NORMAL.

Spontaneous Nystagmus - In Dark

  • All parameters: Absent bilaterally
Finding: No spontaneous nystagmus in dark. NORMAL.

High Frequency Head Shake

  • All parameters: Absent bilaterally
Finding: No post-head-shake nystagmus. This argues against a significant unilateral peripheral vestibular loss.

Hyperventilation

  • All parameters: Absent bilaterally
Finding: No hyperventilation-induced nystagmus. Normal.

5. GAZE TEST

With Fixation (5 positions: Center, Left, Up, Right, Down)

  • All positions: SPV, Amplitude, Fast Phase Direction, Frequency = "-" (absent/not recorded)
Finding: No gaze-evoked nystagmus in any direction with fixation. NORMAL.

Without Fixation (5 positions: Center, Left, Up, Right, Down)

  • All positions: SPV, Amplitude, Fast Phase Direction, Frequency = "-" (absent)
Finding: No gaze-evoked nystagmus without fixation either. NORMAL.
A key caveat: despite these readings showing no formal nystagmus parameters, the raw traces show significant eye movement variability (wide amplitude swings in raw waveforms), which could reflect poor fixation, cooperation issues, or background drift - these warrant careful clinical correlation.

6. POSITIONAL TESTS

Dix-Hallpike Right

PositionFinding
Sit Head Right (initial)No nystagmus
Supine Head Ext. & RightRight eye: Vertical SPV 4.27°/s, Amplitude 4.35°, Frequency 1.25 Hz (Left eye: none)
Return to Sit (Head Right)Left eye: Horizontal SPV -13.78°/s, Amplitude -3.81°, Frequency 0.89 Hz
Interpretation: Positive Dix-Hallpike to the right - nystagmus in the head-hanging right position with a vertical component in the right eye and return nystagmus in the left eye. Pattern is consistent with Right Posterior Canal BPPV (geotropic vertical-torsional nystagmus on right Dix-Hallpike).

Dix-Hallpike Left

PositionFinding
Sit Head Left (initial)No nystagmus
Supine Head Ext. & LeftRight eye: Horizontal SPV -2.84°/s, Amp -2.39°; Vertical SPV 5.32°/s, Amp 3.59°, Fast Phase 236.38°, Freq 1.59 Hz; Left eye: Vertical SPV 5.81°/s, Amp 3.53°, Freq 0.79 Hz
Return to Sit (Head Left)Left eye: Horizontal SPV -15.88°/s, Amp -3.90°, Freq 1.32 Hz
Interpretation: Positive Dix-Hallpike to the left as well. The fast phase direction (236.38°) and the vertical/torsional pattern suggest Left Posterior Canal BPPV, or the possibility of a cupulolithiasis variant given persistent nystagmus. Bilateral Dix-Hallpike positivity suggests bilateral posterior canal BPPV or a central positional disorder.

Yacovino Test (Central BPPV Protocol)

PositionFinding
Supine BeginBoth eyes: Significant nystagmus - Right: Horizontal SPV -11.76°/s, Vertical SPV -53.38°/s, Fast Phase 102.23°, Freq 2.67 Hz; Left: Horizontal SPV -23.15°/s, Vertical SPV -62.10°/s, Fast Phase 119.05°, Freq 2.79 Hz
Supine Head Ext. 90°Left eye: Vertical SPV +14.65°/s, Amp 5.10°, Freq 1.21 Hz (Right eye: none)
Supine Head Flex 45°Right eye: Horizontal SPV 3.35°/s, Amp 1.87°, Freq 0.97 Hz (Left eye: none)
Supine EndNo nystagmus bilaterally
Interpretation: The Yacovino maneuver is designed for anterior canal BPPV or central positional nystagmus. The presence of significant bilateral nystagmus at the beginning of the supine position with a predominantly downbeat/vertical fast phase (102-119°) is strongly suggestive of anterior canal BPPV or a central positional disorder (midline cerebellar lesion). The high SPV values (up to 62°/s) and bilateral nature raise concern for a central etiology.

McClure-Pagnini Test (Horizontal Canal BPPV)

PositionFinding
Sit to SupineNo nystagmus
Right LateralRight eye: Horizontal SPV 6.10°/s, Amp 4.18°, Vertical SPV -5.77°/s, Amp -2.50°, Fast Phase 30.84°, Freq 1.88 Hz
Supine Head Neutral (1st)No nystagmus
Left LateralBoth eyes: Horizontal SPV -7.06°/s (R), -6.73°/s (L); Amp -2.41° (R), -2.07° (L); Freq 1.19 Hz (R), 1.27 Hz (L)
Supine Head Neutral (2nd)No nystagmus
Interpretation: Nystagmus in both lateral positions (right lateral and left lateral) with a horizontal fast phase. This is the typical pattern for Horizontal (Lateral) Canal BPPV. The fact that nystagmus was present in both lateral positions (geotropic pattern - toward the ground in both roll directions) suggests bilateral horizontal canal BPPV (canalithiasis type) or cupulolithiasis of the horizontal canal.

Head Position Tests

PositionFinding
Yaw RightRight eye: Horizontal SPV -6.93°/s, Amp -3.32°, Freq 1.23 Hz
Yaw LeftNo nystagmus
Pitch ForwardNo nystagmus
Pitch BackwardBoth eyes: Complex - Right: H-SPV 1.60°/s, V-SPV 1.98°/s, Fast Phase 349.49°, Freq 2.42 Hz; Left: H-SPV -1.15°/s, V-SPV -17.45°/s, Amp -4.42°, Fast Phase 104.73°, Freq 2.43 Hz
Roll RightRight eye: Vertical SPV -3.96°/s, Amp -1.90°, Freq 0.92 Hz
Roll LeftBoth eyes: H-SPV -8.42°/s (R), -8.32°/s (L); V-SPV +4.83°/s (R), +5.24°/s (L); Fast Phase 217.69° (R), 217.98° (L); Freq 1.95 Hz (R), 2.21 Hz (L)
Interpretation:
  • Yaw Right: Nystagmus in right ear-down position only - could reflect right horizontal canal involvement.
  • Pitch Backward: Bilateral nystagmus with fast phase near 350° (right eye) and 104° (left eye) - predominantly vertical, suggests posterior/anterior canal involvement or central positional nystagmus with head extension.
  • Roll Right: Mild vertical nystagmus in right eye only.
  • Roll Left: Bilateral nystagmus with combined horizontal and vertical components, fast phase at ~218° - suggests left anterior canal or central etiology.

7. SUBJECTIVE VISUAL VERTICAL (SVV)

TrialDeviationDirection
Clockwise - Trial 1Clockwise
Clockwise - Trial 2-1° (Left)Clockwise
Anticlockwise - Trial 1-1° (Left)Anticlockwise
Anticlockwise - Trial 2-1° (Left)Anticlockwise
Blank BG - Trial 1Clockwise
Blank BG - Trial 2Clockwise
Normal SVV range: within ±2.5°
Finding: All deviations are within ±1° - SVV is NORMAL. This argues against significant utricle dysfunction or an acute unilateral otolithic lesion.

Overall Summary & Interpretation

DomainFindingSignificance
SaccadesProlonged latency, reduced precision (left > right)Possible central (brainstem/frontal lobe) dysfunction
Smooth PursuitSeverely reduced gain all directions, both eyesStrong indicator of central (cerebellar/brainstem) pathology
OKNNormal, symmetricReassuring - no major unilateral cortical/peripheral deficit
Spontaneous NystagmusAbsentNormal
Gaze NystagmusAbsentNormal
Head Shake / HyperventilationAbsentNormal
Dix-Hallpike (Right)Positive - Vertical nystagmus on rightRight Posterior Canal BPPV
Dix-Hallpike (Left)Positive - Vertical/torsional nystagmus on leftLeft Posterior Canal BPPV
Yacovino TestPositive with high-velocity downbeat nystagmusAnterior canal BPPV or Central positional disorder
McClure-PagniniPositive bilaterally (horizontal nystagmus)Bilateral Horizontal Canal BPPV
Head Position TestsMultiple positions positiveMulticanal / central involvement
SVVNormal (within ±1°)No utricular lesion

Clinical Impression

This VNG profile in a 66-year-old female reveals two distinct patterns:
  1. Peripheral vestibular component (BPPV): Multiple positional tests are positive - Dix-Hallpike bilaterally, McClure-Pagnini bilaterally, and head position tests. The pattern suggests multicanal BPPV (possibly involving bilateral posterior canals and bilateral horizontal canals simultaneously), which is uncommon but recognized, particularly post-head trauma, in elderly patients, or following vestibular neuritis.
  2. Central vestibular component (suspected): The severely reduced smooth pursuit gain in all directions, prolonged saccadic latency, the pattern of the Yacovino test (high-velocity vertical nystagmus in the supine position), and direction-changing positional nystagmus across multiple head positions collectively point to possible central vestibular dysfunction (cerebellar or brainstem). The normal SVV and absent spontaneous nystagmus do not exclude a central lesion.
Recommended next steps:
  • MRI brain with contrast (posterior fossa focus) to rule out cerebellar or brainstem lesion
  • Canalith repositioning maneuvers (Epley/BBQ roll) for BPPV canals identified
  • Neurological consultation given the central features
  • Consider caloric or vHIT testing to further quantify peripheral vestibular function per canal
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