Gait analysis Ortho in detail

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Gait Analysis - Detailed Orthopaedic Review


1. Definitions and Terminology

Gait is the pattern of locomotion characterized by coordinated limb movements.
  • Step: Distance between successive initial contact (IC) by the two lower limbs (one foot to the opposite foot)
  • Stride: Distance between successive IC by the same lower limb (= two steps; one complete gait cycle)
  • Cadence: Number of steps per unit of time (steps/min)
  • Velocity: Ratio of distance to time (cadence x step length)
  • Walking is defined by always having at least one foot in contact with the ground, plus a period of double-limb support
  • Running differs from walking in that it eliminates double-limb support and introduces a float phase when neither foot touches the ground

2. The Gait Cycle

A complete gait cycle runs from foot strike to successive ipsilateral foot strike.
Gait Cycle Diagram - Step length, stride, stance and swing phases
Fig. 10.1 - Dimensions of the walking cycle showing step length, stride (cycle length), stance, and swing phases with double-limb support intervals. (Miller's Review of Orthopaedics 9th Ed.)
The cycle is divided into two main phases:
PhaseDurationDescription
Stance phase60% of cycleWeight-bearing; from IC to toe-off
Swing phase40% of cycleNon-weight-bearing; from toe-off to next IC
There are two periods of double-limb support (during IC + loading response and during preswing), comprising 20-30% of the cycle; these periods are velocity-dependent.

3. Subdivisions of the Gait Cycle

Stance Phase (60%)

Sub-phaseEventKey Feature
Initial Contact (IC) / Heel StrikeReference foot contacts groundFoot dorsiflexed; tibialis anterior contracts eccentrically
Loading Response (LR)IC of reference foot → initial swing of contralateral footKnee flexes ~15° to dampen load; subtalar joint everted (unlocks transverse tarsal joint for shock absorption)
Midstance (MSt)Initial swing of advancing foot → body's CoG directly over supporting forefootSingle-leg stance; CoG passes over weight-bearing leg
Terminal Stance (TSt)Heel rise → IC of contralateral footGastrocnemius and plantar flexors contract; subtalar joint inverts, rigidifying midfoot for propulsion
Preswing (PSw)IC of contralateral limb → stance foot lifts off groundMaximal subtalar inversion; maximum midfoot rigidity at toe-off

Swing Phase (40%)

Sub-phaseEventKey Feature
Initial Swing (ISw)Foot leaves ground → swinging foot opposite stance footHip flexors propel leg forward
Midswing (MSw)Swinging limb forward → tibia perpendicular to groundTibialis anterior and dorsiflexors contract to clear foot
Terminal Swing (TSw)Tibia perpendicular → foot makes ICHamstrings contract eccentrically to decelerate limb before heel strike

4. Kinetics and Kinematics

Kinetics and Kinematics Graph - Hip, Knee, and Ankle motion through the gait cycle
Fig. 10.2 - Joint angles (hip, knee, ankle) through each phase of the gait cycle with corresponding muscle activity. Red portions = ground reaction force anterior to hip, posterior to knee, and anterior to ankle during stance. (Miller's Review of Orthopaedics 9th Ed.)
Key joint motion values:
  • Hip: ~30° flexion at IC → extends to ~10° extension at TSt → 30°+ flexion during swing
  • Knee: ~5° flexion at IC → 15-18° flexion at LR (shock absorption) → near-full extension at MSt → ~65° flexion in swing
  • Ankle: ~5° plantar flexion at IC → 0° at neutral through MSt → ~10° dorsiflexion at TSt → ~20° plantar flexion at PSw/toe-off → returns to neutral during swing

Ground Reaction Force (GRF)

  • The mean load-bearing vector that changes in magnitude and direction throughout the cycle
  • It determines the moment/torque exerted on each joint
  • During stance: GRF is anterior to the hip, posterior to the knee, and anterior to the ankle

Joint Loading

  • Knee forces: 4-7x body weight during stance; 70% of load passes through the medial compartment
  • During water walking: significant decrease in joint moments due to buoyancy

5. Muscle Actions During Gait

Most muscle activity during gait is eccentric (muscle active while lengthening):
MuscleType of ContractionFunction
Gluteus mediusEccentricControls pelvic tilt during midstance
Gluteus maximusConcentricPowers hip extension
IliopsoasConcentricPowers hip flexion (swing phase)
Hip adductorsEccentricControl lateral sway (late stance)
QuadricepsEccentricStabilize knee at initial contact and preswing
HamstringsEccentricControl rate of knee extension at swing (deceleration at TSw)
Tibialis anteriorEccentric (IC) / Concentric (swing)Slows plantar flexion rate at IC; dorsiflexes ankle in swing
Gastrocnemius-soleusEccentricSlows dorsiflexion rate during stance; powers push-off

6. Determinants of Gait (6 Classic Determinants - Saunders/Inman)

Six processes minimize vertical and lateral displacement of the center of mass, maximizing energy efficiency:
  1. Pelvic rotation - Pelvis externally rotates from IC to preswing, internally during preswing and swing; reduces total vertical displacement needed for limb advancement
  2. Pelvic list (tilt) - Non-weight-bearing contralateral side drops 5°, reducing superior deviation of CoM
  3. Knee flexion at loading - ~15° stance-phase knee flexion dampens impact at initial loading
  4. Foot and ankle motion - Subtalar damping at LR; stability at midstance; propulsion efficiency at push-off
  5. Knee motion - Works with foot/ankle to decrease unnecessary limb motion; flexes at IC, extends at MSt
  6. Control of pelvic lateral displacement - ~5 cm motion over weight-bearing limb; narrows base of support, increases stance stability
Center of mass displacement:
  • Vertical: sinusoidal curve
  • Lateral: sinusoidal curve with amplitude of ~6 cm
  • Body's CoM is located 2 cm anterior to S2
  • Trunk's CoG is just anterior to T10 (~33 cm above hip joints in 184 cm individual)

7. Pathologic Gait Patterns

Factors causing abnormal gait: muscle weakness, neurological conditions, pain, limb deformity, joint disease.

Antalgic Gait

  • Cause: Pain (hip/knee arthritis, stress fracture, infection)
  • Mechanism: Patient shortens the stance phase on the painful limb to reduce load time; contralateral swing phase is more rapid
  • Pattern: Dot-dash cadence (short stance, rapid swing); head dips on affected side

Trendelenburg Gait

  • Cause: Weakness of hip abductors (gluteus medius); positive Trendelenburg sign = pelvis sags >2 cm during single-leg stance
  • Mechanism: Failure to stabilize pelvis during single-limb support; pelvis drops on the unsupported (contralateral) side
  • Pattern: Trunk lurches toward the affected side (compensated Trendelenburg = "waddling" if bilateral)
  • Clinical associations: Superior gluteal nerve injury, polio, OA hip, total hip arthroplasty complications

High-Stepping / Steppage Gait (Foot Drop)

  • Cause: Weakness of ankle dorsiflexors (tibialis anterior); common peroneal nerve palsy, L4/L5 root lesion
  • Mechanism: Inability to clear foot during swing; exaggerated knee and hip flexion to clear foot from ground
  • Pattern: Foot slaps ground at IC; hyperextension moment through knee in stance

Scissor Gait

  • Cause: Overactive/spastic hip adductors (cerebral palsy, spastic diplegia)
  • Mechanism: Adductor overactivity causes knees to cross midline; legs cross like scissors
  • Pattern: Narrow base, knees crossing, typical in spastic CP; often combined with equinus

Crouch Gait

  • Cause: Hamstring contracture, plantar flexor weakness, or cerebral palsy
  • Mechanism: Excessive knee flexion throughout stance; increased dorsiflexion
  • Pattern: "Crouching" posture; increased joint loading

Circumduction Gait

  • Cause: Long limb, stiff knee, abductor shortening/overuse
  • Mechanism: Swinging limb cannot flex normally, so patient circumducts it in an arc laterally to clear the floor

Hip Hiking Gait

  • Cause: Long limb, stiff knee, hamstring weakness
  • Mechanism: Elevation of pelvis on swing side to clear a stiff or long limb

Stiff / Ankylosed Hip Gait

  • Cause: Hip arthrodesis or fusion
  • Pattern: Head rocks to and fro; compensatory lumbosacral motion

Ataxic Gait

  • Cause: Cerebellar pathology
  • Pattern: Wide base, irregular cadence, lurching, incoordinated
Gait TypePrimary CauseKey Feature
AntalgicPainShort stance phase; dot-dash cadence
TrendelenburgAbductor weaknessTrunk lurches to affected side
Steppage/High-steppingFoot drop (peroneal nerve)Exaggerated hip/knee flexion; foot slap
ScissorAdductor spasticity (CP)Knees cross midline
CrouchHamstring contracture / CPExcessive knee flexion throughout
CircumductionStiff knee / long limbLateral arc of swinging limb
Hip hikingStiff knee / long limbPelvis elevates on swing side
AtaxicCerebellar diseaseWide base, irregular, lurching

8. Gait Analysis Methods

Clinical Observation (Most Common)

  • Watch patient walk from front, sides, and back
  • Study one joint at a time: pelvis, hip, knee, ankle, foot
  • Note stride length, cadence, rotational alignment, trunk position, and side-to-side differences

Quantitative Gait Analysis

Modern labs use:
  • High-speed motion capture cameras from multiple angles
  • Retroreflective markers aligned with palpable skeletal landmarks
  • Force platforms (measure GRF)
  • Kinematic data presented as 3D waveforms for each joint
  • Electromyography (EMG) - documents muscle activation timing; identifies out-of-phase firing
  • Pedobarography - plantar pressure/foot pressure mapping
  • Oxygen consumption measurement (energy expenditure)
Quantitative gait analysis in preoperative planning for CP: when given to experienced observers after clinical recommendations were made, the surgical recommendation changed 52% of the time - Campbell's Operative Orthopaedics 15th Ed 2026.

Key Clinical Uses

  • Preoperative planning in cerebral palsy and lower extremity surgery
  • Postoperative outcome assessment
  • Making further recommendations for bracing and physiotherapy
  • Identifying gait deviations and muscle out-of-phase firing patterns

9. Gait Abnormalities by Phase (Summary Table)

PhaseDeviationPossible Cause
IC-MStFoot slapTibialis anterior weakness
IC-MStGenu recurvatumQuadriceps spasticity/shortness; plantar flexor spasticity
IC-MStExcessive trunk extensionGluteus maximus weakness; hip extensor weakness
IC-PSwExcessive trunk flexionHip flexion contracture; quadriceps weakness
IC-PSwCrouch (excessive knee flexion)Hamstring contracture; plantar flexor weakness
IC-PSwMedial femoral rotationMedial hamstring tightness
LR-PSwWide base of supportAbductor contracture
MSt-PSwExcessive foot pronationValgus forefoot; pes planus; tibialis posterior weakness
MSt-PSwBouncing/insufficient push-offAchilles contracture; plantar flexor weakness
SwingSteppage gaitDorsiflexion weakness; equinus deformity
SwingCircumductionAbductor shortening; knee stiffness; long limb
SwingHip hikingHamstring weakness; long limb

10. Metabolic Cost of Gait

  • Metabolic cost of walking increases with proximal-level amputations and is inversely proportional to the length of the residual limb and number of functional joints preserved
  • Body weight is carried 70% by head, neck, trunk, and arms
  • Trunk's CoG is ~33 cm above the hip joints
  • Whole-body CoM is 2 cm anterior to S2

Sources:
  • Miller's Review of Orthopaedics 9th Ed, Chapter 10 (Gait Biomechanics)
  • Campbell's Operative Orthopaedics 15th Ed 2026, Gait Analysis Section
  • Bailey and Love's Short Practice of Surgery 28th Ed, Table 35.1 (Types of limp)
  • Imaging Anatomy: Bones, Joints, Vessels and Nerves (Thieme Atlas), Gait section
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