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Gait Analysis - Detailed Orthopaedic Review
1. Definitions and Terminology
Gait is the pattern of locomotion characterized by coordinated limb movements.
- Step: Distance between successive initial contact (IC) by the two lower limbs (one foot to the opposite foot)
- Stride: Distance between successive IC by the same lower limb (= two steps; one complete gait cycle)
- Cadence: Number of steps per unit of time (steps/min)
- Velocity: Ratio of distance to time (cadence x step length)
- Walking is defined by always having at least one foot in contact with the ground, plus a period of double-limb support
- Running differs from walking in that it eliminates double-limb support and introduces a float phase when neither foot touches the ground
2. The Gait Cycle
A complete gait cycle runs from foot strike to successive ipsilateral foot strike.
Fig. 10.1 - Dimensions of the walking cycle showing step length, stride (cycle length), stance, and swing phases with double-limb support intervals. (Miller's Review of Orthopaedics 9th Ed.)
The cycle is divided into two main phases:
| Phase | Duration | Description |
|---|
| Stance phase | 60% of cycle | Weight-bearing; from IC to toe-off |
| Swing phase | 40% of cycle | Non-weight-bearing; from toe-off to next IC |
There are two periods of double-limb support (during IC + loading response and during preswing), comprising 20-30% of the cycle; these periods are velocity-dependent.
3. Subdivisions of the Gait Cycle
Stance Phase (60%)
| Sub-phase | Event | Key Feature |
|---|
| Initial Contact (IC) / Heel Strike | Reference foot contacts ground | Foot dorsiflexed; tibialis anterior contracts eccentrically |
| Loading Response (LR) | IC of reference foot → initial swing of contralateral foot | Knee flexes ~15° to dampen load; subtalar joint everted (unlocks transverse tarsal joint for shock absorption) |
| Midstance (MSt) | Initial swing of advancing foot → body's CoG directly over supporting forefoot | Single-leg stance; CoG passes over weight-bearing leg |
| Terminal Stance (TSt) | Heel rise → IC of contralateral foot | Gastrocnemius and plantar flexors contract; subtalar joint inverts, rigidifying midfoot for propulsion |
| Preswing (PSw) | IC of contralateral limb → stance foot lifts off ground | Maximal subtalar inversion; maximum midfoot rigidity at toe-off |
Swing Phase (40%)
| Sub-phase | Event | Key Feature |
|---|
| Initial Swing (ISw) | Foot leaves ground → swinging foot opposite stance foot | Hip flexors propel leg forward |
| Midswing (MSw) | Swinging limb forward → tibia perpendicular to ground | Tibialis anterior and dorsiflexors contract to clear foot |
| Terminal Swing (TSw) | Tibia perpendicular → foot makes IC | Hamstrings contract eccentrically to decelerate limb before heel strike |
4. Kinetics and Kinematics
Fig. 10.2 - Joint angles (hip, knee, ankle) through each phase of the gait cycle with corresponding muscle activity. Red portions = ground reaction force anterior to hip, posterior to knee, and anterior to ankle during stance. (Miller's Review of Orthopaedics 9th Ed.)
Key joint motion values:
- Hip: ~30° flexion at IC → extends to ~10° extension at TSt → 30°+ flexion during swing
- Knee: ~5° flexion at IC → 15-18° flexion at LR (shock absorption) → near-full extension at MSt → ~65° flexion in swing
- Ankle: ~5° plantar flexion at IC → 0° at neutral through MSt → ~10° dorsiflexion at TSt → ~20° plantar flexion at PSw/toe-off → returns to neutral during swing
Ground Reaction Force (GRF)
- The mean load-bearing vector that changes in magnitude and direction throughout the cycle
- It determines the moment/torque exerted on each joint
- During stance: GRF is anterior to the hip, posterior to the knee, and anterior to the ankle
Joint Loading
- Knee forces: 4-7x body weight during stance; 70% of load passes through the medial compartment
- During water walking: significant decrease in joint moments due to buoyancy
5. Muscle Actions During Gait
Most muscle activity during gait is eccentric (muscle active while lengthening):
| Muscle | Type of Contraction | Function |
|---|
| Gluteus medius | Eccentric | Controls pelvic tilt during midstance |
| Gluteus maximus | Concentric | Powers hip extension |
| Iliopsoas | Concentric | Powers hip flexion (swing phase) |
| Hip adductors | Eccentric | Control lateral sway (late stance) |
| Quadriceps | Eccentric | Stabilize knee at initial contact and preswing |
| Hamstrings | Eccentric | Control rate of knee extension at swing (deceleration at TSw) |
| Tibialis anterior | Eccentric (IC) / Concentric (swing) | Slows plantar flexion rate at IC; dorsiflexes ankle in swing |
| Gastrocnemius-soleus | Eccentric | Slows dorsiflexion rate during stance; powers push-off |
6. Determinants of Gait (6 Classic Determinants - Saunders/Inman)
Six processes minimize vertical and lateral displacement of the center of mass, maximizing energy efficiency:
- Pelvic rotation - Pelvis externally rotates from IC to preswing, internally during preswing and swing; reduces total vertical displacement needed for limb advancement
- Pelvic list (tilt) - Non-weight-bearing contralateral side drops 5°, reducing superior deviation of CoM
- Knee flexion at loading - ~15° stance-phase knee flexion dampens impact at initial loading
- Foot and ankle motion - Subtalar damping at LR; stability at midstance; propulsion efficiency at push-off
- Knee motion - Works with foot/ankle to decrease unnecessary limb motion; flexes at IC, extends at MSt
- Control of pelvic lateral displacement - ~5 cm motion over weight-bearing limb; narrows base of support, increases stance stability
Center of mass displacement:
- Vertical: sinusoidal curve
- Lateral: sinusoidal curve with amplitude of ~6 cm
- Body's CoM is located 2 cm anterior to S2
- Trunk's CoG is just anterior to T10 (~33 cm above hip joints in 184 cm individual)
7. Pathologic Gait Patterns
Factors causing abnormal gait: muscle weakness, neurological conditions, pain, limb deformity, joint disease.
Antalgic Gait
- Cause: Pain (hip/knee arthritis, stress fracture, infection)
- Mechanism: Patient shortens the stance phase on the painful limb to reduce load time; contralateral swing phase is more rapid
- Pattern: Dot-dash cadence (short stance, rapid swing); head dips on affected side
Trendelenburg Gait
- Cause: Weakness of hip abductors (gluteus medius); positive Trendelenburg sign = pelvis sags >2 cm during single-leg stance
- Mechanism: Failure to stabilize pelvis during single-limb support; pelvis drops on the unsupported (contralateral) side
- Pattern: Trunk lurches toward the affected side (compensated Trendelenburg = "waddling" if bilateral)
- Clinical associations: Superior gluteal nerve injury, polio, OA hip, total hip arthroplasty complications
High-Stepping / Steppage Gait (Foot Drop)
- Cause: Weakness of ankle dorsiflexors (tibialis anterior); common peroneal nerve palsy, L4/L5 root lesion
- Mechanism: Inability to clear foot during swing; exaggerated knee and hip flexion to clear foot from ground
- Pattern: Foot slaps ground at IC; hyperextension moment through knee in stance
Scissor Gait
- Cause: Overactive/spastic hip adductors (cerebral palsy, spastic diplegia)
- Mechanism: Adductor overactivity causes knees to cross midline; legs cross like scissors
- Pattern: Narrow base, knees crossing, typical in spastic CP; often combined with equinus
Crouch Gait
- Cause: Hamstring contracture, plantar flexor weakness, or cerebral palsy
- Mechanism: Excessive knee flexion throughout stance; increased dorsiflexion
- Pattern: "Crouching" posture; increased joint loading
Circumduction Gait
- Cause: Long limb, stiff knee, abductor shortening/overuse
- Mechanism: Swinging limb cannot flex normally, so patient circumducts it in an arc laterally to clear the floor
Hip Hiking Gait
- Cause: Long limb, stiff knee, hamstring weakness
- Mechanism: Elevation of pelvis on swing side to clear a stiff or long limb
Stiff / Ankylosed Hip Gait
- Cause: Hip arthrodesis or fusion
- Pattern: Head rocks to and fro; compensatory lumbosacral motion
Ataxic Gait
- Cause: Cerebellar pathology
- Pattern: Wide base, irregular cadence, lurching, incoordinated
| Gait Type | Primary Cause | Key Feature |
|---|
| Antalgic | Pain | Short stance phase; dot-dash cadence |
| Trendelenburg | Abductor weakness | Trunk lurches to affected side |
| Steppage/High-stepping | Foot drop (peroneal nerve) | Exaggerated hip/knee flexion; foot slap |
| Scissor | Adductor spasticity (CP) | Knees cross midline |
| Crouch | Hamstring contracture / CP | Excessive knee flexion throughout |
| Circumduction | Stiff knee / long limb | Lateral arc of swinging limb |
| Hip hiking | Stiff knee / long limb | Pelvis elevates on swing side |
| Ataxic | Cerebellar disease | Wide base, irregular, lurching |
8. Gait Analysis Methods
Clinical Observation (Most Common)
- Watch patient walk from front, sides, and back
- Study one joint at a time: pelvis, hip, knee, ankle, foot
- Note stride length, cadence, rotational alignment, trunk position, and side-to-side differences
Quantitative Gait Analysis
Modern labs use:
- High-speed motion capture cameras from multiple angles
- Retroreflective markers aligned with palpable skeletal landmarks
- Force platforms (measure GRF)
- Kinematic data presented as 3D waveforms for each joint
- Electromyography (EMG) - documents muscle activation timing; identifies out-of-phase firing
- Pedobarography - plantar pressure/foot pressure mapping
- Oxygen consumption measurement (energy expenditure)
Quantitative gait analysis in preoperative planning for CP: when given to experienced observers after clinical recommendations were made, the surgical recommendation changed 52% of the time - Campbell's Operative Orthopaedics 15th Ed 2026.
Key Clinical Uses
- Preoperative planning in cerebral palsy and lower extremity surgery
- Postoperative outcome assessment
- Making further recommendations for bracing and physiotherapy
- Identifying gait deviations and muscle out-of-phase firing patterns
9. Gait Abnormalities by Phase (Summary Table)
| Phase | Deviation | Possible Cause |
|---|
| IC-MSt | Foot slap | Tibialis anterior weakness |
| IC-MSt | Genu recurvatum | Quadriceps spasticity/shortness; plantar flexor spasticity |
| IC-MSt | Excessive trunk extension | Gluteus maximus weakness; hip extensor weakness |
| IC-PSw | Excessive trunk flexion | Hip flexion contracture; quadriceps weakness |
| IC-PSw | Crouch (excessive knee flexion) | Hamstring contracture; plantar flexor weakness |
| IC-PSw | Medial femoral rotation | Medial hamstring tightness |
| LR-PSw | Wide base of support | Abductor contracture |
| MSt-PSw | Excessive foot pronation | Valgus forefoot; pes planus; tibialis posterior weakness |
| MSt-PSw | Bouncing/insufficient push-off | Achilles contracture; plantar flexor weakness |
| Swing | Steppage gait | Dorsiflexion weakness; equinus deformity |
| Swing | Circumduction | Abductor shortening; knee stiffness; long limb |
| Swing | Hip hiking | Hamstring weakness; long limb |
10. Metabolic Cost of Gait
- Metabolic cost of walking increases with proximal-level amputations and is inversely proportional to the length of the residual limb and number of functional joints preserved
- Body weight is carried 70% by head, neck, trunk, and arms
- Trunk's CoG is ~33 cm above the hip joints
- Whole-body CoM is 2 cm anterior to S2
Sources:
- Miller's Review of Orthopaedics 9th Ed, Chapter 10 (Gait Biomechanics)
- Campbell's Operative Orthopaedics 15th Ed 2026, Gait Analysis Section
- Bailey and Love's Short Practice of Surgery 28th Ed, Table 35.1 (Types of limp)
- Imaging Anatomy: Bones, Joints, Vessels and Nerves (Thieme Atlas), Gait section