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Shoulder Impingement Syndrome - Detailed 10 Marker
Definition
Shoulder impingement syndrome (SIS) is defined as the encroachment of the acromion, coracoacromial ligament, coracoid process, or acromioclavicular (AC) joint on the rotator cuff tendons as they pass beneath the coracoacromial arch during glenohumeral motion. The supraspinatus tendon is most commonly affected, compressed between the greater tuberosity of the humerus and the anteroinferior surface of the acromion.
Originally described by Neer in 1972 after 100 anatomic shoulder dissections, it remains one of the most common causes of non-traumatic shoulder pain.
Anatomy of the Subacromial Space
The subacromial space is bounded:
- Superiorly: Acromion, AC joint, coracoacromial ligament (forming the coracoacromial arch)
- Inferiorly: Humeral head and greater tuberosity
- Contents: Supraspinatus tendon, subacromial bursa, long head of biceps tendon
Normal subacromial space = 9-10 mm. Narrowing below 7 mm is considered pathological.
Types of Acromion (Bigliani/Morrison Classification)
The shape of the acromion plays a major role in impingement. Bigliani and Morrison (1986) classified acromion morphology into three types based on lateral shoulder X-ray (outlet view):
Figure: Three types of acromion morphology. Type I has the least compromise of the supraspinatus outlet; Type III has the highest rate of rotator cuff pathology. (Campbell's Operative Orthopaedics 15th Ed, Fig. 51.24)
| Type | Shape | Description | Impingement Risk |
|---|
| Type I | Flat | Flat undersurface, normal slope | Lowest - rarely associated with impingement |
| Type II | Curved | Gentle concave curve on undersurface | Moderate - most common type (45%), moderately associated with tears |
| Type III | Hooked | Anterior hook (slope 43°, acromial angle <70°) | Highest - strongly associated with rotator cuff tears and severe impingement |
A Type IV (convex) acromion has also been described but is less widely used clinically.
Key notes:
- Genetics play a role in subacromial space shape
- Type III hooked acromions have a reduced supraspinatus outlet and highest rate of rotator cuff pathology
- Acromions with less slope have a propensity toward impingement
- Anterior acromial spurs (traction osteophytes from coracoacromial ligament) can convert a Type I/II into an effectively Type III morphology
Types of Impingement
1. Primary (External) Impingement
Classic Neer-type impingement. Subdivided into:
- Intrinsic primary: Structures passing beneath the arch enlarge (e.g., rotator cuff thickening, calcium deposits, subacromial bursal thickening) - they abut the arch from below
- Extrinsic primary: Space above the cuff is diminished (subacromial spurring, acromial fracture, os acromiale, AC joint osteophytes, greater tuberosity exostoses)
2. Secondary Impingement
Caused by glenohumeral instability allowing abnormal translation of the humeral head (typically anteriorly), bringing the rotator cuff into contact with the coracoacromial arch. Common in younger patients with lax capsules or labral tears.
3. Internal Impingement
Contact of the articular surface of the rotator cuff with the posterosuperior glenoid rim occurs when the arm is in abduction, extension, and external rotation (the "cocked" throwing position). This is a normal phenomenon that becomes pathological in overhead athletes. Associated with glenohumeral internal rotation deficit (GIRD).
4. Subcoracoid Impingement
Impingement between the subscapularis tendon/lesser tuberosity and the coracoid process. Less common; associated with coracoid morphology or post-surgical changes.
Neer's Stages of Impingement (Pathological Progression)
| Stage | Pathology | Typical Age | Reversibility |
|---|
| Stage I | Oedema and haemorrhage in supraspinatus tendon/bursa | <25 years | Fully reversible with conservative treatment |
| Stage II | Fibrosis and tendinitis; irreversible changes begin | 25-40 years | Partial - may need surgery |
| Stage III | Tendon rupture (partial or full thickness), bone changes, anterior acromial spur | >40 years | Irreversible - surgery often required |
Aetiology / Risk Factors
Extrinsic (structural) factors:
- Type III (hooked) acromion
- Acromial osteophytes/spurs
- AC joint osteophytes impinging from above
- Os acromiale
- Subacromial narrowing
Intrinsic (tendon) factors:
- Age-related tendon degeneration: decreased cellularity, fascicular thinning, hypovacularity, dystrophic calcification - likely irreversible
- Degenerative tears associated with narrower subacromial space, larger lateral extension, steeper acromion angulation
- Shear stress from repetitive overhead activities
Dynamic/functional factors:
- Rotator cuff weakness leading to loss of force couples → superior humeral head translation
- Scapular dyskinesia (reduced scapular upward rotation narrows the outlet)
- Kyphosis reducing shoulder elevation
- Overhead sports (swimmers, throwers, racquet sports)
Clinical Features
Symptoms:
- Anterolateral shoulder pain, worsened by overhead activity
- Pain at night, difficulty lying on the affected shoulder
- "Painful arc" - pain between 60° and 120° of abduction
Signs:
- Tenderness over anterolateral acromion and supraspinatus insertion on greater tuberosity
- Painful arc of abduction (60°-120°)
- Weakness in abduction and external rotation (if cuff involved)
- Muscle wasting (supraspinatus/infraspinatus fossa) in chronic cases
Clinical Tests
| Test | Technique | Sensitivity | Specificity | Notes |
|---|
| Neer's sign | Passive forced forward flexion of arm (scapula stabilised) - pain = positive | 68% | 68.7% | Positive in all stages |
| Hawkins-Kennedy sign | Arm at 90° forward flexion, elbow flexed 90°, internal rotation - pain = positive | 71.5% | 66.3% | Most sensitive |
| Painful arc sign | Active abduction 60°-120° causes pain | 73.5% | 81.1% | High overall accuracy |
| Jobe (Supraspinatus) test | Arm at 90° abduction, 30° forward flexion, thumb down, resist downward force | 44.1% | 89.5% | Highly specific |
| Speed test | Resist forward flexion with elbow extended, forearm supinated | 38.3% | 83.3% | For bicipital involvement |
Clinical pearl (Campbell's): If Hawkins-Kennedy + painful arc + infraspinatus (Jobe) test are ALL positive, likelihood of impingement >95%. If all three are negative, likelihood <24%.
Neer's Impingement Test (injection test): Injection of 10 mL lidocaine into subacromial space - if pain is abolished, impingement is confirmed (distinguishes from glenohumeral pathology).
Investigations
Radiographs (X-ray):
- AP and lateral (outlet) views - outlet view best shows acromion type
- Early: normal or hooked acromion
- Late: sclerosis/cysts at anterior acromion and greater tuberosity, narrowed acromiohumeral gap (<7 mm suggests full-thickness tear), superior subluxation of humeral head, anterior acromial traction spur
MRI / MRI Arthrogram:
- Gold standard for soft tissue assessment
- Shows supraspinatus signal changes, partial vs full-thickness tears, bursal thickening
- Arthrogram best for partial-thickness tears
Ultrasound:
- Dynamic assessment, cost-effective, can detect bursal thickening and cuff tears
Management
Conservative (First Line)
- Rest and activity modification - avoid provocative overhead activities
- NSAIDs - anti-inflammatory analgesia
- Physiotherapy - rotator cuff strengthening, scapular stabilisation, posture correction; evidence shows equivalent outcome to surgery at 1, 2, and 5 years
- Subacromial corticosteroid injection - reduces inflammation, provides short-to-medium term relief; also serves as diagnostic injection
- Patient education - gradual return to activity
Surgical
Indicated when 3-6 months of conservative treatment fails.
- Subacromial decompression (acromioplasty): Removal of anteroinferior acromion, resection of coracoacromial ligament, bursectomy. Can be arthroscopic or open. Note: recent evidence questions additional benefit over structured exercise alone for atraumatic tears.
- Rotator cuff repair: For associated full-thickness tears (arthroscopic or mini-open)
- AC joint excision: If AC joint osteophytes contributing
Summary Points for 10 Markers
- Impingement = encroachment on rotator cuff by coracoacromial arch structures
- Bigliani's 3 acromion types: flat (I), curved (II), hooked (III) - Type III highest risk
- Types: primary (intrinsic/extrinsic), secondary (instability), internal (glenoid rim), subcoracoid
- Neer's 3 stages: oedema → fibrosis → tendon rupture
- Most common tendon affected: supraspinatus
- Hallmark symptom: painful arc 60°-120°
- Best combined tests: Hawkins-Kennedy + painful arc + Jobe (>95% likelihood)
- Neer's injection test confirms diagnosis
- MRI is gold standard imaging for soft tissue assessment
- Conservative physiotherapy = surgery outcomes at 5 years; acromioplasty for refractory cases
Sources: Campbell's Operative Orthopaedics 15th Ed 2026, p. 2823-2824; Firestein & Kelley's Textbook of Rheumatology (2-Volume Set), p. 889-891; Bigliani LU, Morrison DS, April EW (1986) - acromion morphology classification