10 most common ecg diagnosis and findings

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Here is a thorough, textbook-grounded breakdown of the 10 most common ECG diagnoses and findings:

10 Most Common ECG Diagnoses and Findings


1. Normal Sinus Rhythm (Baseline Reference)

Before recognizing pathology, every ECG is assessed against normal:
  • Rate: 60-100 bpm
  • P wave: upright in leads I and II, precedes every QRS
  • PR interval: 120-200 ms
  • QRS: <120 ms, axis -30° to +90°
  • QT interval: <450 ms (men), <470 ms (women)

2. Sinus Tachycardia

  • Rate: >100 bpm with a normal P-QRS-T sequence
  • P waves are upright and precede each QRS (1:1 relationship)
  • Most common dysrhythmia seen on ECG overall - present in ~40% of emergency patients with acute illness (e.g. PE, thyrotoxicosis, sepsis)
  • Not a primary arrhythmia - always look for the underlying cause

3. Atrial Fibrillation (AF)

One of the most clinically important and frequently encountered diagnoses:
  • Absent P waves - replaced by irregular, chaotic fibrillatory (f) waves, best seen in V1
  • Irregularly irregular ventricular response (RR intervals vary with no pattern)
  • Narrow QRS unless aberrant conduction or bundle branch block co-exists
  • When AF is associated with pulmonary hypertension, the ECG may show: R/S ratio in V1 >1, right axis deviation, and ST depressions in V1-V3 (Tintinalli's Emergency Medicine)
  • Common in mitral stenosis: ECG shows AF, left atrial enlargement, or right axis deviation

4. Left Ventricular Hypertrophy (LVH)

The most common ECG finding in hypertensive patients and dilated cardiomyopathy:
  • Voltage criteria - Several exist; all have poor sensitivity (30-50%) but good specificity (85-95%)
  • Cornell criterion: S in V3 + R in aVL >28 mm (men) or >20 mm (women)
  • Sokolow-Lyon: S in V1 + R in V5 or V6 >35 mm
  • Associated with ST depression and T wave inversion in lateral leads ("strain pattern")
  • LV hypertrophy + left atrial enlargement are the most common findings in dilated cardiomyopathy (Tintinalli's)

5. Right Bundle Branch Block (RBBB)

ECG hallmarks (from Goldman-Cecil and Braunwald's):
  • QRS duration ≥120 ms
  • rsR' pattern ("M-shape") in V1 - the classic finding
  • Wide, slurred terminal S wave in leads I, V5, V6
  • ST-segment downsloping and T wave inversion in right precordial leads (V1-V3) - discordant with QRS
  • Normal axis; septal Q waves present in lateral leads
  • Can be isolated or combined with left anterior fascicular block (bifascicular block)
RBBB - rSR' pattern in V1, wide S waves in lateral leads
RBBB - note the rSR' in V1 and wide terminal S wave in V5 (Goldman-Cecil Medicine)

6. Left Bundle Branch Block (LBBB)

ECG hallmarks:
  • QRS duration ≥120 ms
  • Broad, notched (M-shaped) complex in I, aVL, and left precordial leads (V5-V6)
  • Small r waves and broad, deep S waves in right precordial leads (V1-V3)
  • ST and T waves discordant (opposite to) the QRS throughout the precordium
  • Axis usually normal or deviated left
  • New LBBB in a patient with chest pain is treated as a STEMI equivalent
LBBB (E) and LPFB (D)
D: Left posterior fascicular block (right axis deviation ~120°). E: LBBB - broad notched R waves in lateral leads, deep S in right precordial leads (Goldman-Cecil Medicine)

7. ST-Elevation Myocardial Infarction (STEMI)

The highest-stakes ECG diagnosis:
  • ST elevation ≥1 mm in ≥2 contiguous limb leads, or ≥2 mm in ≥2 contiguous precordial leads
  • Reciprocal ST depression in opposite leads
  • Hyperacute T waves - earliest finding (tall, broad, asymmetric)
  • Pathological Q waves - develop within hours to days (>40 ms wide or >25% of R wave height)
  • Localizing territory:
    • Inferior (II, III, aVF) = RCA occlusion
    • Anterior (V1-V4) = LAD occlusion
    • Lateral (I, aVL, V5-V6) = LCx occlusion
  • STEMI management target: door-to-balloon <90 min; primary PCI preferred over thrombolysis (Textbook of Family Medicine)
  • New LBBB or posterior STEMI (ST depression V1-V3 with tall R waves) are STEMI equivalents

8. Non-Specific ST-T Wave Changes

The most common "abnormal" finding on routine ECGs:
  • ST depression or T wave inversion that does not meet ischemia criteria
  • T wave flattening or subtle inversion
  • Causes are extensive: ischemia, LVH strain, electrolyte disturbance (hypokalemia), digoxin effect, myocarditis, pericarditis, cardiomyopathy
  • Must always be interpreted in clinical context
  • Sinus tachycardia + nonspecific ST-T changes are the most common ECG findings in pulmonary embolism - they do NOT confirm or exclude the diagnosis

9. First-Degree AV Block (and Fascicular Blocks)

First-degree AV block:
  • PR interval >200 ms (>5 small squares)
  • Every P wave conducts to a QRS - no beats dropped
  • Often benign; seen in athletes, with inferior MI, or drug effect (beta-blockers, digoxin)
Fascicular blocks are also very common:
  • Left anterior fascicular block (LAFB): Left axis deviation (around -60°), normal QRS duration, small Q waves in I/aVL, small R waves in II/III/aVF
  • Left posterior fascicular block (LPFB): Right axis deviation (~+120°), delayed R-wave progression, requires exclusion of other causes of right axis deviation

10. Ventricular / Atrial Premature Beats

  • Premature Ventricular Complexes (PVCs): Wide, bizarre QRS (>120 ms), no preceding P wave, full compensatory pause. Very common - seen in up to 30% of monitored patients. Usually benign in isolation but complex forms (couplets, bigeminy, multifocal) warrant evaluation
  • Premature Atrial Complexes (PACs): Early, narrow QRS preceded by an abnormal P wave; often conducted with a slight PR change or blocked entirely
  • Both are documented in ECG recording tables from Braunwald's Heart Disease and Goldman-Cecil as top findings in long-term Holter monitoring

Summary Table

#DiagnosisKey ECG Feature
1Normal sinus rhythmRate 60-100, regular P-QRS-T
2Sinus tachycardiaRate >100, normal morphology
3Atrial fibrillationNo P waves, irregularly irregular RR
4LVHIncreased voltage ± strain pattern
5RBBBrSR' in V1, wide S in I/V5/V6
6LBBBBroad notched R in I/aVL/V6, deep S in V1
7STEMIST elevation ≥1-2 mm in contiguous leads
8Nonspecific ST-T changesST depression/T inversion, nondiagnostic
9First-degree AV block / fascicular blocksPR >200 ms / axis deviation
10PVCs / PACsWide bizarre QRS / early narrow QRS

Sources: Goldman-Cecil Medicine, Braunwald's Heart Disease, Tintinalli's Emergency Medicine, Textbook of Family Medicine - all via the medical textbook library.
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