This is a hospital discharge summary (page 3 of 5) from SVP — Sardar Vallabhbhai Patel Institute of Medical Sciences and Research, Ahmedabad, for a critically ill postpartum woman. Here is a full plain-language explanation:
Patient Details
| Field | Details |
|---|
| Name | Mrs. Kulsumbanu Mohammadakba Shaikh |
| Age | 33 years, 11 months |
| Gender | Female |
| Date of Admission | 02 March 2026 |
| Date of Discharge | 15 April 2026 |
| Department | General Medicine |
| Treating Unit | Unit A |
She had recently delivered a baby (the document mentions "postpartum uterus" and "IUCD in situ," meaning a contraceptive device was placed after delivery).
Imaging Reports — Plain Explanation
1. USG Both Breasts (31 March 2026) — Breast Ultrasound
- Lactational changes — her breasts show normal changes from breastfeeding (prominent milk ducts)
- No masses or cancer signs — reassuring, no lumps
- Minimal swelling (edema) and water-logging in the area around the nipple of the right breast — minor inflammation, likely related to her overall fluid overload
- Small lymph nodes in both armpits — likely reactive (responding to infection/inflammation), not suspicious for cancer
2. HRCT Thorax (21 March 2026) — High-Resolution CT Scan of the Chest
- Mild cardiomegaly — her heart is slightly enlarged
- Bilateral mild pleural effusions — small amounts of fluid have collected around both lungs
- Basal atelectasis — small collapsed areas at the bottom of the lungs (common with fluid around the lungs)
- Interstitial fluid tracking — fluid seeping into the lung tissue
- Hepatic infarct (right lobe, segments V, VII, VIII) — a portion of her liver has died due to lack of blood supply
- Ascites — fluid collecting inside the abdomen
These findings together point to serious multi-organ involvement.
3. CECT Abdomen (4 March 2026) — Contrast CT of the Abdomen
This is the most critical scan. Key findings:
Liver:
- Hepatomegaly — enlarged liver
- An 8×3×10 cm wedge-shaped dead area (hypodense = no blood supply) in the right lobe → hepatic infarct (a section of liver tissue has died)
Kidneys:
- Bilateral renal cortical necrosis — the outer layer (cortex) of both kidneys shows signs of death/damage from poor blood supply
- Kidney arteries themselves are normal; no stones or hydronephrosis (no blockage)
- This is a very serious finding — it is a known complication of HELLP syndrome and can cause permanent kidney damage or acute kidney failure
Gallbladder:
- Distended (swollen) with a thickened, edematous wall and sludge (8 mm) — likely inflamed due to the overall illness
Fluids and Edema:
- Mild ascites (fluid in abdomen)
- Subcutaneous and intermuscular edema in the abdominal wall — fluid leaking into body tissues
Other organs:
- Uterus is bulky (expected postpartum), IUCD seen in place
- Pancreas, spleen, adrenal glands, bowel, adnexa — all normal
- No abnormal lymph nodes
Clinical Suggestion: HELLP Syndrome — the radiologists explicitly raised this diagnosis.
What is HELLP Syndrome?
HELLP stands for:
- H — Hemolysis (destruction of red blood cells)
- EL — Elevated Liver enzymes (liver damage)
- LP — Low Platelets (bleeding risk)
It is a severe, life-threatening complication of pregnancy/the postpartum period, essentially a severe form of preeclampsia. It causes:
- Damage to the liver (including infarction, as seen here)
- Kidney failure
- Bleeding tendency
- Fluid overload throughout the body
It carries a mortality rate of 7–34% and requires urgent delivery (which had already occurred here) and intensive supportive care.
(Harrison's Principles of Internal Medicine 22E, p. 2490)
Treatment Details
Blood transfusions:
- 4 units of PCV (Packed Cell Volume = red blood cells) given across 4 dates in March 2026 — for severe anemia from hemolysis
- 4 units of PRC (Packed Red Cells) given on 3 March 2026
Hemodialysis (HD):
- 21 cycles of HD (dialysis) performed from 1 March through 14 April 2026 — because her kidneys were failing (renal cortical necrosis)
- Injection EPO (Erythropoietin) given on 10 March 2026 — a hormone injection to stimulate the bone marrow to produce more red blood cells
Drug Reaction:
- She was started on higher antibiotics during admission
- She developed DRESS syndrome (Drug Reaction with Eosinophilia and Systemic Symptoms) — likely due to Levofloxacin (an antibiotic)
- Levofloxacin was stopped
- She was treated with injectable steroids
- Dermatology consultation was obtained and a Drug Card was issued (warning card about the offending drug)
Ascitic Tap (8 March 2026):
- Fluid from her abdomen was drained and tested
- Result: High SAAG, Low Protein ascites
- High SAAG (Serum-Ascites Albumin Gradient >1.1) means the ascites is caused by portal hypertension (high pressure in the liver's blood supply) — consistent with severe liver damage from HELLP
Summary of the Clinical Picture
This patient had a very complicated postpartum course. She likely developed HELLP syndrome after delivery, which led to:
- Liver infarction (a large segment of liver died from loss of blood supply)
- Bilateral renal cortical necrosis → requiring prolonged dialysis (21 sessions over 6 weeks)
- Severe anemia → requiring multiple blood transfusions and EPO
- Pleural effusions and ascites (fluid around lungs and in abdomen) from portal hypertension/hypoalbuminemia
- DRESS drug reaction to Levofloxacin — an additional serious complication during her hospital stay
She was admitted for approximately 6 weeks (2 March – 15 April 2026), reflecting the severity and complexity of her illness. This is page 3 of 5 of her discharge summary, so there are likely more details on other pages.
If you are a family member or the patient herself, I strongly recommend discussing the follow-up kidney function tests, liver recovery, and long-term dialysis needs with the treating nephrology and hepatology teams, as both organ injuries can have lasting effects.