Ecpalin absolute bone conduction test and schwabach test

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Absolute Bone Conduction (ABC) Test and Schwabach Test

Both are tuning fork tests that assess bone conduction (BC) hearing. They are closely related - the ABC test is essentially the formal, clinical version of what the Schwabach test accomplishes.

Absolute Bone Conduction (ABC) Test

The ABC test measures the patient's bone conduction in isolation - that is, it tests how well sound vibrations transmitted through the skull reach the cochlea and auditory nerve, bypassing the outer and middle ear entirely.
Principle:
  • Bone conduction bypasses the external and middle ear and directly stimulates the cochlea.
  • It therefore reflects the sensorineural reserve (cochlear + nerve function).
  • A reduced ABC indicates cochlear or retrocochlear (sensorineural) pathology.
  • A normal or enhanced ABC in the presence of hearing loss points to a conductive lesion (outer/middle ear problem).
Procedure:
  • A tuning fork (typically 512 Hz) is struck and its base placed firmly on the mastoid process.
  • The patient is asked to report when they can no longer hear the tone.
  • The duration or loudness perceived is noted.
Clinical interpretation:
FindingMeaning
Normal ABCNormal sensorineural hearing OR conductive loss (cochlea/nerve intact)
Reduced/diminished ABCSensorineural hearing loss (cochlea or nerve damaged)
Key point: In conductive hearing loss, ABC is preserved or even enhanced - because the masking effect of ambient room noise is removed when the middle ear is blocked, making the bone-conducted tone relatively more prominent.

Schwabach Test

The Schwabach test is a comparative bone conduction test - it compares the patient's bone conduction to that of a known normal-hearing examiner.
Purpose: To compare the patient's bone conduction hearing to that of a person with normal hearing.
Procedure (KJ Lee's Essential Otolaryngology, p. 294):
  • The tuning fork is set into motion and its stem placed alternately on the mastoid process of the patient and the examiner.
  • When the patient no longer hears the sound, the examiner listens to the fork to check if the tone is still audible to them.
  • This alternation continues until a comparison is established.
Important prerequisite: The examiner's own hearing must be confirmed as normal before the test.
Interpretation:
ResultFindingMeaning
Normal SchwabachPatient and examiner stop hearing at the same timeNormal hearing
Diminished (Shortened) SchwabachPatient stops hearing before the examinerSensorineural hearing loss - cochlea/nerve cannot process BC as long as normal
Prolonged (Extended) SchwabachPatient hears longer than the examinerConductive hearing loss - middle ear blockage removes the masking effect of ambient noise, relatively enhancing perception of BC

Comparing Both Tests in Context of Hearing Loss

TestNormalConductive LossSensorineural Loss
ABCNormal BC durationPreserved/prolongedReduced
SchwabachSame as examinerProlonged (hears longer)Diminished (stops sooner)
RinneAC > BC (positive)BC > AC (negative)AC > BC (positive, both reduced)
WeberMidlineLateralizes to poorer (affected) earLateralizes to better ear

Why Conductive Loss Prolongs Bone Conduction?

In conductive hearing loss (e.g., otosclerosis, middle ear effusion, wax), the middle ear cannot transmit ambient environmental noise to the cochlea. This removes the background masking effect that normally partially obscures bone-conducted sounds. As a result, the patient's cochlea perceives the tuning fork vibration for longer than normal - hence prolonged Schwabach.
This is also why in the ABC test, bone conduction appears relatively normal or enhanced in purely conductive loss - the cochlea itself is intact.

Clinical Significance

  • The Schwabach test is particularly useful for differentiating conductive from sensorineural hearing loss using only a tuning fork.
  • It is not commonly used in modern practice because formal audiometry provides more precise, quantifiable results. However, it remains a valid bedside/clinical assessment tool.
  • A classic teaching scenario: a patient with otosclerosis will show prolonged Schwabach + negative Rinne + Weber lateralizing to the affected ear.
Sources: KJ Lee's Essential Otolaryngology, pp. 293-294; Ganong's Review of Medical Physiology, 26th ed., p. 227
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A 22-year-old male presented with right ear discharge for 8 months and gradually progressive hearing loss. The discharge was intermittent, scanty, mucoid, non-foul smelling, and increased after URTI or water entry into the ear. Write rhe case for this

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Clinical Case Write-Up

Case Presentation

Patient: 22-year-old male Presenting Complaints:
  1. Right ear discharge - 8 months duration
  2. Gradually progressive hearing loss in the right ear - 8 months duration

History of Present Illness

A 22-year-old male presented to the ENT OPD with complaints of right ear discharge for 8 months and gradually progressive hearing loss in the same ear over the same period.
The discharge was intermittent in onset, scanty in amount, and mucoid in character. It was non-foul smelling and non-bloodstained. The discharge characteristically increased following upper respiratory tract infections (URTIs) and after water entry into the ear (during bathing/swimming). There was no history of ear pain, tinnitus, vertigo, facial weakness, or headache. No previous similar episodes were reported in the past.
The hearing loss was gradual in onset and progressive in nature, involving the right ear. There was no sudden worsening, no fluctuation in hearing, and no associated tinnitus or aural fullness.
No history of:
  • Ear pain (otalgia)
  • Ear bleeding (haematotympanum)
  • Tinnitus or vertigo
  • Facial palsy or deviation
  • Headache or neck stiffness
  • Previous ear surgeries or trauma
  • Aural polyp noted by the patient

Past Medical History

  • No significant past medical history
  • No history of chronic nasal obstruction, allergic rhinitis, or recurrent URTIs (though discharge worsens with URTIs, suggesting underlying Eustachian tube dysfunction)
  • No history of diabetes mellitus, hypertension, or immunosuppression

General Examination

  • Well-built, well-nourished, afebrile
  • No pallor, icterus, cyanosis, clubbing, lymphadenopathy, or edema
  • Vital signs within normal limits

ENT Examination

External Ear

  • Pinna: normal shape, size, and position; no tenderness
  • Pre- and post-auricular areas: no swelling, redness, or sinus
  • External auditory canal (EAC): no signs of active discharge or stenosis

Otoscopic / Endoscopic Findings (Right Ear)

The otoendoscopic image reveals:
  • Tympanic membrane (TM): Thickened, dull, and pearly white in appearance with loss of normal translucency
  • Perforation: A central perforation located in the antero-inferior and postero-inferior quadrants (inferior pars tensa), approximately 30-40% of the total TM area - medium to large in size
  • Perforation margins: Smooth, rounded, and fully epithelialized - indicating a chronic, inactive (dry), stable perforation
  • Tympanosclerosis: Prominent chalky-white calcified plaque visible in the posterior quadrants of the TM (myringosclerosis) - consistent with longstanding chronic inflammation
  • Middle ear: Promontory visible through the perforation; middle ear mucosa appears dry, pale, and healthy - no active granulation tissue or mucosal edema
  • Malleus: Handle visible; umbo terminating near the superior-posterior edge of the perforation; mildly retracted
  • No cholesteatoma: No squamous debris, keratin pearls, or retraction pockets seen
  • Left ear: Normal (not shown)

Diagnosis

Chronic Suppurative Otitis Media (CSOM) - Tubotympanic (Safe/Mucosal) Type, Right Ear - Inactive (Dry) Phase

Basis of Diagnosis

FeatureSignificance
Intermittent, mucoid, non-foul dischargeTypical of tubotympanic/mucosal CSOM - mucus from metaplastic middle ear mucosa
Discharge worsening with URTIReflects Eustachian tube pathway - nasopharyngeal organisms ascend during URTIs
Discharge worsening with water entryWater contamination through the perforation activates middle ear infection
Non-foul smelling dischargeArgues against cholesteatoma/bony disease (unsafe type)
Central perforation in pars tensaHallmark of tubotympanic (safe) CSOM
No attic/pars flaccida perforationRules out atticoantral (unsafe) disease/cholesteatoma
Tympanosclerosis on TMSequela of chronic recurrent inflammation in the tympanic membrane
Conductive hearing loss (gradual)Due to: (a) perforation reducing TM surface area, (b) middle ear mucosal disease, (c) tympanosclerosis
No cholesteatoma featuresConfirms safe/benign type
Currently dry earInactive phase

Differential Diagnosis

  1. CSOM - Atticoantral (Unsafe) Type - Excluded by: central (not marginal/attic) perforation, mucoid (not foul-smelling, scanty, non-cheesy) discharge, no evidence of cholesteatoma on endoscopy
  2. Otitis Media with Effusion (Glue Ear) - Excluded by: presence of frank discharge through perforation
  3. Acute on Chronic Suppurative Otitis Media - Possible during exacerbations (URTI/water exposure), but currently in dry phase
  4. Tuberculous Otitis Media - Excluded by: no multiple perforations, no pale granulations, no painless discharge in atypical pattern

Investigations Planned

  1. Audiometry (Pure Tone Audiogram): Expected to show conductive hearing loss in the right ear (low-frequency > high-frequency loss); assess air-bone gap
  2. Tympanometry: Type B (flat) or type C curve expected due to perforation
  3. Throat swab / Ear swab for C&S: During active discharge phase - to identify organisms (commonly Pseudomonas aeruginosa, Staphylococcus aureus, Proteus, E. coli)
  4. High-Resolution CT Temporal Bone (HRCT): To assess:
    • Extent of middle ear disease
    • Ossicular chain integrity
    • Mastoid pneumatization
    • Any occult cholesteatoma
    • Tympanosclerotic involvement of ossicles
  5. Nasal examination / PNS X-ray or CT: To evaluate for adenoid hypertrophy, DNS, or chronic rhinosinusitis contributing to Eustachian tube dysfunction

Management

Conservative (Medical) Management - Inactive Phase

  • Ear toilet: aural cleaning and drying
  • Strict water precautions: use of ear plugs during bathing; avoid swimming
  • Treat underlying nasal/nasopharyngeal pathology (contributing to Eustachian tube dysfunction)
  • Topical antibiotic ear drops (ciprofloxacin/ofloxacin) during active discharge episodes
  • Systemic antibiotics during acute exacerbations

Surgical Management (Definitive)

  • Myringoplasty (Type I Tympanoplasty): Repair of the central perforation using a temporalis fascia graft - indicated once the ear has been dry for at least 6-8 weeks
    • Goals: close the perforation, prevent recurrent infections, and improve hearing
    • Approach: underlay or overlay technique
  • Tympanoplasty (Type II or higher): If ossicular chain involvement (erosion or fixation by tympanosclerosis) is found on intraoperative assessment
  • Tympanosclerotic plaques causing ossicular fixation may require ossiculoplasty

Expected Audiological Findings

TestExpected Result
Pure Tone AudiogramConductive hearing loss, right ear; air-bone gap present
Weber TestLateralizes to the right (affected) ear
Rinne TestNegative Rinne (BC > AC) on right
Schwabach TestProlonged (patient hears BC longer than examiner)
TympanometryType B (flat) or perforated TM pattern

Prognosis

The tubotympanic (safe) type carries a good prognosis with appropriate management. The disease does not invade bone and serious intracranial or extracranial complications are rare unless a virulent superinfection occurs. Successful myringoplasty results in elimination of discharge and improvement in conductive hearing loss in the majority of cases.
Sources: Shambaugh - Surgery of the Ear (Chapter 26, CSOM types, Table 26-2 & 26-3); Cummings Otolaryngology, p. 3072; KJ Lee's Essential Otolaryngology
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