Give a descriptiptive answer on normal labour, stages of labour and obstructed labour

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normal labour stages of labour obstructed labour clinical management 2024

I now have enough information to provide a thorough, well-structured answer. The medical library does not contain a dedicated obstetrics textbook, so I'll synthesize from my knowledge base confirmed by web sources.

Normal Labour, Stages of Labour, and Obstructed Labour


1. Normal Labour - Definition and Prerequisites

Normal (physiological) labour is defined as the process by which regular, spontaneous uterine contractions cause progressive cervical effacement and dilatation, ultimately resulting in the expulsion of the fetus and placenta through the birth canal.
WHO criteria for normal labour:
  • Spontaneous onset between 37-42 completed weeks of gestation
  • Single fetus in cephalic (vertex) presentation
  • No complications before or during labour
  • Spontaneous vaginal delivery
  • Healthy mother and baby at the end

The "Three Ps" Framework

Labour progress depends on the interaction of three key factors:
FactorComponents
PowersUterine contractions + maternal bearing-down efforts (secondary powers)
PassageBony pelvis (inlet, midpelvis, outlet) + soft tissues (cervix, vagina, perineum)
PassengerFetus - size, presentation, position, lie, and attitude

2. Physiology of Uterine Contractions

Contractions originate from the pacemaker region at the cornual junction of the uterus. They have:
  • Fundal dominance: the contraction starts at the fundus and sweeps down
  • Triple descending gradient: the contraction is strongest and longest at the fundus, progressively weakening downward
  • Polarity: the upper uterine segment (UUS) contracts and retracts; the lower uterine segment (LUS) dilates and thins passively
As labour progresses, the retraction ring (physiological retraction ring / Bandl's ring in pathology) forms at the junction of the UUS and LUS.

3. Stages of Labour

Labour is classically divided into three stages (some include a fourth).

Stage 1 - Cervical Effacement and Dilatation

Definition: From the onset of true labour to full cervical dilatation (10 cm).
Divided into two phases:
a) Latent Phase
  • Cervix effaces and dilates from 0 to 4 cm (Friedman) or 0-6 cm (newer ACOG 2014 criteria)
  • Irregular, mild contractions that gradually become more regular
  • Duration: up to 20 hours in nulliparae, up to 14 hours in multiparae
  • Prolonged latent phase: >20 hours (nullipara) or >14 hours (multipara)
b) Active Phase
  • Cervical dilatation from 4-6 cm (or 6 cm by ACOG) to 10 cm
  • Regular, strong contractions: typically 3-5 per 10 minutes, lasting 45-60 seconds, intensity 50-75 mmHg
  • Friedman's curve - expected dilatation rate: ≥1.2 cm/hour (nullipara) and ≥1.5 cm/hour (multipara)
  • More recent WHO/ACOG data shows: ≥1 cm/hour after 6 cm dilatation
  • Fetal descent begins in this phase, particularly in multiparae
Physiological changes in Stage 1:
  • Cervix effaces (shortens) before dilating (especially in nulliparae)
  • Forewaters (bag of membranes) may rupture spontaneously ("show")
  • Bloody show (pink-stained mucus) from displacement of cervical mucus plug
  • Engagement of fetal head typically precedes labour onset in nulliparae

Stage 2 - Delivery of the Fetus

Definition: From full cervical dilatation (10 cm) to complete delivery of the baby.
Duration:
  • Nullipara: up to 2 hours (3 hours with epidural)
  • Multipara: up to 1 hour (2 hours with epidural)
  • Prolonged Stage 2: exceeding these limits
Mechanism of Normal Labour (Cardinal Movements)
In a well-flexed cephalic vertex presentation in a gynecoid pelvis, the fetus undergoes a series of movements:
MovementDescription
1. EngagementBiparietal diameter (BPD) passes through the pelvic inlet; head enters at the transverse or oblique diameter
2. DescentProgressive downward movement of the fetal presenting part throughout all stages
3. FlexionHead flexes so chin approaches chest; suboccipito-bregmatic (9.5 cm) replaces larger diameters
4. Internal RotationHead rotates from transverse/oblique to anteroposterior in the midpelvis; occiput comes to lie under the symphysis pubis (OA position)
5. ExtensionHead extends under the pubic symphysis; occiput, bregma, face, and chin successively sweep over the perineum
6. RestitutionAfter delivery of head, it rotates back to its natural alignment with the shoulders
7. External RotationShoulders rotate internally in the pelvis; the head (externally) rotates further to the transverse
8. ExpulsionAnterior shoulder delivers first under the symphysis, then posterior shoulder, then body by lateral flexion
Monitoring in Stage 2:
  • Fetal heart rate (FHR) monitoring - auscultate after every contraction, or CTG continuously for high-risk
  • Maternal vital signs, progress of descent (station)
  • Perineal management - controlled delivery of head, episiotomy if indicated

Stage 3 - Delivery of the Placenta

Definition: From delivery of the baby to expulsion of the placenta and membranes.
Duration: Normally within 30 minutes; prolonged if >30 minutes without active management.
Phases of placental separation:
  1. Contraction of uterus after fetal delivery - uterus becomes globular and hard
  2. Gush of blood as retroplacental hematoma forms
  3. Lengthening of cord as placenta descends
  4. Expulsion of placenta - Schultze mechanism (central separation, shiny fetal surface first, ~80%) or Matthews-Duncan mechanism (marginal separation, maternal surface first, ~20%)
Active Management of Third Stage (AMTSL):
  • Oxytocin 10 IU IM/IV within 1 minute of birth (first-line agent)
  • Controlled cord traction (Brandt-Andrews manoeuvre) after signs of separation
  • Uterine massage after placenta delivery
  • Reduces postpartum haemorrhage (PPH) risk by ~60%
Signs of placental separation:
  • Uterus rises and becomes globular (Calkin's sign)
  • Sudden gush of blood
  • Cord lengthens at vulva
  • Fundus rises in the abdomen

Stage 4 (Immediate Postpartum Period)

  • Not universally classified as a formal stage, but clinically important
  • The first 1-2 hours following placental delivery
  • Monitored for primary PPH, uterine atony, perineal lacerations, vital signs
  • Uterus should remain firmly contracted (size of a grapefruit at umbilicus level)

4. Monitoring Labour - The Partograph

The partograph (WHO partograph) is the standard tool for monitoring labour:
  • Records: cervical dilatation, fetal descent, fetal heart rate, uterine contractions, maternal vitals, medications
  • Alert line: expected normal progress (1 cm/hr) from active phase onset
  • Action line: 4 hours to the right of the alert line
  • Crossing the action line signals the need for intervention

5. Obstructed Labour

Definition

Obstructed labour occurs when, despite strong uterine contractions, the fetus cannot descend through the birth canal due to a mechanical obstruction. The presenting part fails to advance despite adequate (or seemingly adequate) uterine powers.
It is distinct from "prolonged labour" (which is time-based) - obstruction is a mechanical failure of the passenger/passage relationship.

Causes of Obstruction

Fetal causes (Passenger):
  • Malpresentation: brow presentation (commonest malpresentation causing obstruction), face presentation (mentoposterior), shoulder presentation (transverse lie, completely obstructs)
  • Malposition: persistent occipito-posterior (OP), deep transverse arrest
  • Large baby (absolute or relative macrosomia)
  • Hydrocephalus, fetal anomalies
  • Locked twins
Maternal causes (Passage):
  • Contracted pelvis (pelvic inlet, midpelvis, or outlet contraction)
    • Causes: rickets, malnutrition, pelvic fractures
  • Cephalopelvic disproportion (CPD)
  • Cervical fibroid, ovarian cyst in the pelvis
  • Previous pelvic surgery/scar
  • Atresia or stenosis of the vagina/cervix

Clinical Features of Obstructed Labour

General signs:
  • Exhausted, anxious, dehydrated mother
  • Fever, tachycardia
  • Ketonuria, oliguria
  • Prolonged duration of labour
Abdominal signs:
  • Uterus tightly moulded over the fetus (loss of fetal parts palpability)
  • Bandl's pathological retraction ring: a palpable transverse groove/band across the lower abdomen at the junction of the UUS and LUS, indicating impending uterine rupture. The UUS becomes thick and short while the LUS becomes thin and stretched. This is a danger sign.
  • Round ligaments tense and tender (appearing as visible cords)
  • LUS tender on palpation, may show "hourglass" shape
Vaginal signs:
  • Presenting part deeply impacted, immovable
  • Excessive caput succedaneum (scalp oedema) and moulding (overlapping of skull bones) - Grade 3 moulding (bones overriding and not reducible) is ominous
  • Oedematous, congested cervix and vagina
  • Foetid liquor if membranes ruptured for >18 hours (risk of chorioamnionitis)
Bladder signs:
  • Urinary retention or haematuria (from bladder compression between fetal head and symphysis pubis) - indicates impending vesicovaginal fistula (VVF)

Grades of Moulding

GradeFinding
+Bones touching but not overlapping
++Bones overlapping but easily reducible
+++Bones overlapping and not reducible - OBSTRUCTED

Complications of Obstructed Labour

Maternal complications:
ComplicationMechanism
Uterine ruptureUUS/LUS tears, catastrophic haemorrhage, life-threatening
Vesicovaginal fistula (VVF)Bladder compressed between fetal head and symphysis; ischaemic necrosis
Rectovaginal fistula (RVF)Less common, posterior compartment compression
Postpartum haemorrhageUterine atony, tears, ruptured uterus
Sepsis/puerperal infectionProlonged labour, ruptured membranes, multiple VEs
Maternal deathFrom rupture, haemorrhage, or sepsis
Fetal complications:
  • Fresh stillbirth (the most common outcome in neglected cases)
  • Birth asphyxia - hypoxic ischaemic encephalopathy (HIE)
  • Tentorial tears, intracranial haemorrhage from moulding
  • Severe caput and moulding

Management of Obstructed Labour

Immediate resuscitation:
  1. IV access - two large-bore cannulae
  2. IV fluids (Ringer's lactate/crystalloid) for hydration and to correct dehydration/shock
  3. Catheterize bladder (relieves compression, monitors urine output)
  4. Broad-spectrum antibiotics (ampicillin + gentamicin + metronidazole)
  5. Blood grouping and cross-matching
Definitive management (depends on cause, stage, and fetal viability):
SituationManagement
Live fetus, vaginal delivery not feasibleEmergency caesarean section (most common intervention)
Dead fetus, vaginal delivery possibleDestructive/instrumental operations (craniotomy, decapitation) - in resource-limited settings
Brow presentationC-section (brow cannot deliver vaginally unless it converts)
Face - mentoanteriorVaginal delivery possible; mentoposterior requires C-section
Shoulder presentation / transverse lieC-section
Deep transverse arrestVacuum/forceps if criteria met; otherwise C-section
Note on Bandl's Ring: Once a pathological retraction ring is identified, caesarean section must be performed immediately. Attempting further vaginal manoeuvres risks complete uterine rupture.

Prevention of Obstructed Labour

  • Skilled birth attendance
  • Antenatal care with pelvic assessment
  • Use of the partograph with early recognition of poor progress
  • Timely referral from community/primary care
  • Addressing CPD early (elective C-section if trial of labour unsafe)
  • Management of malpresentation before/early in labour (ECV for breech)

Summary Table - Stages at a Glance

StageDefinitionNormal Duration (Nullipara / Multipara)Key Events
Stage 1 (Latent)Onset of labour to 4-6 cm dilatationUp to 20 hrs / 14 hrsEffacement, slow dilatation
Stage 1 (Active)4-6 cm to full dilatation (10 cm)6-12 hrs / 4-6 hrsRapid dilatation, descent begins
Stage 2Full dilatation to birth of babyUp to 2 hrs / up to 1 hrCardinal movements, delivery
Stage 3Birth of baby to placental deliveryUp to 30 minPlacental separation, expulsion
Stage 4First 1-2 hrs postpartum1-2 hoursMonitoring for PPH, involution

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