Heart Failure Examination OSCE Checklist
1. Preparation
- Hand hygiene
- Introduce yourself, confirm patient identity
- Explain examination and obtain consent
- Offer chaperone
- Ask about pain or breathlessness and position patient at 45°
- Expose from waist to knees while maintaining dignity
2. General inspection
Look from end of bed for:
- Breathlessness, inability to lie flat
- Oxygen, IV lines, diuretics, GTN infusion, monitoring
- Cyanosis, pallor, cachexia
- Peripheral edema
- Evidence of previous cardiac surgery, such as sternotomy scar
- Fluid balance chart or medications if available
Assess observations: pulse, BP, respiratory rate, SpO₂, temperature.
3. Hands and arms
- Peripheral cyanosis
- Capillary refill time and peripheral temperature
- Pulse: rate, rhythm, volume
- Collapsing pulse if significant aortic regurgitation suspected
- Tremor, nicotine staining
- Radial-radial delay
- Measure blood pressure
4. Face
- Conjunctival pallor
- Central cyanosis
- Xanthelasma or corneal arcus
- Dentition, if infective endocarditis is a concern
5. Neck
- Assess JVP at 45°:
- Height
- Waveform
- Hepatojugular or abdominojugular reflux
- Check carotid pulse, one side at a time
- Listen for carotid bruits only if appropriate
An elevated JVP, especially with a positive hepatojugular reflux, supports systemic venous congestion. Braunwald’s Heart Disease, p. 936.
6. Precordium
Inspect
- Scars: sternotomy, pacemaker/ICD, thoracotomy
- Visible apex beat or heaves
Palpate
- Apex beat: location and character
- Displaced, diffuse apex suggests cardiomegaly/LV dilatation
- Parasternal heave: right ventricular hypertrophy or pulmonary hypertension
- Thrills
Auscultate
- S1 and S2
- S3 gallop: suggests volume overload and systolic dysfunction
- S4: stiff ventricle, e.g. hypertensive heart disease
- Murmurs:
- Mitral regurgitation may cause or worsen HF
- Aortic stenosis is an important cause
- Tricuspid regurgitation may accompany right-sided failure
7. Lungs
- Inspect respiratory effort and rate
- Percuss lung bases for pleural effusion
- Auscultate posteriorly for:
- Bibasal fine crackles
- Wheeze, sometimes termed cardiac asthma
- Reduced air entry and dullness at bases from pleural effusions
Absence of crackles does not exclude congestion, particularly in chronic HF.
8. Abdomen
- Inspect for distension
- Palpate for tender hepatomegaly
- Assess for ascites
- Consider abdominal aortic aneurysm palpation if relevant
9. Legs and sacrum
- Check for bilateral pitting edema:
- Ankles in mobile patients
- Sacrum in bedbound patients
- Assess peripheral pulses
- Look for chronic venous insufficiency, ulcers, or signs suggesting an alternative cause of edema
Right-sided congestion classically produces edema, hepatomegaly, ascites, and raised JVP. The Washington Manual of Medical Therapeutics, physical examination section.
10. Complete the examination
Say:
“To complete my assessment, I would check the observations and fluid balance, review the ECG, chest radiograph, echocardiogram, BNP/NT-proBNP, renal function, electrolytes, full blood count, liver function, thyroid function, and urinalysis. I would also perform a focused neurological and peripheral vascular examination where indicated.”
OSCE summary statement
“This patient has clinical features of heart failure with congestion, including [raised JVP / bibasal crackles / peripheral edema / displaced apex / S3]. I would assess whether this is acute or chronic, identify a precipitant such as ischemia, arrhythmia, infection, uncontrolled hypertension, medication non-adherence, renal impairment, or valvular disease, and arrange urgent management if they are hypoxic, hypotensive, or in pulmonary edema.”
Red flags requiring urgent escalation
- Severe respiratory distress or hypoxia
- Pink frothy sputum
- Hypotension, confusion, cold peripheries
- New chest pain or suspected acute coronary syndrome
- New rapid atrial fibrillation or other unstable arrhythmia
- Markedly reduced urine output
The core examination sequence of general assessment, JVP, precordial signs, pulmonary crackles, hepatomegaly/ascites, and peripheral edema aligns with the clinical HF assessment described in Braunwald’s Heart Disease, pp. 935-936.