Questions or template to clerk a DFU patient

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Diabetic Foot Ulcer (DFU) Clerking Template


1. PRESENTING COMPLAINT

  • Location of ulcer/wound on foot
  • Duration present
  • How it was first noticed (or went unnoticed due to neuropathy)
  • Triggering factor (ill-fitting footwear, minor trauma, spontaneous)

2. HISTORY OF PRESENTING COMPLAINT

Wound characteristics

  • Size (width, depth)
  • Discharge — purulent, serous, bloody, none?
  • Odour
  • Change over time — improving, worsening, stable?
  • Previous episodes of ulceration at the same or other sites

Symptoms of infection

  • Redness / erythema — extent from wound edge
  • Warmth
  • Swelling / induration
  • Pain or tenderness (note: absence does NOT exclude infection in neuropathic feet)
  • Purulent discharge
  • Crepitation (gas in tissue)
  • Fever, rigors, sweating (systemic features → SIRS)

Neuropathy symptoms

  • Loss of sensation / numbness in feet or legs
  • Paraesthesiae (tingling, burning, "pins and needles")
  • Burning pain, worse at night
  • Balance problems, falls
  • Has patient noticed the ulcer themselves, or was it found by someone else?

Peripheral vascular disease symptoms

  • Intermittent claudication — distance, muscle group (calf, thigh, buttock)
  • Rest pain (worse lying, relieved hanging foot down)
  • Colour changes — pallor on elevation, dependent rubor
  • Non-healing cuts / previous ulcers elsewhere
  • History of coronary artery disease / stroke / TIA (markers of systemic atherosclerosis)

Structural / biomechanical factors

  • Foot deformity: bunions (hallux valgus), hammer toes, claw toes, Charcot foot
  • History of previous amputation (toe, ray, partial foot)
  • Callus formation over pressure points
  • Footwear: tight shoes, ill-fitting insoles, walking barefoot

3. DIABETIC HISTORY

  • Type of diabetes (Type 1 / Type 2) and duration
  • Current glycaemic control — last HbA1c, home glucose readings
  • Previous DFUs or amputations
  • Known microvascular complications: nephropathy (CKD/dialysis), retinopathy, neuropathy
  • Known macrovascular complications: IHD, stroke, PVD
  • Current diabetes medications (insulin, oral agents)

4. PAST MEDICAL HISTORY

  • Hypertension
  • Dyslipidaemia
  • CKD / end-stage renal disease (ESRD) — worsens small vessel disease
  • Immunosuppression (transplant, chronic steroids — increases infection severity)
  • Previous cardiac or vascular surgery
  • Osteomyelitis — prior episodes at same site?

5. DRUG HISTORY & ALLERGIES

  • Insulin / oral hypoglycaemics
  • Antiplatelets (aspirin, clopidogrel)
  • Statins
  • Antihypertensives
  • Antibiotics — recent courses (guides microbial resistance)
  • Anticoagulants
  • Immunosuppressants
  • Allergies (especially antibiotic allergies — critical for DFI treatment planning)

6. SOCIAL HISTORY

  • Smoking (current, ex, pack-years) — key PAD risk factor
  • Alcohol
  • Functional status: mobilising independently, wheelchair, housebound?
  • Home support: can patient do daily wound inspection? Is there a carer?
  • Access to follow-up (daily follow-up essential for mild infections treated outpatient)
  • Footwear / occupation / activity level
  • Nutrition — diet history, weight loss, malnutrition risk

7. SYSTEMS REVIEW

  • Constitutional: fever, weight loss, malaise
  • Cardiovascular: chest pain, dyspnoea, palpitations
  • Renal: decreased urine output, oedema (wound maceration may be fluid-overload related)
  • Respiratory: dyspnoea (relevant if surgical debridement / anaesthesia planned)

8. EXAMINATION

Vital signs

  • Temperature (>38°C or <36°C = SIRS criterion)
  • Heart rate (>90 bpm = SIRS criterion)
  • Respiratory rate (>20/min = SIRS criterion)
  • Blood pressure
  • Capillary blood glucose

Wound assessment (WIfI — Wound)

GradeDescription
0No ulcer / pre-ulceration
1Shallow ulcer, no exposed bone (except distal phalanx), no gangrene
2Exposed bone or gangrene limited to digits, or shallow heel ulcer
3Extensive ulcer/gangrene, or full-thickness heel ulcer
  • Document: location, size (cm), depth (probe with back of cotton swab — probe-to-bone test)
  • Debride callus before full assessment
  • Assess: base colour (granulating / sloughy / necrotic / bone visible), edges, surrounding skin, discharge, odour
  • Signs of infection: erythema (measure extent from wound edge), warmth, swelling, purulence, crepitation

Neurological examination

  • 10-g monofilament test (4 sites on each foot)
  • Vibration sensation — 128 Hz tuning fork (great toe)
  • Ankle reflexes
  • Proprioception

Vascular examination (WIfI — Ischaemia)

GradeABIToe Pressure
0≥0.80≥60 mmHg
10.60–0.7940–59 mmHg
20.40–0.5930–39 mmHg
3<0.40<30 mmHg
  • Femoral, popliteal, dorsalis pedis, posterior tibial pulses
  • Capillary refill time
  • Skin: pallor, dependent rubor, hair loss, shiny skin, trophic changes
  • Ankle-brachial index (ABI) — note: may be falsely elevated due to medial calcinosis in diabetics; toe pressures more reliable
  • Pulse volume recordings (PVRs)

Foot structure

  • Deformities: hallux valgus, hammer/claw toes, Charcot joint
  • Calluses — location
  • Prior amputation sites
  • Both feet examined

IWGDF/IDSA Infection Classification (WIfI — foot Infection)

GradeFeatures
1 – UninfectedNo signs of infection
2 – Mild≥2 local signs (swelling, erythema ≤2 cm, warmth, pain, purulence); no systemic features
3 – ModerateErythema ≥2 cm from wound OR deeper tissue/bone involvement; no SIRS
4 – SevereAny infection with ≥2 SIRS criteria (fever, tachycardia, tachypnoea, leukocytosis/penia)
Add "(O)"If osteomyelitis confirmed or suspected

9. INVESTIGATIONS

Bedside

  • Capillary blood glucose / point-of-care HbA1c
  • Probe-to-bone test (positive = high PPV for osteomyelitis)

Bloods

  • FBC (leukocytosis, anaemia)
  • CRP, ESR (support diagnosis of infection; elevated levels correlate with severity)
  • U&E / eGFR (renal function — affects antibiotic dosing; CKD worsens prognosis)
  • LFTs
  • HbA1c
  • Lipid profile
  • Coagulation (if surgical intervention planned)
  • Blood cultures (if SIRS/fever present)

Microbiology

  • Deep tissue swab or tissue biopsy (NOT superficial swab — avoids colonisers)
  • Bone culture if osteomyelitis suspected (guides antibiotic duration and selection)

Imaging

  • Plain x-ray foot (3 views): bone erosion, foreign bodies, soft tissue gas, Charcot changes, prior amputations, heterotopic ossification
  • MRI foot: gold standard for early osteomyelitis detection
  • CT: identifies deeper abscesses when MRI unavailable
  • Angiography (CT angiogram / formal angiogram) if revascularisation being considered

Vascular

  • ABI + toe pressures
  • Transcutaneous oxygen measurement (TcPO₂) — <40 mmHg indicates impaired wound healing and potential indication for hyperbaric oxygen

10. SUMMARY IMPRESSION

Formulate using the WIfI classification:
  • Wound grade (0–3)
  • Ischaemia grade (0–3)
  • fI (foot Infection) grade (1–4)
These three grades together predict:
  1. Risk of major lower extremity amputation at 1 year
  2. Likely benefit of revascularisation

11. MANAGEMENT PLAN (document thinking)

  • Glycaemic optimisation
  • Wound care: moist environment, debridement of callus/necrotic tissue, offloading (orthotic device, total contact cast)
  • Antibiotics: oral for mild (gram-positive cover); IV broad-spectrum for moderate–severe (e.g., piperacillin-tazobactam + vancomycin)
  • Osteomyelitis: 6-week antibiotic course if no surgical resection
  • Surgical referral: urgent if necrotising infection, deep abscess, compartment syndrome, or limb ischaemia
  • Vascular surgery referral: if ischaemia grade ≥1 with non-healing wound at 4–6 weeks
  • Hyperbaric oxygen: consider if TcPO₂ <40 mmHg and wound not healing
  • Daily follow-up for outpatients; hospitalisation if systemic features or inadequate home support
  • Long-term follow-up: minimum 1 year to confirm remission

Sources: Sabiston Textbook of Surgery 21e (Diabetic Foot Infections, p. 643–644); Current Surgical Therapy 14e (Diabetic Foot, p. 1186–1188); Rosen's Emergency Medicine 9e (The Diabetic Foot, p. 2540)
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