Summary
Most results are reassuring. Two findings need follow-up:
- Fasting glucose: 112 mg/dL - mildly high
- GGT: 124 U/L - elevated (lab reference 0-38)
The kidney markers, most liver markers, proteins, bilirubin, and electrolytes are within the lab range. The CBC is listed as pending and is not shown in these screenshots.
| Test | Result | Interpretation |
|---|
| Fasting glucose | 112 mg/dL | Mildly raised. If the sample was after at least 8 hours fasting, this falls in the prediabetes / impaired fasting glucose range of 100-125 mg/dL. It is not in the diabetes range, which begins at 126 mg/dL or higher. See ADA diagnostic criteria. |
| GGT | 124 U/L | About 3 times the lab upper limit. GGT is a sensitive but nonspecific liver and bile-duct enzyme. |
| AST (SGOT) | 18 U/L | Normal |
| ALT (SGPT) | 21 U/L | Normal |
| ALP | 92 U/L | Normal |
| Bilirubin, total/direct | 0.37 / 0.15 mg/dL | Normal |
| Creatinine | 0.85 mg/dL | Normal |
| eGFR | 80 mL/min/1.73 m², category G2 | Generally acceptable. An eGFR of 60 or above does not diagnose chronic kidney disease by itself; kidney damage, such as persistent urine protein/albumin, would also need to be present. |
| Urea / BUN | 25 / 11.68 mg/dL | Normal |
| Uric acid | 3.7 mg/dL | Normal |
| Sodium / potassium / chloride | 136 / 3.7 / 102 mEq/L | Normal, with sodium and calcium at the lower end of normal |
| Calcium / phosphorus | 8.7 / 3.53 mg/dL | Normal |
| Albumin / total protein | 4.53 / 6.9 g/dL | Normal |
What the elevated GGT may mean
Because AST, ALT, ALP, and bilirubin are normal, this is an isolated GGT elevation. It does not by itself prove liver damage, obstruction, or a serious condition. Common possibilities include:
- Alcohol intake, including regular or recent intake
- Fatty liver related to excess weight, insulin resistance, elevated triglycerides, or prediabetes
- Medicines or supplements, including some anticonvulsants, antibiotics, herbal preparations, and other drugs
- Less commonly, liver or bile-duct disease
GGT is commonly elevated in hepatobiliary disease and alcohol-related liver disease, but it is nonspecific. In cholestasis, GGT usually rises alongside alkaline phosphatase, which is
not the pattern here.
AASLD guidance on liver enzymes discusses using the overall pattern of liver tests rather than GGT alone.
Sensible next steps
Arrange a non-urgent appointment with the prescribing clinician/GP to review:
- HbA1c - shows average glucose over roughly the prior 2-3 months.
- Repeat fasting glucose - especially if fasting duration was uncertain, illness was present, or there was a high-carbohydrate meal/alcohol the prior day.
- Repeat liver panel including GGT after reviewing alcohol, all prescribed medicines, over-the-counter drugs, and herbal or nutritional supplements.
- Consider a lipid profile, blood pressure, waist/weight assessment, and discussion of fatty liver risk.
- If GGT remains elevated or rises, the clinician may consider hepatitis screening and an abdominal ultrasound based on history and examination.
For the glucose level, practical measures such as limiting sugary drinks and refined carbohydrates, regular physical activity, weight reduction if applicable, and balanced meals can reduce diabetes risk.
When to seek prompt medical care
Seek medical assessment sooner if there is yellowing of eyes/skin, dark urine, pale stools, significant itching, persistent upper-right abdominal pain, fever, vomiting, confusion, or marked weakness.
Bottom line: kidney function and the main liver-function indicators are currently reassuring. The report suggests prediabetes-range fasting glucose and an isolated elevated GGT that warrants follow-up and review of alcohol, medicines, metabolic risk, and possibly fatty liver.