ATLS (ABCDE)

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Here is a comprehensive overview of the ATLS Primary Survey — ABCDE, drawn from Miller's Anesthesia and Mulholland & Greenfield's Surgery.

ATLS Primary Survey: ABCDE

The primary survey is the structured, sequential assessment of the trauma patient designed to identify and treat immediately life-threatening problems in order of priority. It must be repeated whenever the patient's condition changes.
ATLS ABCDE Flowchart
Simplified assessment and management of the trauma patient. Adapted from the ACS ATLS curriculum. — Miller's Anesthesia, 10e

A — Airway (with C-spine protection)

Rationale: Hypoxia is the most immediate threat to life — permanent brain injury and death occur within 5–10 minutes of hypoxic arrest.
Assessment:
  • Vocal response (talking = patent airway)
  • Auscultation
  • Look for obstruction: blood, secretions, foreign bodies, facial/mandibular/neck trauma, altered consciousness
Causes of obstruction in trauma:
  • Direct injury to face, mandible, or neck
  • Hemorrhage in nasopharynx, sinuses, or mouth
  • Diminished consciousness (TBI, intoxication, analgesia)
  • Aspiration of gastric contents, blood, or foreign body (dentures, broken teeth)
  • Esophageal intubation
Interventions (escalating):
  1. Chin lift / jaw thrust
  2. Suction + oral/nasal airway
  3. Bag-valve-mask with 100% O₂
  4. Endotracheal intubation (confirm with capnography immediately — esophageal intubation is common and devastating)
  5. Surgical cricothyroidotomy if all else fails — this takes absolute priority over all other procedures
C-spine immobilization is maintained throughout unless injury has been excluded.

B — Breathing & Ventilation

Rationale: A patent airway does not guarantee adequate ventilation. Oxygenation AND CO₂ clearance must be assessed.
Assessment:
  • Pulse oximetry
  • Arterial blood gas
  • Chest X-ray
  • Inspect: respiratory rate, symmetry of chest rise, tracheal deviation, crepitus, subcutaneous emphysema
Causes of inadequate ventilation in trauma:
  • Tension pneumothorax
  • Open pneumothorax
  • Hemothorax
  • Flail chest / pulmonary contusion
  • Direct tracheobronchial injury
  • Cervical spine injury (phrenic nerve)
  • Bronchospasm (smoke / toxic gas inhalation)
Interventions:
  • Supplemental O₂ for all trauma patients
  • Mechanical ventilation if needed
  • Needle decompression + tube thoracostomy for pneumo/hemothorax
  • Seal open chest wounds (three-sided dressing)

C — Circulation & Hemorrhage Control

Rationale: Hemorrhage is the #1 cause of preventable death in the first 6 hours after trauma. Shock is presumed hemorrhagic until proven otherwise.
Assessment:
  • Vital signs: HR, BP, pulse pressure (narrowed = early shock)
  • Capillary refill
  • Skin: pallor, diaphoresis
  • Response to fluid bolus
  • Labs: CBC, coagulation studies, type & crossmatch
  • FAST (Focused Assessment with Sonography in Trauma)
  • Pelvic plain films
Symptoms of shock: Pallor · Diaphoresis · Agitation or obtundation · Hypotension · Tachycardia · Prolonged capillary refill · Diminished urine output · Narrowed pulse pressure
The 5 sites of exsanguinating hemorrhage:
SiteDetection
Pleural spaceCXR, auscultation
Intra-abdominalFAST, DPA
Pelvis / retroperitonealPelvic XR
Long bone fractures / soft tissueClinical exam
ExternalDirect visualization
Interventions:
  • Large-bore IV access × 2 (or IO/central if needed)
  • Uncrossmatched blood (O-negative) if in extremis
  • Activate Massive Transfusion Protocol (MTP) early — target 1:1:1 ratio (pRBC:FFP:platelets)
  • Direct pressure on wounds
  • Tourniquet for extremity hemorrhage
  • Pelvic binder for open-book pelvic fractures
  • REBOA (Resuscitative Endovascular Balloon Occlusion of the Aorta) at trauma centers
  • ED thoracotomy if indicated
  • Emergent OR for hemorrhage control
Avoid the lethal triad: Hypothermia + Acidosis + Coagulopathy (Trauma-Associated Coagulopathy, TAC)

D — Disability (Neurological Status)

Rationale: Rapid, reproducible assessment of CNS function to detect TBI or spinal injury early — delays in evacuating epidural/subdural hematomas worsen outcomes.
Assessment:
  • GCS (Glasgow Coma Scale) — Eye (1–4) + Verbal (1–5) + Motor (1–6) = 3–15
  • Pupil size, reactivity, and symmetry
  • Sensation and motor function in all four extremities
  • Pressure stimuli: fingertip pressure, trapezius pinch, or supraorbital notch pressure
GCSInterpretation
13–15Mild TBI
9–12Moderate TBI
≤ 8Severe TBI — intubate
Abnormal motor responses:
  • Abnormal flexion (decorticate): Slow stereotyped arm across chest, forearm rotating, thumb clenched, leg extended
  • Normal flexion: Rapid, variable, arm moves away from body
Interventions:
  • Any significant neurological deficit → immediate head CT
  • Support oxygenation and perfusion (secondary brain injury prevention)
  • Cervical spine films / CT
  • Neurosurgery for operative lesions (epidural/subdural hematoma)
  • ICP monitoring if indicated

E — Exposure & Environment

Rationale: Complete examination requires full exposure; hypothermia must be actively prevented (it worsens coagulopathy and outcomes).
Assessment:
  • Completely undress the patient (cut off clothing)
  • Full log-roll to examine the back
  • Look for: wounds, deformities, bruising, burns, signs of penetrating injury
Interventions:
  • Warm blankets and IV fluids to prevent hypothermia
  • Laboratory studies
  • ECG
  • Plain films and CT as indicated
  • Detailed history (AMPLE: Allergies, Medications, Past medical history, Last meal, Events/mechanism)

After the Primary Survey: Secondary Survey

The secondary survey — a head-to-toe examination — only begins once:
  1. The primary survey is complete
  2. Resuscitation is underway
  3. Vital functions are improving
The ABCDE survey must be reassessed continuously throughout care. Any deterioration restarts the sequence from A.

Sources: Miller's Anesthesia 10e, Ch. 62; Mulholland & Greenfield's Surgery 7e, Ch. 4

Fracture basics

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Here is a comprehensive overview of Fracture Basics, drawn from Rosen's Emergency Medicine, Miller's Review of Orthopaedics, Rockwood & Green's Fractures in Adults, and Junqueira's Basic Histology.

Fracture Basics

A fracture is a break in the continuity of bone. History of trauma, pain, tenderness, swelling, loss of function, abnormal motion, and deformity all suggest a fracture.

1. Fracture Description Framework

Every fracture should be described systematically:
CategoryDescriptors
Skin integrityOpen vs. closed
Anatomic locationBone, side (L/R), region (proximal/middle/distal third)
Fracture line directionTransverse, oblique, spiral, comminuted
CompletenessComplete (both cortices) vs. incomplete (one cortex)
PositionDisplacement, angulation, rotation, shortening
Articular involvementIntra- vs. extra-articular (% of surface)
Special typesAvulsion, impaction, depression, compression, stress, pathologic
By convention, the distal fragment is described relative to the proximal fragment.

2. Fracture Line Types

Types of Fractures: (A) Transverse, (B) Oblique, (C) Spiral, (D) Comminuted
Types of Fractures. (A) Transverse. (B) Oblique. (C) Spiral. (D) Comminuted. — Rosen's Emergency Medicine
TypeMechanismNotes
TransverseDirect/bending forcePerpendicular to long axis
ObliqueAngulation forceRuns obliquely along shaft
SpiralRotational/torque forceEncircles shaft; may suggest non-accidental injury in children
ComminutedHigh-energy>2 fragments
SegmentalHigh-energyTwo separate fracture levels; isolates a segment
AvulsionMuscle/ligament pullFragment torn at tendon/ligament insertion
StressRepetitive loading on normal bone
InsufficiencyNormal load on weakened boneOsteoporosis, radiation, metabolic disease
PathologicMinimal trauma through diseased boneTumour, Paget's, infection

3. Open vs. Closed

  • Closed: Skin and soft tissue overlying fracture are intact
  • Open (compound): Fracture communicates with external environment — may not be immediately obvious
    • If a wound is near the fracture and doubt exists, treat as open
    • Do NOT probe with blunt swabs to determine communication — unreliable and potentially harmful
    • Open fractures require urgent surgical debridement and IV antibiotics

4. Displacement & Angulation

  • Displacement: Fragments deviate from their normal position (described in mm or % bone width)
  • Angulation: Deviation of the long axis; described by the direction the apex points
    • Valgus: Apex toward midline
    • Varus: Apex away from midline
  • Rotation: Distal fragment rotated along the bone axis — may only be apparent clinically (e.g., finger scissoring on flexion)
  • Shortening: Overlap or impaction of fragments

5. Fracture Healing

Phases of Secondary Bone Healing

Bone Fracture Repair Stages
Stages of bone fracture repair. — Junqueira's Basic Histology, 17e
StageWhat Happens
1. Inflammation / HematomaTorn blood vessels bleed → fracture hematoma forms; macrophages phagocytose debris; cytokines (IL-1β, IL-6, TNF-α) recruit progenitor cells
2. Soft (fibrocartilaginous) callusPeriosteum and endosteum proliferate; MSCs differentiate; fibrocartilage procallus bridges the gap; regenerating blood vessels invade
3. Hard (bony) callusFibrocartilage undergoes endochondral ossification → woven bone callus
4. RemodelingWoven bone replaced by lamellar bone; Wolff's law dictates structure along stress lines; original shape and strength restored

Primary vs. Secondary Healing

Primary (Direct)Secondary (Indirect)
ConditionAbsolute stability, no gapRelative stability, some movement
MechanismHaversian remodeling (cutting cones)Callus formation (endochondral + intramembranous ossification)
FixationCompression plateCasting, IM nail, external fixator
CallusNone visible on X-rayVisible periosteal callus

Key Factors Affecting Healing

  • Pro-healing: Good vascularity, periosteum intact, stable fixation, BMPs (BMP-2, BMP-7)
  • Impair healing: NSAIDs (inhibit COX-2, impair endochondral ossification), smoking, corticosteroids, infection, diabetes, poor nutrition
  • Bone grafts:
    • Osteoconductive — scaffold (e.g., demineralized bone matrix)
    • Osteoinductive — stimulate bone formation (e.g., BMP)
    • Osteogenic — contain living bone-forming cells (e.g., autograft)

6. Complications

Early

ComplicationKey Points
Vascular injuryCheck distal pulses; ABI if concerned
Nerve injurySpecific nerves at risk per fracture (e.g., radial nerve in humeral shaft fractures, median nerve in distal radius fractures)
Compartment syndromePain out of proportion + tense compartment; tibia most common; emergency fasciotomy if pressure >30 mmHg or within 30 mmHg of diastolic
Fat embolismLong bone / pelvic fractures; triad: hypoxia + confusion + petechiae
InfectionEspecially open fractures

Late

ComplicationKey Points
MalunionHealed in poor position
NonunionFailure to heal by 6 months; hypertrophic (vascular, needs more stability) vs. atrophic (avascular, needs biology)
Delayed unionHealing slower than expected
Avascular necrosisDisrupted blood supply; common: femoral head, scaphoid, talus
Post-traumatic arthritisIntra-articular fractures
RefractureEspecially after hardware removal

7. Principles of Fracture Management

The goals are: Reduce → Hold → Rehabilitate
PrincipleOptions
ReductionClosed (manipulation) or open (surgical)
ImmobilizationCast/splint · External fixator · IM nail · Plate & screws
RehabilitationEarly mobilisation to prevent stiffness and muscle atrophy
Rule: If in doubt about fracture stability or open communication — immobilise, image in two planes (orthogonal views), and get orthopaedic input.

Sources: Rosen's Emergency Medicine, Ch. 41; Miller's Review of Orthopaedics 9e; Rockwood & Green's Fractures in Adults 10e; Junqueira's Basic Histology 17e

Shoulder dislocation

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Here is a comprehensive overview of Shoulder Dislocation, drawn from Rockwood & Green's Fractures in Adults, Roberts & Hedges' Clinical Procedures in Emergency Medicine, Schwartz's Principles of Surgery, and the THIEME Atlas of Anatomy.

Shoulder Dislocation

Anatomy & Why the Shoulder Dislocates So Easily

Glenohumeral joint anatomy — anterior and posterior views
Glenohumeral joint — anterior (a) and posterior (b) views. — THIEME Atlas of Anatomy
The glenohumeral joint is the most mobile joint in the body — and consequently the most commonly dislocated (~45% of all dislocations). The glenoid cavity is 3–4× smaller than the humeral head; the glenoid labrum deepens it by only ~5 mm. The joint capsule and ligaments are inherently weak, making the rotator cuff the primary dynamic stabilizer.

Types of Dislocation

DirectionFrequencyMechanismArm Position
Anterior (anteroinferior)~95%Forced external rotation + abduction (FOOSH, throwing, seizure)Slight abduction, external rotation
Posterior~2–4%Forced internal rotation (seizure, electrocution, direct blow)Internal rotation + adduction ("sling position")
Inferior (Luxatio Erecta)RareHyperabduction forceArm locked overhead in hyperabduction

Anterior Dislocation

Clinical Features

  • Posterior sulcus visible (loss of normal rounded contour)
  • Arm held in slight abduction and external rotation
  • Inability to place palm of affected hand on contralateral shoulder
  • Palpable fullness anteriorly (humeral head)

Imaging

  • AP view — humeral head not in glenoid fossa
  • Axillary (glenoid) view — gold standard; confirms direction
  • Y-view (scapular lateral) — useful when axillary view not tolerated

Associated Injuries

InjuryDetails
Bankart lesionTear of anteroinferior glenoid labrum ± avulsion of glenoid rim (bony Bankart) — the essential lesion of shoulder instability
Hill-Sachs lesionImpaction fracture of posterior humeral head against glenoid rim — seen in 33% of primary and 62% of recurrent dislocations
Axillary nerve injury13.5% incidence; test sensation over deltoid (regimental badge area); 90% recover with expectant management
Axillary artery injuryCheck capillary refill and radial pulse; more common in elderly
Rotator cuff tearUp to 38% incidence; much higher in patients >40 years
Greater tuberosity fractureIf displaced >1 cm post-reduction → rotator cuff tear likely → orthopaedic consult
HAGL lesionHumeral Avulsion of GlenoHumeral Ligaments

Posterior Dislocation

Clinical Features (Easily Missed!)

  • Arm in internal rotation + adduction ("sling position")
  • Loss of external rotation and forward flexion
  • No obvious deformity — diagnosis commonly delayed
  • Causes: seizures, electrocution, direct posterior blow

Imaging

  • Often missed on AP view alone — the humeral head may appear falsely normal
  • Axillary view is mandatory — shows posterior displacement
  • Trough sign on AP — dense sclerotic line on humeral head (reverse Hill-Sachs impaction)
  • Fracture of the lesser tuberosity = posterior dislocation until proven otherwise

Associated Injuries

  • Reverse Bankart fracture (posterior glenoid rim)
  • Reverse Hill-Sachs lesion (anterior humeral head)
  • Posterior capsule and labrum tear
  • 65% have an associated injury overall

Inferior Dislocation — Luxatio Erecta

  • Arm locked in marked hyperabduction with forearm lying on or behind the head
  • Associated with neurovascular injury (brachial plexus, axillary vessels) and rotator cuff tears
  • Reduction: Overhead traction in abduction with cephalad pressure on the humeral head; assistant applies countertraction caudally via sheet over the shoulder

Reduction Techniques (Anterior Dislocation)

Premedication (procedural sedation or intra-articular lidocaine block) improves muscle relaxation and success.
TechniqueMethodNotes
External RotationArm adducted, elbow flexed 90°; slowly externally rotate forearm to bed level. No traction.Low force; high patient tolerance; good for first-line attempt
StimsonPatient prone, arm hanging off stretcher; 5 kg weight attached; 20–30 min. Add scapular manipulation.Low force; requires patient cooperation
Scapular ManipulationRotate inferior tip of scapula medially and dorsally while assistant provides tractionCan combine with Stimson; patient prone or seated
Spaso TechniqueSupine; lift arm vertically toward ceiling + gentle vertical traction + gentle ER; 87.5% success rateFast, single-operator; equivalent efficacy to external rotation
Milch TechniqueAbduct arm overhead, apply gentle traction + slight ER; push humeral head into glenoid if needed
Traction-CountertractionTwo sheets — one around axilla (assistant countertraction), one around forearm (operator traction); gentle adduction while second assistant applies lateral tractionClassic technique; requires 2 assistants
Best-of-BothPatient seated sideways; downward force on flexed forearm + scapular manipulation simultaneouslyCombines two effective methods
Signs of successful reduction:
  • Audible/palpable "clunk"
  • Immediate pain relief
  • Restoration of round shoulder contour
  • Patient can place palm of affected hand on contralateral shoulder
Post-reduction: Repeat neurovascular exam + post-reduction radiographs. Immobilise with sling/shoulder immobiliser pending orthopaedic follow-up.

Posterior Dislocation Reduction

  • Traction on internally rotated and adducted arm + anteriorly directed pressure on posterior humeral head
  • Generous premedication + countertraction via axillary sheet
  • If head locked on posterior glenoid → add lateral traction on upper humerus
  • Impression defect >20% of articular surface → open reduction required
  • Post-reduction: Check patient can place palm on opposite shoulder; orthopedic consultation for all posterior dislocations

Post-Reduction Management

FactorRecommendation
ImmobilisationSling/shoulder immobiliser; prolonged immobilisation does not reduce recurrence rates (>1 week not beneficial per meta-analysis)
Position of immobilisationSome evidence favours external rotation (maximises labral contact with glenoid rim vs. internal rotation which increases labral detachment)
Age <40Higher recurrence risk (up to 70–90% in teenagers); consider early orthopaedic referral for discussion of surgical stabilisation
Age >60Early mobilisation to prevent stiffness and adhesive capsulitis; follow up at 5–7 days
ImagingMRI to assess soft tissue (labrum, rotator cuff, capsule)

Recurrent Instability & Surgery

  • Arthroscopic Bankart repair — gold standard for most cases (anteroinferior capsulolabral complex)
  • Latarjet procedure — coracoid transfer to anterior glenoid rim; for large bony glenoid defects (>25% glenoid bone loss) or engaging Hill-Sachs lesions
  • Remplissage — arthroscopic infraspinatus tenodesis into Hill-Sachs defect; reduces engagement
  • Open Bankart repair — reserved for revision or complex cases

Sources: Rockwood & Green's Fractures in Adults 10e, Ch. 35; Roberts & Hedges' Clinical Procedures in Emergency Medicine; Schwartz's Principles of Surgery 11e; THIEME Atlas of Anatomy — General Anatomy & Musculoskeletal System

Fracture of humerus

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Here is a comprehensive overview of Fractures of the Humerus, covering proximal, shaft, and distal segments, drawn from Rockwood & Green's, Schwartz's Principles of Surgery, and Miller's Review of Orthopaedics.

Fractures of the Humerus

The humerus is divided into three regions for clinical purposes: proximal, shaft (diaphyseal), and distal. Each has a distinct epidemiology, classification, nerve injury risk, and management.

1. Proximal Humerus Fractures

Epidemiology

  • Most common in elderly women after low-energy falls onto the shoulder
  • Also occur in young patients after high-energy trauma
  • Account for ~5% of all fractures; 3rd most common fragility fracture after hip and distal radius

Anatomy — The Neer 4-Part System

Four-part proximal humerus fracture
Four-part proximal humeral fracture. Parts: 1 = greater tuberosity, 2 = humeral head, 3 = articular segment, 4 = humeral shaft. — Schwartz's Principles of Surgery 11e
Neer's classification divides the proximal humerus into 4 parts:
  1. Humeral head (articular segment)
  2. Greater tuberosity (supraspinatus, infraspinatus, teres minor insertions)
  3. Lesser tuberosity (subscapularis insertion)
  4. Humeral shaft
A part is considered "displaced" when separated by >1 cm or >45° angulation.
Neer ClassificationDescriptionFrequency
1-partAny fracture, no segment displaced~80%
2-partOne segment displacedCommon
3-partTwo segments displacedLess common
4-partAll four segments separatedRare; high AVN risk
Despite wide use, inter-observer reliability of the Neer system is moderate. CT is recommended for complex patterns.

Blood Supply & AVN Risk

  • The humeral head blood supply comes predominantly via the anterior humeral circumflex artery (arcuate artery) and posterior circumflex artery
  • 4-part fractures and fracture-dislocations carry >30% risk of avascular necrosis (AVN) of the humeral head

Treatment

Fracture TypeManagement
Minimally displaced (1-part)Sling immobilisation; pendulum exercises by 2 weeks; physio within 2 weeks to prevent stiffness
2-part greater tuberosityIf displaced >5 mm → ORIF (especially in overhead athletes); if <5 mm → conservative
2-part surgical neckClosed reduction + sling, or ORIF if unstable
3-partORIF with locking plate ± tension band
4-part / head-splitting / fracture-dislocationHemiarthroplasty or reverse total shoulder arthroplasty (RTSA) (favoured in elderly with osteoporosis); ORIF in young patients with good bone stock
Elderly + osteoporosis + comminutedRTSA gaining popularity
Post-op: Early pendulum exercises → passive ROM → active ROM → strengthening

2. Humeral Shaft (Diaphyseal) Fractures

Mechanisms

  • Direct blow, fall, high-energy trauma
  • Torsional force (spiral fracture pattern)
  • Pathologic fracture (metastasis is common here)

Key Nerve at Risk: Radial Nerve

Radial nerve and wrist drop from humeral shaft fracture
Radial nerve injury at the humeral shaft causing wrist drop. — Schwartz's Principles of Surgery 11e
  • The radial nerve runs in the spiral (radial) groove on the posterior surface of the humerus
  • Incidence of radial nerve palsy: ~11–18% of all humeral shaft fractures
  • Holstein-Lewis fracture: Spiral fracture of the distal third of the shaft — highest risk of radial nerve entrapment/injury
  • The vast majority of radial nerve palsies are neuropraxias (stretch/contusion) → spontaneous recovery expected within 3–4 months
  • EMG at 6–8 weeks helps assess recovery; if no recovery by 3–4 months → surgical exploration

Acceptable Alignment for Non-Operative Treatment

ParameterAcceptable Limit
Anterior angulation< 20°
Varus/valgus angulation< 30°
Shortening< 3 cm
Radial nerve palsy is NOT a contraindication to conservative management (except in open fractures, where nerve must be explored)

Treatment

ScenarioManagement
Most closed fracturesCoaptation splint initially → functional brace (plastic clamshell with Velcro) within 1–2 weeks; gentle motion started within 1–2 weeks
Unacceptable angulationORIF with plate (more stable, allows early weight-bearing)
Intramedullary nailAlternative; risk of shoulder pain at nail entry site
Open fracture + radial nerve palsySurgical exploration of nerve mandatory
Radial nerve palsy (closed fracture)Expectant management; EMG to monitor; explore if no recovery by 3–4 months

3. Distal Humerus Fractures

Types

TypeDescription
SupracondylarAbove the elbow joint; does not involve articular surface — most common in adults after a fall
TranscondylarThrough both condyles
Intercondylar (bicondylar)Intra-articular "T" or "Y" pattern; most complex
Single condyle (lateral/medial)Lateral = Milch classification; can cause cubitus valgus + tardy ulnar nerve palsy
Capitellum / trochleaCoronal shear fractures
EpicondyleAvulsion fractures

Nerve Injuries at the Distal Humerus

FractureNerve at Risk
Supracondylar (extension type, children)Anterior interosseous nerve (branch of median) — unable to make "OK sign"
SupracondylarRadial nerve, median nerve
Medial epicondyleUlnar nerve
Lateral condyle malunion → cubitus valgusTardy ulnar nerve palsy

Vascular Injury

  • Supracondylar fractures can injure or tether the brachial artery
  • Check: radial pulse, capillary refill, and "pink pulseless hand" (pulseless but perfused via collaterals) vs. true ischaemia

Treatment

FractureManagement
Minimally displaced supracondylarPosterior long arm splint at 90° elbow flexion
Displaced / most distal humerusORIF with anatomically contoured dual plates (orthogonal or parallel plating); goal = anatomic articular reduction + early ROM
Severely comminuted (elderly)Total elbow arthroplasty (TEA)
Intra-articular fracturesAnatomic reduction of joint surface is paramount; early motion to prevent stiffness
Elbow fractures are notorious for stiffness — early range of motion is critical to a good outcome.

Summary Table

RegionKey Nerve RiskKey ClassificationPrimary Non-Op RxPrimary Op Rx
ProximalAxillary nerveNeer (4-part)Sling + early pendulumsORIF locking plate / RTSA
ShaftRadial nerve (spiral groove)AO/OTAFunctional bracePlate fixation / IM nail
DistalAIN / ulnar / radialAO/OTAPosterior splint (min. displaced)Dual plate ORIF / TEA

Sources: Schwartz's Principles of Surgery 11e, Ch. 43; Rockwood & Green's Fractures in Adults 10e; Miller's Review of Orthopaedics 9e

Hip fracture

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Here is a comprehensive overview of Hip Fractures, drawn from Rockwood & Green's Fractures in Adults, Mulholland & Greenfield's Surgery, Grainger & Allison's Diagnostic Radiology, and Morgan & Mikhail's Clinical Anesthesiology.

Hip Fracture

Epidemiology & Public Health Impact

Hip fractures are a major public health crisis:
  • Bimodal distribution: rare high-energy fractures in the young; overwhelmingly common fragility fractures in the elderly (mean age ~80 years, female:male ~3:1)
  • 15–20% mortality within 3 months; 20–25% mortality at 1 year for trochanteric fractures
  • 30–50% lose a degree of functional independence
  • ~4.5 million fractures/year worldwide projected by 2050 (up from 1.3 million in 1990)
  • Strongly associated with osteoporosis, falls, sarcopenia, polypharmacy, and frailty

Classification by Anatomical Location

Proximal femur fracture classification — intracapsular, basal, trochanteric, subtrochanteric
Basic classification of proximal femur fractures. — Rockwood & Green's Fractures in Adults 10e
The critical division is intracapsular vs. extracapsular — this determines the risk of avascular necrosis (AVN) and dictates surgical strategy.
TypeLocationAVN RiskKey Issue
Intracapsular (femoral neck)Subcapital, transcervical, basicervicalHigh (15–35% displaced)Blood supply to femoral head at risk
Extracapsular — TrochantericInter-trochanteric regionLowMechanical instability, varus collapse
Extracapsular — Subtrochanteric≤5 cm distal to lesser trochanterLowHigh bending forces; challenging fixation

1. Intracapsular (Femoral Neck) Fractures

Blood Supply

The femoral head receives blood from retinacular vessels (branches of the medial and lateral circumflex femoral arteries) that run along the femoral neck. Displacement tears these vessels → AVN of the femoral head in 15–35% of displaced fractures.

Garden Classification

Garden Classification of Hip Fractures
Garden Classification. — Grainger & Allison's Diagnostic Radiology
Garden GradeDescriptionAVN Risk
IUndisplaced incomplete (impacted in valgus)Low
IIComplete, no displacementLow
IIIComplete, varus angulationHigh
IVCompletely displacedHigh
Clinically simplified as: Undisplaced (I + II) vs. Displaced (III + IV)

Treatment

Patient GroupFractureTreatment
Young patient (<60 yrs)Undisplaced (Garden I/II)Internal fixation — multiple cannulated screws or dynamic hip screw (DHS); preserve the femoral head
Young patientDisplaced (Garden III/IV)Urgent ORIF (emergency to restore blood supply); if irreducible → hemiarthroplasty
Elderly, independently mobileAny displacementTotal Hip Arthroplasty (THA) — better functional outcomes than hemiarthroplasty in active patients
Elderly, low demand / frailDisplacedHemiarthroplasty (cemented preferred for stability and pain)
Poor bone stockUndisplacedHemiarthroplasty/THA rather than fixation
Key principle: Displaced intracapsular fractures in the elderly are generally best treated with arthroplasty rather than fixation (high failure and revision rate with fixation due to AVN and non-union).

2. Extracapsular — Trochanteric Fractures

These fractures have a good blood supply but are mechanically unstable — they collapse into varus without surgical fixation.

OTA/AO Classification

Trochanteric fracture classification — A1, A2, A3
OTA/AO Classification of trochanteric hip fractures. — Rockwood & Green's Fractures in Adults 10e
AO TypePatternStabilityLateral Wall
31.A1Two-part; fracture through trochantersStableIntact
31.A2Comminuted; lesser trochanter detached; ≥3 main fragmentsUnstableIntact but compromised
31.A3Reverse oblique or transverse; fracture line extends laterallyVery unstableIncompetent

Treatment

PatternDevice
A1 / A2 (intact lateral wall)Sliding Hip Screw (Dynamic Hip Screw, DHS) + side plate; allows controlled collapse and impaction
A3 / reverse obliquity / subtrochanteric extension / compromised lateral wallIntramedullary nail (cephalomedullary nail, e.g., PFNA, Gamma nail) — controls rotation, resists bending, allows weight-bearing
Key point for DHS: Screw tip must be close to the articular surface (tip-apex distance <25 mm) to prevent cut-out.

3. Subtrochanteric Fractures

  • Region of highest mechanical stress in the skeleton (bending + compressive forces)
  • Occur at medial calcar — area of cortical stress concentration
  • Associated with bisphosphonate therapy (atypical subtrochanteric fractures with prodromal thigh pain and a "banana" lateral bow on plain X-ray)
  • Treatment: Intramedullary nail (long, to the knee, to avoid stress risers)

Clinical Presentation

FeatureFinding
HistoryFall from standing height (elderly); high-energy in young
PainGroin, hip, inner thigh; referred to knee
Limb positionShortened + externally rotated (displaced); neutral position (undisplaced)
Inability to weight-bearTypical, but undisplaced fractures may still walk
TendernessOver greater trochanter or groin
~15% of hip fractures are occult on plain X-ray — use MRI (gold standard) or CT if high clinical suspicion with normal X-ray.

Perioperative Management

Timing of Surgery

  • Target: within 24–48 hours of admission (once medically optimised)
  • Delay >48 hours associated with higher mortality, increased pressure sores, DVT, pneumonia
  • Brief delay acceptable for reversing anticoagulation or optimising significant cardiorespiratory comorbidity

Anaesthesia

  • Spinal vs. general: No significant difference in 60-day mortality or delirium in large prospective studies; slightly shorter hospital stay with regional anaesthesia
  • Regional analgesia: Fascia iliaca block or 3-in-1 femoral nerve block — effective pre- and post-operative pain control; avoids respiratory depression of intrathecal opioids

Perioperative Considerations

  • Thromboprophylaxis: LMWH or direct oral anticoagulants post-op; mechanical prophylaxis (TED stockings, pneumatic compression) pre-op
  • Delirium prevention: Avoid anticholinergics, benzodiazepines; early mobilisation; adequate analgesia; sleep hygiene
  • Orthogeriatric co-management: Geriatrician involvement from admission reduces mortality and complications
  • Bone health: Osteoporosis treatment (bisphosphonates, denosumab) must be initiated prior to discharge — a hip fracture is a sentinel event for future fractures

Falls Risk Factors to Address

  • Polypharmacy review (antihypertensives, tranquillisers, alcohol)
  • Visual correction
  • Balance and strength physiotherapy
  • Home hazard assessment

Complications

ComplicationNotes
AVN of femoral headIntracapsular fractures; 15–35% of displaced; may present months–years later
Non-unionMore common with displaced intracapsular fractures treated with fixation
Fixation failure / screw cut-outDHS: tip-apex distance >25 mm; osteoporosis
DVT / PEHigh risk; mandatory prophylaxis
PneumoniaFrom immobility; early mobilisation is protective
DeliriumVery common post-op in elderly hip fracture patients
Pressure soresFrom prolonged bed rest
Mortality~20–25% at 1 year

Summary Decision Framework

Hip Fracture
│
├── Intracapsular (femoral neck)
│   ├── Undisplaced (Garden I/II)
│   │   ├── Young → Internal fixation (cannulated screws)
│   │   └── Elderly / poor bone → Hemiarthroplasty
│   └── Displaced (Garden III/IV)
│       ├── Young → Urgent ORIF (or hemi if irreducible)
│       ├── Active elderly → THA
│       └── Frail elderly → Hemiarthroplasty (cemented)
│
└── Extracapsular
    ├── Trochanteric (A1/A2, intact lateral wall) → DHS
    ├── Trochanteric (A3 / reverse oblique) → IM nail
    └── Subtrochanteric → Long IM nail

Sources: Rockwood & Green's Fractures in Adults 10e, Ch. 54; Mulholland & Greenfield's Surgery 7e, Ch. 28; Grainger & Allison's Diagnostic Radiology; Morgan & Mikhail's Clinical Anesthesiology 7e, Ch. 38

Femur fracture

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Here is a comprehensive overview of Femur Fractures, covering the shaft and distal femur in detail, drawn from Rockwood & Green's Fractures in Adults 10e.

Femur Fractures

The femur is the largest, strongest bone in the body. Fracturing it requires substantial force — making it a marker of high-energy trauma and a cause of significant haemorrhage, systemic injury, and mortality.

Overview: Regions of the Femur

RegionCovered In
Proximal femur (femoral neck, trochanteric, subtrochanteric)← See Hip Fracture topic
Femoral shaft (diaphysis)This section
Distal femur (supracondylar/condylar)This section

Part 1: Femoral Shaft (Diaphyseal) Fractures

Epidemiology & Mechanism

Femoral shaft fractures show a bimodal distribution:
  • Young males (15–25 years): High-energy trauma — MVA, motorcycle accidents, falls from height, pedestrian injuries, gunshot wounds
  • Elderly females (>75 years): Low-energy falls on osteoporotic bone
  • Atypical femur fractures (50–70 years): Bisphosphonate-associated; transverse subtrochanteric or midshaft fractures with prodromal thigh pain and lateral cortical thickening ("beaking")
Fracture patterns by force:
  • Bending force → transverse ± butterfly fragment
  • Rotational force → spiral or oblique
  • Axial force → associated hip/knee injuries
  • Higher energy → comminution

Haemorrhage

The femoral shaft lies within the thigh musculature — closed femoral shaft fractures can result in 1.0–1.5 L of blood loss into the thigh. In multiply injured patients, this is a major contributor to haemorrhagic shock. Stabilise early.

Associated Injuries

In a review of 26,357 femur fractures:
  • Lung injury: 18.9%
  • Intracranial injury: 13.5%
  • Liver: 6.2%
  • Tibia/fibula fractures: 20.5%
  • Ribs/sternum: 19.1%
  • Ipsilateral femoral neck fracture: 5.8% — missed in 20–50% of cases initially
Always image the full femur, including the hip and knee. Protocol: internal rotation AP hip view + fine-cut CT of the hip (2 mm axial/sagittal) + post-op radiographs → 91% reduction in missed femoral neck fractures.

Classification — Winquist & Hansen (Comminution)

GradeComminutionCortical Contact
0None100%
ISmall butterfly <25%≥75%
IIButterfly ~25–50%≥50%
IIIButterfly ~50–75%Minimal
IVComplete; no cortical contactNone (segmental)
Higher grade = requires statically locked nail to prevent shortening and rotation.

Treatment

Timing: Stabilise the femur within 24 hours in multiply-injured patients → reduces pulmonary complications (Bone et al.).

1. Temporary Stabilisation

MethodUse
Hare traction splint (skin traction)Prehospital / field — restore length, reduce pain
Skeletal traction (distal femur or proximal tibia pin)In-hospital bridge to definitive surgery in polytrauma or haemodynamically unstable patients
External fixationDamage control orthopaedics — polytrauma, open fracture with contamination, vascular injury needing repair, medullary contamination
Skeletal traction pin placement:
  • Distal femur pin: placed medial → lateral (avoids femoral artery at adductor hiatus)
  • Proximal tibia pin: placed lateral → medial (avoids peroneal nerve)
  • Weight: ~15% of body weight (15–20 lb)

2. Definitive Fixation

Intramedullary Nailing (IMN) — gold standard for most femoral shaft fractures
ParameterDetail
Entry pointAntegrade (piriformis fossa or greater trochanter tip) or Retrograde (via knee, intercondylar notch)
Reamed vs. unreamedReamed preferred — improves union rates; reaming does not increase mortality in trauma patients
LockingStatic locking (proximal + distal interlocking screws) — controls length and rotation in comminuted fractures
Weight bearingImmediate weight-bearing allowed after static locking even in comminuted fractures
Indications for IMN:
  • Most closed and open femoral shaft fractures
  • Segmental and comminuted fractures
  • Multiple trauma (within 24 hours when stable)
Relative contraindications to IMN:
  • Canal too narrow (unable to ream)
  • Pre-existing implant filling the canal (e.g., THA)
  • Associated ipsilateral femoral neck fracture (use separate fixation)
  • Open growth plates (piriformis entry avoided; trochanteric entry used)
  • Severe polytrauma with chest/head injury → temporary external fixation first
Plate Fixation (less common for shaft):
  • Used when IMN is contraindicated or in certain fracture patterns
  • Open approach (lateral): extensive dissection → higher blood loss, infection risk
  • MIPO (minimally invasive plate osteosynthesis): smaller incisions, preserves blood supply, but higher risk of malreduction
  • Plate is not load-bearing → immediate weight-bearing carries implant failure risk

Part 2: Distal Femur (Supracondylar/Condylar) Fractures

Definition

The distal 15 cm of the femur, from the condyles to the junction of metaphysis and diaphysis.

Epidemiology

  • Bimodal: Young (high-energy) and elderly (fragility fracture)
  • Elderly distal femur fracture = analogous to hip fracture in terms of morbidity; 5-year mortality >50%
  • Often complicates total knee arthroplasty (periprosthetic fracture)

OTA/AO Classification

OTA/AO Classification of Distal Femur Fractures (33A–C)
OTA/AO classification of distal femur fractures. — Rockwood & Green's Fractures in Adults 10e
TypeDescription
33-A (Extra-articular)Supracondylar; joint surface not involved
33-B (Partial articular)One condyle involved; rest of shaft in continuity
33-C (Complete articular)Bicondylar; complete separation from shaft ("T" or "Y" fracture)
Each type is subdivided 1–3 by increasing comminution.
Hoffa fracture: Coronal plane fracture through a single condyle — found in 40% of intercondylar fractures; frequently missed on plain X-ray — CT mandatory.

Key Anatomy

  • Distal femur is trapezoidal on end — implants placed anteriorly on AP view may protrude posteriorly and cause pain
  • Popliteal artery lies posterior to the distal femur — at risk in displaced fractures
  • Peroneal and tibial nerves in the popliteal fossa — check neurovascular status

Deforming Force

The gastrocnemius pulls the distal fragment into hyperflexion/posterior angulation — this must be corrected at reduction (knee flexion during surgery reduces this deformity).

Treatment

FractureManagement
Minimally displaced extra-articularPosterior long-arm splint with knee at 90° → early motion
Most displaced / articular fracturesORIF — lateral locking plate (± medial plate for very comminuted cases); goal = anatomic articular reduction + stable fixation + early ROM
Retrograde IM nailGood for extra-articular and some partial articular fractures; also useful in periprosthetic fractures above TKA
Periprosthetic above TKARetrograde nail if adequate distal space; locked plating if not
Severely comminuted, elderlyTotal Knee Arthroplasty (TKA) or distal femur replacement (megaprosthesis)
Intra-articular fractures: Anatomic reduction of the joint surface is paramount — tolerances <2 mm step-off — to reduce risk of post-traumatic arthritis.
Stiffness is the most common complication of distal femur fractures — early range of motion is critical.

Complications Summary

ComplicationNotes
Haemorrhagic shock1–1.5 L blood loss in closed shaft fracture
Fat embolism syndromeEspecially with bilateral or high-energy femur fractures; triad: hypoxia + petechiae + confusion
Missed ipsilateral neck fractureUp to 50% missed on initial presentation — use CT protocol
MalunionEspecially with non-operative treatment; leg length discrepancy, angular or rotational deformity
Non-unionUnion rates with IMN ~97–100%; higher with plate and open fractures
Knee stiffnessMost common after distal femur fractures and prolonged traction
Post-traumatic arthritisIntra-articular distal femur fractures
InfectionOpen fractures; pin tract infections with external fixation (>50%)
Implant failurePlates especially prone in osteoporotic bone without early mobilisation

Sources: Rockwood & Green's Fractures in Adults 10e, Ch. 57 (Femoral Shaft), Ch. 58–59 (Distal Femur); Mulholland & Greenfield's Surgery 7e
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