Explain 10 heart disease with ECG change and ECG images of those disease

Reading File
Finding Sources
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ECG changes myocardial infarction ST elevation

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atrial fibrillation ECG irregular rhythm no P waves

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complete heart block third degree AV block ECG

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Wolff-Parkinson-White syndrome ECG delta wave short PR

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hypertrophic cardiomyopathy ECG left ventricular hypertrophy

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pericarditis ECG saddle shaped ST elevation diffuse

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Brugada syndrome ECG coved ST elevation V1 V2 V3

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long QT syndrome ECG prolonged QT interval torsades de pointes

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pulmonary embolism ECG S1Q3T3 right heart strain sinus tachycardia

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dilated cardiomyopathy ECG left bundle branch block poor R wave progression

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hyperkalemia ECG peaked T waves wide QRS sine wave

Here is a comprehensive guide to 10 heart diseases and their characteristic ECG changes, with clinical ECG images for each.

10 Heart Diseases with ECG Changes


1. πŸ«€ ST-Elevation Myocardial Infarction (STEMI)

ECG Changes

  • Hyperacute T waves β€” earliest sign (peaked, broad T waves)
  • ST-segment elevation β‰₯1 mm in β‰₯2 contiguous leads (convex/tombstone morphology)
  • Reciprocal ST depression in opposing leads
  • Pathological Q waves β€” develop within hours (>25% of R wave height, >40 ms wide)
  • T-wave inversion in the evolving/chronic phase
TerritoryLeads with ST ElevationArtery
AnteriorV1–V4LAD
InferiorII, III, aVFRCA
LateralI, aVL, V5–V6LCx
PosteriorV1–V3 (tall R + ST depression)RCA/LCx

ECG Image β€” Anterior STEMI (LAD occlusion)

Anterior STEMI ECG
12-lead ECG showing convex ST elevation in V1–V6 with hyperacute T waves and reciprocal inferior ST depression. Classic proximal LAD occlusion pattern.

ECG Image β€” Inferior STEMI with posterior extension

Inferior STEMI ECG
ST elevation in II, III, aVF with reciprocal depression in I and aVL; tall R waves and ST depression V1–V3 indicating posterior involvement.

2. πŸ’“ Atrial Fibrillation (AF)

ECG Changes

  • Absent P waves β€” replaced by chaotic fibrillatory (f) waves, most visible in V1
  • Irregularly irregular RR intervals β€” hallmark feature
  • Narrow QRS complexes (unless aberrant conduction or pre-excitation)
  • Rate: can be rapid (>100 bpm), controlled, or slow
  • Fine vs. coarse f waves (coarse may mimic flutter)

ECG Image β€” Atrial Fibrillation

Atrial Fibrillation ECG
Classic AF: completely irregular R-R intervals, absent P waves, fine f waves most prominent in V1. Rapid ventricular response ~126 bpm.

3. πŸ”‡ Complete (Third-Degree) AV Heart Block

ECG Changes

  • Complete AV dissociation β€” P waves and QRS complexes are totally independent
  • Atrial rate > ventricular escape rate
  • Regular P-P intervals and regular R-R intervals but NO consistent relationship
  • Wide QRS (>120 ms) if ventricular escape focus; narrow if junctional escape
  • Bradycardia β€” escape rate 20–40 bpm (ventricular) or 40–60 bpm (junctional)

ECG Image β€” Complete Heart Block

Complete AV Block ECG
Third-degree AV block: P waves march through QRS complexes with no fixed PR interval. Wide ventricular escape complexes at a slow rate.

4. ⚑ Wolff-Parkinson-White (WPW) Syndrome

ECG Changes

  • Short PR interval (<120 ms) β€” rapid conduction through accessory pathway
  • Delta wave β€” slurred upstroke at onset of QRS (pre-excitation of ventricle)
  • Wide QRS β€” total duration >120 ms (delta + normal conduction fusion)
  • Secondary ST-T changes β€” discordant from QRS
  • Delta wave polarity localizes the accessory pathway
  • Risk: AF with pre-excitation can cause ventricular fibrillation

ECG Image β€” WPW Syndrome

WPW ECG
Classic WPW triad: short PR interval, prominent delta waves (slurred initial QRS), and broadened QRS complexes. Pathway localized by delta wave polarity.

5. 🧱 Hypertrophic Cardiomyopathy (HCM)

ECG Changes

  • Left ventricular hypertrophy (LVH) β€” increased QRS voltage (Sokolow-Lyon: S V1 + R V5 > 35 mm)
  • Deep, symmetric T-wave inversions β€” especially lateral and precordial leads; "giant" negative T waves in apical HCM (Yamaguchi pattern)
  • Strain pattern β€” ST depression with T-wave inversion in V4–V6, I, aVL
  • Absence of septal Q waves in I, aVL, V5–V6 (abnormal septal depolarization)
  • Abnormal Q waves in inferior/lateral leads mimicking infarction

ECG Image β€” HCM with Apical Variant

HCM ECG
Apical HCM (Yamaguchi syndrome): high-voltage QRS in V4–V6 with giant deep T-wave inversions in V2–V5 β€” classic "ace of spades" apical morphology.

ECG Image β€” Septal HCM with strain pattern

HCM Septal ECG
Septal HCM: LVH voltage criteria, ST depression and deep T inversions V2–V6, aVL, and a positive T wave in aVR (high arrhythmic risk marker).

6. πŸ”₯ Acute Pericarditis

ECG Changes (Classically Evolves Through 4 Stages)

StageECG Finding
IDiffuse concave (saddle-shaped) ST elevation in all leads except aVR/V1; PR depression
IIST returns to baseline; PR depression persists
IIIT-wave inversion (diffuse)
IVECG normalizes
  • PR segment depression β€” most specific finding (due to atrial epicarditis); reciprocal PR elevation in aVR
  • Spodick's sign β€” downward slope of TP segment
  • ST elevation is diffuse (not territory-specific), and there are no reciprocal ST depressions (unlike STEMI)

ECG Image β€” Acute Pericarditis

Pericarditis ECG
Diffuse saddle-shaped ST elevation in I, II, III, aVF, V2–V6 with PR depression in II and Spodick's sign. Sinus tachycardia from systemic inflammation.

7. 🧬 Brugada Syndrome

ECG Changes

Three ECG patterns recognized:
PatternST MorphologyDiagnostic
Type 1 (coved)J-point β‰₯2 mm, convex/coved ST descending to inverted TDiagnostic of Brugada syndrome
Type 2 (saddle-back)J-point β‰₯2 mm, saddle-back ST with positive TNot diagnostic alone
Type 3<2 mm elevation, saddle-backNot diagnostic
  • Location: V1–V3 (or V1–V2 with leads placed in 3rd/2nd intercostal space)
  • Spontaneous Type 1 is most significant; provocation with ajmaline/flecainide can unmask
  • Risk of polymorphic VT and sudden cardiac death, especially at rest/night

ECG Image β€” Brugada Type 1

Brugada ECG
Classic Type 1 Brugada coved pattern: J-point elevation β‰₯2 mm in V1–V2 with convex, downward-sloping ST descending into inverted T wave.

8. πŸ” Long QT Syndrome (LQTS)

ECG Changes

  • Prolonged QTc interval β€” QTc >440 ms (men), >460 ms (women); >500 ms = high risk
  • T-wave abnormalities β€” broad, notched, or biphasic T waves
  • T-wave alternans β€” beat-to-beat variation in T-wave morphology (severe LQTS)
  • Torsades de Pointes (TdP) β€” polymorphic VT that "twists" around the isoelectric line; triggered by R-on-T
LQTS SubtypeT-Wave PatternTrigger
LQT1Broad-based T waveExercise
LQT2Notched/bifid T waveAuditory stimuli
LQT3Late-onset, peaked T waveRest/sleep

ECG Image β€” LQTS with Torsades de Pointes

Long QT ECG
Marked QTc prolongation with R-on-T phenomenon triggering classic torsades de pointes β€” polymorphic VT twisting around the isoelectric baseline.

9. 🩸 Acute Pulmonary Embolism (PE)

ECG Changes

  • Sinus tachycardia β€” most common finding (80%)
  • S1Q3T3 pattern (McGinn-White sign) β€” S wave in I, Q wave and inverted T in III
  • Right bundle branch block (RBBB) β€” complete or incomplete, due to acute RV pressure overload
  • T-wave inversions V1–V4 β€” RV strain pattern
  • Right axis deviation
  • P pulmonale β€” peaked P waves in II (right atrial enlargement)
  • Atrial arrhythmias β€” AF, flutter from RV dilation

ECG Image β€” Pulmonary Embolism with S1Q3T3

PE ECG
PE with right heart strain: sinus tachycardia, S1Q3T3 pattern (prominent S in I, Q and inverted T in III), T inversions V1–V3, incomplete RBBB morphology.

10. 🫁 Dilated Cardiomyopathy (DCM)

ECG Changes

  • Left bundle branch block (LBBB) β€” broad notched R waves in I, aVL, V5–V6; deep S in V1–V3; >120 ms QRS
  • Left ventricular hypertrophy β€” increased voltage criteria
  • Poor R-wave progression in precordial leads (V1–V4 fail to develop normal R waves)
  • Non-specific ST-T changes β€” ST depression, T-wave flattening or inversion
  • Pathological Q waves β€” from fibrosis/scar mimicking infarction
  • Atrial fibrillation or flutter β€” from atrial dilatation
  • Ventricular ectopy β€” frequent PVCs, NSVT
  • Prolonged PR interval (1st degree AV block)

ECG Image β€” Dilated Cardiomyopathy with LBBB

DCM LBBB ECG
LBBB in DCM: wide QRS >120 ms, broad monophasic R waves in I/aVL/V5–V6, deep S waves in V1–V3, discordant ST-T changes. Often an indication for CRT.

Quick Reference Summary Table

#DiseaseKey ECG Finding
1STEMIST elevation (convex) + reciprocal depression + Q waves
2Atrial FibrillationIrregular RR, absent P waves, f-waves
3Complete AV BlockP-QRS dissociation, bradycardia, escape rhythm
4WPW SyndromeShort PR + delta wave + wide QRS
5Hypertrophic CardiomyopathyLVH + giant T inversions (apical) or strain pattern
6Acute PericarditisDiffuse saddle ST elevation + PR depression
7Brugada SyndromeCoved ST elevation V1–V3 + inverted T wave
8Long QT SyndromeProlonged QTc + T-wave abnormalities + TdP
9Pulmonary EmbolismS1Q3T3 + sinus tachycardia + RBBB + RV strain
10Dilated CardiomyopathyLBBB + poor R progression + arrhythmias

Sources: Braunwald's Heart Disease (10th ed.), Rosen's Emergency Medicine; ECG images from PMC Clinical VQA database.
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