Learn ecg 10 common

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ECG electrocardiogram common patterns interpretation

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normal sinus rhythm ECG waveform PQRST

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atrial fibrillation ECG irregularly irregular rhythm

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STEMI ST elevation myocardial infarction ECG 12-lead

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ventricular tachycardia fibrillation ECG wide complex

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left bundle branch block right bundle branch block ECG

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complete heart block third degree AV block ECG

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atrial flutter sawtooth waves ECG

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sinus bradycardia tachycardia ECG rate

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Wolff-Parkinson-White WPW delta wave ECG short PR

I now have rich content from the medical library and images for all 10 common ECG patterns. Let me compile the comprehensive guide.

πŸ«€ 10 Common ECG Patterns β€” A Complete Learning Guide


How to Read an ECG: The Basics First

Paper speed: 25 mm/sec β†’ each small box = 0.04 s; each large box = 0.2 s Heart rate (quick): 300 Γ· number of large boxes between R waves
The PQRST sequence:
  • P wave β€” atrial depolarization (SA node β†’ atria)
  • PR interval β€” AV nodal conduction delay (normal: 0.12–0.20 s)
  • QRS complex β€” ventricular depolarization (normal: < 0.12 s)
  • ST segment β€” early repolarization (should be isoelectric)
  • T wave β€” ventricular repolarization
"Electrical signals are transmitted from the Purkinje system to individual ventricular cardiomyocytes. The spread of depolarization to the ventricular myocardium is exhibited as the QRS complex on the ECG. Depolarization is followed by ventricular repolarization and the appearance of the T wave." β€” Miller's Anesthesia, 10e

1. Normal Sinus Rhythm (NSR)

Rate: 60–100 bpm Key features:
  • Regular P waves before every QRS (upright in II, inverted in aVR)
  • PR interval 0.12–0.20 s
  • Narrow QRS < 0.12 s
  • Regular R-R intervals
Normal sinus rhythm ECG showing classic PQRST complex
Clinical significance: The baseline from which all abnormalities are compared.

2. Sinus Bradycardia

Rate: < 60 bpm Key features:
  • Normal P-QRS-T morphology
  • Regular rhythm, just slower
  • All other intervals normal
Sinus bradycardia ECG at approximately 45 bpm with narrow QRS
Causes: Athletes, hypothyroidism, beta-blockers, inferior MI, increased vagal tone Treat if: Symptomatic (dizziness, syncope, hypotension) β†’ Atropine, then pacing

3. Atrial Fibrillation (AF)

Rate: Ventricular rate variable (usually 100–160 bpm if uncontrolled) Key features:
  • No P waves β€” replaced by chaotic fibrillatory (f) waves, best seen in V1
  • Irregularly irregular R-R intervals (hallmark)
  • Narrow QRS (unless aberrant conduction)
Atrial fibrillation ECG showing irregularly irregular rhythm, absent P waves, fibrillatory baseline
Causes: Hypertension, valvular disease, hyperthyroidism, alcohol Key risks: Stroke (thrombus in LAA) β†’ anticoagulate if CHAβ‚‚DSβ‚‚-VASc β‰₯ 2 (men) / β‰₯ 3 (women) Treatment: Rate control (beta-blocker, diltiazem) or rhythm control (cardioversion, amiodarone)

4. Atrial Flutter

Rate: Atrial ~300 bpm; ventricular usually 150 bpm (2:1 block) Key features:
  • Classic sawtooth flutter waves (F-waves) at 300/min β€” most visible in II, III, aVF
  • Regular ventricular rate (fixed AV conduction ratio, e.g., 2:1, 3:1, 4:1)
  • No isoelectric baseline between F-waves
Atrial flutter ECG showing classic sawtooth F-waves in inferior leads, regular ventricular response
Mechanism: Macro-reentrant circuit around tricuspid valve (cavotricuspid isthmus) Treatment: Rate control; cardioversion; catheter ablation (highly effective)

5. ST-Elevation MI (STEMI)

Key features:
  • ST elevation β‰₯ 1 mm in β‰₯ 2 contiguous limb leads, or β‰₯ 2 mm in V1–V3
  • Reciprocal ST depression in opposite leads
  • Hyperacute (peaked) T waves early; Q waves develop later
  • Location tells you the artery:
TerritoryLeadsArtery
AnteriorV1–V4LAD
InferiorII, III, aVFRCA
LateralI, aVL, V5–V6LCx
PosteriorST depression V1–V3RCA/LCx
Anterior STEMI ECG showing ST elevation V1-V6 with tombstoning morphology and reciprocal changes inferiorly
Inferolateral STEMI showing ST elevation in II, III, aVF and lateral leads with reciprocal ST depression in I, aVL
Emergency action: Activate cath lab β€” door-to-balloon ≀ 90 min

6. Left Bundle Branch Block (LBBB)

Key features:
  • Broad QRS > 0.12 s (usually > 0.14 s)
  • Broad, notched R in I, aVL, V5–V6 ("M" or "W" pattern)
  • Deep QS in V1 (no r wave)
  • Secondary ST/T discordance (T wave opposite QRS direction)
  • New LBBB + chest pain = treat as STEMI equivalent
LBBB ECG showing wide QRS, broad notched R waves in lateral leads, QS in V1
Causes: Hypertension, dilated cardiomyopathy, anterior MI, cardiac surgery

7. Right Bundle Branch Block (RBBB)

Key features:
  • Broad QRS > 0.12 s
  • rSR' ("rabbit ears") in V1 β€” terminal R' wave
  • Broad/slurred S wave in I, V5, V6
  • T-wave inversion in V1–V3 (secondary change)
RBBB ECG showing rSR' pattern in V1, wide S waves in lateral leads, left axis deviation
Causes: Often normal variant; also PE (acute RBBB), RV hypertrophy, anterior MI Memory: LBBB = WiLLiaM (W in V1, M in V5), RBBB = MaRRoW (M in V1, W in V5)

8. Third-Degree (Complete) Heart Block

Key features:
  • Complete AV dissociation β€” P waves and QRS complexes are independent
  • Atrial rate > ventricular rate
  • Regular P-P intervals, regular R-R intervals (but NO relationship between them)
  • Escape rhythm: junctional (narrow QRS ~40–60 bpm) or ventricular (wide QRS ~20–40 bpm)
Complete heart block ECG showing total AV dissociation with independent P waves and slow escape rhythm
Third-degree heart block with narrow-complex junctional escape rhythm at 36 bpm
Causes: Inferior MI (RCA), Lyme disease, drugs (digoxin, beta-blockers), fibrosis Treatment: Atropine (temporary), urgent transvenous/permanent pacemaker

9. Ventricular Tachycardia / Fibrillation

Ventricular Tachycardia (VT)

  • Rate: 100–250 bpm
  • Broad QRS > 0.12 s (usually > 0.14 s)
  • Regular (monomorphic VT) or irregular (polymorphic VT/Torsades)
  • AV dissociation, fusion beats, capture beats (diagnostic)

Ventricular Fibrillation (VF)

  • Chaotic, irregular waveforms β€” no discernible P, QRS, or T
  • No cardiac output β†’ cardiac arrest
Ventricular fibrillation ECG showing chaotic irregular wide-complex waveforms
Torsades de Pointes showing polymorphic VT with twisting QRS axis around isoelectric line
VT: Amiodarone, cardioversion if unstable; VF: Immediate defibrillation Torsades (prolonged QT): Stop offending drug, IV Magnesium 2 g

10. Wolff-Parkinson-White (WPW) Syndrome

Key features:
  • Short PR interval < 0.12 s (bypasses AV nodal delay)
  • Delta wave β€” slurred upstroke of QRS (pre-excitation)
  • Widened QRS (> 0.10 s due to delta wave)
  • Risk of rapid AF over accessory pathway β†’ hemodynamic collapse
WPW ECG showing classic short PR interval, delta waves, and widened QRS complexes
DANGER: Never give AV-nodal blocking drugs (adenosine, digoxin, verapamil, beta-blockers) in WPW + AF β†’ can precipitate VF Treatment: Procainamide (acute); radiofrequency ablation of accessory pathway (definitive)

Quick Reference Summary Table

#PatternRateP WavesQRSKey Feature
1Normal Sinus Rhythm60–100Normal, uprightNarrowBaseline normal
2Sinus Bradycardia< 60NormalNarrowSlow but regular
3Atrial FibrillationVariableAbsent (f-waves)NarrowIrregularly irregular
4Atrial Flutter~150 (2:1)Sawtooth F-wavesNarrowRegular saw pattern
5STEMIVariableNormalNarrow (initially)ST elevation + reciprocal ↓
6LBBBVariableNormalWide (M in V5)No r in V1, M in V6
7RBBBVariableNormalWide (rSR' V1)Rabbit ears V1, S wave I/V6
83Β° Heart BlockAtrial > VentDissociatedWide (escape)P & QRS independent
9VT/VF100–250/chaoticOften noneWide/chaoticEmergency β€” shock/defib
10WPWVariableNormalWide (delta)Short PR + delta wave

A Systematic Approach (Never Miss Anything)

  1. Rate β€” 300 Γ· large boxes between R-R
  2. Rhythm β€” regular or irregular?
  3. Axis β€” normal (I ↑, aVF ↑), LAD, RAD
  4. P waves β€” present? morphology? 1:1 with QRS?
  5. PR interval β€” short (< 0.12), normal, long (> 0.20)?
  6. QRS duration β€” narrow (< 0.12) or wide (> 0.12)?
  7. ST segment β€” elevation, depression, or flat?
  8. T waves β€” upright, inverted, peaked?
  9. QT interval β€” corrected QTc (normal < 440 ms men, < 460 ms women)

Sources: Miller's Anesthesia 10e; Fuster and Hurst's The Heart 15e; Tintinalli's Emergency Medicine; Braunwald's Heart Disease; Harrison's Principles of Internal Medicine 21e
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