Explain me 10 common heart diseases with ECG changes and ECG images of those diseases

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ECG myocardial infarction ST elevation changes

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atrial fibrillation ECG irregular rhythm no P waves

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heart block complete AV block ECG third degree

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ventricular tachycardia ECG wide complex tachycardia

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left ventricular hypertrophy ECG voltage criteria

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pulmonary embolism ECG S1Q3T3 right heart strain

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Wolff-Parkinson-White syndrome delta wave short PR ECG

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pericarditis ECG diffuse ST elevation saddle shape PR depression

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hyperkalemia ECG peaked T waves wide QRS sine wave

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dilated cardiomyopathy heart failure ECG left bundle branch block poor R wave progression

Here is a comprehensive guide to 10 common heart diseases with their ECG changes and ECG images.

10 Common Heart Diseases: ECG Changes & Images


1. πŸ«€ Acute Myocardial Infarction (STEMI) β€” Anterior

ECG Changes:

  • Hyperacute T waves (earliest sign β€” tall, peaked, broad-based)
  • ST-segment elevation β‰₯1 mm in β‰₯2 contiguous leads (convex "tombstone" morphology)
  • Pathological Q waves (>40 ms wide, >25% of QRS height) β€” indicate necrosis
  • Reciprocal ST depression in opposing leads
  • T-wave inversion in later stages
  • Lead distribution: V1–V4 (anterior/LAD territory), V5–V6 + I + aVL (anterolateral), II/III/aVF (inferior/RCA or LCx)

Anatomical Correlation:

TerritoryLeads with ST elevationArtery
AnteriorV1–V4LAD (proximal)
AnterolateralV1–V6, I, aVLLAD (proximal)
InferiorII, III, aVFRCA or LCx
PosteriorV1–V3 (depression + tall R)LCx or RCA

ECG Image β€” Anterolateral STEMI:

Anterolateral STEMI β€” ST elevation V1–V6, I, aVL with reciprocal inferior depression

ECG Image β€” Inferior STEMI (with posterior involvement):

Inferior STEMI β€” ST elevation II, III, aVF; posterior changes V1–V3

2. β€οΈβ€πŸ©Ή Atrial Fibrillation (AF)

ECG Changes:

  • No visible P waves β€” replaced by chaotic fibrillatory (f) waves, best seen in V1 and II
  • Irregularly irregular R-R intervals (hallmark sign)
  • Narrow QRS (unless aberrant conduction/bundle branch block)
  • Ventricular rate: uncontrolled (>100 bpm), controlled (60–100), or bradycardic (<60)
  • Fine vs. coarse AF: coarse f-waves can mimic atrial flutter

ECG Image β€” Atrial Fibrillation (fast ventricular response):

AF β€” irregularly irregular rhythm, absent P waves, fine fibrillatory baseline

ECG Image β€” Coarse AF:

Coarse AF β€” prominent fibrillatory waves in V1, irregular RR intervals

3. πŸ›‘ Complete (Third-Degree) AV Block

ECG Changes:

  • Complete AV dissociation β€” P waves and QRS complexes are completely independent
  • P waves occur at a faster regular rate (atrial rate)
  • QRS complexes occur at a slower, regular escape rate (ventricular rate)
  • Wide QRS (>120 ms) if escape rhythm is ventricular (infra-Hisian); narrow QRS if junctional escape
  • No fixed PR interval β€” P waves "march through" QRS complexes
  • Typically presents with bradycardia (ventricular rate 30–45 bpm)

ECG Image β€” Complete (3rd Degree) AV Block:

Complete AV block β€” AV dissociation, wide QRS escape rhythm, independent P waves

ECG Image β€” 3rd Degree Block (junctional escape, narrow QRS):

Complete heart block β€” narrow complex escape, P waves unrelated to QRS

4. ⚑ Ventricular Tachycardia (VT)

ECG Changes:

  • Wide QRS (>120 ms) at rate >100 bpm
  • Monomorphic VT: uniform QRS morphology, regular rhythm
  • AV dissociation (P waves unrelated to QRS) β€” pathognomonic when seen
  • Fusion beats and capture beats (Dressler beats) β€” confirm ventricular origin
  • Concordance: all precordial leads positive (positive) or negative (negative) β€” strongly suggests VT
  • Northwest axis (extreme left axis deviation)
  • Rate typically 140–220 bpm

ECG Image β€” Monomorphic VT (wide complex tachycardia):

Monomorphic VT β€” wide QRS, rapid rate, positive concordance precordial leads

ECG Image β€” Wide Complex Tachycardia (VT):

Wide complex tachycardia β€” markedly wide QRS, rapid irregular rhythm

5. 🫁 Pulmonary Embolism (PE)

ECG Changes:

  • Sinus tachycardia β€” most common finding (>50% of cases)
  • Classic S1Q3T3 pattern: deep S in I, Q wave in III, T-wave inversion in III
  • Right bundle branch block (RBBB) β€” complete or incomplete
  • T-wave inversions in V1–V4 (right ventricular strain)
  • Right axis deviation
  • P pulmonale (tall peaked P waves in II if right atrial strain)
  • New atrial fibrillation

ECG Image β€” PE with S1Q3T3 (annotated):

Pulmonary embolism ECG β€” S1Q3T3 pattern, sinus tachycardia, RV strain

ECG Image β€” PE with RV strain (RBBB + precordial T-wave inversions):

PE ECG β€” S1Q3T3, T-wave inversions V1–V3, incomplete RBBB

6. πŸ’‰ Left Ventricular Hypertrophy (LVH)

ECG Changes:

Voltage criteria (increased QRS amplitudes due to increased muscle mass):
  • Sokolow-Lyon: S in V1 + R in V5 or V6 β‰₯35 mm
  • Cornell voltage: R in aVL + S in V3 >28 mm (men) or >20 mm (women)
  • Cornell product: >2440 mmΒ·ms
Associated changes (strain pattern):
  • ST depression + T-wave inversion in lateral leads (I, aVL, V5–V6) β€” "strain pattern"
  • Left axis deviation
  • Left atrial enlargement (broad/notched P waves, negative terminal component in V1)

ECG Image β€” LVH with Cornell voltage criteria:

LVH ECG β€” high voltage in aVL/V3, lateral T-wave inversions (strain pattern)

7. πŸ”₯ Acute Pericarditis

ECG Changes (classically evolve in 4 stages):

  • Stage I: Diffuse, concave (saddle-shaped) ST elevation in most leads (except aVR and V1); PR depression (most specific finding); reciprocal changes in aVR (ST depression + PR elevation)
  • Stage II: ST returns to baseline, T waves flatten
  • Stage III: T-wave inversions develop diffusely
  • Stage IV: Normalization (weeks to months)
  • Spodick's sign: downsloping TP segment (early and specific)
  • Key differentiator from STEMI: diffuse (not territorial) + concave morphology + PR depression

ECG Image β€” Acute Pericarditis (diffuse saddle ST + PR depression):

Pericarditis ECG β€” diffuse concave ST elevation, PR depression leads II/V4–V6, PR elevation aVR

ECG Image β€” Pericarditis with Spodick's sign:

Pericarditis β€” widespread saddle-shape ST, PR depression, downsloping TP (Spodick's)

8. ⚑ Wolff-Parkinson-White Syndrome (WPW)

ECG Changes:

  • Short PR interval (<120 ms) β€” accessory pathway bypasses AV node delay
  • Delta wave β€” slurred upstroke at start of QRS (initial ventricular pre-excitation)
  • Wide QRS (>120 ms) β€” due to fusion of pre-excited and normally conducted depolarization
  • Pseudo-infarct Q waves (negative delta waves in inferior or lateral leads can mimic MI)
  • Secondary ST-T changes discordant to QRS direction
  • Accessory pathway localization based on delta wave polarity in each lead

Risk: WPW + AF β†’ rapid conduction via accessory pathway β†’ ventricular fibrillation

ECG Image β€” WPW (classic delta waves + short PR):

WPW ECG β€” short PR, delta waves, widened QRS in multiple leads

ECG Image β€” WPW (posteroseptal pathway, pseudoinfarct Q waves inferior):

WPW β€” negative delta waves II, III, aVF (pseudoinfarct), left-sided pathway

9. πŸ§‚ Hyperkalemia

ECG Changes (progressive with rising K⁺):

K⁺ levelECG change
5.5–6.5 mmol/LTall, narrow, peaked ("tented") T waves (earliest sign)
6.5–7.5 mmol/LFlattened/absent P waves (atrial standstill), prolonged PR
7.5–8.5 mmol/LWide QRS (intraventricular conduction delay), deepening S waves
>8.5 mmol/LSine wave pattern (merged QRS-T), VF/asystole risk

ECG Image β€” Severe Hyperkalemia (peaked T waves + wide QRS + sine wave):

Hyperkalemia ECG β€” peaked T waves, wide QRS, absent P waves, sine wave pattern (K 9.2)

ECG Image β€” Early-Moderate Hyperkalemia (peaked T waves):

Hyperkalemia β€” tall tented T waves precordial leads, normal PR, no QRS widening yet

10. πŸ«€ Dilated Cardiomyopathy (DCM)

ECG Changes:

  • Left Bundle Branch Block (LBBB): broad notched R waves in I, aVL, V5–V6; deep S waves in V1; QRS >120 ms; secondary ST-T changes discordant to QRS
  • Poor R wave progression (V1–V4) β€” mimics anterior infarction
  • Left axis deviation
  • Left atrial enlargement β€” broad, bifid P waves (P mitrale)
  • Sinus tachycardia (compensatory)
  • Non-specific intraventricular conduction delay (IVCD)
  • Ventricular ectopy / premature ventricular complexes (PVCs)
  • Low voltage (if coexisting pericardial effusion)

ECG Image β€” DCM with LBBB:

Dilated cardiomyopathy ECG β€” complete LBBB, poor R wave progression, lateral ST-T changes

ECG Image β€” DCM (echocardiogram + LBBB ECG):

DCM β€” apical 4-chamber echo showing dilated LV; ECG with poor R progression and LBBB

Quick Reference Summary Table

#DiseaseKey ECG Finding(s)
1Acute MI (STEMI)ST elevation (convex), Q waves, T-wave inversion, reciprocal depression
2Atrial FibrillationAbsent P waves, irregularly irregular QRS, fibrillatory baseline
3Complete AV BlockAV dissociation, slow escape rhythm, no PR relationship
4Ventricular TachycardiaWide QRS >120 ms, rate >100, AV dissociation, concordance
5Pulmonary EmbolismS1Q3T3, sinus tachycardia, RBBB, V1–V4 T-wave inversions
6LV HypertrophyTall R/deep S voltages, lateral strain pattern, LAD
7Acute PericarditisDiffuse concave ST↑, PR depression, Spodick's sign
8WPW SyndromeShort PR, delta wave, wide QRS, pseudo-Q waves
9HyperkalemiaPeaked T waves β†’ absent P β†’ wide QRS β†’ sine wave
10Dilated CardiomyopathyLBBB, poor R progression, PVCs, left atrial enlargement

Clinical Pearl: Always interpret ECG findings in clinical context. Many findings overlap (e.g., LBBB can obscure STEMI β€” use Sgarbossa criteria; WPW can mimic STEMI or AF).
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