Orris
attachment

Interpret this ecg..rbs 544mg/dl

This ECG photo is not perfectly clear, so this is a cautious read, not a definitive diagnosis.
What I can make out:
  • Rhythm appears sinus tachycardia (rate roughly around 120-130/min).
  • QRS appears narrow.
  • There seems to be diffuse ST depression/T-wave abnormalities (especially in lateral/precordial leads), with no obvious classic ST-elevation MI pattern in this image.
  • Machine print on the sheet appears to mention T-wave abnormality / possible ischemic change (not fully legible).
Given RBS 544 mg/dL, this is high risk for diabetic emergency (DKA/HHS) plus electrolyte disturbance, which can itself cause ECG changes and arrhythmia.
What to do now (urgent):
  1. Go to emergency care immediately / ICU-level monitoring.
  2. Immediate labs: serum potassium, bicarbonate, anion gap, venous/arterial blood gas, serum ketones (or beta-hydroxybutyrate), sodium (corrected), creatinine, osmolality, troponin.
  3. Repeat 12-lead ECG after initial stabilization and potassium correction.
  4. Continuous cardiac monitoring.
  5. Treat per DKA/HHS protocol (fluids, insulin, potassium-guided replacement) under physician supervision.
Important:
  • If there is chest pain, breathlessness, sweating, confusion, vomiting, abdominal pain, drowsiness, or low BP, treat as a medical emergency right now.
  • Hyperglycemia this severe with ECG abnormalities should not be managed at home.
If you want, I can give you a quick structured ER order set/checklist for DKA-HHS + cardiac risk in this exact scenario.
attachment

Abg of this patient

ABG interpretation from your slip:
Key values I can read
  • pH: 7.452
  • PaCO2: 27.8 mmHg (low)
  • HCO3-: 19.4 mmol/L (low)
  • Base excess: about -3.4 to -4.6
  • Anion gap: 15 (mildly elevated/borderline)
  • Lactate: 0.9 mmol/L (normal)
  • Glucose: 441 mg/dL (very high)
  • Na+: 132 mmol/L (mild low; corrected Na is higher after glucose correction)
  • K+: 3.9 mmol/L
  • PaO2: 70 mmHg on FiO2 21% (mild hypoxemia)
  • sO2: 95%
Acid-base conclusion
  • Mixed disorder:
    • Primary respiratory alkalosis (low CO2, alkalemic pH)
    • With concurrent mild metabolic acidosis (low HCO3-, negative BE, AG ~15)
  • Not classic severe DKA on this ABG because pH is not acidemic and bicarbonate is only mildly low.
  • Could represent early/mild ketoacidosis or hyperglycemic state with hyperventilation, so ketones/beta-hydroxybutyrate are still essential.
Clinical interpretation with glucose > 400
  • This is severe hyperglycemia with mixed acid-base disturbance.
  • Need urgent evaluation for:
    • DKA/HHS overlap
    • precipitating cause (infection, MI, stroke, missed insulin, etc.)
  • Potassium is currently normal, but can fall rapidly after insulin, so frequent monitoring is mandatory.
Immediate next steps
  1. Serum ketones or beta-hydroxybutyrate urgently.
  2. Repeat venous/arterial gas and BMP every 2-4 hours.
  3. Measured serum osmolality, urea/creatinine, urine ketones.
  4. Start protocolized IV fluids and insulin under monitored care.
  5. Continuous ECG and electrolyte-guided potassium replacement.
So in one line: ABG shows respiratory alkalosis with coexisting mild high-anion-gap metabolic acidosis in the setting of marked hyperglycemia, concerning for early/mixed diabetic metabolic emergency rather than frank advanced DKA.

She is icu..gabe ivf..insulin infusion..can I give one tab ivabrad

Short answer: do not give ivabradine now for this ICU tachycardia unless a cardiologist/intensivist has a specific indication.
Why:
  • In hyperglycemic crisis, tachycardia is usually physiologic (dehydration, stress hormones, pain, infection, acidosis/electrolyte shifts).
  • First-line is to treat cause: fluids, insulin, potassium/electrolytes, infection/pain/fever.
  • Ivabradine is not standard treatment for sinus tachycardia in DKA/HHS and may mask clinical status.
  • It can cause bradycardia, conduction issues, and interacts with some ICU drugs.
What to do instead now:
  1. Continue protocolized IVF + insulin infusion.
  2. Check and correct K/Mg/Phos frequently.
  3. Reassess volume status, urine output, MAP, lactate, temperature, infection markers.
  4. Repeat ECG after stabilization and when HR improves.
  5. If persistent problematic tachycardia after correction, discuss targeted rate control with ICU/cardiology.
If she is unstable (hypotension, chest pain, ischemic ECG change, shock, altered sensorium), manage per ICU emergency pathway immediately.

This patient is a diabetic kidney disease case but urea creatinine are normal...how to give fluid

For this case (severe hyperglycemia, ICU, likely DKA/HHS spectrum), fluid should be careful but not withheld just because she has diabetic kidney disease history.
Practical ICU approach:

1) Initial fluid choice

  • Prefer balanced crystalloid (Ringer lactate or Plasma-Lyte) if available.
  • Alternative: 0.9% saline.

2) Initial rate (if no shock/pulmonary edema)

  • Start around 250-500 mL/hour.
  • If clearly hypovolemic and BP soft, can give a cautious bolus (eg 500 mL) then reassess.
  • Avoid automatic large boluses in DKD-risk patients.

3) Reassess every 1-2 hours

Use bedside endpoints:
  • MAP (target >=65), HR trend
  • Urine output (target >=0.5 mL/kg/hr)
  • Lung exam, SpO2, work of breathing
  • JVP/IVC/bedside ultrasound if available
  • Cumulative fluid balance and body weight
If signs of overload (crepitations, rising O2 need, B-lines, high JVP):
  • Slow/hold fluids
  • Continue insulin
  • Consider diuretic only if volume overloaded and perfusing adequately (ICU decision)

4) Sodium-guided fluid after first phase

  • Calculate corrected Na in hyperglycemia.
  • If corrected Na is low/normal: continue isotonic fluid.
  • If corrected Na high: switch to 0.45% saline (carefully).

5) When glucose reaches ~200-250 mg/dL

  • Add dextrose-containing fluid (eg D5 with appropriate saline) and continue insulin infusion to clear ketosis/prevent rebound.

6) Electrolyte safety (very important)

  • Check K every 2-4 hours.
  • Keep K around 4-5 mEq/L.
  • Replace K early because insulin will drop serum K quickly.

7) “Normal creatinine” does NOT rule out DKD risk

  • DKD can exist with near-normal creatinine (early disease, reduced reserve).
  • So titrate fluid by dynamic response, not by diagnosis label alone.
A simple starting order pattern (adapt to patient):
  • Balanced crystalloid 300 mL/hr
  • Hourly vitals + urine output
  • ABG/electrolytes/glucose every 2-4 hr
  • Reduce to 150-200 mL/hr once perfusion improves
  • Add dextrose fluid when glucose ~250 and continue insulin protocol
If you share weight, BP/MAP, urine output, crackles/O2 requirement, I can give you a tighter hour-by-hour fluid plan.
attachment

Cxr of this dka pt

From this photo of the film (limited quality), my bedside read is:
  • Cardiomediastinal silhouette: not enlarged.
  • Lungs: no obvious focal lobar consolidation.
  • No clear pleural effusion.
  • No visible pneumothorax.
  • Mild bibasal linear/interstitial crowding could be positional/low-volume film rather than frank edema.
So this CXR does not show obvious pulmonary edema or major pneumonia on this image.
How this helps your DKA management:
  • You can continue cautious IV fluids with close reassessment.
  • Keep monitoring for evolving overload (new crackles, rising O2 need, B-lines on lung US), since CXR can lag behind clinical fluid changes.
  • If fever/leukocytosis or sepsis concern persists, repeat imaging/ultrasound and cultures despite this near-clear film.
If you want, I can give a practical ICU fluid titration table (next 6–12 hours) based on HR, MAP, urine output, SpO2, and lung findings.
This is a shared conversation. Sign in to Orris to start your own chat.