Here is everything you need to know about varicose veins for clinical presentation and viva, compiled from S Das and Pye's Surgical Handicraft:
VARICOSE VEINS - Complete Viva & Clinical Guide
1. DEFINITION
A vein is called varicose when it is dilated, tortuous, and elongated. It is unique to humans - a consequence of the erect posture. Veins of the lower limb are most commonly affected; others include spermatic veins (varicocele), oesophageal veins (oesophageal varices), and haemorrhoidal veins (piles).
2. APPLIED ANATOMY (High-yield for viva)
Long (Great) Saphenous Vein (LSV)
- Longest vein in the body
- Begins at the medial end of the dorsal venous arch of the foot
- Runs along the medial side of the leg, passes behind the medial condyle of the tibia and femur
- Passes through the saphenous opening (fossa ovalis) in the deep fascia to join the femoral vein at the saphenofemoral junction (SFJ), 3.5 cm below and lateral to the pubic tubercle
- Tributaries at SFJ: superficial circumflex iliac, superficial epigastric, superficial external pudendal, and deep external pudendal veins
Short (Small) Saphenous Vein (SSV)
- Begins behind the lateral malleolus
- Runs up the midline of the calf
- Joins the popliteal vein at the saphenopopliteal junction (SPJ) in the popliteal fossa
Perforating (Communicating) Veins
- Connect the superficial to the deep system
- Valves normally allow flow only from superficial to deep
- Important perforators: Dodd's (lower thigh), Boyd's (below knee), Cockett's (medial calf - most important clinically)
3. AETIOLOGY & CLASSIFICATION
Primary Varicose Veins
- Exact cause unknown
- Valve incompetence - either of the main trunk or communicating veins
- Weak venous walls allowing dilatation, which then causes secondary valve incompetence
- Very rarely: congenital absence of valves
Secondary Varicose Veins
| Mechanism | Causes |
|---|
| Obstruction to venous outflow | Pregnancy, fibroid, ovarian cyst, pelvic cancer (cervix/uterus/ovary/rectum), abdominal lymphadenopathy, ascites, iliac vein thrombosis, retroperitoneal fibrosis |
| Destruction of valves | Deep vein thrombosis (post-thrombotic syndrome) |
| High-pressure flow | Arteriovenous fistula |
Risk Factors
- Sex: Women > Men (ratio 10:1); hormonal factor (progesterone?)
- Age: Middle-aged most commonly
- Occupation: Prolonged standing - tram drivers, policemen
- Pregnancy: Hormonal + mechanical compression
- Obesity
- Family history: Strong hereditary component
- Previous DVT
Viva tip: Varicose veins are NOT found in other animals - it is the "penalty of erect posture" (S Das)
4. SYMPTOMS
The most common symptom is aching pain in the leg:
- Worse towards end of the day
- Worse on prolonged standing
- Relieved by lying down / elevation
Other symptoms:
- Bursting pain while walking - suggests associated DVT
- Night cramps
- Ankle swelling (worse at end of day)
- Itching of the skin
- Cosmetic concern (the veins are unsightly)
- Varicose ulcer (medial malleolus area)
Viva tip: S Das emphasises that "it is not the varicose veins which produce the symptoms, but it is the disordered psychology which is the root of all evils." Asymptomatic varicose veins on one side and severe symptoms with minimal veins on the other side is possible.
5. CLINICAL EXAMINATION
A. INSPECTION (Patient standing)
- Distribution of varicosities - long saphenous (medial) vs short saphenous (posterior/lateral) system
- Skin changes:
- Pigmentation (haemosiderin deposition - brownish discolouration)
- Varicose eczema
- Lipodermatosclerosis (thickened, indurated, fibrosed skin - "inverted champagne bottle" leg)
- Atrophie blanche (white scarring)
- Varicose ulcer - medial malleolus / gaiter area
- Oedema of the ankle
- Saphenous varix - bluish swelling below inguinal ligament (may mimic femoral hernia)
B. PALPATION
- Temperature - may be raised over varicosities
- Tenderness along varicosed veins
- Fascial defects - palpable gaps/pits in deep fascia at sites of incompetent perforators (best felt with patient lying down and leg elevated)
- Cough impulse at SFJ - if positive, suggests saphenofemoral incompetence (Morrissey's test)
- Saphena varix - disappears on lying down; fluid thrill transmissible from below
C. PERCUSSION
Schwartz Test - Tap the varicose vein below; feel a percussion wave transmitted upward at the saphenous opening. Confirms continuity of the dilated column of blood (absent valves between the two fingers).
D. AUSCULTATION
- Limited value except in arteriovenous fistula - continuous machinery murmur heard
E. Regional lymph nodes
- Inguinal nodes only enlarged if there is an infected varicose ulcer
F. Examine the other limb
- Always examine both limbs for bilateral involvement
6. SPECIAL CLINICAL TESTS (Most Viva-important)
1. Trendelenburg Test (Brodie-Trendelenburg)
Purpose: To identify the site of valvular incompetence (SFJ vs perforators)
Method:
- Patient lies supine; elevate leg to empty veins
- Apply tourniquet just below SFJ at groin
- Patient stands up
Results:
| Finding | Meaning |
|---|
| Veins remain empty with tourniquet; fill rapidly when released | Trendelenburg I positive - SFJ incompetence (Long saphenous incompetence) |
| Veins fill from below even with tourniquet in place | Trendelenburg II positive - Incompetent perforators below tourniquet level |
| Both findings | Double positive - incompetence at both sites |
2. Multiple Tourniquet Test
- Three tourniquets at upper thigh, just above knee, upper calf
- Localises the level of perforator incompetence by observing which segment fills
3. Perthes' Test
Purpose: Assess patency and function of deep veins - MUST be done before surgery
Method (Modified): Tourniquet at upper thigh (tight enough to block superficial reflux) → ask patient to walk quickly
Results:
- Deep veins patent → varicose veins shrink (calf muscle pump works)
- Deep veins blocked → varicose veins become more distended + crampy pain
Critical viva point: If Perthes' test is positive (deep veins obstructed), surgical stripping of superficial veins is contraindicated - the patient would lose their only venous drainage.
Original Perthes' test: Wrap leg in elastic bandage; walk → severe crampy pain = deep vein thrombosis
4. Schwartz Test (Percussion Test)
Purpose: Confirm continuous column of blood in long-standing varicosity
Method: One hand taps/percusses the vein in the lower leg; other hand placed at saphenous opening
Positive: A transmitted impulse felt at the groin = absent or incompetent valves between the two hands
5. Morrissey's Cough Impulse Test
Purpose: Detect saphenofemoral incompetence / saphenous varix
Method: Elevate leg → empty veins → patient stands → cough forcibly
Positive: Expansile impulse felt at saphenous opening = incompetent saphenofemoral valve
6. Pratt's Test
Purpose: Locate positions of incompetent perforators in the leg
Method:
- Apply Esmarch bandage from toes to groin (empty veins)
- Apply tourniquet at groin
- Remove Esmarch bandage from below
- Re-apply Esmarch bandage from groin downwards
- "Blow outs" appear at the sites of incompetent perforators as the bandage passes them
7. Fegan's Method
Purpose: Locate perforators
Method: Mark excessive bulges on standing → lie patient down, elevate leg → palpate along marked lines for fascial defects (pits/gaps) in deep fascia - these are the perforator sites
8. Short Saphenous Incompetence Test
- Mark the saphenopopliteal junction with a pen (patient standing)
- Elevate leg to empty SSV; apply thumb pressure to the mark
- Patient stands; release pressure
- Immediate filling of SSV = SSV incompetence
7. GENERAL EXAMINATION
Abdominal Examination
Most important general examination in varicose veins - to exclude secondary causes:
- Pregnant uterus
- Intrapelvic tumour: fibroid, ovarian cyst, cancer of cervix/rectum
- Abdominal lymphadenopathy
8. COMPLICATIONS
| Complication | Key Points |
|---|
| Haemorrhage | Profuse bleeding from minor trauma due to high venous pressure; treated by leg elevation |
| Superficial thrombophlebitis | Vein becomes tender, firm, red, warm; pyrexia; thrombus firmly attached so low PE risk |
| Venous ulcer | More due to DVT than varicose veins alone; medial malleolus ("gaiter area"); shallow, sloping edges, pink/fibrous floor, seropurulent discharge |
| Pigmentation | Haemosiderin deposition from RBC extravasation |
| Varicose eczema | Itching, weeping dermatitis |
| Lipodermatosclerosis | Skin thickened, fibrosed, pigmented - due to fibrin deposition from high venous pressure |
| Calcification of vein | Phlebolithiasis |
| Periostitis | In long-standing ulcer over tibia |
| Equinus deformity | From long-standing ulcer - patient walks on toes to relieve pain → Achilles tendon shortens |
| Marjolin's ulcer | Malignant change (squamous cell carcinoma) in edge of chronic venous ulcer - raised, everted edge, enlarged inguinal nodes |
9. VARICOSE ULCER - Detailed Features (Viva favourite)
- Site: Lower third of leg, medial side (gaiter area), around medial malleolus; never above junction of middle and lower thirds
- Shape: Irregular, ragged edges
- Edge: Sloping, pale purple-blue in colour
- Floor: Pink granulation tissue (in acute); white fibrous tissue (in chronic)
- Discharge: Seropurulent with trace of blood
- Surroundings: Induration, tenderness, pigmentation, lipodermatosclerosis
- Healing sign: Faint blue rim of advancing epithelium at margin
- Compared to arterial ulcer: Venous ulcers are painless/less painful at rest; arterial ulcers are extremely painful
10. INVESTIGATIONS
| Investigation | Purpose |
|---|
| Duplex Doppler ultrasound | Gold standard; B-mode + Doppler - shows anatomy and confirms/locates incompetence; identifies DVT |
| Doppler ultrasound (handheld) | Probe over femoral vein at groin; squeeze calf → should produce roar; absent roar = DVT between calf and groin |
| Ascending phlebography (venography) | Contrast injected into dorsal foot vein; shows deep veins; identifies DVT (filling defect / non-filling) |
| 125I-labelled fibrinogen scan | Detects early/asymptomatic DVT; >20% increase in radioactivity at any point = thrombus |
| Ultrasound / CT abdomen | For suspected secondary varicose veins (abdominal tumours) |
11. TREATMENT
A. Conservative
- Compression stockings/elastic support bandages - for elderly, unfit, mild cases, pregnancy
- Lifestyle advice: avoid prolonged standing, elevate legs, weight loss
- Exercise - calf muscle pump activity
B. Injection Sclerotherapy (Fegan's technique)
- Sclerosant: Ethanolamine oleate, STD (sodium tetradecyl sulphate), hypertonic saline
- Principle: Inject sclerosant into empty vein → cause endothelial damage → thrombosis → fibrosis of vein
- Method (Fegan's): Perforator sites identified → patient standing (veins full) → injected → immediately compressed with foam pad + crêpe bandage for 6 weeks
- Indication: Small/medium varicosities, incompetent perforators, recurrent varices after surgery
- Complication: Skin necrosis (if extravasation), allergic reaction, phlebitis, DVT
- Disadvantage: Does not remove the source of incompetence; higher recurrence than surgery
C. Surgical Treatment (Trendelenburg's Operation + Stripping)
Indications for surgery:
- Saphenofemoral junction incompetence
- Recurrence after sclerotherapy
- Failure of conservative treatment
- Complications (ulcer, bleeding)
- Patient preference
Prerequisites: Perthes' test must be negative (deep veins must be patent)
Steps of operation (Long saphenous system):
- Saphenofemoral disconnection (Trendelenburg's operation): Groin incision → SFJ identified → LSV ligated flush with femoral vein → all tributaries at SFJ ligated and divided (to prevent recurrence)
- Stripping of LSV: LSV stripped from groin to just below knee (not to ankle - lower LSV rarely diseased; saphenous nerve runs with it below knee and stripping risks saphenous nerve injury)
- Stab avulsion (multiple phlebectomies): Small (2-3 mm) stab incisions over marked varicosities; veins avulsed with a hook
For short saphenous system:
- Saphenopopliteal disconnection in popliteal fossa
- SSV stripped/avulsed
D. Endovenous Treatments (Modern)
- Endovenous Laser Ablation (EVLA): Laser fibre introduced into vein; thermal damage causes closure
- Radiofrequency Ablation (RFA): Similar principle with radiofrequency energy
- Foam sclerotherapy: Sclerosant mixed with air to create foam - greater surface contact
- Now preferred over open surgery in many centres
12. CAUSES OF VARICOSE ULCER NON-HEALING
- Arterial disease (check ABPI - ankle-brachial pressure index)
- Osteomyelitis of underlying tibia
- Malignant change (Marjolin's ulcer)
- Infection
- Poor compliance with compression
13. KEY VIVA QUESTIONS & ANSWERS
Q: Why are varicose veins not seen in other animals?
A: Unique to humans due to erect posture; hydrostatic column of blood from heart to feet imposes maximum pressure (~100 cmH2O) on the venous valves.
Q: Why is Perthes' test done before surgery?
A: If deep veins are blocked (post-thrombotic), the superficial varicose veins serve as the only collateral drainage for the limb. Stripping them would cause severe venous hypertension, worsening oedema, and potential venous gangrene.
Q: What is the most important general examination in varicose veins?
A: Abdominal examination - to exclude secondary causes (pregnancy, pelvic tumour, lymphadenopathy).
Q: What is a saphenous varix and how does it differ from a femoral hernia?
A: Saphenous varix is a blow-out at the SFJ due to severe incompetence. Like femoral hernia it appears at the femoral triangle. Differences: saphenous varix disappears on lying down, has a fluid thrill on tapping lower varices, no cough impulse transmitted into it (whereas femoral hernia has a cough impulse and does not disappear on lying flat).
Q: What is Marjolin's ulcer?
A: Squamous cell carcinoma arising in the edge of a chronic venous ulcer. Suspect when: long-standing ulcer changes character, edge becomes raised/everted/irregular, inguinal lymph nodes enlarge. Requires biopsy.
Q: What is lipodermatosclerosis?
A: Skin and subcutaneous tissue becomes thickened, indurated, fibrosed, and pigmented due to chronic venous hypertension. Mechanism: high venous pressure causes fibrin deposition around capillaries and activates white cells, leading to tissue ischaemia and fibrosis.
Q: What are the tributaries of the long saphenous vein at the SFJ?
A: Superficial circumflex iliac, superficial epigastric, superficial external pudendal, and deep external pudendal veins. All must be ligated during Trendelenburg's operation to prevent recurrence.
Q: Why is the LSV stripped only to the knee and not to the ankle?
A: Below the knee, the saphenous nerve runs alongside the LSV. Stripping below the knee carries high risk of saphenous nerve injury causing medial leg numbness.
Q: What is the commonest complication of varicose veins?
A: Haemorrhage (from minor trauma to a dilated high-pressure vein). Easily controlled by leg elevation.
Sources: S Das - A Manual on Clinical Surgery 13th Edition; Pye's Surgical Handicraft 22nd Edition