I now have excellent images for all 10 ECG patterns. Let me compile the comprehensive educational response.
10 Common ECG Patterns in Heart Disease
1. ST-Elevation Myocardial Infarction (STEMI)
Key ECG Features:
- ST-segment elevation ≥ 1 mm in ≥2 contiguous leads (≥2 mm in V1–V3)
- Hyperacute (tall, peaked) T-waves in early phase
- Pathological Q waves develop within hours (necrosis)
- Reciprocal ST depression in mirror leads
- "Tombstone" or convex-upward (coved) ST morphology in severe LAD occlusion
Localization by leads:
| Territory | Elevated leads | Artery |
|---|
| Anterior | V1–V4 | LAD |
| Inferior | II, III, aVF | RCA |
| Lateral | I, aVL, V5–V6 | LCx |
2. Atrial Fibrillation (AF)
Key ECG Features:
- Absent P waves — replaced by irregular fibrillatory (f) waves, best seen in V1
- Irregularly irregular R-R intervals — the hallmark
- Narrow QRS unless aberrant conduction present
- Ventricular rate varies (60–170 bpm in untreated AF)
Clinical context: Most common sustained arrhythmia. Causes include hypertension, valvular disease, thyrotoxicosis, heart failure, alcohol.
3. Ventricular Fibrillation (VF)
Key ECG Features:
- Chaotic, disorganized waveforms — no identifiable P, QRS, or T waves
- Completely irregular amplitude and frequency
- Coarse VF: higher amplitude fibrillatory waves (more recent onset, better chance of defibrillation success)
- Fine VF: low amplitude, harder to distinguish from asystole
Emergency: Immediately lethal without defibrillation. Shockable rhythm in ACLS.
4. Ventricular Tachycardia (VT)
Key ECG Features:
- Wide QRS complexes (>120 ms), typically >140–160 ms
- Rapid regular rate — typically 100–250 bpm
- No preceding P waves (or AV dissociation when visible)
- Capture beats and fusion beats are pathognomonic
- Positive or negative concordance in precordial leads
Types:
- Monomorphic VT: uniform QRS morphology (scar-related in structural heart disease)
- Polymorphic VT: varying QRS morphology (ischemia, long QT)
5. Complete (Third-Degree) AV Block
Key ECG Features:
- Complete AV dissociation — P waves and QRS complexes fire independently
- P rate > QRS rate (e.g., atrial rate 70–80 bpm; ventricular escape 30–50 bpm)
- No fixed PR interval — P waves "march through" QRS complexes
- Wide escape QRS if infra-nodal origin; narrow if junctional escape
Clinical significance: Causes syncope, hemodynamic compromise; requires pacemaker.
6. Left Bundle Branch Block (LBBB)
Key ECG Features:
- Wide QRS ≥ 120 ms
- Broad, notched "M-shaped" R-wave in lateral leads (I, aVL, V5, V6)
- Deep broad S-wave (QS pattern) in V1–V3
- Absent septal Q waves in lateral leads
- Discordant ST-T changes: T waves opposite to QRS vector
Significance: New LBBB in chest pain may indicate acute MI (Sgarbossa criteria apply). Associated with cardiomyopathy, hypertension, aortic stenosis.
7. Right Bundle Branch Block (RBBB)
Key ECG Features:
- Wide QRS ≥ 120 ms
- rSR' ("rabbit ears") pattern in V1–V3 — classic sign
- Broad, slurred S-waves in lateral leads (I, aVL, V5, V6)
- T-wave inversion in V1–V3 (secondary repolarization change)
Significance: Isolated RBBB may be normal variant; new RBBB with anterior MI indicates proximal LAD occlusion (Brugada-type); also seen in pulmonary embolism, congenital heart disease.
8. Wolff-Parkinson-White (WPW) Syndrome
Key ECG Features:
- Short PR interval (< 120 ms) — accessory pathway bypasses AV node
- Delta wave — slurred, slow upstroke at start of QRS
- Widened QRS (> 110 ms) due to pre-excitation
- Secondary ST-T changes discordant to delta wave
- Pseudo-infarct Q waves in inferior leads with posteroseptal pathway
Risk: Pre-excited AF can degenerate to VF → sudden cardiac death. Definitive treatment is catheter ablation.
9. Long QT Syndrome (LQTS)
Key ECG Features:
- Prolonged QTc (corrected for heart rate):
- Normal: ≤ 440 ms (men), ≤ 460 ms (women)
- Borderline: 440–470 ms
- Definite: > 500 ms = high risk for Torsades de Pointes
- Broad, notched T-waves or T-wave alternans (LQT2)
- Deep, notched T-U waves (LQT3)
- Can precipitate polymorphic VT (Torsades de Pointes)
Causes: Congenital (KCNQ1, KCNH2, SCN5A mutations), drugs (antiarrhythmics, antibiotics, antipsychotics), electrolyte disturbances (hypokalemia, hypomagnesemia).
10. Left Ventricular Hypertrophy (LVH) with Strain
Key ECG Features:
- High voltage QRS — Sokolow-Lyon criteria: S in V1 + R in V5 or V6 ≥ 35 mm
- Left ventricular strain pattern: ST depression + T-wave inversion in lateral leads (I, aVL, V4–V6)
- Left axis deviation
- Prolonged QRS (but usually < 120 ms)
- May show P-mitrale (bifid P in II, notched) if associated left atrial enlargement
Causes: Long-standing hypertension, aortic stenosis, hypertrophic cardiomyopathy.
Quick Reference Summary
| # | Pattern | Rate | Rhythm | P wave | QRS | Key Feature |
|---|
| 1 | STEMI | Normal | Regular | Normal | Normal | ST elevation + Q waves |
| 2 | Atrial Fibrillation | Variable | Irregular | Absent | Narrow | f-waves, irregular RR |
| 3 | Ventricular Fibrillation | — | Chaotic | Absent | None | Wavy baseline |
| 4 | Ventricular Tachycardia | 100–250 | Regular | Dissociated | Wide | Capture/fusion beats |
| 5 | 3rd Degree AV Block | Slow | Regular | Regular, dissociated | Wide escape | Complete AV dissociation |
| 6 | LBBB | Normal | Regular | Normal | ≥120 ms | M-wave lateral, QS V1–V3 |
| 7 | RBBB | Normal | Regular | Normal | ≥120 ms | rSR' V1, S-wave lateral |
| 8 | WPW | Normal | Regular | Normal | Widened | Short PR + delta wave |
| 9 | Long QT | Normal | Regular | Normal | Normal | QTc > 500 ms |
| 10 | LVH/Strain | Normal | Regular | Normal | High voltage | ST depression lateral leads |
Images sourced from peer-reviewed clinical ECG datasets (PMC Clinical VQA). All ECGs displayed at standard 25 mm/s paper speed and 10 mm/mV calibration unless noted.