Based on all retrieved sources, here is the curated list of diagrams to draw for a university theory exam on Aggressive Periodontitis:
Diagrams to Draw - Aggressive Periodontitis (University Theory Exam)
1. Classification Diagram (Flow Chart / Table)
Draw: A branching diagram showing the 1999 AAP classification
Aggressive Periodontitis
├── Localized Aggressive Periodontitis (LAP)
└── Generalized Aggressive Periodontitis (GAP)
Include former names (Localized Juvenile Periodontitis, Generalized Early-Onset Periodontitis, Rapidly Progressive Periodontitis).
Source: 1999 AAP International Workshop for Classification of Periodontal Diseases; Sherris & Ryan's Medical Microbiology, 8th ed.
2. Primary & Secondary Features (Diagnostic Criteria Table)
Draw: A two-column table or labeled box
| Primary Features | Secondary Features |
|---|
| Non-contributory medical history | Scanty microbial deposits inconsistent with severity |
| Rapid attachment loss & bone destruction | Elevated A. actinomycetemcomitans / P. gingivalis |
| Familial aggregation | Phagocyte abnormalities (PMN chemotaxis defect) |
| Hyper-responsive macrophage phenotype (elevated PGE2, IL-1β) |
Source: Lang et al. (1999); Sherris & Ryan's Medical Microbiology, 8th ed., p. 1509
3. Pattern of Bone Loss - LAP vs GAP (Radiographic Diagram)
Draw: Schematic dental arch (upper and lower) showing:
- LAP: Arc-shaped / "mirror image" vertical bone loss around first molars and incisors only ("arc cratering")
- GAP: Generalized horizontal and vertical bone loss affecting most teeth
Label: molar-incisor pattern, attachment loss, alveolar bone crest.
Source: Carranza's Clinical Periodontology; Dimensions of Dental Hygiene review
4. Pathogenesis Flow Diagram
Draw: A cascade/arrow diagram:
Genetic predisposition (familial aggregation)
↓
PMN chemotaxis & phagocytic defect
↓
Colonization by A. actinomycetemcomitans
(leukotoxin, collagenase, LPS, cytolethal distending toxin)
↓
Hyper-responsive macrophage phenotype
(↑IL-1β, ↑PGE2, ↑TNF-α)
↓
Collagen destruction + Alveolar bone resorption
↓
Rapid attachment loss & deep periodontal pockets
Source: Sherris & Ryan's Medical Microbiology, 8th ed., p. 1509-1510; Etiology & Pathogenesis of Aggressive Periodontitis (Mani et al., GIJHSR 2018)
5. Microbiology Diagram - Key Pathogens
Draw: A labeled diagram of the subgingival plaque with organisms highlighted:
- Primary: Aggregatibacter actinomycetemcomitans (Aa) - present in 97% of cases
- Secondary: Porphyromonas gingivalis, Prevotella intermedia, Capnocytophaga spp., Eikenella corrodens
Note virulence factors of Aa: leukotoxin, collagenase, endotoxin (LPS), cytolethal distending toxin (CDT), epitheliotoxin.
Source: Sherris & Ryan's Medical Microbiology, 8th ed., p. 1509-1510; Mani et al. 2018
6. PMN Defect Diagram (Immunopathogenesis)
Draw: Comparison schematic:
- Normal PMN: Chemotaxis toward bacteria, phagocytosis, killing
- AgP PMN: Impaired chemotaxis, reduced phagocytic function → bacteria evade → uncontrolled tissue destruction
Source: Mani et al., GIJHSR 2018; AAP Consensus Report 1999
7. Clinical Features Diagram - Intraoral Drawing (LAP)
Draw: Labeled intraoral diagram showing:
- Deep periodontal pockets at first molars & incisors
- Minimal plaque/calculus (clinically healthy-looking gingiva)
- Distolabial migration of maxillary incisors with diastema formation
- Increasing mobility of affected teeth
- "Arc-cratered" vertical bone loss on radiograph
Source: Dimensions of Dental Hygiene; PMC3265097 (Generalized AP case reports)
8. Disease Activity Pattern - GAP (Graph)
Draw: A time vs. attachment loss graph showing episodic "burst" pattern:
- Periods of rapid active destruction (bursts)
- Alternating periods of quiescence (no clinical signs despite deep pockets)
Label: active phase, quiescent phase, cumulative attachment loss.
Source: PMC3265097 - Generalized Aggressive Periodontitis and Its Treatment Options
9. Comparison Table: LAP vs. GAP vs. Chronic Periodontitis
Draw: A 3-column comparison table covering:
- Age of onset
- Teeth affected
- Bone loss pattern
- Plaque/calculus
- Microbiology
- PMN function
- Familial tendency
Source: Sherris & Ryan's Medical Microbiology, 8th ed.; Carranza's Clinical Periodontology
10. Treatment Algorithm (Flow Chart)
Draw: Management steps:
Diagnosis (clinical + radiographic)
↓
Phase I: Cause-related therapy
- Scaling & root planing (SRP)
- Systemic antibiotics (Amoxicillin + Metronidazole)
↓
Re-evaluation (6-8 weeks)
↓
Phase II: Surgical therapy (if pockets remain)
- Osseous surgery / Bone grafts / GTR
↓
Phase III: Supportive periodontal therapy (SPT)
- Family screening
Source: PMC3265097; Carranza's Clinical Periodontology
Priority diagrams for exam (if time limited): #3 (bone loss pattern), #4 (pathogenesis cascade), #2 (primary/secondary features table), and #7 (intraoral clinical features) are the most commonly requested in university theory papers.