Clinical anatomy. physiology of the pharynx. Pharyngeal spaces. Acute diseases of the pharynx (primary and secondary angina). Peritonsillar abscess. in detailed medical answer based on ent

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Clinical Anatomy & Physiology of the Pharynx — Acute Pharyngeal Disease — Peritonsillar Abscess


I. CLINICAL ANATOMY OF THE PHARYNX

Definition and Overview

The pharynx is a musculofascial half-cylinder that links the oral and nasal cavities in the head to the larynx and esophagus in the neck. Its cavity serves as a common pathway for air and food. It is attached superiorly to the base of the skull and is continuous inferiorly at approximately the level of vertebra C6 with the top of the esophagus.
Pharynx diagram
Fig. Pharynx — showing relationships of nasal and oral cavities, pharynx, larynx, and trachea. (Gray's Anatomy for Students)

Subdivisions of the Pharynx

The pharynx is divided into three regions based on anterior relationships:
RegionAnterior OpeningLevel
NasopharynxPosterior choanae of nasal cavitiesSkull base → soft palate
OropharynxOropharyngeal isthmus (posterior oral cavity opening)Soft palate → hyoid
Laryngopharynx (Hypopharynx)Laryngeal inlet (superior aperture of larynx)Hyoid → C6/esophagus
Additional relationships:
  • The pharyngotympanic (Eustachian) tubes open into the lateral walls of the nasopharynx
  • The pharynx is separated from the vertebral column by the thin retropharyngeal space containing loose connective tissue
  • The soft palate acts as a "flutter valve": elevates to seal the nasopharynx from oropharynx; depresses to seal the oral cavity from the oropharynx
Gray's Anatomy for Students, p. 1180

Skeletal Framework

The superior and anterior margins of the pharyngeal wall attach to bone and cartilage. The two walls are united posteriorly in the midline by the pharyngeal raphe — a cord-like ligament descending from the pharyngeal tubercle on the base of the skull to C6, where it blends with the esophageal wall.
The line of skull attachment is C-shaped, beginning at the posterior margin of the medial pterygoid plate, crossing inferior to the pharyngotympanic tube, running onto the petrous temporal bone, and meeting at the midline pharyngeal tubercle.

Anterior Vertical Line of Attachment (Three Parts):

  1. First part: Medial pterygoid plate → pterygoid hamulus → pterygomandibular raphe → mandible
  2. Second part: Stylohyoid ligament → lesser horn of hyoid → body of hyoid → greater horn of hyoid
  3. Third part: Posterior aspect of thyroid cartilage and cricoid cartilage
Gray's Anatomy for Students, p. 1181

Muscles of the Pharynx

Constrictors (outer circular layer):
  • Superior constrictor — attaches to pterygomandibular raphe; overlaps the middle constrictor; most relevant clinically — the peritonsillar space lies between the tonsil capsule and this muscle
  • Middle constrictor — from hyoid bone; fans out posteriorly
  • Inferior constrictor — from thyroid and cricoid cartilages; its lowest fibers (cricopharyngeus) form the upper esophageal sphincter
Elevators (inner longitudinal layer):
  • Stylopharyngeus — CN IX (glossopharyngeal)
  • Salpingopharyngeus
  • Palatopharyngeus
The constrictor muscles overlap like stacked cups — superior overlapped by middle, middle overlapped by inferior.

Nasopharynx

The roof and posterior wall form a continuous surface attached to the base of the skull and the anterior arch of C1–C2. Key structures:
  • Pharyngeal tonsil (adenoids) — lymphoid tissue on the roof/posterior wall
  • Torus tubarius — cartilaginous elevation marking the Eustachian tube opening
  • Fossa of Rosenmüller — lateral recess posterior to the torus; most common site of nasopharyngeal carcinoma
  • Salpingopharyngeal fold — runs inferiorly from torus
  • Pharyngeal isthmus — junction with oropharynx, closed by soft palate elevation

Oropharynx

Extends from the soft palate to the upper border of the epiglottis. Contains:
  • Palatine tonsils — large ovoid lymphoid masses on each lateral wall, between the palatoglossal arch (anterior pillar) and the palatopharyngeal arch (posterior pillar)
  • Posterior one-third of the tongue (base of tongue / lingual tonsils)
  • Valleculae — recesses between the base of tongue and epiglottis

Laryngopharynx (Hypopharynx)

Extends from the epiglottis to C6/cricoid cartilage. Contains:
  • Piriform fossae — paired lateral recesses that channel food around the laryngeal inlet into the esophagus; common site of foreign body impaction and carcinoma
  • Postcricoid region — posterior to the larynx; connects to the esophagus
  • Cricopharyngeus muscle — the "upper esophageal sphincter" at C6

Waldeyer's Tonsillar Ring

A ring of lymphoid tissue surrounding the pharyngeal inlet:
  • Pharyngeal tonsil (adenoid) — roof of nasopharynx (midline)
  • Tubal tonsils — near Eustachian tube opening
  • Palatine tonsils — lateral oropharynx (the clinically most prominent)
  • Lingual tonsils — posterior tongue
Gray's Anatomy for Students, p. 1188

Blood Supply

Arterial:
  • Upper pharynx: ascending pharyngeal artery, ascending palatine and tonsillar branches of the facial artery, branches of maxillary and lingual arteries (all from external carotid)
  • Lower pharynx: pharyngeal branches of inferior thyroid artery (from thyrocervical trunk)
Venous:
  • Pharyngeal venous plexus → internal jugular vein

Nerve Supply

  • Motor (all constrictors + most elevators): pharyngeal branch of vagus (CN X) via pharyngeal plexus
  • Sensory: CN IX (glossopharyngeal) for oropharynx; CN X for laryngopharynx; CN V2 for nasopharynx
  • Stylopharyngeus: CN IX alone

Lymphatic Drainage

  • Nasopharynx and posterior pharynx → retropharyngeal nodes → deep cervical chain
  • Tonsils and oropharynx → jugulodigastric (tonsillar) node — the sentinel node for tonsillar infection/malignancy

II. PHYSIOLOGY OF THE PHARYNX

Functions

  1. Respiratory conduit — transmits air between nasal/oral cavities and larynx
  2. Alimentary conduit — directs food/liquid from oral cavity to esophagus
  3. Phonation — acts as a resonating chamber
  4. Eustachian tube equalization — nasopharynx communicates pressure to middle ear
  5. Immune defense — Waldeyer's ring provides first-line mucosal immune surveillance

Swallowing (Deglutition)

Swallowing occurs in three phases:
1. Oral phase (voluntary): food bolus formed, tongue propels it posteriorly into oropharynx
2. Pharyngeal phase (involuntary, CN IX/X/XII coordinated):
  • Soft palate elevates → closes pharyngeal isthmus → prevents nasal regurgitation
  • Hyoid bone and larynx elevate anterosuperiorly
  • Laryngeal inlet closes (epiglottis folds, arytenoids approximate, true cords adduct) → airway protected
  • Pharyngeal constrictors contract in peristaltic wave superiorly → inferiorly
  • Upper esophageal sphincter (cricopharyngeus) relaxes
A person cannot breathe and swallow simultaneously because the airway is closed at two sites: the pharyngeal isthmus and the larynx.Gray's Anatomy for Students, p. 1188
3. Esophageal phase: bolus enters esophagus; peristalsis continues to stomach

III. PHARYNGEAL SPACES

The pharynx is surrounded by fascial spaces that are clinically critical because infection can spread rapidly through them. Understanding their boundaries guides surgical drainage.

Fascial Layers of the Neck

Superficial layer (investing fascia): envelopes SCM, trapezius, submandibular gland, parotid
Deep cervical fascia:
  • Middle layer (visceral/pretracheal): envelopes pharynx, larynx, trachea, esophagus, thyroid
  • Deep layer: two sublayers:
    • Alar layer: base of skull → mediastinum; covers sympathetic trunk
    • Prevertebral layer: base of skull → coccyx; covers vertebral bodies, paraspinous muscles
    • ⚠️ Danger space = space between alar and prevertebral layers → extends from base of skull to mediastinum → rapid infection spread

Key Pharyngeal Spaces

1. Peritonsillar Space

  • Medial: palatine tonsil (capsule)
  • Lateral: superior constrictor muscle
  • Contents: loose areolar tissue, tonsillar branches of lingual/facial/ascending pharyngeal vessels
  • Clinical significance: site of peritonsillar abscess — the most common deep space infection of the head and neck

2. Parapharyngeal Space (Lateral Pharyngeal Space)

  • Superior: base of skull (middle cranial fossa)
  • Inferior: hyoid bone
  • Anterior: pterygomandibular raphe
  • Posterior: prevertebral fascia
  • Medial: pharyngobasilar fascia / superior constrictor
  • Lateral: deep lobe of parotid, mandible, medial pterygoid
  • Divided by styloid process into:
    • Prestyloid (muscular) compartment: fat, lymph nodes, internal maxillary artery, inferior alveolar/lingual/auriculotemporal nerves, pterygoid muscles, deep parotid lobe
    • Poststyloid (neurovascular) compartment: internal carotid artery, internal jugular vein, CN IX, X, XI, XII, cervical sympathetic chain
  • Clinical significance: Peritonsillar abscess can rupture through superior constrictor into this space; spread here causes Horner syndrome (sympathetic chain), cranial nerve palsies

3. Retropharyngeal Space (Retrovisceral Space)

  • Superior: base of skull
  • Inferior: posterior mediastinum (T1–T2)
  • Anterior: posterior pharyngeal wall (constrictor muscles)
  • Posterior: alar layer of deep cervical fascia
  • Contents: retropharyngeal lymph nodes (most prominent in children < 5 years), loose areolar tissue
  • Clinical significance: retropharyngeal abscess — most common in children; on lateral neck X-ray: >5 mm prevertebral soft tissue at C2 (child) or >7 mm (adult); can spread to mediastinum

4. Danger Space

  • Between alar and prevertebral layers of deep cervical fascia
  • Extends from skull base to diaphragm (T12)
  • Has no natural barriers → direct conduit for mediastinitis

5. Prevertebral Space

  • Behind prevertebral fascia
  • Contains paraspinous and scalene muscles, vertebral bodies, phrenic nerve, brachial plexus
  • Extends to coccyx

6. Submandibular (Submaxillary) Space

  • Bounded by floor of mouth mucosa (superior), digastric muscle (inferior), skin/platysma/mandible (lateral)
  • Divided by mylohyoid into sublingual (supramylohyoid) and submaxillary (inframylohyoid) compartments
  • Ludwig's angina = bilateral cellulitis of this space; floor-of-mouth edema, tongue swelling, airway emergency

7. Visceral (Pretracheal) Space

  • Superior: hyoid bone
  • Inferior: T4 (arch of aorta)
  • Contains: pharynx, esophagus, larynx, trachea, thyroid

8. Carotid Sheath Space

  • Envelopes carotid artery, internal jugular vein, vagus nerve
  • Called "The Lincoln Highway of the Neck" — a potential avenue for rapid spread of infection from skull base to thorax
K.J. Lee's Essential Otolaryngology, pp. 699–704

IV. ACUTE DISEASES OF THE PHARYNX

A. Acute Pharyngitis — Primary Angina

Definition: Acute inflammation of the pharyngeal mucosa and submucosal tissues, most commonly infectious. "Primary angina" refers to pharyngitis/tonsillitis where the pharynx itself is the primary site of infection (not secondary to systemic disease).

Viral Causes (Most Common Overall)

VirusClinical Feature
RhinovirusMost common cause of common cold
CoronavirusCommon cold
ParainfluenzaCommon cold, croup
Influenza A & BWidespread outbreaks, systemic illness
AdenovirusPharyngoconjunctival fever (pharyngitis + conjunctivitis)
Coxsackie AHerpangina (vesicular eruption on soft palate/anterior pillar + fever); hand-foot-mouth disease
Epstein-Barr virus (EBV)Infectious mononucleosis — exudative tonsillitis + lymphadenopathy + splenomegaly + atypical lymphocytosis
Cytomegalovirus (CMV)CMV mononucleosis
HSV types 1 & 2Pharyngitis, gingivostomatitis
HIVAcute retroviral syndrome (primary HIV infection) — consider in sexually active patients

Bacterial Causes

OrganismClinical Syndrome
Group A β-hemolytic Streptococcus (GABHS) S. pyogenesMost important bacterial cause; pharyngitis, tonsillitis, scarlet fever
Non-group A β-hemolytic Streptococcus (B, C, G)Pharyngitis, tonsillitis
Neisseria gonorrhoeaeSTI-related pharyngitis
Corynebacterium diphtheriaeDiphtheria — gray pseudomembrane, bull-neck, myocarditis, neuropathy
Arcanobacterium haemolyticumPharyngitis + scarlatiniform rash in adolescents
Mixed anaerobes + spirochetesVincent's angina (acute necrotizing ulcerative pharyngitis) — fusospirochetal infection
Francisella tularensisUlceroglandular tularemia
K.J. Lee's Essential Otolaryngology, pp. 684–686

GABHS Pharyngitis — Clinical Features and Diagnosis

Centor Criteria (used to estimate probability of GABHS):
  1. Tonsillar exudate
  2. Tender anterior cervical lymphadenopathy
  3. Fever (>38°C / 100.4°F)
  4. Absence of cough
Score 3–4: empiric treatment OR confirm with rapid strep test/culture Score 0–1: viral etiology likely; no antibiotic needed
Complications of untreated GABHS:
  • Non-suppurative: Acute rheumatic fever (ARF), post-streptococcal glomerulonephritis, PANDAS
  • Suppurative: peritonsillar abscess, parapharyngeal abscess, otitis media, sinusitis, mastoiditis
Treatment: Penicillin V (first-line) × 10 days; amoxicillin; clindamycin or macrolide if penicillin-allergic

B. Secondary Angina (Pharyngitis as Manifestation of Systemic Disease)

"Secondary angina" refers to pharyngeal inflammation that is a manifestation of an underlying systemic process rather than a primary pharyngeal infection:
ConditionPharyngeal Findings
Infectious mononucleosis (EBV)Severe exudative tonsillitis, palatal petechiae, uvular edema, posterior cervical adenopathy, hepatosplenomegaly
AgranulocytosisNecrotizing tonsillopharyngitis in context of absent neutrophils (drug-induced, leukemia)
Leukemia / lymphomaTonsillar enlargement, ulceration, without purulent exudate
DiphtheriaGrayish pseudomembrane (bleeds on removal), bull-neck adenopathy; toxin → cardiac/neurologic complications
SyphilisSecondary syphilis → mucous patches, painless pharyngeal lesions
Scarlet feverGABHS toxin → strawberry tongue, sandpaper rash, pharyngitis
HIV acute retroviral syndromeMononucleosis-like pharyngitis, rash, generalized lymphadenopathy
GERD/LPRChronic posterior pharyngitis, cobblestoning

C. Acute Tonsillitis

Acute inflammation of the palatine tonsils specifically.
Pathogens: GABHS (most important), adenovirus, EBV; other β-hemolytic streptococci
Clinical features:
  • Severe sore throat, odynophagia, dysphagia, fever
  • Tonsillar enlargement, erythema, white/yellow exudate (follicular or membranous)
  • Jugulodigastric lymphadenopathy (tender "tonsillar node")
  • Halitosis, trismus if extending
Grading of tonsillar hypertrophy (Friedman / Mallampati):
  • Grade 1: tonsils within tonsillar fossa
  • Grade 2: tonsils extending to pillars
  • Grade 3: tonsils beyond pillars
  • Grade 4: tonsils touching ("kissing tonsils")
Indications for tonsillectomy (Paradise criteria):
  • ≥7 episodes in 1 year, OR
  • ≥5 episodes/year for 2 consecutive years, OR
  • ≥3 episodes/year for 3 consecutive years
  • 2 weeks school/work missed in 1 year
  • Upper airway obstruction / sleep-disordered breathing
  • Peritonsillar abscess
  • Suspicion of malignancy (unilateral tonsillar enlargement)
  • Recurrent febrile seizures from tonsillitis
  • Diphtheria carrier
K.J. Lee's Essential Otolaryngology, p. 611–613

V. PERITONSILLAR ABSCESS

Definition

A peritonsillar abscess (PTA)quinsy — is an accumulation of pus in the peritonsillar space: deep to the tonsil capsule, between the palatine tonsil and the superior constrictor muscle. It is the most common deep space infection of the head and neck.

Pathogenesis

PTA develops as a complication of:
  1. Acute tonsillitis — spreading infection through the tonsil capsule
  2. Weber's glands (minor salivary glands in the superior peritonsillar space) — possibly a primary focus of infection
  3. Infection follows: tonsillitis → peritonsillar cellulitis → peritonsillar abscess
Microbiology: Typically polymicrobial:
  • Aerobic: Streptococcus pyogenes (GABHS), Staphylococcus aureus (including MRSA)
  • Anaerobic: Bacteroides spp., Fusobacterium spp.
The most common aerobic organisms are Streptococcus pyogenes (GABHS) and Staphylococcus aureus. The most common anaerobes are Bacteroides and Fusobacterium.Pfenninger & Fowler's Procedures for Primary Care
Location: Most commonly superior pole of the tonsil (where Weber's glands are concentrated); occasionally middle or inferior pole.

Clinical Presentation

Symptom/SignMechanism
Severe unilateral sore throatPeritonsillar space inflammation
Trismus (difficulty opening mouth)Spasm of medial pterygoid muscle due to adjacent inflammation
Hot potato voice (muffled, "plummy")Alteration of pharyngeal resonance by peritonsillar swelling
Dysphagia/odynophagiaOropharyngeal swelling
Drooling (pooling of saliva)Inability to swallow
Uvular deviation to contralateral sidePeritonsillar pus displaces soft palate and uvula across midline
Tonsil displaced medially and inferiorlyPus in superior peritonsillar space pushes tonsil
Bulging of soft palateInferior extension of pus under soft palate
Fever, chills, malaiseSystemic infectious response
Neck stiffness / ipsilateral neck painAdjacent parapharyngeal involvement
Foetor orisAnaerobic bacteria
Key distinguishing sign: unilateral peritonsillar bulge with uvular deviation away from the affected side (contrast with parapharyngeal abscess, where the tonsil is displaced medially but the soft palate may be less affected).

Imaging

  • CT neck with IV contrast: Gold standard for confirming abscess vs. cellulite (phlegmon), delineating extent, ruling out parapharyngeal extension
    • CT can identify which neck spaces require drainage, though still misidentifies abscess vs. phlegmon in ~25% of explorations
  • Intraoral ultrasound: Growing evidence for real-time guidance during aspiration
  • Plain lateral neck X-ray: Limited for PTA specifically (more useful for retropharyngeal abscess)

Management

1. Drainage (Primary Treatment)

Three approaches:
a. Needle aspiration: First-line in adults and cooperative adolescents
  • Insert 18-gauge needle at the point of maximal fluctuance (usually superior pole)
  • Aspirate pus; send for culture
  • Can repeat if re-accumulates
b. Incision and drainage (I&D): More thorough
  • Local anesthesia; incision at superior pole through mucosa and capsule
  • Blunt dissection with hemostat into abscess cavity
  • CPT code: 42700
c. Quinsy tonsillectomy (tonsillectomy in the acute setting):
  • Indications: recurrent PTA, patient already under general anesthesia, massive swelling limiting visualization, poor cooperation
  • Disadvantage: higher hemorrhage risk in acute infection
Peritonsillar abscess (PTA) may spontaneously drain, progress to involve the deep neck space or sepsis, or even lead to airway obstruction. The most important part of treatment is drainage.Textbook of Family Medicine, 9e

2. Antibiotics

  • Empiric coverage for GABHS and oral anaerobes
  • Penicillin G IV or Amoxicillin-clavulanate
  • Clindamycin (good anaerobic coverage, especially if MRSA concern)
  • Metronidazole can be added for anaerobic coverage

3. Supportive

  • IV fluids (often dehydrated from odynophagia)
  • Corticosteroids (dexamethasone) — reduce edema, symptom duration
  • Analgesics
  • Airway monitoring — if significant trismus and swelling, awake fiberoptic intubation or tracheostomy may be required

Complications

ComplicationMechanism
Parapharyngeal abscessRupture of PTA through superior constrictor into lateral pharyngeal space
Airway obstructionMassive edema or downward extension
DehydrationSevere odynophagia preventing oral intake
Internal carotid artery erosion / pseudoaneurysmParapharyngeal extension → vascular involvement
Internal jugular vein thrombophlebitis (Lemierre's syndrome)Bacteremia → septic thrombophlebitis of IJV; Fusobacterium necrophorum classically
Mediastinitis / descending necrotizing mediastinitisSpread via danger space — life-threatening
Brain abscessHematogenous spread or direct extension
Aspiration pneumoniaAbscess rupture with aspiration
Post-streptococcal nephritisStreptococcal antigen deposition
EndocarditisBacteremia
SepsisSystemic infection
Complications include: dehydration, airway obstruction, parapharyngeal abscess (due to rupture through superior constrictor), venous involvement (thrombosis, phlebitis, bacteremia, endocarditis), arterial involvement (thrombosis, hemorrhage, pseudoaneurysms), mediastinitis, brain abscess, aspiration pneumonia.K.J. Lee's Essential Otolaryngology

Differential Diagnosis of PTA

ConditionKey Differentiating Feature
Peritonsillar cellulitisNo fluctuance; less trismus; no abscess on imaging
Parapharyngeal abscessTonsil displaced medially but no peritonsillar bulge; neck swelling; CN palsy
Retropharyngeal abscessPosterior pharyngeal wall bulge; stiff neck; dysphagia; fever in child
EpiglottitisDysphagia, drooling, inspiratory stridor; "thumbprint sign" on lateral X-ray
Tonsillar malignancyNo fever, firm, non-fluctuant unilateral tonsillar mass; no response to antibiotics
Infectious mononucleosisBilateral tonsils, posterior cervical adenopathy, splenomegaly; Monospot positive

VI. SUMMARY TABLE — DEEP PHARYNGEAL SPACE INFECTIONS

SpaceCommon SourceClinical CluesDrainage Route
PeritonsillarTonsillitisUvular deviation, hot potato voice, trismusTransoral needle/I&D
ParapharyngealPTA rupture, dental, parotidLateral neck swelling, trismus, CN palsiesTransoral or transcervical
RetropharyngealURI, adenitis (children)Posterior pharyngeal bulge, stiff neck, dysphagiaTransoral or transcervical
SubmandibularDental (2nd/3rd molar)Floor of mouth induration, Ludwig's anginaTranscervical
Danger spaceExtension from aboveMediastinitis, rapid deteriorationMediastinal drainage

Sources

  • K.J. Lee's Essential Otolaryngology, 12th ed. — Chapters 36–37 (pharyngeal spaces, infections, PTA)
  • Cummings Otolaryngology Head and Neck Surgery — Chapters 96, 100 (pharyngeal anatomy, radiology)
  • Gray's Anatomy for Students — Chapter 8 (pharynx, p. 1180–1188)
  • Pfenninger & Fowler's Procedures for Primary Care — Peritonsillar abscess drainage
  • Textbook of Family Medicine, 9e — Peritonsillar abscess management

Physiology of the pharynx. Pharyngeal spaces The relationship of the pharynx to the dental system and gastrointestinal tract List the boundaries of the pharyngeal space. Formation of the pharynx List the methods of examination of the pharynx. Tonsils of Waldeyer´s ring (location) Structure of pharyngeal wall (layers) Relation pharynx with ear Parts, boundaries of the pharynx and lining epithelium? Nasopharyngeal tonsil, structure and function Lymphatic drainage of the pharynx Tubal tonsils, structure, location and function Anatomy of palatine tonsils (blood supply, venous and lymphatic drainage) The elements belonging to the upper part of the pharynx. The blood supply of the pharynx. The elements belonging to the middle floor of the pharynx. The innervation of the pharynx. Methods of examination of the pharynx. Pharyngoscopic picture in normal. Pharyngoscopic picture in follicular tonsillitis. Pharyngoscopic picture in parenchymaous tonsillitis. Pharyngoscopic picture in membranous tonsillitis. Pharyngoscopic picture in peritonsillar abscess. Method of treatment of peritonsillar abscess. Differential diagnosis of acute tonsillitis and faucial diphtheria. Examination of chronic tonsillitis Zenker´s diverticulum (Sac) Causes and complications of chronic tonsillitis. Faucial diphtheria. Aetiology, clinical features, complication, treatment Atrophic pharyngitis. Clinical features and treatment Ludwig´s angina. Aetiology, clinical features, complication, treatment Types of chronic tonsillitis. Differential diagnosis of chronic compensated and decompensated tonsillitis? Complications of chronic tonsillitis Conservative treatment of chronic tonsillitis. Pharyngoscopic picture in chronic tonsillitis. Local signs of chronic tonsillitis on pharyngoscopy. Local signs of acute pharyngitis on pharyngoscopy. Technique of tonsillectomy Technique of adenotomy Indications for tonsillectomy. Indication for adenotomy. Complications after tonsillectomy Diagnosis and clinical feature of adenoids? Chronic hypertrophic pharyngitis - clinic, diagnosis and treatment. Chronic catarrhal pharyngitis - clinic, diagnosis and treatment. Chronic atrophic pharyngitis - clinic, diagnosis and treatment. Angiofibroma of nasopharynx (clinic, diagnosis and treatment). Benign tumors of the pharynx. Malignant tumors of the pharynx. in detail answer based on ent

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