Here is a detailed explanation of this ECG report.
ECG Report - Full Explanation
Patient & Technical Details
| Parameter | Value |
|---|
| Date/Time | 30-05-2026, 14:32:15 |
| Machine | CardioTouch (Bionet Co.) |
| Speed | 25 mm/sec |
| Gain | 10 mm/mV |
| Filters | Base 0.2 Hz, LPF 150 Hz, AC 50 Hz |
Measured Intervals
| Measurement | Value | Normal Range |
|---|
| Heart Rate | 187 bpm | 60-100 bpm |
| PR/RR Interval | 124/321 ms | 120-200 ms (PR) |
| QRS Duration | 116 ms | <120 ms |
| QT/QTc | 208/360 ms | <440 ms (QTc) |
| P-R-T Axes | -129 / -28 / -131 | QRS axis -28 is normal |
Computer Analysis Results (Key Findings)
The machine has flagged 3 critical findings, all marked ## (to be finally confirmed by physician):
1. Atrial Flutter
What it is: A rapid, regular atrial arrhythmia caused by a macro-reentrant circuit typically confined to the right atrium, traveling counterclockwise around the tricuspid annulus. The atria fire at ~300 beats/min, with the ventricles typically responding at half that rate (2:1 block = ~150 bpm).
Why the rate is 187 bpm here: At a heart rate of 187 bpm, this is consistent with atrial flutter with a variable or near 2:1 AV conduction ratio. The atrial flutter rate (~300 bpm) divided by 1.6 gives this ventricular rate, suggesting some variable block or a slightly atypical flutter circuit.
What to look for on the tracing:
- Sawtooth (flutter) waves - negative in leads II, III, and aVF; positive in V1 (typical counterclockwise flutter)
- No distinct P waves - replaced by continuous flutter waves
- Regular or regularly-irregular QRS complexes
As described in Goldman-Cecil Medicine: "Typical atrial flutter can be recognized by a 'sawtooth' P wave morphology, which is predominantly negative in leads II, III, and aVF and positive in V1 (counterclockwise atrial flutter)."
2. Normal QRS Axis
- The P-R-T axis shows QRS axis of -28 degrees - this is at the borderline of normal/mild left axis deviation, but the machine has classified it as normal.
- Normal axis range is -30 to +90 degrees, so -28 is just within normal limits.
3. ST Abnormality - Possible Transmuural Anterolateral Injury
This is the most clinically urgent finding.
The machine reports: "ST abnormality, possible transmural injury (anterolateral)"
- SV1/RV5/R+S: 0.00/0.83/0.83 mV
What "transmural anterolateral injury" means:
- ST elevation in the anterolateral leads (I, aVL, V4-V6) suggests acute full-thickness myocardial injury - the pattern of an ST-elevation MI (STEMI) involving the territory supplied by the left anterior descending (LAD) or left circumflex artery.
- "Transmural" = injury extending through the full thickness of the myocardial wall.
Important caveat: ST changes during atrial flutter can be secondary (rate-related) rather than primary ischemic. At a heart rate of 187 bpm, demand ischemia and rate-related ST depression/elevation are possible even without coronary artery occlusion. The machine itself flags this as "possible" and requires physician confirmation.
Summary: What This ECG Tells Us
RHYTHM: Atrial Flutter (~300 atrial / ~187 ventricular bpm)
RATE: 187 bpm — markedly elevated
AXIS: Normal (-28°)
ST: Possible anterolateral transmural injury
OVERALL: MARKEDLY ABNORMAL ECG
Clinical Priority
This is a markedly abnormal ECG (as stated on the report). The combination of:
- Rapid atrial flutter with fast ventricular rate (187 bpm)
- ST changes suggesting possible anterolateral ischemia/injury
...requires urgent physician review. The two major differential considerations are:
- Rate-related ST changes - ST abnormalities are common at very high rates and may resolve with rate control (cardioversion or AV nodal blockade)
- Acute STEMI - If ST elevation persists after rate control, acute coronary syndrome must be ruled out urgently (troponins, repeat ECG, clinical context)
Immediate management steps typically include:
- Confirm the rhythm clinically (12-lead review by cardiologist)
- Rate control or cardioversion for atrial flutter
- Serial troponins and repeat ECG after rate control
- Echocardiography if wall motion abnormality is suspected
Disclaimer: This analysis is educational. The report itself states "to be finally confirmed by physician." This ECG must be urgently reviewed by the treating clinician - it must not be interpreted in isolation without clinical context.
- Goldman-Cecil Medicine, p. 573 | Braunwald's Heart Disease, p. 423