Hi so my team and i are presenting on Gatrointestinal bleeding for undergraduate year 3 surgical posting and i will be talking about anatomy upper and lower gastrointestinal tract, its going to be less than 5 minutesof presentation what i should cover, provide links of images i should use too
upper gastrointestinal tract anatomy diagram
lower gastrointestinal tract colon anatomy diagram
GI tract anatomy esophagus stomach duodenum small bowel colon labeled surgical
This distinction drives everything clinically — how the patient presents (haematemesis vs. rectal bleeding), how you investigate, and how you manage.
| Structure | Key Anatomical Points for GI Bleeding |
|---|---|
| Oesophagus | Lower 1/3 → portal hypertension → varices (submucosal venous plexus drains via azygos → portal) |
| Gastro-oesophageal junction | Mallory-Weiss tears occur at the mucosal junction here |
| Stomach | Rich blood supply: left/right gastric, left/right gastroepiploic, short gastric arteries. Peptic ulcer disease → posterior DU can erode gastroduodenal artery (GDA) |
| Duodenum | 1st part = bulb (most common PUD site). Posterior D1 is directly adjacent to the GDA — the most dangerous arterial bleeding source in UGIB |
| Structure | Key Anatomical Points for GI Bleeding |
|---|---|
| Small bowel (jejunum/ileum) | Long vasa recta from SMA; difficult endoscopic access — LGIB here is rare but serious |
| Colon | Two distinct arterial territories — critical for understanding bleeding patterns |
| Rectum | Dual supply: superior rectal (IMA) + middle/inferior rectal (internal iliac) |
| Anal canal | Internal haemorrhoids = dilated superior rectal venous plexus (above dentate line); external haemorrhoids = inferior rectal plexus |


| Section | Time |
|---|---|
| Ligament of Treitz — the dividing line | 45 sec |
| Upper GI anatomy + blood supply | 1.5 min |
| Lower GI anatomy + vascular territories | 1.5 min |
| Clinical correlation (why anatomy = presentation) | 1 min |
| Total | ~4 min 45 sec |
My friends are presenting on the pathophysiology of gi bleeding, how to avoid clashing if i havenslidenumber 4 or should i omit it
| Instead of saying... | Say this (anatomy framing)... |
|---|---|
| "Blood is digested over 8 hours causing melaena" | "Melaena indicates a source proximal to the splenic flexure — the transit distance is long enough for digestion" |
| "Portal hypertension causes varices" | "Varices occur at portosystemic anastomotic sites — GOJ, rectal plexus, umbilical veins — where portal and systemic circulations connect" |
| "Haematochezia means LGIB" | "Bright red blood per rectum localises the source to the distal colon or rectum — short transit, no digestion" |
"I'll cover what the symptoms tell us about where the bleeding is anatomically — the why it bleeds is coming up in the next section."
Whats my overall script for slide 1 to 4
"Good morning everyone. I'll be covering the anatomy of the gastrointestinal tract as it relates to GI bleeding.Before we talk about any specific structure, there is one anatomical landmark that underpins everything in GI bleeding — the ligament of Treitz, also called the suspensory muscle of the duodenum. It marks the junction between the fourth part of the duodenum and the jejunum.This single structure divides GI bleeding into two categories. Everything proximal to it — the oesophagus, stomach, and duodenum — is upper GI bleeding. Everything distal — the jejunum, ileum, colon, rectum, and anus — is lower GI bleeding.Keep that landmark in mind, because every structure I discuss from here sits on one side of that line."
"Starting with the upper GI tract.The oesophagus is the first structure of note. Its lower third is where you find oesophageal varices — these develop in the submucosal venous plexus at the gastro-oesophageal junction, a site where the portal and systemic circulations meet. The gastro-oesophageal junction itself is also where Mallory-Weiss tears occur — linear mucosal splits at that junction.Moving into the stomach — it has an extraordinarily rich arterial supply from the coeliac axis: the left and right gastric arteries along the lesser curve, the left and right gastroepiploic arteries along the greater curve, and the short gastric arteries supplying the fundus. This is why gastric bleeding can be brisk and difficult to control.Finally the duodenum, specifically the first part or the bulb — this is the most common site for peptic ulcer disease. What makes it surgically important is the gastroduodenal artery, which runs directly posterior to the first part of the duodenum. A posterior duodenal ulcer that erodes into the GDA produces one of the most catastrophic bleeds we see — high volume, arterial, and potentially life-threatening."
"Now the lower GI tract, distal to the ligament of Treitz.The small bowel — jejunum and ileum — is supplied by the superior mesenteric artery via long vasa recta. Bleeding here is uncommon but serious, and it's notoriously difficult to access endoscopically.The colon is where most lower GI bleeding originates, and its anatomy is best understood through two arterial territories — which you can see on this diagram.(point to image)The superior mesenteric artery supplies the small bowel, caecum, ascending colon, and proximal two-thirds of the transverse colon — shown in red. The inferior mesenteric artery supplies the distal transverse, descending, and sigmoid colon — shown in green. The rectum has a dual supply from the IMA above and the internal iliac arteries below — shown in blue.Two points on the colon worth highlighting: the splenic flexure and the rectosigmoid junction are watershed zones — where the terminal branches of two arterial systems meet. These areas are most vulnerable to ischaemia when blood pressure drops.Finally the anal canal — internal haemorrhoids arise from the superior rectal venous plexus above the dentate line. External haemorrhoids arise from the inferior rectal plexus below it. A simple anatomical line — the dentate line — separates them."
"So to bring it all together — and I want to be clear this is purely the anatomical perspective, my colleagues will be explaining the mechanisms behind why these sites bleed.When a patient presents with haematemesis, you are dealing with a source proximal to the ligament of Treitz — the oesophagus, stomach, or duodenum.Melaena — dark, tarry stool — tells you the source is anatomically far enough proximal that blood undergoes digestion during transit. That typically means proximal to the splenic flexure.Bright red blood per rectum localises the source to the distal colon or rectum — short transit, no time for digestion.And knowing the arterial supply — coeliac axis for the upper tract, SMA and IMA for the lower — tells the surgical team where to look on CT angiography and where to target for embolisation.So the anatomy does not just describe the structures — it tells you where the patient is bleeding from before you even pick up a scope.I'll hand over now to my colleague who will cover the pathophysiology."
| Slide | Content | Time |
|---|---|---|
| 1 | Ligament of Treitz | ~45 sec |
| 2 | Upper GI anatomy | ~1 min 30 sec |
| 3 | Lower GI anatomy | ~1 min 30 sec |
| 4 | Anatomy maps the source | ~1 min |
| Total | ~4 min 45 sec |