How do i separate this outline to 8 people, first of all is this outline okay for undergraduates or should I be for Bailey and love oriented *Seminar 2: Upper Gastro intestinal Bleed* 1. Anatomy of Upper gastro-intestinal tract 2. Causes of upper GI Bleed 3. Emergency Management of acute GI bleeding episode 4. Brief discussion of management of hemorrhage due to Ulcers (Oesophageal Gastric Duodenal)/ Erosions (Oesophageal Gastric Duodenal) / Mallory–Weiss tear / Oesophageal varices / Tumour (Oesophageal Gastric)/ Vascular lesions /any other lesions eg. Hemobilia etc. by remaining speakers
Asking for Preferences
Here's my honest assessment and the split:
Is the outline appropriate for undergraduates?
Yes — the outline is well-suited for early clinical undergraduates (Year 3–4), and you do NOT need Bailey & Love depth. Bailey & Love is a surgical reference text aimed at trainees and surgeons. For a 40-minute undergraduate seminar, you want:
- Conceptual understanding, not operative technique
- Recognise → stabilise → investigate → refer thinking
- Clinical principles over procedural detail
The one adjustment I'd suggest: point 4 is currently one massive topic assigned to "remaining speakers." It needs to be broken into individual speaker slots — which is exactly what the split below does.
Division Among 8 Speakers (~5 min each)
| # | Speaker | Topic | Key Points to Cover |
|---|---|---|---|
| 1 | Speaker 1 | Anatomy of the Upper GI Tract | Oesophagus, stomach, duodenum up to ligament of Treitz; blood supply (left gastric, splenic, hepatic arteries); venous drainage & portosystemic connections |
| 2 | Speaker 2 | Causes & Classification of Upper GI Bleed | Variceal vs. non-variceal; common causes by frequency (PUD most common); Rockall / Blatchford score mention as risk tools |
| 3 | Speaker 3 | Emergency Management — Resuscitation | ABCDE approach; 2 large-bore IV lines, fluid resuscitation, blood transfusion targets; nil by mouth, catheter, monitoring |
| 4 | Speaker 4 | Emergency Management — Investigation & Escalation | Bloods (FBC, U&E, coagulation, G&S), NG tube debate, urgent OGD timing, surgical/ITU escalation |
| 5 | Speaker 5 | Peptic Ulcer Disease Bleeding (Gastric & Duodenal) | Causes (H. pylori, NSAIDs), Forrest classification, endoscopic haemostasis (injection, clips, APC), PPI infusion |
| 6 | Speaker 6 | Oesophageal Varices & Portal Hypertension | Pathophysiology of portal HTN, Sengstaken-Blakemore tube, terlipressin, banding vs. sclerotherapy, TIPSS mention |
| 7 | Speaker 7 | Mallory–Weiss Tear & Oesophageal/Gastric Erosions | Mechanism (vomiting → longitudinal mucosal tear), usually self-limiting, when intervention needed; erosions vs. ulcers distinction |
| 8 | Speaker 8 | Tumours, Vascular Lesions & Rare Causes | Oesophageal/gastric malignancy as bleeding source; angiodysplasia, Dieulafoy lesion, haemobilia; brief overview only |
Practical Tips
- Speakers 1–2 set the foundation — everyone else builds on them, so they should present first.
- Speakers 3–4 work as a pair covering emergency management and flow naturally together.
- Speakers 5–8 each own a specific cause — they should use a consistent mini-template: What is it → Why it bleeds → How you manage it.
- For a 5-minute slot, 2–4 slides maximum per speaker is the right target.
- Avoid operative surgical detail (e.g., oversewing vessels, Whipple) — that's Bailey & Love territory and beyond scope.
Any repetition in this flow or anyone getting too much of work
Good question. Here's an honest audit:
Repetition Risks
| Overlap | Between | Fix |
|---|---|---|
| "Emergency management" could bleed into cause-specific management | Speakers 3–4 vs. Speakers 5–8 | Speakers 3–4 stick to generic resuscitation only — no cause-specific treatment. Speakers 5–8 own their specific management. |
| Erosions vs. ulcers distinction | Speaker 5 (PUD) and Speaker 7 (erosions) | Speaker 5 defines ulcers, Speaker 7 defines erosions — but they must not both explain the difference. Assign that definition clearly to Speaker 7 only. |
| Portal hypertension pathophysiology | Could creep into Speaker 2 (causes) and Speaker 6 (varices) | Speaker 2 names varices as a cause only. Speaker 6 explains the pathophysiology. Draw that line explicitly. |
Workload Imbalance
| Speaker | Load Assessment |
|---|---|
| Speaker 8 | Overloaded — tumours (2 sites), vascular lesions (angiodysplasia, Dieulafoy), AND rare causes (haemobilia, etc.) is too much for 5 minutes |
| Speaker 7 | Underloaded — Mallory–Weiss + erosions is relatively light content |
| Speakers 3 & 4 | Roughly equal but could overlap on "when to scope" — clarify that Speaker 4 owns OGD timing |
Suggested Fixes
1. Redistribute Speaker 8's load onto Speaker 7:
Speaker 7 (revised): Mallory–Weiss tear + Erosions + Vascular lesions (angiodysplasia, Dieulafoy) Speaker 8 (revised): Tumours (oesophageal + gastric) + Rare causes (haemobilia, aortoenteric fistula) — this is now more balanced since tumours alone carry enough clinical content for 5 minutes
2. Give each cause-specific speaker (5–8) a shared template:
- What is it / how common?
- Why/how it bleeds
- One or two key management points
This prevents some speakers going deep on pathophysiology while others rush through management.
Revised Clean Split
| # | Speaker | Topic |
|---|---|---|
| 1 | Speaker 1 | Anatomy of upper GI tract |
| 2 | Speaker 2 | Causes & classification (overview only, no management) |
| 3 | Speaker 3 | Emergency resuscitation (ABCDE, IV access, transfusion) |
| 4 | Speaker 4 | Investigations, OGD timing & escalation |
| 5 | Speaker 5 | Peptic ulcer bleeding (gastric + duodenal) |
| 6 | Speaker 6 | Oesophageal varices |
| 7 | Speaker 7 | Mallory–Weiss + Erosions + Vascular lesions |
| 8 | Speaker 8 | Tumours (oesophageal/gastric) + Rare causes |
This gives a cleaner, more balanced flow with no significant repetition if each speaker respects their lane.
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