Recurrent patella dislocation

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Recurrent Patellar Dislocation

Definition and Epidemiology

Recurrent patellar dislocation refers to repeated lateral displacement of the patella out of the trochlear groove. It most commonly affects adolescents and young adults engaged in athletic activities. Recurrence after an initial dislocation is reported in 15% to 75% of patients, with risk of recurrence particularly high in the skeletally immature (two thirds of dislocations in younger patients will recur).
  • Campbell's Operative Orthopaedics 15th Ed 2026 (Ch. 52)
  • Miller's Review of Orthopaedics 9th Edition

Pathomechanics

Patellar instability results from a valgus-producing force, either direct (blow to the medial patella) or indirect (twisting with planted foot). It is inherently multifactorial with a genetic predisposition.
Static stabilizers - MPFL (primary), medial retinaculum Dynamic stabilizers - Vastus medialis obliquus (VMO) Bony stabilizers - Trochlear groove (active from ~30° flexion onward)
The MPFL and VMO are the critical stabilizers in 0-20 degrees of flexion. Beyond 30 degrees the patella engages the trochlear groove and bony stability predominates. When patella alta or dysplasia is present, this bony engagement is delayed or absent, predisposing to instability.
With dislocation, the MPFL tears (most commonly at its patellar insertion on MRI) and chondral/osteochondral injury can occur at the medial patellar facet (shear during reduction) and/or the lateral femoral condyle (impaction during dislocation).

Risk Factors for Recurrence

FactorThreshold
Young age< 15 years
Female sex-
Trochlear dysplasiaSulcus angle > 145°, depth ≥ 3 mm
Patella altaInsall index > 1.3
Elevated TT-TG distance> 20 mm (normal 9-13 mm)
Ligamentous laxity / hypermobility-
Positive family history-
Femoral anteversion / external tibial torsionThigh-foot angle > 30°
Genu valgum-
The classic triad of highest risk is trochlear dysplasia + patella alta + skeletal immaturity.
  • Campbell's Operative Orthopaedics 15th Ed 2026

Trochlear Dysplasia - Dejour Classification

The most important anatomical risk factor. Classified on lateral X-ray by the crossing sign (trochlear groove line crossing the anterior femoral cortex) and on CT/MRI.
Dejour Classification Type C showing crossing sign, double contour, lateral convexity and medial hypoplasia
Dejour classification of trochlear dysplasia (from Miller's Review of Orthopaedics)
  • Type A: Shallow trochlea (sulcus angle > 145°)
  • Type B: Flat or convex trochlea + supratrochlear spur
  • Type C: Asymmetric - lateral convexity + medial hypoplasia + double contour sign
  • Type D: Combines B + C features (cliff pattern)

Clinical Assessment

History:
  • Twisting mechanism or planted-foot rotation
  • Audible/palpable "pop" followed by giving way - often confused with ACL injury in young patients
  • Spontaneous reduction with knee extension is common
  • Ongoing anterior knee pain ± instability
Physical Examination:
  • Diffuse peripatellar swelling and tenderness (worse medially - MPFL injury)
  • Positive patellar apprehension test (lateral translation at 0-30° flexion)
  • J-sign: patella tracks laterally in full extension and reduces abruptly as flexion begins
  • Lateral patellar glide > 3 quadrants (incompetent MPFL)
  • Assess Q angle (> 20° abnormal), foot progression angle, femoral anteversion
  • Quadriceps atrophy (thigh circumference)
  • Examine for systemic hypermobility (Beighton score)

Imaging

X-rays (weight-bearing + axial views):
  • Lateral view: crossing sign, trochlear bump (supratrochlear spur), patella alta assessment
  • Merchant/axial view: sulcus angle (normal ~137°), congruence angle (normal −8°), lateral patellar tilt
  • Patellar height indices: Insall-Salvati ratio (>1.2 = alta), Caton-Deschamps index (>1.2 = alta)
CT:
  • Gold standard for TT-TG distance (tibial tubercle to trochlear groove distance)
    • Normal: 9-13 mm
    • 15-20 mm: borderline
    • 20 mm: clearly abnormal, associated with instability
  • Measures rotational deformities (femoral anteversion, tibial torsion)
MRI:
  • Best for MPFL integrity (usually torn at patellar insertion)
  • Characteristic "kissing contusion": lateral femoral condyle + medial patella bruising
  • Articular cartilage damage assessment
  • Can estimate TT-TG (but tends to underestimate vs. CT)

Management

Non-operative (First-line for first-time dislocation)

  • Short immobilization (1-3 weeks), then patellar stabilizing brace
  • Physiotherapy: VMO strengthening, hip abductor and core strengthening, proprioception training
  • Activity modification; weight loss if applicable
  • Return to sport: 6-12 weeks when functional milestones are met
  • In patients with hypermobile joints, conservative management is the mainstay - surgery outcomes are poor in this group

Indications for Surgery

Urgent/acute:
  • Associated osteochondral fracture (loose body)
Elective - strong indications:
  • Recurrent instability (multiple events)
  • First-time dislocation in high-risk anatomy (trochlear dysplasia + patella alta + young age)
  • Contralateral recurrent instability

Surgical Treatment

The treatment framework is guided by the underlying pathoanatomy: containment (bony), alignment, and soft-tissue imbalance.

Treatment Table (Campbell's)

PathologyFindingProcedure
Patella altaInsall index > 1.3Distalization osteotomy
Trochlear dysplasiaCrossing sign, sulcus angle > 145°MPFL reconstruction ± Trochleoplasty
Patellar dysplasia (Wiberg C)-MPFL reconstruction
Lateral TT displacementTT-TG > 20 mmTibial tubercle anteromedialization
Femoral anteversion / genu valgumThigh-foot angle > 30°Rotational osteotomy / epiphysiodesis
VMO dysfunction (dynamic)TT-TG < 20 mmRehabilitation
Incompetent MPFL / hyperlaxityLateral glide 3+ quadrantsMPFL reconstruction
Excessive lateral tightnessLateral tilt on axial viewsLateral release only

Key Procedures

1. MPFL Reconstruction (Workhorse procedure - "Low risk, high reward")

  • Primary procedure for recurrent MPFL deficiency ± trochlear dysplasia
  • Graft: gracilis or semitendinosus autograft (or allograft)
  • Femoral attachment at the Schottle point (1 mm anterior to posterior cortex line, 2.5 mm distal to posterior medial femoral condyle, proximal to Blumensaat line)
  • Tunnel placement errors:
    • Too proximal = tight in flexion
    • Too distal = loose in flexion
  • Key complication: medial patellar instability or medial facet OA from overtightening
MPFL repair and adductor magnus tendon reconstruction technique - medial knee anatomy
MPFL: A - anatomy showing patellofemoral and patellotibial ligaments. B - ligament reattachment. C - retinaculum duplication after reconstruction. (Campbell's 15th Ed)

2. Tibial Tubercle Anteromedialization (Fulkerson / Elmslie-Trillat)

  • Indicated when TT-TG > 20 mm (or > 15 mm with trochlear dysplasia)
  • Elmslie-Trillat: pure medialization - quicker healing, lower fracture risk
  • Fulkerson: oblique osteotomy providing anteromedialization (30° = 1 mm anterior per 2 mm medial; 45° = 1:1 ratio)
    • Best for lateral/distal facet chondral damage
    • Contraindicated for proximal/medial facet arthritis
  • Often combined with MPFL reconstruction

3. Trochleoplasty ("High risk, high reward")

  • Indicated for severe trochlear dysplasia (convex/flat trochlea) unresponsive to other procedures
  • Low recurrence rate but significant risk: osteonecrosis, arthrofibrosis, progressive DJD of lateral facet

4. 3-in-1 Procedure (Oliva) - for open physes / skeletally immature

  • Lateral release + VMO advancement + transfer of medial third of patellar tendon to MCL
  • Used when tibial tubercle transfer is unsafe (open physes)
  • Steps: lateral retinacular release → mobilization of VMO insertion → medial 1/3 patellar tendon detached distally, transferred medially and fixed with suture anchors at 45° → VMO advanced 10 mm distally

5. Lateral Release - indicated ONLY for excessive lateral pressure syndrome

  • Not indicated as isolated treatment for patellar instability
  • May actually increase risk of both medial and lateral subluxation if used in isolation

Special Considerations

Pediatric / Open Physes

  • Tibial tubercle osteotomy is contraindicated (physeal injury risk)
  • MPFL reconstruction must avoid the physis
  • 3-in-1 soft-tissue procedure or MQTFL (medial quadriceps tendon-femoral ligament) reconstruction preferred
  • Epiphysiodesis for angular deformities

Hypermobility Syndromes

  • Patellar instability in hypermobile joints is primarily managed conservatively
  • Joint hypermobility is a poor prognostic indicator for operative outcomes
  • Surgery should not be the mainstay of treatment in this group

"Miserable Malalignment Syndrome"

  • Combination of femoral anteversion + genu valgum + external tibial torsion + pronated feet
  • Exacerbates patellar instability in adolescents
  • May require rotational osteotomy in severe cases

Complications of Recurrent Dislocation

  • Articular cartilage damage (medial patellar facet - most common site)
  • Osteochondral fractures and loose bodies
  • Progressive patellofemoral osteoarthritis
  • Ongoing anterior knee pain
  • Quadriceps inhibition and muscle atrophy

Recent Evidence (2024-2026)

A 2024 systematic review (PMID 39260775) confirmed that distal femoral osteotomies improve recurrent patellar instability in patients with genu valgum, supporting the role of addressing coronal plane deformity.
A 2025 systematic review (PMID 40744396) found that females experience higher rates of recurrent dislocation following MPFL reconstruction, highlighting the need for careful patient selection and potentially addressing additional anatomic risk factors (trochlear dysplasia, patella alta) in female patients rather than isolated MPFL reconstruction.

Sources: Campbell's Operative Orthopaedics 15th Ed 2026 (Ch. 38 & 52); Miller's Review of Orthopaedics 9th Ed; Rheumatology 2-Volume Set 2022 (Elsevier)

Ctev

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Congenital Talipes Equinovarus (CTEV / Clubfoot)

Definition

CTEV is a complex congenital foot deformity present at birth characterized by four components summarized by the mnemonic CAVE:
ComponentDescription
C - CavusHigh arch (plantar flexion of forefoot on hindfoot)
A - AdductusForefoot adduction (medial deviation)
V - VarusHindfoot varus (inversion)
E - EquinusPlantarflexion deformity at ankle
The deformity is corrected in exactly this CAVE order during treatment.

Epidemiology

  • Incidence: ~1 in 1,000 live births
  • Boys affected twice as often as girls
  • 50% of cases are bilateral
  • Most common serious congenital foot anomaly

Pathoanatomy

The primary structural abnormality is talar neck deformity - medial and plantar deviation of the talar neck, with secondary:
  • Medial rotation of the calcaneus under the talus
  • Medial displacement of the navicular (onto the medial aspect of the talar head)
  • Medial displacement of the cuboid
  • Shortening/contracture of: Achilles tendon, tibialis posterior, FHL, FDL, intrinsic muscles, joint capsules, ligaments, and plantar fascia
The anterior tibial artery is often absent or diminutive - this is an important surgical consideration (dorsalis pedis may be insufficient as sole blood supply).

Etiology

TypeDetails
Idiopathic (majority)Genetic predisposition via PITX1-TBX4 transcriptional pathway
NeurogenicMyelomeningocele (30-50% of MMC patients have clubfoot), spinal cord anomalies
SyndromicArthrogryposis, diastrophic dwarfism, prune-belly syndrome, tibial hemimelia
Constriction bandStreeter dysplasia
Syndromic and neurogenic clubfeet are more rigid, have higher recurrence rates, and respond less well to conservative management than idiopathic forms.

Clinical Assessment

History

  • Visible foot deformity at birth
  • Family history (genetic predisposition)
  • Prenatal diagnosis often possible on USS (~ 20 weeks)

Examination

  • Rigid vs. positional (postural) clubfoot - distinguish by gentle manipulation
  • Check for associated spine (MMC), hip dysplasia
  • Assess skin creases: deep medial and posterior creases indicate severity
  • In older/untreated: weight-bearing on dorsolateral foot, callosities, short leg, calf atrophy

Severity Scoring

Pirani Score (0-6, most widely used)

Six clinical signs, each scored 0 (normal), 0.5 (moderate), or 1 (severe):
Midfoot signs (0-3):
  1. Curved lateral border
  2. Medial crease
  3. Lateral talar head coverage
Hindfoot signs (0-3): 4. Posterior crease 5. Rigid equinus 6. Empty heel
Pirani Classification scoring chart showing all 6 signs graded 0, 0.5, and 1
Pirani classification - Campbell's Operative Orthopaedics 15th Ed

Dimeglio Classification (0-20)

  • Grade I (Benign): 0-5 pts
  • Grade II (Moderate): 6-10 pts
  • Grade III (Severe): 11-15 pts
  • Grade IV (Very Severe): 16-20 pts
Both Pirani and Dimeglio scores correlate with the number of Ponseti casts required.

Radiographic Features

Minimal ossification in the neonate makes X-rays less useful in early infancy. When obtained:
  • AP view (Kite angle - talocalcaneal angle): Normal 20-40°; reduced (<20°) in CTEV due to parallelism of talus and calcaneus
  • Lateral/Turco view (dorsiflexion lateral): Talocalcaneal angle < 35° (normal)
  • Talus-1st metatarsal angle (AP): Negative in CTEV (normally 0-20°)
  • Key radiographic sign: "Parallelism" - talus and calcaneus axes run parallel rather than diverging

Treatment

Treatment begins as early as possible - ideally within the first 2-4 weeks of life.

1. Ponseti Method (Gold Standard - First-line)

Principles:
  • Serial weekly gentle manipulation and long-leg plaster casting
  • Corrects deformities in the CAVE sequence
  • Uses the lateral talar head as a fulcrum for adduction/varus correction
  • Supinates forefoot (do NOT pronate - creates rocker-bottom)
Casting sequence:
Cast No.Correction Achieved
1st castCorrects cavus - supinate forefoot, dorsiflex 1st ray
2nd-4th castsCorrect adductus and varus - lateral pressure on distal talar head as fulcrum, abduct forefoot
5th-6th castCorrect equinus - dorsiflexion (after tenotomy)
  • Total: typically 5-6 casts required
  • Final cast placed in 70° of abduction
  • >90% success in children under 2 years, even after previous unsuccessful treatment
Percutaneous Achilles Tenotomy:
  • Required in ~90% of patients to fully correct equinus
  • Done under local anesthesia (office/clinic) before final cast application
  • Prevents rocker-bottom deformity (do not dorsiflex through midfoot before varus corrected)
  • Cast then worn for 3 more weeks post-tenotomy
Maintenance Phase - Foot Abduction Brace (Denis Browne / FAB):
  • Mandatory - most important factor in preventing recurrence
  • Full-time for 3 months, then during sleep and naps for 3-4 years
  • Brace: shoes set at 60-70° abduction bilaterally (40° for unilateral)
  • Recurrence is most strongly associated with brace non-compliance
Recurrence rates: 10-30%; most recurrences can be retreated with recasting ± Achilles tenotomy
Modifications showing equivalent results:
  • Accelerated biweekly casting (faster correction)
  • Fiberglass casting = plaster results
  • Bracing to age 4 may be superior to stopping at age 3

2. Anterior Tibial Tendon Transfer (ATTT)

  • Indicated for dynamic supination deformity (residual or recurrent varus driven by anterior tibialis over-activity)
  • Transfer of the anterior tibial tendon from its lateral cuneiform insertion to the middle/lateral cuneiform or cuboid
  • Usually combined with recasting
  • Typically performed at age 2.5-4 years (when child begins walking consistently)

3. Operative Treatment

Reserved for resistant/refractory clubfeet (~5% of idiopathic cases) or older children at presentation.

Posteromedial Release (PMR) - Turco / Cincinnati approach

  • Tendon lengthening (Achilles, tibialis posterior, FHL, FDL) + capsulotomy (subtalar, tibiotalar, talonavicular joints) + realignment
  • Posterior tibial artery must be carefully protected (anterior tibial artery often absent)
  • Typically performed at 6-12 months
  • Now largely replaced by Ponseti method; reserved for true failures

For Older Children (3-10 years) with Residual Deformity:

  • Medial opening-wedge osteotomy or lateral column shortening (cuboid decancellation/cuneiform osteotomy) - for residual adductus
  • Calcaneal osteotomy - for residual varus/valgus

For Late-Presenting / Severe Neglected Clubfoot:

  • Triple arthrodesis (subtalar + talonavicular + calcaneocuboid) - for children presenting late (adolescence/adults)
    • Contraindicated in insensate feet (risk of ulceration from rigidity)
  • Talectomy - salvage for severely deformed rigid clubfoot in older child
    • Complete talus removal required (any remnant regrows and causes recurrence)
    • Achilles tendon may also need resection after talectomy
  • Ilizarov frame - for complex recurrent/neglected deformities

Complications

ComplicationCause
RecurrenceMost common; primarily from brace non-compliance
Rocker-bottom deformityDorsiflexion through midfoot before varus corrected
Flat-top talusAggressive dorsiflexion causing talar dome flattening
OvercorrectionExcessive lateral column procedures
Dorsal bunionAfter clubfoot surgery; from strong tibialis anterior + FHB/abductor hallucis imbalance
Vascular injuryPMR - anterior tibial artery often absent
Wound dehiscencePosterior skin under tension (especially in MMC clubfeet)
Avascular necrosis of talusExtensive surgical dissection

Special Populations

Myelomeningocele-associated Clubfoot

  • 30-50% of MMC children affected; deformity is more rigid, resembles arthrogryposis
  • Ponseti method still first-line but: 68% early relapse, 33% require surgical release, 10% no improvement
  • Surgery: radical posteromedial-lateral release via Cincinnati incision at 10-12 months
  • Active anterior tibial tendon should be tenotomized to prevent recurrent supination
  • Insensate feet are a contraindication to triple arthrodesis

Arthrogryposis

  • Very rigid; requires more aggressive casting and higher surgical rates

Recent Evidence (2024-2026)


Sources: Campbell's Operative Orthopaedics 15th Ed 2026 (Ch. 31 & 36); Miller's Review of Orthopaedics 9th Edition; Schwartz's Principles of Surgery 11th Ed

Acl reconstruction

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ACL Reconstruction

Background

Primary repair of mid-substance ACL tears routinely fails (myofibroblasts coat the torn ends, preventing primary healing). Reconstruction - replacing the ACL with a graft - is therefore the standard for patients who want to return to pivoting/cutting sports.
Goals of surgery:
  • Restore normal knee kinematics (anterior translation + rotation)
  • Return patient to full activity
  • Prevent secondary meniscal/chondral injury and progressive arthrosis

Indications for Reconstruction

  • Young, active patients wishing to return to pivoting/cutting sports
  • Acute ACL tear with concomitant repairable meniscal tear
  • Chronic ACL insufficiency with functional instability
  • First-time dislocation with osteochondral fracture (relative)
  • Pediatric patients (special considerations apply - physeal-sparing techniques)
Timing: Early reconstruction is preferred for return to sport and decreased risk of secondary injury. However, full ROM must be achieved before surgery to minimize arthrofibrosis risk - reconstruction in the acute inflammatory phase increases stiffness risk significantly.

Graft Selection

Biomechanical Strength Comparison (Ultimate Load to Failure)

GraftStrength (N)Stiffness (N/mm)
Native ACL2160242
Central 1/3 patellar tendon (BPTB)2977455
Quadruple hamstring (ST+G)4140807
Quadriceps tendon2353326
Native PCL1867-
  • Campbell's Operative Orthopaedics 15th Ed 2026, Table 56.2

1. Bone-Patellar Tendon-Bone (BPTB) Autograft

Advantages:
  • Bone-to-bone healing (fastest incorporation - ~6 weeks)
  • High strength and stiffness
  • Lower retear rate in young athletes
  • More likely to achieve: normal Lachman, normal pivot shift, KT-1000 side-to-side difference < 3 mm
Disadvantages:
  • Anterior knee pain (kneeling pain - "kneeler's pain")
  • Patellar tendinitis
  • Patellar fracture risk (especially if graft too deep)
  • Greater risk of extension loss
  • Higher patellofemoral pain
  • Some studies show higher arthritis risk at 5-7 years vs. hamstring

2. Four-Strand Hamstring (Semitendinosus ± Gracilis) Autograft

Advantages:
  • Highest biomechanical strength (quadrupled)
  • Less anterior knee pain/kneeling pain
  • Less extension loss
  • Lower patellofemoral crepitance
Disadvantages:
  • Graft creep/relaxation over time (more than ligamentous grafts)
  • Slower soft-tissue tunnel incorporation (2-3 weeks longer than bone plug)
  • Hamstring weakness (~20% loss of knee flexion/internal rotation strength)
  • Saphenous nerve branch injury risk
  • Must be ≥ 8 mm diameter - smaller grafts have significantly higher revision rates (0.82× lower revision risk per 0.5 mm increase from 7-9 mm)

3. Quadriceps Tendon Autograft

  • Growing in popularity, especially with bone plug from patella
  • Strength intermediate between BPTB and hamstring
  • Avoids anterior knee pain of BPTB while providing bone-to-bone fixation option
  • Patellar fracture risk similar to BPTB

4. Allograft

Advantages: Less donor morbidity, better cosmesis, shorter operative time, preserves extensor mechanism
Disadvantages:
  • Failure rates 2-5 times higher than autograft in young/active patients
  • Slower incorporation into bone tunnels
  • Not recommended in young patients (age < 34 years)
  • Acceptable in older/lower-demand patients (no difference in retear > age 34)
  • Infection risk: Clostridium, hepatitis, HIV (1:1.5-6 million)
  • Chemically processed/irradiated allografts have higher failure rates than fresh-frozen
Bottom line: BPTB and hamstring autografts produce equivalent long-term outcomes (laxity, ROM, subjective scores, OA rates at 10-17 years). BPTB may have slightly lower early retear; hamstring has less anterior knee pain.

Surgical Technique

Tunnel Placement - The Critical Variable

Femoral tunnel:
  • Anatomic reconstruction targets the center of the femoral ACL footprint (10- or 2-o'clock position, or "low and deep" on the posterior condyle)
  • Achieved via independent femoral drilling (outside-in or accessory medial portal) rather than transtibial technique
  • More horizontal graft position reduces rotational instability
  • Single-bundle vs. double-bundle: currently no difference in patient-reported outcomes
Tibial tunnel:
  • Aim: center of the tibial ACL footprint (lateral to PCL, anterior to posterior horn of lateral meniscus)
  • Angle: typically 55-65° to the tibial plateau

Fixation Methods

  • Bone-to-bone (BPTB): Interference screws - provide excellent fixation if properly placed
  • Soft-tissue grafts (hamstring): Interference screws (BioScrew) + tunnel compaction + secondary fixation (cortical buttons, cross-pins)
  • Bone plug incorporation: ~6 weeks; soft-tissue graft incorporation: 8-12 weeks
  • Tunnel widening: aperture widening occurs up to 6 weeks, then narrows by 12 weeks

Anterolateral Ligament (ALL) Reconstruction - Augmentation

  • Indicated for high-risk patients: 3+ pivot shift, hyperextension > 5°, high-demand athletes
  • ACL + ALL reconstruction reduces failure rate to < 3%, which is:
    • 2.5× lower than isolated BPTB
    • 3.1× lower than isolated hamstring reconstruction
  • Medial meniscal repair failure also 2× lower with combined reconstruction
  • Technique: gracilis autograft fixed 8 mm proximal and 4.3 mm posterior to lateral epicondyle (femur), and 22 mm posterior to Gerdy's tubercle (tibia)

Physeal-Sparing Techniques (Pediatric)

  • All-epiphyseal or combined physeal-sparing techniques to avoid growth disturbance
  • Soft-tissue fixation preferred over bone plugs crossing physis
  • MQTFL/MPFL-type reconstruction approaches used in some cases

Complications

Tunnel Malposition (Most Common Technical Error)

ACL Reconstruction Complications: femoral tunnel malposition, tibial tunnel malposition, interference screw divergence, vertical femoral tunnel, femoral tunnel blowout
Common complications of ACL reconstruction - Miller's Review of Orthopaedics 9th Ed
ErrorConsequence
Femoral tunnel too anteriorTight in flexion → flexion loss
Femoral tunnel too posteriorLax in extension → instability
Tibial tunnel too anteriorRoof impingement with extension, tight in flexion
Tibial tunnel too posteriorPCL impingement
Vertical graftAP stability preserved but rotationally unstable (pivot shift not corrected)
Femoral tunnel blowoutRisk at flexion < 70° if drilling with knee insufficiently flexed

Interference Screw Errors

  • Femoral divergence > 30°: reduced pullout strength
  • Tibial divergence > 15°: reduced pullout strength

Other Complications

ComplicationKey Points
ArthrofibrosisMost common overall complication; risk minimized by achieving full ROM before surgery; avoid reconstruction in acute phase
Graft failure/retearTechnical (tunnel malposition, vertical graft), biological (poor incorporation), patient factors (young age, early return to sport)
InfectionIrrigation + debridement with graft retention successful in ~85% of cases
Patellar fractureBPTB harvest; avoided with careful technique
Saphenous nerve injuryHamstring harvest
Donor site painBPTB: anterior knee/kneeling pain; hamstring: flexion weakness
OA progressionRadiographic OA appears in > 50% of patients 10-20 years post-reconstruction; ACL reconstruction does NOT reduce OA risk

Rehabilitation

Key Principles

PhaseFocus
ImmediateEarly knee extension (heel elevated), immediate partial weight bearing with crutches, cold therapy
Early (0-6 weeks)Full ROM - emphasis on extension; avoid open chain extension (OKC) which stresses graft especially near full extension
StrengtheningClosed chain exercises preferred (lower patellofemoral pain, better KT-1000 scores, higher satisfaction); hamstring-dominant exercises early
ProprioceptionBegin within first 2 weeks; combine with strength training
BracingPostoperative bracing NOT proven beneficial except in downhill skiers

Return to Sport Criteria

  • Minimum 6 months after surgery (animal studies suggest 6-12 months for graft maturation)
  • 80% quadriceps strength symmetry vs. contralateral limb
  • Early return to high-level sport is a significant risk factor for ACL reconstruction failure
  • Prehabilitation (pre-operative rehab) improves short- and long-term outcomes

Blood Flow Restriction (BFR) Training

  • Air tourniquet proximally during therapy
  • Low-intensity exercise with vascular occlusion promotes muscle hypertrophy
  • Attenuates quadriceps atrophy post-operatively

Exercise Guidance

  • Avoid: Open kinetic chain (OKC) extension in last degrees of extension, especially in first 6 weeks
  • Prefer: Closed kinetic chain (CKC) exercises (squats, leg press, cycling)
  • CPM machines: not routinely recommended (no proven benefit)
  • No difference between accelerated and standard rehabilitation programs

Results

MetricData
Stability restoration90-95% achieve KT-1000 side-to-side difference ≤ 3 mm
OA at 10-20 years> 50% develop radiographic OA (reconstruction does not prevent this)
Young ageIndependent risk factor for reconstruction failure
Pivot shift preop 3+Significantly increases failure rate

Revision ACL Reconstruction

Causes of graft failure to consider:
  1. Technical: tunnel malposition (most common), vertical graft placement, poor fixation
  2. Missed concomitant injuries: posterolateral corner, MCL, ALL
  3. Alignment: varus alignment or excessive posterior tibial slope increases graft stress
  4. Biological: poor graft incorporation
  5. Patient factors: young age, early return to sport, hyperextension laxity > 5°
Workup: prior operative notes, CT for tunnel assessment, full-length alignment films.

Recent Evidence (2023-2026)


Sources: Campbell's Operative Orthopaedics 15th Ed 2026 (Ch. 50 & 56); Miller's Review of Orthopaedics 9th Edition; Rockwood and Green's Fractures 10th Ed 2025
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