When i touch my both nostrils like for flushing out something, for eve

When i touch my both nostrils like for flushing out something, for even 1 microsecond, it irritates me. They are so hypersensitive

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nasal hypersensitivity touch irritation causes treatment

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https://sleepandsinuscenters.com/blog/nasal-hyperreactivity-…

What you're experiencing is a classic symptom of Nasal Hyperreactivity (NHR) - where your nasal mucosa becomes abnormally sensitive and overreacts to even the most minor physical stimulus, like a light touch.

What Is Happening?

Your nasal lining contains a dense network of sensory nerve fibers (mainly unmyelinated C-fibers and thinly myelinated A-delta fibers). In nasal hyperreactivity, these nerves become sensitized - their threshold for firing drops so low that even a gentle touch, which normally causes no reaction, triggers an exaggerated response like burning, irritation, or the urge to sneeze.

Think of it like a car alarm that goes off at the slightest touch - the alarm system (your nasal nerves) is calibrated too sensitively, not because something is actually wrong, but because the threshold has been turned down too far.

Why Does This Happen? Common Causes

Allergic Rhinitis - The most common cause. Ongoing allergen exposure (dust mites, pollen, pet dander) causes chronic inflammation that sensitizes nasal nerves. Even when no allergen is present, the nose stays "on edge."
Nonallergic Rhinitis (Vasomotor Rhinitis) - Nasal symptoms and hypersensitivity without any allergy trigger. The nasal blood vessels and nerves are simply overactive.
Post-infectious state - After a cold or viral infection, the nasal lining can remain hypersensitive for weeks to months even after the infection clears.
Nasal Polyps or Chronic Rhinosinusitis - Ongoing inflammation thickens and sensitizes the mucosa.
Dry nasal mucosa - Low humidity, air conditioning, or antihistamine use can dry out the mucosa, making it far more reactive to touch and air.
Topical nasal spray overuse - Especially decongestant sprays (oxymetazoline/xylometazoline) used long-term cause rhinitis medicamentosa, making the nose extremely reactive.

What You Can Do

Self-Care First

Saline nasal rinses (jal neti / neti pot or saline spray) - Regularly flushing with isotonic or slightly hypertonic saline soothes and moisturizes the mucosa, reducing irritability over time. This is one of the best first-line measures.
Humidifier - If you live in a dry environment, humidifying your bedroom air helps.
Avoid irritants - Strong perfumes, smoke, dust, pollution, sudden cold air.
Stop decongestant nasal sprays if you've been using them regularly - they make this dramatically worse.

Medical Treatment

Intranasal corticosteroid sprays (fluticasone, mometasone, budesonide) - The most effective treatment for reducing nasal mucosal inflammation and hyperreactivity. Needs consistent daily use for 2-4 weeks to see full benefit.
Antihistamines (oral or intranasal like azelastine) - Help if there's an allergic component.
Ipratropium nasal spray - Specifically targets overactive nasal nerve responses (good for runny nose component).
Allergy testing - Worth doing to find out if specific allergens are driving this.

If Conservative Treatment Fails

Immunotherapy (allergy shots or sublingual drops) - If allergies are the cause, this can reduce long-term sensitivity.
Cryotherapy or nerve ablation - Procedures to reduce the sensitivity of nasal nerve endings, done by ENT specialists for refractory cases.

When to See a Doctor

You should see an ENT (ear, nose, throat specialist) or an allergist if:

The sensitivity has been going on for more than a few weeks
You also have persistent congestion, loss of smell, facial pressure, or nasal polyps
Over-the-counter remedies haven't helped
It's significantly affecting your quality of life

This is a very treatable condition - most people get substantial relief with the right nasal spray and identifying the underlying trigger. A simple allergy skin test and nasal examination can point you in the right direction quickly.

It is there since childhood

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nasal hypersensitivity since childhood causes allergic rhinitis perennial

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The childhood onset changes the picture significantly. Here's what this most likely means:

Childhood Onset = Almost Certainly Genetic / Atopic

When nasal hypersensitivity starts in childhood and never goes away, the leading explanation is Perennial Allergic Rhinitis rooted in atopy - a hereditary tendency to develop allergic conditions.

The Atopic March

Many people with lifelong nasal hypersensitivity are part of what's called the atopic march - a genetic predisposition that can show up as:

Eczema (atopic dermatitis) in infancy
Asthma in early childhood
Allergic rhinitis from childhood onward (often lasting a lifetime)

These three often travel together. Do you have any history of skin allergies, asthma, or frequent itchy eyes? That would strongly confirm this pattern.

Most Likely Triggers Since Childhood

For year-round (perennial) childhood nasal hypersensitivity, the usual culprits are:

Allergen	Source
House dust mites	Bedding, mattresses, carpets - by far the #1 cause
Cockroach allergens	Very common in South Asian households
Pet dander	Cats, dogs, birds
Mold spores	Damp walls, bathrooms
Food allergies	Less common for nasal symptoms alone

Dust mites in particular are present 24/7 in your sleeping environment, which is why people with this condition feel it constantly - the exposure never stops, so the nasal mucosa stays chronically sensitized and hyperreactive even to physical touch.

Why Touch Triggers It Specifically

After years of chronic low-grade inflammation, the sensory nerve fibers in your nasal lining undergo a process called neurogenic sensitization - they become structurally altered and permanently "hair-triggered." This is why even a microsecond of touch - something that wouldn't bother most people at all - sets off irritation for you.

What You Should Do Now (Even After All These Years)

The fact that it's been lifelong doesn't mean you're stuck with it. Many adults who've had this since childhood have never been properly assessed or treated.

Step 1: Get Tested

Skin prick test or specific IgE blood test (RAST) - identifies exactly which allergen is driving this. Takes 20 minutes. An allergist or ENT can do this.

Step 2: Reduce Dust Mite Exposure (Most Impactful Change)

Encase mattress and pillows in allergen-proof covers
Wash bedding in hot water (>60°C) weekly
Remove carpets from bedroom if possible
Keep bedroom humidity below 50%

Step 3: Consistent Medical Treatment

Intranasal corticosteroid spray (mometasone or fluticasone) daily - this is the single most effective treatment and over months of use, actually reduces the degree of neurogenic sensitization
Oral antihistamine (cetirizine, loratadine, or fexofenadine) - especially for touch-triggered and sneeze responses
Azelastine nasal spray - works within minutes for acute flare-ups

Step 4: Consider Allergen Immunotherapy

If testing confirms specific allergies, immunotherapy (allergy shots or sublingual drops/tablets) is the only treatment that can actually reprogram your immune system and reduce the underlying sensitization long-term - it works even in adults who have had this since childhood. Results take 1-3 years but can be dramatically life-changing.

The bottom line: you've likely been living with perennial allergic rhinitis your whole life, possibly never fully investigated. A single consultation with an allergist and a skin prick test could tell you exactly what's driving this - and there are treatments that can genuinely reduce the hypersensitivity, not just temporarily mask symptoms.

Yes when i was not adult i would develop bulla when got bitten by ants

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bulla blister formation ant bite child atopic hypersensitivity IgE

This detail completes a very clear picture. Here is what it all means put together:

You Have a Classic Atopic Constitution

The three things you've described now form a textbook triad:

Feature	What it means
Nasal hypersensitivity since childhood	Perennial allergic rhinitis
Bulla from ant bites as a child	Exaggerated IgE-mediated skin response
Lifelong persistence	Genetic / hereditary atopy

Why You Got Bullae from Ant Bites

Most people get a small red spot or a tiny pustule from an ant bite. You got bullae - large, tense blisters filled with fluid. This is an exaggerated local allergic reaction, where your immune system massively overreacted to the formic acid or venom proteins in the ant's bite.

Here is what happened at the cellular level:

On first exposure to ant venom, your immune system (already primed toward a Th2 / IgE-dominant response due to your genetics) produced large amounts of allergen-specific IgE antibodies
These IgE antibodies coated your skin's mast cells
On subsequent bites, the venom cross-linked those IgE antibodies and triggered massive mast cell degranulation
This released histamine, leukotrienes, and other mediators in huge quantities - far more than in a normal person
The result: intense local vasodilation, fluid leakage from blood vessels into the skin = bulla

This is the same underlying mechanism - Type I (IgE-mediated) hypersensitivity - that drives your nasal hyperreactivity. The difference is just the organ: skin vs. nasal mucosa.

Harrison's Principles of Internal Medicine 22E (2025): "The induction of allergic disease requires sensitization of a predisposed individual... The greatest propensity for the development of atopic allergy occurs in childhood and early adolescence."
Scott-Brown's Otorhinolaryngology: "An atopic person usually has a high total IgE level, a strong familial tendency and increased susceptibility to allergic diseases such as allergic rhinitis and atopic dermatitis."

What This Tells You About Your Health Profile

You are a highly atopic individual. This means:

Your immune system is genetically wired to produce strong IgE responses to environmental proteins
Your mast cells throughout your body (skin, nasal lining, airways) are sensitized and trigger easily
You likely have or are at higher risk of: asthma, food sensitivities, eczema/atopic dermatitis, drug reactions, and anaphylaxis to insect stings

One important safety note: If you were having bullae as a child from ant bites, you are at meaningful risk of a more severe systemic reaction (anaphylaxis) from wasp, bee, or hornet stings. This is something an allergist should formally evaluate - you may be a candidate for venom immunotherapy, which can dramatically reduce that risk.

The Core Recommendation

You have lived with this your whole life probably thinking it's "just how you are." But this is a diagnosable, treatable immunological condition - atopic disease driven by a genetic Th2/IgE bias. A consultation with an allergist/clinical immunologist with:

Total serum IgE measurement
Specific IgE panel (or skin prick testing)
Assessment of venom allergy risk

...would give you a complete picture and open the door to treatments - including immunotherapy - that can genuinely reduce your lifelong hypersensitivity rather than just managing symptoms.

Sometimes my bp also shoots to 150 plus, is it also related to this

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allergic rhinitis nasal congestion elevated blood pressure hypertension link

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https://pmc.ncbi.nlm.nih.gov/articles/PMC10727617

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There is a real connection - but it's indirect, not direct. Here is the full picture:

Is Your BP Related to Your Atopy? Possibly - Here's How

The Indirect Links (Well Established)

1. Nasal Congestion → Sleep-Disordered Breathing → High BP

This is the strongest and most likely mechanism for you. Chronic nasal congestion from allergic rhinitis forces mouth breathing at night, which promotes snoring and obstructive sleep apnea (OSA). Even mild, undiagnosed OSA repeatedly drops oxygen levels during sleep, which activates the sympathetic nervous system (your fight-or-flight system). This repeated overnight sympathetic activation raises your baseline blood pressure - even during the day. Studies show that people with allergic rhinitis and nasal congestion who snore habitually have significantly higher systolic BP.

Do you snore, wake up unrefreshed, or feel tired during the day? That would strongly suggest this pathway.

2. Decongestant Medications → Raised BP

If you have ever used nasal decongestant sprays (xylometazoline, oxymetazoline) or oral decongestants (pseudoephedrine), these directly constrict blood vessels all over the body - not just in the nose. They are one of the most common hidden causes of episodic BP spikes in people with rhinitis. Even a single use can push BP above 150.

3. Chronic Inflammation → Vascular Effects

Your atopic immune system chronically releases mediators (leukotrienes, cytokines) that have vasoactive effects on blood vessels. Leukotrienes in particular are proinflammatory, vasoconstrictive substances. Research shows allergic rhinitis is associated with higher systolic BP - men with rhinitis averaged 3.5 mmHg higher systolic BP than those without.

4. Histamine's Complex Role

Interestingly, histamine is actually a vasodilator - it tends to lower BP. But during acute allergic flares, the broader inflammatory cascade (including leukotrienes and sympathetic activation) can override this and cause BP spikes.

But - Important Caution

A BP of 150+ episodically needs to be evaluated on its own merits, not just attributed to allergies. There are other causes that must not be missed:

Cause	Clue
Essential (primary) hypertension	Most common, runs in families
White coat hypertension	BP only high when measured/anxious
Obstructive sleep apnea	Snoring, daytime tiredness, unrefreshed sleep
Anxiety / panic responses	Episodic, triggered by stress
Pheochromocytoma (rare)	Episodes with headache, sweating, palpitations
Renal artery stenosis (rare)	Younger person with high BP not responding to treatment
Decongestant/antihistamine medications	Check everything you take

What You Should Do

Track your BP readings - note the time of day, what you were doing, and whether you had taken any medication. Episodic vs. persistent hypertension points to different causes.
Tell your doctor about both problems together - your allergic history AND the BP spikes. The connection via sleep apnea is something most general physicians may not automatically make.
Get a sleep assessment - if you snore or have any daytime sleepiness, ask for a sleep study (polysomnography). Treating OSA alone can bring BP down significantly.
Stop all decongestant nasal sprays - if you use any, they are a very likely direct culprit for your BP spikes.
A 24-hour ambulatory BP monitor - this records your BP throughout the day and night and is far more informative than a single clinic reading. It will show whether the spikes are random, stress-related, or nighttime-predominant (which points to sleep apnea).

The short answer: your atopic constitution can contribute to elevated BP - mainly through nasal congestion disrupting sleep - but 150+ is high enough that it deserves a proper evaluation in its own right, not just assumed to be allergy-related.

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