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Here is Urinary Incontinence explained simply, directly from Bailey and Love's Short Practice of Surgery, 28th Edition:
Urinary Incontinence - Bailey and Love
What Is It?
Urinary incontinence (UI) is simply the involuntary (unintentional) leakage of urine. It affects 25-45% of men and women and becomes more common with age. It has a significant impact on quality of life.
Types (Classification)
| Type | What Happens | Easy Memory |
|---|
| Stress UI (SUI) | Leakage on effort - coughing, sneezing, laughing, exercise | "Stress = physical stress on bladder" |
| Urgency UI (UUI) | Leakage with a sudden, strong urge to urinate that's hard to hold | "Gotta go NOW" |
| Mixed UI (MUI) | Both SUI and UUI together | "Mix of both" |
| Overflow / Chronic retention | Bladder doesn't empty fully; urine leaks out when full | "Cup overflowing" |
| Continuous UI | Constant leaking - suspect fistula or ectopic ureter | "Non-stop drip" |
| Nocturnal enuresis | Leakage during sleep (bedwetting) | "Night time leak" |
| Functional UI | Can't reach the toilet in time due to mobility/cognitive problems, but the bladder itself is normal | "Problem getting there, not the bladder" |
Overactive Bladder (OAB) = urgency + frequency + nocturia, with or without leakage. Can be neurogenic or idiopathic.
How Continence Normally Works (Pathogenesis)
Staying dry depends on 4 things:
- Normal bladder compliance - bladder expands without pressure rising (thanks to its elastic, low-collagen wall)
- Intact urethral sphincter - keeps the outlet closed
- Pelvic floor support - a "hammock" of fascia and muscles (pubocervical fascia + levator ani) that holds the bladder neck in place
- Mucosal seal of the urethra - the soft tissue lining that creates a watertight closure
Why SUI happens:
- The pelvic floor "hammock" becomes lax (from childbirth, ageing, menopause)
- The urethra drops out of its normal position
- During coughing/sneezing, raised intra-abdominal pressure is NOT transmitted to the urethra - so it can't resist the pressure, and urine leaks
- Intrinsic sphincter deficiency (ISD) - the sphincter muscle itself is weak (from previous surgery, radiation, nerve damage, oestrogen loss)
- Most women with SUI have BOTH laxity and ISD to varying degrees
Why UUI/OAB happens:
- Detrusor (bladder muscle) overactivity - the muscle contracts before the bladder is full
- Reduced bladder compliance from fibrosis, neurological causes, or chronic obstruction
Risk Factors
Predisposing (structural):
- Family history
- Ectopic ureter, urinary tract fistulae, urethral diverticulum
- Neurological conditions - spina bifida, spinal cord injury, Parkinson's, stroke, multiple sclerosis
- Pregnancy and childbirth (vaginal delivery)
- Pelvic surgery or radiotherapy
- Chronic bladder inflammation causing fibrosis (TB cystitis, ketamine cystitis, interstitial cystitis)
Exacerbating (making it worse):
- Age
- Obesity
- Raised intra-abdominal pressure (chronic cough, constipation, heavy lifting)
- Menopause (oestrogen loss weakens urethral mucosa)
- Loop diuretics, certain medications
- Urinary tract infection (UTI)
- Diabetes mellitus
- Immobility
In men, the most common cause of SUI is radical prostatectomy for prostate cancer.
Symptoms to Ask About
Storage symptoms:
- Frequency (>8 times/day)
- Urgency
- Nocturia (>1 void/night)
- Leakage with exertion/coughing
Voiding symptoms:
- Hesitancy, slow stream, incomplete emptying, straining
Post-micturition:
- Post-void dribble, feeling of incomplete emptying
Also ask about: haematuria (in women >40 years, must rule out malignancy)
Investigations
| Investigation | Purpose |
|---|
| Bladder diary + symptom/QOL questionnaire | Baseline - how often, how much, triggers |
| Urine dipstick + MSU | Rule out UTI, haematuria |
| Digital examination | Pelvic floor tone, prolapse, masses |
| Post-void residual (PVR) ultrasound | Is bladder emptying properly? |
| Urodynamics | For MUI, voiding dysfunction, neurological causes, treatment failure, or recurrence after surgery |
| KUB ultrasound | For recurrent UTI or haematuria |
| Cystoscopy | If bladder pathology suspected |
Management
Treatment is tailored to the type of incontinence and is stepped:
OAB / Urgency UI
| Step | Treatment |
|---|
| Conservative | Limit fluids (avoid caffeine), bladder training (bladder drill) for ≥6 weeks, pelvic floor exercises (8 contractions x 3/day for ≥3 months), review medications |
| Medical | Anticholinergics: oxybutynin, tolterodine (side effects: dry mouth, constipation - avoid oxybutynin in elderly due to cognitive risk); β3-agonist: mirabegron; Desmopressin for nocturia |
| Surgical | Intravesical botulinum toxin A; Neuromodulation (tibial nerve or sacral nerve stimulation); Augmentation cystoplasty (risks: bowel disturbance, metabolic acidosis, mucus retention, UTI, malignancy); Urinary diversion (last resort) |
Stress UI (SUI)
| Step | Treatment |
|---|
| Conservative | Pelvic floor exercises (≥8 contractions x 3/day for ≥3 months), manage chronic cough, ISC if poor emptying |
| Medical | Duloxetine (SNRI) - only when conservative failed AND surgery contraindicated/declined |
| Surgical | Colposuspension (bladder neck suspension); Autologous rectus fascial sling; Retropubic mid-urethral mesh sling; Periurethral bulking agents; Artificial urinary sphincter |
Do NOT offer: anterior colporrhaphy, needle suspension, paravaginal defect repair, porcine dermis sling, or Marshall-Marchetti-Krantz procedure for SUI.
Summary Box (Bailey and Love Key Points)
- UI should be classified as UUI, SUI, or MUI
- Initial management for ALL types = behavioural modification + bladder training + pelvic floor muscle training (PFMT)
- OAB: first-line medical = anticholinergics; if failed = botulinum toxin A
- For "end-stage" incontinence failing all measures: urinary diversion (ileal conduit) as last resort
Quick Memory Trick
Types: S-U-M-C-N-O-F
Stress - Urgency - Mixed - Continuous - Nocturnal - Overflow - Functional
Management (all types start with): Pelvic Floor + Behaviour
Then add medical → then surgical
- Bailey and Love's Short Practice of Surgery, 28th Edition (Chapter 83 & 87)