Spasticity Management: Physiotherapy
What is Spasticity?
Spasticity is a velocity-dependent increase in muscle tone resulting from upper motor neuron (UMN) lesions (stroke, spinal cord injury, multiple sclerosis, cerebral palsy, TBI). It is a component of the upper motor neuron syndrome and manifests as increased resistance to passive movement, clonus, spasms, and exaggerated deep tendon reflexes.
Assessment Tools
| Scale | Description |
|---|
| Modified Ashworth Scale (MAS) | 0–4 scale grading resistance to passive movement; most widely used in clinical practice |
| Tardieu Scale | Measures spasticity at two speeds; more specific for velocity-dependent component |
| Penn Spasm Frequency Scale | Quantifies frequency of spasms |
| Functional independence / ADL scales | FIM, Barthel Index — contextualise functional impact |
The Tardieu Scale is considered more specific than the MAS for true neurological spasticity (differentiating contracture from spasticity).
Physiotherapy Interventions
1. Stretching
- Sustained/prolonged passive stretching is the cornerstone of PT for spasticity
- Reduces muscle stiffness, maintains range of motion (ROM), and may modulate reflex excitability
- Should be performed daily — brief stretches (30 seconds) have limited benefit; prolonged holds (20–30 minutes) show better outcomes
- Both manual stretching and self-stretching programmes are used
2. Positioning and Postural Management
- Correct positioning reduces abnormal reflex patterns (e.g., inhibitory positioning in stroke)
- 24-hour postural management programmes (supine, sitting, standing) are recommended, especially in severe spasticity
- Tilt-table standing has evidence for reducing lower limb spasticity in SCI and stroke
3. Splinting and Orthotics
- Static splints (resting hand splints, ankle-foot orthoses [AFOs]): maintain joint position and prevent contracture
- Dynamic/serial splinting: progressively increases ROM over time
- AFOs are particularly important in managing equinovarus foot deformity post-stroke
- Serial casting: used in fixed or developing contractures — evidence supports short-term ROM gains
4. Exercise and Active Movement
- Active movement and task-specific training may reduce spasticity via reciprocal inhibition and normalization of supraspinal drive
- Strengthening the antagonist muscle can help modulate spastic agonist tone
- Aquatic/hydrotherapy: warm water reduces muscle tone and allows easier active movement
- Constraint-Induced Movement Therapy (CIMT): forces use of the affected limb in upper limb spasticity post-stroke
5. Neurophysiological Approaches
- Bobath/Neurodevelopmental Treatment (NDT): uses handling techniques to inhibit spastic patterns and facilitate normal movement; widely used though evidence base is debated
- Proprioceptive Neuromuscular Facilitation (PNF): techniques such as contract-relax and hold-relax used to reduce tone and improve ROM
6. Electrical Stimulation
- Transcutaneous Electrical Nerve Stimulation (TENS): applied to spastic muscles or antagonists; evidence for short-term tone reduction
- Functional Electrical Stimulation (FES): stimulates antagonist muscles to reduce agonist spasticity through reciprocal inhibition; also improves motor function
- Neuromuscular Electrical Stimulation (NMES): evidence supports reduction in spasticity in upper limb post-stroke
7. Extracorporeal Shock Wave Therapy (ESWT)
- Evidence shows statistically significant improvement in MAS scores at short-term (1–2 weeks), mid-term (3–4 weeks), and long-term (up to 12 weeks) follow-up compared with sham or conventional physiotherapy
- However, the mean differences in MAS scores were not considered clinically significant (MD: −0.43 to −0.81), per evidence reviewed in stroke rehabilitation guidelines (Management of Stroke Rehabilitation, p. 73)
- May be used as an adjunct, particularly for focal upper and lower limb spasticity
8. Vibration Therapy
- Focal muscle vibration (FMV) and whole-body vibration (WBV): applied to spastic muscles, may reduce tonic vibration reflex and transiently decrease tone
- Short-term effects are documented; long-term benefit requires further study
9. Trigger Avoidance and Environmental Modification
- Identifying and removing spasticity triggers is critical:
- Urinary tract infections
- Pressure sores / skin breakdown
- Constipation/fecal impaction
- Pain, tight clothing, bladder distension
- Posture and temperature changes
- Addressing triggers often results in immediate reduction in spasticity (Harrison's, p. 12626)
Integration with Pharmacological Management
Physiotherapy works synergistically with medical management. PT is typically combined with:
| Drug | Dose Range | Notes |
|---|
| Baclofen | 20–120 mg/day | First-line oral agent; GABA-B agonist |
| Tizanidine | 8–32 mg/day | Alpha-2 agonist; useful for nocturnal spasms |
| Diazepam | 2–40 mg/day | Sedating; second-line |
| Dantrolene | 25–400 mg/day | Acts peripherally on muscle |
| Botulinum toxin A | Focal injection | Best combined with intensive PT; allows window of opportunity for stretching and retraining |
| Intrathecal baclofen (ITB) | Pump | Severe generalized spasticity; PT critical for functional gains post-implant |
Botulinum toxin injections are particularly important in the PT context: the 2–3 month window of reduced tone following injection should be actively used for intensive stretching, serial casting, and motor relearning.
Goal-Setting in Physiotherapy
Goals should be functional and patient-centred, not purely tone-reduction focused:
- Improving ease of caregiving (passive ROM for hygiene, dressing)
- Reducing pain from spasms
- Improving gait pattern and walking speed
- Facilitating active use of limb
- Preventing contracture and skin complications
Use of Goal Attainment Scaling (GAS) is recommended alongside standard scales to capture meaningful functional outcomes.
Summary: PT Intervention Pyramid
┌──────────────────────┐
│ FES / ESWT / Vibra- │ Adjunct modalities
│ tion / ESWT │
├──────────────────────┤
│ Splinting / Casting │ Prevent contracture
├──────────────────────┤
│ Exercise / Active │ Motor relearning
│ Movement / CIMT │
├──────────────────────┤
│ Positioning / │ 24-hr management
│ Postural mgmt │
├──────────────────────┤
│ Stretching │ Daily cornerstone
└──────────────────────┘
Key principle: Spasticity is not always harmful — it may assist standing or walking in some patients. PT goals should target functionally problematic spasticity rather than tone reduction for its own sake.