Diagnosis and treatment for hyperhidrosis

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hyperhidrosis diagnosis treatment International Hyperhidrosis Society clinical guidelines

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https://www.sweathelp.org/about-hyperhidrosis/diagnosis-guid…

Hyperhidrosis is sweating beyond what is needed for temperature control. It may be primary focal or secondary to a medication or illness.

Diagnosis

1. Decide whether it is primary or secondary

Primary focal hyperhidrosis typically:

Begins in childhood or adolescence
Affects both sides similarly, most often the palms, soles, underarms, face/scalp
Occurs at least weekly and disrupts daily activities
Often has a family history
Stops during sleep
May be triggered by emotion, heat, or exercise

A commonly used clinical definition is focal visible excessive sweating for at least 6 months without an apparent cause, plus at least two features above. Dermatology 2-Volume Set, 5e, lines 703-716.

Secondary hyperhidrosis is more likely when sweating:

Is new in adulthood, generalized, asymmetric, or occurs at night
Coincides with fever, weight loss, palpitations, tremor, flushing, pain, or other systemic symptoms
Started after a new drug or substance

Potential causes include fever/infection, hyperthyroidism, hypoglycemia or diabetes, menopause, pregnancy, lymphoma, Parkinson disease, pheochromocytoma, alcohol/substance withdrawal, and medications. Andrews' Diseases of the Skin, lines 213-219.

2. Clinical assessment

A clinician should ask about:

Distribution and duration of sweating, triggers, and sleep sweating
Family history
Medication and substance use, including antidepressants, opioids, hypoglycemic medicines, and others
Symptoms suggesting endocrine disease, infection, malignancy, neurologic disease, or menopause

Examination targets the involved areas and signs of systemic disease. Testing is not routinely required for classic primary focal hyperhidrosis, but may include glucose testing, thyroid tests, CBC, and other targeted investigations if secondary causes are suspected. The International Hyperhidrosis Society diagnostic guidance emphasizes ruling out secondary causes first.

Severity can be tracked using the Hyperhidrosis Disease Severity Scale:

1-2: tolerable or only occasionally interferes
3-4: barely tolerable or intolerable, frequently/constantly interferes

Treatment

Treatment is selected by body site, severity, prior response, side effects, and patient preference.

First-line measures

Use a clinical-strength antiperspirant, usually aluminum chloride or aluminum chloride hexahydrate.
Apply to completely dry skin at night, wash off in the morning; use nightly initially, then reduce to maintenance frequency once controlled.
Skin irritation is common. Apply only to intact, dry skin and discuss a low-potency topical steroid briefly with a clinician if dermatitis occurs.
Wear breathable fabrics, change socks/clothes as needed, and use absorbent shoe inserts for plantar sweating.

Textbook first-line topical options include over-the-counter antiperspirants and aluminum chloride hexahydrate 10%-35%. Fitzpatrick's Dermatology, lines 2864-2868.

Site-specific escalation

Site	Usual next options
Underarms	Prescription topical anticholinergic, botulinum toxin injections, microwave thermolysis in suitable patients
Palms/soles	Tap-water iontophoresis, then botulinum toxin if needed
Face/scalp	Carefully selected topical anticholinergic or botulinum toxin by an experienced clinician
Multiple sites/generalized symptoms	Treat underlying cause if present; consider oral medication under medical supervision

Iontophoresis: Particularly effective for palms and soles. Hands or feet are placed in shallow water trays while a mild electrical current is passed. Initial frequent sessions are followed by maintenance. It may be unsuitable with certain implanted electrical devices or during pregnancy, so confirm with the treating clinician.

Botulinum toxin injections: Effective for axillary and palmar sweating and can also be used for other focal sites. Benefits are temporary, so repeat treatment is required. Palmar injections can be uncomfortable and can cause temporary hand weakness. Goldman-Cecil Medicine, line 1354.

Topical anticholinergics: Options vary by country and include glycopyrronium or sofpironium preparations, particularly for axillary disease. Possible adverse effects include dry mouth, blurred vision, and pupil dilation if the medicine reaches the eyes. Wash hands after applying.

Oral anticholinergics: Glycopyrrolate or oxybutynin can help widespread or refractory focal sweating, but require clinician prescription and monitoring. Side effects can include dry mouth, constipation, urinary retention, blurred vision, fast heart rate, overheating, and cognitive effects. They may be inappropriate in some people, including those with narrow-angle glaucoma or urinary retention.

Procedures for severe refractory disease:

Axillary microwave thermolysis or local sweat-gland procedures in selected cases
Endoscopic thoracic sympathectomy for very severe, treatment-resistant palmar hyperhidrosis only. It can cause persistent compensatory sweating elsewhere, so it should be considered cautiously.

When to seek prompt medical review

Arrange a medical assessment promptly if sweating is new or rapidly worsening, mainly occurs at night, is generalized, or comes with:

Fever, unexplained weight loss, swollen lymph nodes
Chest pain, shortness of breath, fainting
Palpitations, tremor, or heat intolerance
Confusion, weakness, or symptoms of low blood sugar

For most people with the typical focal pattern, a dermatologist can confirm the diagnosis and build a stepwise treatment plan. Current treatment algorithms are available through the International Hyperhidrosis Society.

Recent evidence note: a 2025 systematic review found evidence supporting sofpironium for primary hyperhidrosis, primarily axillary disease (PMID: 39668771). Recent reviews also show that primary hyperhidrosis is associated with anxiety and depression, so screening for its quality-of-life impact is appropriate (PMIDs: 39351777, 42458641).

Full body hyperhidrosis

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generalized hyperhidrosis treatment

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generalized hyperhidrosis evaluation secondary causes management clinical guidance

Full-body sweating is generalized hyperhidrosis. Unlike sweating confined to palms, soles, or underarms, it is more often caused by a medication, hormonal change, or another medical condition, so it should be evaluated rather than treated only with antiperspirants.

Arrange a medical appointment soon

A clinician should review:

When it began and whether it happens during sleep
New medicines or dose changes, including antidepressants, opioids, steroids, diabetes medicines, alcohol/substance use, and caffeine
Fever, weight loss, cough, pain, palpitations, tremor, flushing, diarrhea, anxiety, or menopausal symptoms
Diabetes or low-blood-sugar episodes, thyroid disease, infection risks, and neurologic symptoms

Typical initial testing is guided by your history, but often includes blood glucose/HbA1c and thyroid-function tests. Additional tests may be needed for infection, malignancy, endocrine disorders, or medication effects. Causes include hyperthyroidism, diabetes/hypoglycemia, menopause, pregnancy, infections, lymphoma, Parkinson disease, endocrine tumors, and drugs. Andrews' Diseases of the Skin, generalized hyperhidrosis section, lines 213-219.

Get urgent care today if sweating comes with

Chest pain, shortness of breath, fainting, or confusion
Fever with severe illness
Shakiness, weakness, confusion, or loss of consciousness suggesting low blood sugar
Drenching night sweats plus unintentional weight loss, persistent fever, or enlarged lymph nodes

Treatment

Treat the cause if one is found. This is the main treatment for generalized hyperhidrosis, including adjusting a triggering medicine only with the prescriber.
Practical relief: breathable and moisture-wicking clothing, cool environment, hydration and electrolyte replacement if sweating is heavy, and antiperspirant on the most troublesome areas.
If no reversible cause is found and symptoms remain severe, a dermatologist or primary-care clinician may consider oral sweat-reducing medicines, usually anticholinergic drugs such as glycopyrrolate or oxybutynin. These can cause dry mouth, constipation, blurred vision, urinary retention, overheating, and sometimes cognitive side effects, so they need individual prescribing and follow-up.
Botox, iontophoresis, and topical treatments are more useful for specific focal areas than for whole-body sweating.

Textbook guidance notes that secondary generalized hyperhidrosis is commonly associated with systemic illness or drugs, and that antiperspirants are initial symptom treatment, while systemic medicines may be considered under supervision. Goldman-Cecil Medicine, Hyperhidrosis section, line 1354. The International Hyperhidrosis Society guidance similarly recommends identifying and treating the underlying cause first.

If you share your age, duration, whether you have night sweats, and any medicines you take, I can help identify the most likely causes to discuss with your clinician.

Whom should we consult

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generalized hyperhidrosis which specialist primary care dermatologist endocrinologist

Start with a general physician / primary-care doctor (internal medicine or family medicine). For full-body sweating, they should first look for a medication-related or medical cause and arrange targeted tests.

They may refer you to:

Dermatologist: if no underlying cause is found, or for treatment of persistent hyperhidrosis.
Endocrinologist: if there are signs or test results suggesting thyroid disease, diabetes or repeated low blood sugar, menopause-related issues, or another hormonal disorder.
Other specialists only if your history or test results point to infection, neurologic disease, or another specific cause.

Do not begin oral medicines for sweating on your own. Generalized hyperhidrosis is commonly secondary to drugs or systemic illness. Goldman-Cecil Medicine, Sweating Disorders, p. 1242. The International Hyperhidrosis Society likewise recommends primary care first, followed by dermatology when needed.

Keratosis Pilaris treatment

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keratosis pilaris treatment guidance urea lactic acid topical retinoid dermatology

Keratosis pilaris (KP) causes harmless rough, small follicular bumps, usually on the upper arms, thighs, buttocks, or cheeks. It cannot be permanently cured, but regular skin care usually improves roughness and redness.

Simple treatment routine

1. Moisturize daily

After a short lukewarm shower, pat skin partly dry and apply a cream within 5 minutes. Choose one product containing:

Urea 10%-20%
Lactic acid / ammonium lactate 5%-12%
Salicylic acid 1%-2%
Glycolic acid or another alpha-hydroxy acid

Use once daily initially. If your skin tolerates it, increase as directed on the product. Urea- and lactate-based creams are commonly recommended treatments. Textbook of Family Medicine, 9e, Keratosis Pilaris section.

2. Avoid irritation

Use a mild fragrance-free cleanser.
Keep showers short and lukewarm.
Do not scrub hard, pick the bumps, or use harsh exfoliating gloves or abrasive scrubs.
Use loose, soft clothing if friction worsens the bumps.

3. If it remains rough after 6-8 weeks

A dermatologist may prescribe a topical retinoid such as adapalene, tretinoin, or tazarotene. Start only a few nights per week because these products can cause dryness and irritation. Do not use prescription retinoids during pregnancy or when trying to conceive unless your prescribing clinician specifically advises it.

4. If it is itchy, red, or inflamed

A short course of a suitable topical steroid may be recommended by a clinician. Avoid using steroid creams regularly or long term without medical advice.

Expectations

Improvement is gradual, often over several weeks. Treatment must be continued intermittently because the bumps commonly return if treatment stops. The 2025 systematic review on KP treatment supports keratolytic topical treatments and recognizes that evidence for other approaches, including laser procedures, is more limited (PMID: 41277649). DermNet guidance also lists urea, lactic acid, salicylic acid, alpha-hydroxy acids, and topical retinoids as options.

See a dermatologist if it is painful, pus-filled, leaving scars, associated with major eczema, or there is no improvement after 2-3 months of consistent care.

Urea 10%-20% Lactic acid / ammonium lactate 5%-12% Salicylic acid 1%-2% Glycolic acid or another alpha-hydroxy acid List the products which have this formulation

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Hyperhidrosis is sweating beyond what is needed for temperature control. It may be primary focal or secondary to a medication or illness.

Diagnosis

1. Decide whether it is primary or secondary

Primary focal hyperhidrosis typically:

Begins in childhood or adolescence
Affects both sides similarly, most often the palms, soles, underarms, face/scalp
Occurs at least weekly and disrupts daily activities
Often has a family history
Stops during sleep
May be triggered by emotion, heat, or exercise

A commonly used clinical definition is focal visible excessive sweating for at least 6 months without an apparent cause, plus at least two features above. Dermatology 2-Volume Set, 5e, lines 703-716.

Secondary hyperhidrosis is more likely when sweating:

Is new in adulthood, generalized, asymmetric, or occurs at night
Coincides with fever, weight loss, palpitations, tremor, flushing, pain, or other systemic symptoms
Started after a new drug or substance

Potential causes include fever/infection, hyperthyroidism, hypoglycemia or diabetes, menopause, pregnancy, lymphoma, Parkinson disease, pheochromocytoma, alcohol/substance withdrawal, and medications. Andrews' Diseases of the Skin, lines 213-219.

2. Clinical assessment

A clinician should ask about:

Distribution and duration of sweating, triggers, and sleep sweating
Family history
Medication and substance use, including antidepressants, opioids, hypoglycemic medicines, and others
Symptoms suggesting endocrine disease, infection, malignancy, neurologic disease, or menopause

Examination targets the involved areas and signs of systemic disease. Testing is not routinely required for classic primary focal hyperhidrosis, but may include glucose testing, thyroid tests, CBC, and other targeted investigations if secondary causes are suspected. The International Hyperhidrosis Society diagnostic guidance emphasizes ruling out secondary causes first.

Severity can be tracked using the Hyperhidrosis Disease Severity Scale:

1-2: tolerable or only occasionally interferes
3-4: barely tolerable or intolerable, frequently/constantly interferes

Treatment

Treatment is selected by body site, severity, prior response, side effects, and patient preference.

First-line measures

Use a clinical-strength antiperspirant, usually aluminum chloride or aluminum chloride hexahydrate.
Apply to completely dry skin at night, wash off in the morning; use nightly initially, then reduce to maintenance frequency once controlled.
Skin irritation is common. Apply only to intact, dry skin and discuss a low-potency topical steroid briefly with a clinician if dermatitis occurs.
Wear breathable fabrics, change socks/clothes as needed, and use absorbent shoe inserts for plantar sweating.

Textbook first-line topical options include over-the-counter antiperspirants and aluminum chloride hexahydrate 10%-35%. Fitzpatrick's Dermatology, lines 2864-2868.

Site-specific escalation

Site	Usual next options
Underarms	Prescription topical anticholinergic, botulinum toxin injections, microwave thermolysis in suitable patients
Palms/soles	Tap-water iontophoresis, then botulinum toxin if needed
Face/scalp	Carefully selected topical anticholinergic or botulinum toxin by an experienced clinician
Multiple sites/generalized symptoms	Treat underlying cause if present; consider oral medication under medical supervision

Iontophoresis: Particularly effective for palms and soles. Hands or feet are placed in shallow water trays while a mild electrical current is passed. Initial frequent sessions are followed by maintenance. It may be unsuitable with certain implanted electrical devices or during pregnancy, so confirm with the treating clinician.

Botulinum toxin injections: Effective for axillary and palmar sweating and can also be used for other focal sites. Benefits are temporary, so repeat treatment is required. Palmar injections can be uncomfortable and can cause temporary hand weakness. Goldman-Cecil Medicine, line 1354.

Topical anticholinergics: Options vary by country and include glycopyrronium or sofpironium preparations, particularly for axillary disease. Possible adverse effects include dry mouth, blurred vision, and pupil dilation if the medicine reaches the eyes. Wash hands after applying.

Oral anticholinergics: Glycopyrrolate or oxybutynin can help widespread or refractory focal sweating, but require clinician prescription and monitoring. Side effects can include dry mouth, constipation, urinary retention, blurred vision, fast heart rate, overheating, and cognitive effects. They may be inappropriate in some people, including those with narrow-angle glaucoma or urinary retention.

Procedures for severe refractory disease:

Axillary microwave thermolysis or local sweat-gland procedures in selected cases
Endoscopic thoracic sympathectomy for very severe, treatment-resistant palmar hyperhidrosis only. It can cause persistent compensatory sweating elsewhere, so it should be considered cautiously.

When to seek prompt medical review

Arrange a medical assessment promptly if sweating is new or rapidly worsening, mainly occurs at night, is generalized, or comes with:

Fever, unexplained weight loss, swollen lymph nodes
Chest pain, shortness of breath, fainting
Palpitations, tremor, or heat intolerance
Confusion, weakness, or symptoms of low blood sugar

For most people with the typical focal pattern, a dermatologist can confirm the diagnosis and build a stepwise treatment plan. Current treatment algorithms are available through the International Hyperhidrosis Society.

Recent evidence note: a 2025 systematic review found evidence supporting sofpironium for primary hyperhidrosis, primarily axillary disease (PMID: 39668771). Recent reviews also show that primary hyperhidrosis is associated with anxiety and depression, so screening for its quality-of-life impact is appropriate (PMIDs: 39351777, 42458641).Full-body sweating is generalized hyperhidrosis. Unlike sweating confined to palms, soles, or underarms, it is more often caused by a medication, hormonal change, or another medical condition, so it should be evaluated rather than treated only with antiperspirants.

Arrange a medical appointment soon

A clinician should review:

When it began and whether it happens during sleep
New medicines or dose changes, including antidepressants, opioids, steroids, diabetes medicines, alcohol/substance use, and caffeine
Fever, weight loss, cough, pain, palpitations, tremor, flushing, diarrhea, anxiety, or menopausal symptoms
Diabetes or low-blood-sugar episodes, thyroid disease, infection risks, and neurologic symptoms

Typical initial testing is guided by your history, but often includes blood glucose/HbA1c and thyroid-function tests. Additional tests may be needed for infection, malignancy, endocrine disorders, or medication effects. Causes include hyperthyroidism, diabetes/hypoglycemia, menopause, pregnancy, infections, lymphoma, Parkinson disease, endocrine tumors, and drugs. Andrews' Diseases of the Skin, generalized hyperhidrosis section, lines 213-219.

Get urgent care today if sweating comes with

Chest pain, shortness of breath, fainting, or confusion
Fever with severe illness
Shakiness, weakness, confusion, or loss of consciousness suggesting low blood sugar
Drenching night sweats plus unintentional weight loss, persistent fever, or enlarged lymph nodes

Treatment

Treat the cause if one is found. This is the main treatment for generalized hyperhidrosis, including adjusting a triggering medicine only with the prescriber.
Practical relief: breathable and moisture-wicking clothing, cool environment, hydration and electrolyte replacement if sweating is heavy, and antiperspirant on the most troublesome areas.
If no reversible cause is found and symptoms remain severe, a dermatologist or primary-care clinician may consider oral sweat-reducing medicines, usually anticholinergic drugs such as glycopyrrolate or oxybutynin. These can cause dry mouth, constipation, blurred vision, urinary retention, overheating, and sometimes cognitive side effects, so they need individual prescribing and follow-up.
Botox, iontophoresis, and topical treatments are more useful for specific focal areas than for whole-body sweating.

Textbook guidance notes that secondary generalized hyperhidrosis is commonly associated with systemic illness or drugs, and that antiperspirants are initial symptom treatment, while systemic medicines may be considered under supervision. Goldman-Cecil Medicine, Hyperhidrosis section, line 1354. The International Hyperhidrosis Society guidance similarly recommends identifying and treating the underlying cause first.

If you share your age, duration, whether you have night sweats, and any medicines you take, I can help identify the most likely causes to discuss with your clinician.Start with a general physician / primary-care doctor (internal medicine or family medicine). For full-body sweating, they should first look for a medication-related or medical cause and arrange targeted tests.

They may refer you to:

Dermatologist: if no underlying cause is found, or for treatment of persistent hyperhidrosis.
Endocrinologist: if there are signs or test results suggesting thyroid disease, diabetes or repeated low blood sugar, menopause-related issues, or another hormonal disorder.
Other specialists only if your history or test results point to infection, neurologic disease, or another specific cause.

Do not begin oral medicines for sweating on your own. Generalized hyperhidrosis is commonly secondary to drugs or systemic illness. Goldman-Cecil Medicine, Sweating Disorders, p. 1242. The International Hyperhidrosis Society likewise recommends primary care first, followed by dermatology when needed.Keratosis pilaris (KP) causes harmless rough, small follicular bumps, usually on the upper arms, thighs, buttocks, or cheeks. It cannot be permanently cured, but regular skin care usually improves roughness and redness.

Simple treatment routine

1. Moisturize daily

After a short lukewarm shower, pat skin partly dry and apply a cream within 5 minutes. Choose one product containing:

Urea 10%-20%
Lactic acid / ammonium lactate 5%-12%
Salicylic acid 1%-2%
Glycolic acid or another alpha-hydroxy acid

Use once daily initially. If your skin tolerates it, increase as directed on the product. Urea- and lactate-based creams are commonly recommended treatments. Textbook of Family Medicine, 9e, Keratosis Pilaris section.

2. Avoid irritation

Use a mild fragrance-free cleanser.
Keep showers short and lukewarm.
Do not scrub hard, pick the bumps, or use harsh exfoliating gloves or abrasive scrubs.
Use loose, soft clothing if friction worsens the bumps.

3. If it remains rough after 6-8 weeks

A dermatologist may prescribe a topical retinoid such as adapalene, tretinoin, or tazarotene. Start only a few nights per week because these products can cause dryness and irritation. Do not use prescription retinoids during pregnancy or when trying to conceive unless your prescribing clinician specifically advises it.

4. If it is itchy, red, or inflamed

A short course of a suitable topical steroid may be recommended by a clinician. Avoid using steroid creams regularly or long term without medical advice.

Expectations

Improvement is gradual, often over several weeks. Treatment must be continued intermittently because the bumps commonly return if treatment stops. The 2025 systematic review on KP treatment supports keratolytic topical treatments and recognizes that evidence for other approaches, including laser procedures, is more limited (PMID: 41277649). DermNet guidance also lists urea, lactic acid, salicylic acid, alpha-hydroxy acids, and topical retinoids as options.

See a dermatologist if it is painful, pus-filled, leaving scars, associated with major eczema, or there is no improvement after 2-3 months of consistent care.Product availability and strengths vary by country.Which country are you buying these products in? Brand names and available strengths differ substantially between India, the US, UK, and other countries.

List urea, lactic-acid, and salicylic-acid products available locally

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Please tell me your country and city so I can list products actually available locally. For example: “Mumbai, India” or “London, UK.”

Hyderabad india

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India keratosis pilaris urea 10% cream product composition Ureaderm Moisturex Hyderabad

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India lactic acid ammonium lactate 12% lotion product composition keratosis pilaris

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India salicylic acid 2% body lotion cream keratosis pilaris product composition

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keratosis pilaris treatment

For keratosis pilaris on the body in Hyderabad, these are commonly listed by Indian online pharmacies. Local stock and prescription requirements can vary, so confirm availability with your nearby pharmacy or through delivery to your PIN code.

Active ingredient	Products to ask for	Notes
Urea 10%	Cetaphil PRO Urea 10% Smoothing Repair Lotion	Contains 10% urea and also lactic acid/sodium lactate. Fragrance-free and a sensible first option if your skin is sensitive. See the ingredient listing.
Urea 10%	Urea-10 Cream	A urea-based emollient. Check the outer pack to confirm it is urea 10% and whether any additional active ingredients are included.
Urea 20% plus lactic acid	Aqurea 20 Moisturizing Urea Cream	Stronger option for very rough skin. Contains urea and lactic acid. It may sting more than a 10% product, so patch-test first.
Urea plus lactic acid	Moisturex Cream	Contains urea, lactic acid, propylene glycol, and liquid paraffin, according to the product listing. Exact concentrations should be checked on the pack.
Lactic acid 12%	Lacsoft 12% Cream or Lotion	Lactic acid 12% keratolytic moisturizer. It is a practical KP option but may irritate sensitive, eczema-prone, or freshly shaved skin. Apollo's listing identifies it as lactic acid 12%.
Lactic acid 10% plus BHA 1%	Wishcare 10% AHA + 1% BHA Body Lotion	A cosmetic body lotion marketed for rough and bumpy skin. Use cautiously at first because it contains two exfoliating acids. Product listing.
Salicylic acid 2%	Saliac 2% Lotion	A salicylic-acid keratolytic product listed for KP and other thick, scaly skin conditions. Product details.
Salicylic acid 2%	Salgrow 2% Lotion	Similar 2% salicylic-acid option, also listed for KP. Product details.
Salicylic acid product	Saslic DS Cream	Another pharmacy option for hyperkeratotic conditions including KP. Confirm the exact salicylic-acid concentration on the pack or with the pharmacist before purchase. Product details.

A simple starting choice

Pick only one initially, not all together:

Sensitive or dry skin: Cetaphil PRO Urea 10% or Moisturex.
Typical rough bumps: Lacsoft 12% lotion/cream.
Very rough bumps, non-sensitive skin: Saliac 2% lotion or Aqurea 20, preferably after dermatologist advice.

Apply a thin layer to damp body skin once daily after bathing. If there is no burning, redness, or peeling after 1-2 weeks, continue. Do not apply acids to the face, broken skin, freshly shaved skin, or active eczema unless a dermatologist advises it. Avoid combining lactic acid, salicylic acid, and a retinoid on the same area initially.

Stop and seek advice if there is significant burning, swelling, cracking, or persistent rash. A dermatologist is best if the bumps are painful, pus-filled, scarred, or not improving after 8-12 weeks.

Can I get the exact picture of Moisturex with urea and lactic acid

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Moisturex Cream urea lactic acid India product image

Yes. This is the Moisturex Cream to look for. The label should state urea, lactic acid, propylene glycol, and liquid paraffin.

Confirm the ingredient list on the pack before buying, as Moisturex has multiple products. The correct product is listed as Moisturex Cream with urea and lactic acid, not Moisturex Hydra gel cream.