Teach me Audiometry in detail from scratch for my third year pg exam of ENT. Also include various types of Audiometry in detail

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audiogram types conductive sensorineural mixed hearing loss

I now have comprehensive content from the ENT textbooks. Let me compile this into a detailed, exam-ready guide.

🎧 Audiometry β€” Complete PG ENT Exam Guide

Sources: K J Lee's Essential Otolaryngology | Cummings Otolaryngology Head and Neck Surgery

1. INTRODUCTION & BASIC CONCEPTS

Audiometry is the science of measuring hearing sensitivity and diagnosing auditory disorders. It forms the cornerstone of any otological evaluation.

The Decibel (dB) Scale

  • dB SPL (Sound Pressure Level): Absolute physical measure of sound intensity; reference 0.0002 dynes/cmΒ²
  • dB HL (Hearing Level): Clinically used; calibrated to average normal hearing thresholds at each frequency. "0 dB HL" = what a normal young adult barely hears at that frequency
  • dB SL (Sensation Level): Intensity above the patient's own threshold

Frequency Range

  • Human hearing: 20 Hz – 20,000 Hz
  • Speech range: 500–3000 Hz (most critical)
  • Standard audiometric test frequencies: 250, 500, 1000, 2000, 4000, 8000 Hz
  • Inter-octave frequencies (1500, 3000, 6000 Hz) tested when β‰₯25 dB difference exists between adjacent octaves

Classification of Hearing Loss by Severity (dB HL)

DegreeThreshold
Normal≀25 dB HL (adults); ≀15 dB HL (children)
Mild26–40 dB HL
Moderate41–55 dB HL
Moderately severe56–70 dB HL
Severe71–90 dB HL
Profound>90 dB HL

2. PURE-TONE AUDIOMETRY (PTA)

Principle

The foundation of audiometric testing. Measures hearing sensitivity to pure tones presented at specific frequencies.
Threshold = the lowest level at which a patient responds at least 50% of the time

Test Conditions

  • Sound-treated booth (quiet enough to measure 0 dB HL thresholds)
  • Calibrated equipment (complete calibration annually)
  • Trained audiologist
  • Otoscopic inspection prior to testing
  • Clear instructions, patient comfort

Methods of Threshold Determination

  • Modified Hughson-Westlake method (ascending): Standard clinical approach β€” decrease in 10 dB steps, increase in 5 dB steps; threshold = lowest level with β‰₯2/3 correct responses on ascent
  • Test sequence: Better ear first β†’ AC β†’ BC if needed

Air Conduction (AC)

  • Via earphones (supra-aural or insert)
  • Tests entire auditory pathway: outer ear β†’ middle ear β†’ cochlea β†’ auditory nerve β†’ brainstem
  • Symbols: O (right, unmasked), X (left, unmasked), β–³ (right, masked), β–‘ (left, masked)

Bone Conduction (BC)

  • Via bone oscillator placed on mastoid or forehead
  • Bypasses outer and middle ear β€” tests only cochlea β†’ auditory nerve β†’ brainstem
  • Symbols: < (right, unmasked), > (left, unmasked), [ (right, masked), ] (left, masked)

Air-Bone Gap (ABG)

  • ABG = AC threshold βˆ’ BC threshold
  • ABG β‰₯ 10 dB is clinically significant
  • Reflects conductive component of hearing loss (outer/middle ear pathology)

3. THE AUDIOGRAM β€” INTERPRETATION

Standard Symbols Table

SymbolMeaning
ORight ear, AC, unmasked
XLeft ear, AC, unmasked
β–³Right ear, AC, masked
β–‘Left ear, AC, masked
<Right ear, BC, unmasked
>Left ear, BC, unmasked
[Right ear, BC, masked
]Left ear, BC, masked

Types of Audiograms

1. Normal Hearing
  • All AC thresholds ≀25 dB HL (adults)
  • No ABG; BC = AC
2. Conductive Hearing Loss (CHL)
  • AC thresholds elevated (>25 dB HL)
  • BC thresholds normal (≀25 dB HL)
  • Significant ABG (β‰₯10 dB)
  • Causes: Otitis media with effusion, otosclerosis, tympanic membrane perforation, ossicular chain disruption
3. Sensorineural Hearing Loss (SNHL)
  • Both AC and BC thresholds equally elevated
  • No ABG (≀10 dB)
  • Causes: Noise-induced, presbycusis, MΓ©niΓ¨re's disease, ototoxicity, acoustic neuroma
4. Mixed Hearing Loss
  • Both AC and BC thresholds elevated
  • AC > BC β†’ ABG present
  • Cause: Combination of conductive + sensorineural pathology

Special Audiometric Patterns

PatternDescriptionCondition
Carhart notchBC dip at 2000 HzOtosclerosis (impedance mismatch)
4 kHz notchAC dip at 4000 HzNoise-induced hearing loss (NIHL)
FlatEqual loss across all frequenciesOtitis media with effusion
Cookie-biteMid-frequency loss (500–2000 Hz)Genetic/hereditary SNHL
Low-frequency lossRising audiogramMénière's disease (early)
High-frequency lossFalling audiogramPresbycusis, ototoxicity

Pure-Tone Average (PTA)

PTA = (AC at 500 + 1000 + 2000 Hz) Γ· 3
  • Used to verify agreement with speech threshold (should be within Β±10 dB)

4. MASKING

Why Mask?

When testing one ear, the sound can cross over through the skull and be heard by the non-test ear (NTE), giving a false response.

Interaural Attenuation (IA)

The reduction in sound as it travels from one side of the skull to the other:
  • Supra-aural earphones (AC): 40–65 dB depending on frequency
  • Insert earphones (AC): 70–90 dB (much less need for masking)
  • Bone conduction: 0–10 dB (essentially no attenuation β†’ mask BC almost always)

Rules for When to Mask

Rule A – Air Conduction: Mask the NTE when:
AC stimulus level to test ear exceeds BC threshold of NTE by β‰₯40 dB (supra-aural) or β‰₯70 dB (insert earphones)
Rule B – Bone Conduction: Mask the NTE when:
There is an ABG >10 dB in the test ear

Type of Masker Used

  • For pure tones: Narrow-band noise (NBN) centered around the test frequency
  • For speech: Speech-spectrum noise

5. SPEECH AUDIOMETRY

Speech audiometry assesses how well a patient processes speech, providing functional information beyond what pure-tone testing shows.

Components

A. Speech Awareness/Detection Threshold (SAT/SDT)

  • Lowest level at which patient detects speech (not necessarily understands)
  • Should be within 10 dB of the best pure-tone threshold anywhere in 250–8000 Hz range
  • Used when SRT cannot be obtained (children, language barriers, disabilities)

B. Speech Recognition Threshold (SRT)

  • Lowest level at which patient can repeat spondaic words correctly 50% of the time
  • Spondee = two-syllable word with equal stress on both syllables (e.g., railroad, sidewalk, eardrum, icecream)
  • Key rule: SRT should be within Β±10 dB of the PTA
  • Used to confirm pure-tone thresholds

C. Word Recognition Score (WRS) / Speech Discrimination Score (SDS)

  • Percentage of phonetically balanced (PB) words correctly repeated at suprathreshold level (25–35 dB SL above SRT)
  • Uses PB word lists (phonemes occur at same proportion as in normal English discourse)
  • Presented via recorded material (CDs/digital) β€” NOT monitored live voice (less reliable)

WRS Interpretation Table

ScoreInterpretation
90–100%Normal
76–88%Slight difficulty
60–74%Moderate difficulty
40–58%Poor
≀40%Very poor

Clinical Significance of WRS

  • Rollover: WRS decreases at higher intensities β†’ suggests retrocochlear lesion (acoustic neuroma)
  • Disproportionately poor WRS relative to pure-tone loss β†’ suggests retrocochlear pathology
  • In MΓ©niΓ¨re's disease: WRS may fluctuate and be surprisingly poor during attacks

6. IMMITTANCE (IMPEDANCE) AUDIOMETRY

Objective tests of middle ear function. Does NOT require a behavioral response from the patient. Based on the concept of acoustic impedance/admittance at the tympanic membrane.
Impedance = energy rejected; Admittance = energy accepted. "Immittance" encompasses both.

6A. TYMPANOMETRY

Principle: Measures mobility (compliance) of the middle ear system as a function of applied air pressure in the ear canal.
  • Probe tone: 226 Hz (standard); 1000 Hz in infants <6 months (more accurate in compliant neonatal canals)
  • Compliance expressed as acoustic admittance (mmho or cmΒ³/mL)
  • Pressure expressed in daPa (dekapascals)
Tympanometric peak pressure (TPP): Pressure at which TM is most mobile = middle ear pressure Normal peak pressure: βˆ’100 to +100 daPa (adults)

Tympanogram Types (Jerger Classification)

Tympanogram types A, B, C, As, Ad
TypeShapeCompliancePressureClinical Association
ANormal sharp peakNormalNormal (βˆ’100 to +100 daPa)Normal middle ear
As (A-shallow)Low/shallow peakReducedNormalOtosclerosis, tympanosclerosis, malleus fixation
Ad (A-deep)Very high peakIncreased (hypercompliant)NormalOssicular discontinuity, flaccid TM
BFlat, no peakMinimalN/AOME (fluid in middle ear), TM perforation (large ECV)
CPeak shifted negativeNormal< βˆ’100 daPaEustachian tube dysfunction, early OME
ECV (Equivalent Ear Canal Volume):
  • Normal: 0.6–1.5 cmΒ³ (adults); 0.4–1.0 cmΒ³ (children)
  • High ECV + Type B = TM perforation or patent PE tube
  • Low ECV = probe blocked against canal wall

6B. ACOUSTIC REFLEX TESTING

Principle: Loud sounds (β‰₯70 dB SL) trigger contraction of the stapedius muscle (CN VII), stiffening the ossicular chain β†’ measured as change in compliance.
Reflex arc: Sound β†’ CN VIII β†’ Superior olivary complex β†’ CN VII nucleus β†’ Stapedius muscle
Types of Reflexes:
  • Ipsilateral reflex: Stimulus and probe in same ear
  • Contralateral reflex: Stimulus in one ear, probe in other
Normal Acoustic Reflex Threshold (ART): 70–100 dB HL for pure tones

Acoustic Reflex Interpretation

FindingClinical Significance
Absent ipsilateral + contralateralCHL (probe ear), severe SNHL, CN VII lesion
Absent contralateral onlyLesion in crossed CN VIII/brainstem pathway
Present reflex with CHLRules out significant middle ear pathology
Reflex threshold elevatedModerate-severe SNHL
Diphasic (on-off) reflexEarly stapedial fixation (otosclerosis)
Absent reflex (with normal audiogram)Retrocochlear lesion

Acoustic Reflex Decay

  • Sustained tone at 10 dB above ART for 10 seconds
  • Positive decay (>50% decrease in amplitude in ≀5 seconds) β†’ CN VIII/retrocochlear lesion
  • Key test for acoustic neuroma screening

7. OTOACOUSTIC EMISSIONS (OAE)

Principle: The outer hair cells (OHC) of the cochlea not only receive sound but actively vibrate, generating sounds that travel backward through the middle ear to the ear canal, where they can be recorded.
Pathway: OHC β†’ Basilar membrane β†’ Cochlear fluids β†’ Oval window β†’ Ossicles β†’ TM β†’ External canal β†’ Microphone
Key Facts:
  • Tests OHC function specifically β€” does not test inner hair cells, CN VIII, or central pathways
  • Objective, non-invasive, no behavioral response needed
  • Absent OAEs + normal ABR = central/neural lesion (Auditory Neuropathy)
  • Present OAEs = cochlear hearing ≀30–40 dB HL (if retrocochlear is intact)
  • Contraindicated/affected by: Middle ear pathology (fluid, perforation), ear canal debris

Types of OAE

A. Spontaneous OAE (SOAE)

  • Present without any stimulus
  • Found in ~35–60% of normally hearing individuals
  • Absence is non-diagnostic (many normal ears have none)
  • Rarely used clinically

B. Evoked OAE (EOAE)

i. Transient-Evoked OAE (TEOAE)
  • Stimulus: Click or brief tone burst
  • Response: Broad-frequency response (up to 4000 Hz)
  • Present = cochlear function ≀30–40 dB HL
  • Primary screening tool β€” used in Universal Neonatal Hearing Screening (UNHS)
  • Low stimulus level (<30 dB SPL); requires quiet environment
ii. Distortion Product OAE (DPOAE)
  • Stimulus: Two simultaneous pure tones (F1 and F2, with F2/F1 ratio ~1.2)
  • The healthy cochlea generates a distortion product at 2F1–F2 frequency
  • Stimulus levels: 55–65 dB SPL
  • More frequency-specific than TEOAE
  • Can sometimes be recorded even in moderate-to-severe hearing loss
  • Used to monitor ototoxic drug effects (can detect early cochlear damage before threshold shifts occur)

OAE Summary Table

FeatureTEOAEDPOAE
StimulusClickTwo tones (F1, F2)
Main product2F1–F22F1–F2
Frequency specificityLowHigh
Clinical useNeonatal screeningOtotoxicity monitoring, frequency-specific cochlear assessment
Threshold correlate>30–40 dB HL β†’ absentMore sensitive

8. ELECTRICAL RESPONSE AUDIOMETRY (ERA) / AUDITORY EVOKED POTENTIALS (AEP)

ERA records electrical potentials from the auditory pathway in response to sound. Used when behavioral tests are unreliable (infants, difficult-to-test patients).

Classification by Latency

CategoryLatencyTests
Short-latency (<10 ms)Cochlea + brainstemABR, ECoG
Middle-latency (10–50 ms)Thalamo-corticalMLR
Long-latency (>50 ms)CorticalCERA (N1-P2), P300, MMN

8A. AUDITORY BRAINSTEM RESPONSE (ABR) / BERA / BAER

The most clinically important ERA.
Principle: Click or tone-burst stimuli evoke 5 waves (I–V) within 10 ms, representing sequential activation of auditory neural generators.
Stimulus: Click (broad spectrum, 2–4 kHz region) or tone burst (frequency-specific) Recording: Surface electrodes (vertex positive, mastoid/earlobe negative)

ABR Wave Generators

WaveGenerator
IDistal end of CN VIII (cochlear)
IIProximal CN VIII
IIICaudal brainstem (trapezoid body, superior olivary complex)
IVSuperior olivary complex
VLateral lemniscus β†’ inferior colliculus
VI, VIIInferior colliculus
Most reliable waves: I, III, and V The I–V inter-peak latency is the key measure (~4 ms)

ABR β€” Clinical Applications

ApplicationDetails
Threshold ABREstimates hearing threshold (wave V tracked to lowest level); used in infants, non-cooperative patients
Neurologic ABRHigh intensity (80–95 dB nHL); detects retrocochlear lesions; acoustic neuroma screening (90% sensitivity, 80% specificity)
Intraoperative monitoringDuring posterior fossa/acoustic neuroma surgery
Neonatal screeningAutomated ABR (AABR) used in UNHS

ABR β€” Not Affected By:

Sedation, anesthesia, sleep state, most drugs β†’ ideal for testing infants and uncooperative patients

ABR β€” Affected By:

Age, sex, body temperature, degree of hearing loss

ABR Abnormalities

FindingInterpretation
Absent all wavesProfound HL or no auditory function
Wave I present, V absent/delayedRetrocochlear lesion
Prolonged I–V IPLAcoustic neuroma, demyelination
No wave, but CM presentAuditory neuropathy spectrum disorder

8B. ELECTROCOCHLEOGRAPHY (ECoG / ECochG)

Principle: Records electrical potentials from the cochlea and proximal CN VIII.
Three potentials recorded:
  1. Summating Potential (SP): DC potential from outer hair cells (>50%) and inner hair cells; represents receptor potential during sound stimulation
  2. Action Potential (AP / N1): From spiral ganglia and distal CN VIII afferents; equivalent to Wave I of ABR
  3. Cochlear Microphonic (CM): AC potential from outer hair cells; mirrors the acoustic waveform exactly
Key Measurement: SP/AP Ratio
  • Normal: < 0.4 (or <40%)
  • Elevated SP/AP ratio (>0.4) β†’ MΓ©niΓ¨re's disease (endolymphatic hydrops)
  • CM is used to differentiate auditory neuropathy
Electrode placement:
  • Transtympanic (needle on promontory) β€” most accurate
  • Extratympanic (ear canal) β€” less invasive

8C. AUDITORY STEADY-STATE RESPONSE (ASSR)

  • Uses modulated tones at specific frequencies (500, 1000, 2000, 4000 Hz)
  • Allows frequency-specific threshold estimation
  • Can test multiple frequencies simultaneously
  • Useful for fitting hearing aids in infants (predicts audiogram shape)
  • Less affected by stimulus artifact than ABR clicks

8D. MIDDLE LATENCY RESPONSE (MLR)

  • Latency: 10–50 ms
  • Waves: Po, Na, Pa, Nb, Pb
  • Generators: Thalamus (Na), Primary auditory cortex (Pa)
  • Less commonly used clinically
  • Useful in assessing auditory processing disorders and cortical hearing loss

8E. CORTICAL ELECTRIC RESPONSE AUDIOMETRY (CERA) β€” N1-P2

  • Long-latency response (>50 ms)
  • Generated in auditory cortex
  • Waves: P1, N1, P2, N2
  • Used for threshold estimation in cooperative patients, medicolegal assessment

8F. AUDITORY P300

  • Latency ~300 ms
  • Cognitive/event-related potential
  • Tests auditory discrimination and cognitive processing
  • Abnormal in dementia, auditory processing disorders
  • Generator: Hippocampus (medial temporal lobe)

9. BEHAVIORAL AUDIOMETRY (Pediatric)

Since young children cannot perform standard audiometry, age-appropriate behavioral tests are used.

Age-Based Approach

AgeMethod
0–6 monthsBehavioral Observation Audiometry (BOA)
6–30 monthsVisual Reinforcement Audiometry (VRA) / COR
30 months – 5 yearsConditioned Play Audiometry (CPA)
5 years+Standard pure-tone audiometry

A. Behavioral Observation Audiometry (BOA)

  • Age: ≀6 months (neurodevelopmental age)
  • No conditioning; observer watches for reflex responses (eye widening, startle, pause in activity, head turn)
  • Sound field via loudspeakers or earphones
  • Minimal Response Level (MRL) obtained β€” typically suprathreshold (not a true threshold)
  • Limitations: Response fatigues rapidly, high inter-subject variability, unreliable

B. Visual Reinforcement Audiometry (VRA)

  • Age: 6–30 months
  • Child is operantly conditioned to turn head toward sound source, rewarded by animated toy/video
  • Only requires head turn toward sound (does not need to localize direction)
  • Insert earphones preferred (avoids collapsing canal)
  • Conditioned Orienting Response (COR): Variant requiring correct direction localization (right sound β†’ right head turn)

C. Conditioned Play Audiometry (CPA)

  • Age: 30 months and above
  • Game-based: Each tone β†’ child performs play activity (drops block in bucket, places ring on peg)
  • The game itself is the reinforcer
  • By this age, most children tolerate earphones β†’ ear-specific thresholds possible

10. SITE-OF-LESION TESTING

Tests to differentiate cochlear (sensory) vs. retrocochlear (neural) hearing loss.

Modern (Still Used)

TestPrincipleCochlear findingRetrocochlear finding
Acoustic reflex decaySustained tone causes reflex to decayNo decayDecay >50% in ≀5 sec
ABRInter-peak latencyNormal or mildly elevatedProlonged I–V IPL; absent waves
OAE + ABR combinationOHC vs neural functionOAE absent + ABR present (sensory)OAE present + ABR abnormal (neural = ANSD)

Historical (No Longer Routinely Used)

TestPrincipleCochlearNeural
ABLB (Alternate Binaural Loudness Balance)Balance loudness between earsRecruitment presentNo recruitment
SISI (Short Increment Sensitivity Index)Detects 1-dB incrementsScore >70% (cochlear)Score <30% (neural)
Tone Decay TestAuditory adaptation to continuous tone<25 dB decay>25 dB decay
Bekesy AudiometryPulsed vs. continuous tone tracingsType IIType III/IV

Bekesy Types

TypeTracingMeaning
IContinuous β‰ˆ Pulsed (overlap)Normal
IIContinuous 10–20 dB below pulsedCochlear lesion
IIIContinuous drops dramatically >20 dBNeural lesion (severe fatigue)
IVContinuous consistently >20 dB below pulsedNeural lesion
VContinuous better than pulsedFunctional/nonorganic hearing loss

11. AUDITORY NEUROPATHY SPECTRUM DISORDER (ANSD)

A distinct pattern:
  • OAEs: Present (OHC intact)
  • Cochlear Microphonic: Present
  • ABR: Absent or severely abnormal (neural dyssynchrony)
  • May or may not show threshold loss on PTA
  • Speech understanding disproportionately poor
  • Causes: Selective inner hair cell loss, synaptic dysfunction, CN VIII dyssynchrony
  • Management: Cochlear implant (benefits more than hearing aids)

12. DIAGNOSTIC FLOWCHART (Harrison's Approach)

Hearing loss diagnostic algorithm

13. QUICK REVISION β€” HIGH-YIELD POINTS FOR PG EXAM

TopicKey Fact
Spondee2-syllable, equal-stress word (railroad, eardrum)
SRT vs PTAShould agree within Β±10 dB
Carhart notchBC dip at 2000 Hz β†’ otosclerosis
4 kHz notchNoise-induced hearing loss
Masking BCRequired when ABG >10 dB
Type B tympanogramOME (normal ECV) vs. perforation (high ECV)
Type AsOtosclerosis, tympanosclerosis
Type AdOssicular discontinuity
OAE absent + ABR normalNot possible if cochlear; suggests neural
SP/AP ratio >0.4Ménière's disease (ECoG)
ABR wave V generatorLateral lemniscus / inferior colliculus
ABR not affected bySedation, anesthesia, sleep
Rollover on WRSRetrocochlear (acoustic neuroma)
Diphasic acoustic reflexEarly otosclerosis
ANSD patternOAE present + ABR absent
VRA age6–30 months
CPA age30 months +
BOA limitationResponse extinguishes quickly, suprathreshold only
DPOAE clinical useOtotoxicity monitoring, frequency-specific OHC testing
AABRUsed in neonatal universal hearing screening

14. SUMMARY OF AUDIOMETRY TYPES

TypeObjective/SubjectiveWhat It TestsKey Use
Pure-Tone AudiometrySubjectiveThreshold sensitivity, AC & BCFoundation of hearing assessment
Speech Audiometry (SRT, WRS)SubjectiveSpeech detection, recognition, discriminationFunctional hearing, retrocochlear
TympanometryObjectiveMiddle ear pressure & complianceOME, otosclerosis, perforation
Acoustic ReflexObjectiveStapedius reflex arc integrityCN VII/VIII, retrocochlear
OAE (TEOAE, DPOAE)ObjectiveOHC functionNeonatal screening, ototoxicity
ABR/BERAObjectiveAuditory nerve + brainstemThreshold in infants, acoustic neuroma
ECoGObjectiveCochlear potentials (SP, AP, CM)Ménière's, ANSD
ASSRObjectiveFrequency-specific cochlear sensitivityInfant hearing aid fitting
MLRObjectiveThalamo-cortical pathwaysAuditory processing
CERA / P300ObjectiveCortical processingMedicolegal, cognitive assessment
BOABehavioralGross hearing in infants<6 months
VRA / CORBehavioral + conditioningThreshold in toddlers6–30 months
CPABehavioral + conditioningThreshold in young children30+ months
Bekesy AudiometrySubjective (automated)Auditory fatigue/adaptationSite of lesion (historical)
SISISubjectiveRecruitmentSite of lesion (historical)
Tone DecaySubjectiveNeural fatigueSite of lesion (historical)

Compiled from: K J Lee's Essential Otolaryngology (9781260122237) and Cummings Otolaryngology Head and Neck Surgery (9780323612173)
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