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Blunt Abdominal Trauma (BAT)
Epidemiology & Common Mechanisms
Motor vehicle collisions account for the majority of cases. Falls from height, direct blows, sports injuries, and blast injuries make up the rest. The abdomen is one of the most vulnerable anatomic regions in blunt trauma, and injuries are frequently occult - physical examination alone is unreliable, especially in patients with altered mental status, intoxication, distracting injuries, or spinal cord injury. - Rosen's Emergency Medicine
Most commonly injured organs:
- Spleen - most common; isolated in ~2/3 of splenic injuries
- Liver - second most common
- Kidney - third
- Small bowel / hollow viscus - less common (1-5% of BAT admissions) but frequently missed
Initial Assessment - ATLS Framework
The primary approach follows the xABCDE pattern (updated ATLS):
- xA - eXsanguinating hemorrhage control first
- A - Airway
- B - Breathing
- C - Circulation (two large-bore IV lines, blood transfusion if needed)
- D - Disability (GCS, pupils)
- E - Exposure/Environment (undress completely, logroll for back)
The hemodynamic status is the single most important driver of the subsequent workup and management pathway. Everything diverges at this fork.
Diagnostic Tools
1. FAST / eFAST (First-Line Bedside Tool)
FAST (Focused Assessment with Sonography for Trauma) is now the standard adjunct to the primary survey. It is performed bedside, simultaneously with resuscitation, and evaluates four windows:
| Window | Structure Examined |
|---|
| Subxiphoid | Pericardium / cardiac tamponade |
| Right subcostal | Hepatorenal fossa (Morrison's pouch) |
| Left subcostal | Splenorenal fossa |
| Suprapubic | Pelvis / pouch of Douglas |
Extended FAST (eFAST) adds bilateral thoracic views to detect hemothorax and pneumothorax.
- Sensitivity: 73-88% | Specificity: 98-100% | Accuracy: 96-98%
- Requires ≥200 mL of free fluid for detection
- Cannot evaluate the retroperitoneum - a major limitation
- Can be repeated serially with any hemodynamic change
- Positive FAST in an unstable patient → immediate laparotomy
- Positive FAST in a stable patient → proceed to CT for characterization
Current practice is trending toward "Complete Abdominal Sonography for Trauma" (CAST) in stable patients to allow more selective CT use (BJS 2025).
2. CT Abdomen/Pelvis with IV Contrast (Definitive Imaging in Stable Patients)
CT has essentially become the primary method for comprehensive evaluation of hemodynamically stable BAT patients. Key points:
- Performed with IV contrast in arterial and portal venous phases - best demonstrates solid organ perfusion and active bleeding
- Negative predictive value up to 99.63% for significant intraabdominal injury
- Identifies solid organ injury (spleen, liver, kidney), retroperitoneal hematoma, and active extravasation ("vascular blush")
- Less sensitive for hollow viscus / bowel injury - CT signs include bowel wall thickening, mesenteric stranding, free fluid without solid organ injury, pneumoperitoneum
- A hemodynamically unstable patient should not be taken to CT unless resuscitative measures can continue simultaneously and the facility can provide true "scoop and scan"
3. Diagnostic Peritoneal Lavage / Aspirate (DPL/DPA)
DPL has been largely supplanted by FAST and CT. Remaining indications:
- FAST unavailable or technically inadequate
- Suspected occult bowel injury not detectable by CT
- Rapid triage when other modalities unavailable
DPL positive criteria:
| Finding | Threshold |
|---|
| Gross blood on aspiration | >10 mL |
| RBCs in lavage effluent | >100,000/mL |
| WBCs in lavage effluent | >500/mL |
| Bacteria, bile, or GI contents | Any amount |
Pitfalls: high false-positive rate, cannot evaluate retroperitoneum, risk of iatrogenic injury. Always place Foley catheter and NGT first. Use a supraumbilical approach in pelvic fracture or pregnancy.
Management Algorithm
Fig. 38.8 - Rosen's Emergency Medicine: BAT Algorithm. IPH = intraperitoneal hemorrhage; DPA = diagnostic peritoneal aspirate; SPEs = serial physical examinations.
Unstable Patient + Positive FAST/DPA → Immediate Laparotomy
Clinical indications for immediate laparotomy after blunt trauma (Rosen's, Table 38.2):
| Indication | Pitfall |
|---|
| Unstable vitals with suspected abdominal injury | Alternate shock sources (pelvic fracture, tension pneumothorax) |
| Unequivocal peritoneal irritation | May be unreliable |
| Pneumoperitoneum | Can come from non-abdominal sources |
| Evidence of diaphragmatic injury | Nonspecific |
| Significant GI bleeding | Uncommon |
Stable Patient → CT then Selective NOM or Surgery
Organ-Specific Management
Spleen
The most commonly injured organ. Clinical signs: LUQ pain, LUQ tenderness, hypotension, Kehr's sign (referred pain to the left shoulder on deep inspiration from diaphragmatic irritation by splenic hematoma).
AAST Spleen Injury Scale:
| Grade | Description |
|---|
| I | Subcapsular hematoma <10% surface area; capsular tear <1 cm depth |
| II | Subcapsular hematoma 10-50%; intraparenchymal <5 cm; laceration 1-3 cm depth |
| III | Subcapsular hematoma >50% or expanding; laceration >3 cm or involving trabecular vessels |
| IV | Laceration involving segmental/hilar vessels with >25% devascularization |
| V | Shattered spleen; hilar vascular injury devascularizing entire spleen |
(Advance one grade for multiple injuries, up to Grade III)
NOM (Nonoperative Management): ~60-80% of blunt splenic injuries can be managed nonoperatively at Level I/II trauma centers. Success rate ~95% when criteria are met.
Criteria for NOM:
- Hemodynamic stability
- No peritoneal signs
- No hollow viscus injury
- No free extravasation of IV contrast from splenic parenchyma
Risk factors for NOM failure:
- Grade III-V injury
- Age >55 years
- Moderate-large hemoperitoneum
- Subcapsular hematoma (risk of delayed rupture at 6-8 days)
- Portal hypertension / cirrhosis
- Contrast "blush" on CT (pseudoaneurysm) - present in ~2/3 of NOM failures
Angioembolization: Used for contained contrast blush (pseudoaneurysm) to selectively occlude splenic arterial branches; reduces NOM failure rate. About 20% of patients initially on NOM eventually require intervention.
Operative management: Splenorrhaphy (repair) preferred when possible at surgery. If splenectomy is unavoidable: post-splenectomy vaccines (pneumococcus, meningococcus, Hib) and prophylactic antibiotics to prevent Overwhelming Post-Splenectomy Infection (OPSI).
Liver
Second most commonly injured organ. The right lobe is injured more frequently due to its larger size.
AAST Liver Injury Scale:
| Grade | Description |
|---|
| I | Subcapsular hematoma <10%; capsular tear <1 cm depth |
| II | Subcapsular hematoma 10-50%; intraparenchymal <10 cm; laceration 1-3 cm depth, <10 cm length |
| III | Subcapsular hematoma >50% or expanding; laceration >3 cm depth |
| IV | Parenchymal disruption 25-75% of hepatic lobe or 1-3 Couinaud segments |
| V | Disruption >75% hepatic lobe or >3 Couinaud segments; juxtahepatic venous injury |
| VI | Hepatic avulsion |
NOM: Selective NOM is the standard of care for hemodynamically stable patients. Similar to the spleen: NOM of liver and spleen combined has a 95% success rate at centers with adequate resources.
- Angiographic embolization has similarly reduced morbidity and mortality for arterial liver injuries
- Complications of NOM: delayed hemorrhage, biloma, abscess, hemobilia
Operative management: Damage control surgery (packing + temporary closure) for Grade IV-V or hemodynamically unstable. Hepatorrhaphy or partial hepatectomy for definitive repair.
Kidney
AAST Kidney Injury Scale: Grades I-V (contusion/hematoma through vascular pedicle injury).
- CT with IV contrast is the imaging modality of choice for renal injury evaluation
- Hematuria (macro or micro) is a common but not universal finding
- Most renal injuries (Grades I-III) are managed nonoperatively with bed rest and monitoring
- Grade IV-V: often require angioembolization or operative repair/nephrectomy
- Retroperitoneal hematoma from renal injury may not be visible on FAST
Hollow Viscus (Bowel & Mesentery)
Incidence: 1-5% of BAT admissions - the diagnosis most likely to be missed.
High-risk mechanisms/signs:
- Lap belt / seatbelt sign (abdominal wall ecchymosis) - highly suspicious
- Handlebar injury
- "Chance fracture" of lumbar spine (flexion-distraction) - associated with bowel injury in ~50%
CT findings (often subtle):
- Overt: pneumoperitoneum, extravasation of oral contrast
- Subtle: bowel wall thickening, mesenteric fat stranding, free fluid without solid organ injury
Management: Any perforation requires surgical repair. Serial abdominal examinations are a critical adjunct for patients in whom exam is reliable. DPL can evaluate for occult bowel injury when CT is non-diagnostic. Hollow viscus injury must be ruled out before committing to NOM of solid organ injuries.
Pancreas
- Rare (2-12% of BAT), but frequently missed - CT sensitivity for pancreatic injury is lower
- Suspect with epigastric pain, rising amylase/lipase after upper abdominal blow
- MRCP or ERCP may be needed to evaluate ductal injury
- Isolated contusions: NOM with pancreatic rest
- Ductal disruption (Grade III+): surgical drainage or resection
Special Scenarios
Pelvic Fracture + BAT
Pelvic fractures complicate management by creating an additional retroperitoneal hemorrhage source not visible on FAST. Algorithm:
- Unstable + FAST positive for hemoperitoneum → laparotomy (pelvic packing if needed)
- Unstable + FAST negative → pelvic angioembolization or preperitoneal pelvic packing
- Stable → CT to differentiate intraperitoneal vs retroperitoneal hemorrhage
Head Injury + BAT
Closed head trauma is the scenario most vulnerable to delayed diagnosis of bowel perforation - impaired neurologic exam masks evolving peritonitis. Serial imaging and DPL have a higher threshold of use in this population.
Seatbelt Sign
Patients with visible abdominal wall ecchymosis from a lap belt should be admitted for observation and serial examination. Detection of intraperitoneal fluid by FAST or CT mandates DPL to determine the nature of the fluid, or exploratory laparotomy.
Key Laboratory Studies
- CBC - baseline Hgb/Hct (may be normal initially due to redistribution)
- Comprehensive metabolic panel - liver enzymes (ALT/AST elevated in liver injury), BUN/Cr
- Amylase/Lipase - pancreatic injury screen (low sensitivity alone)
- Urinalysis - hematuria suggests genitourinary injury
- Coagulation studies (PT/PTT/INR, fibrinogen) - guides damage control resuscitation
- Type and Screen / Crossmatch
- Lactate / Base deficit - severity of shock and resuscitation endpoint
Damage Control Surgery (DCS) Principles
For the hemodynamically unstable patient who goes to the OR:
- Hemorrhage control - packing, vascular clamps
- Contamination control - bowel clamps, temporary closure
- ICU resuscitation - correction of "lethal triad" (hypothermia, acidosis, coagulopathy)
- Definitive repair - return to OR at 24-72 hours once physiology normalized
Modern damage control resuscitation uses balanced transfusion (1:1:1 ratio of packed RBCs : FFP : platelets) and avoids large-volume crystalloid which worsens coagulopathy and abdominal compartment syndrome.
Sources: Rosen's Emergency Medicine (8th ed., Chapter 38); Current Surgical Therapy 14e; Sabiston Textbook of Surgery; Maingot's Abdominal Operations; BJS 2024 Management Review; EAST Practice Management Guidelines